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The Plant Cell, ISSN 1040-4651, 10/2014, Volume 26, Issue 10, pp. 4084 - 4101
Selective autophagy has been extensively studied in various organisms, but knowledge regarding its functions in plants, particularly in organelle turnover, is... 
Proteins | Mesophyll cells | Starvation | RESEARCH ARTICLES | Cellular senescence | Plants | Plastids | Vacuoles | Chlorophylls | Plant cells | Seedlings | RIBULOSE-1,5-BISPHOSPHATE CARBOXYLASE/OXYGENASE | SYSTEM | FLORAL-DIP | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHLOROPLASTS | SELECTIVE AUTOPHAGY | PLANT SCIENCES | CELL BIOLOGY | LEAF SENESCENCE | PLANT AUTOPHAGY | DEGRADATION | ENDOPLASMIC-RETICULUM | STRESS | Vesicular Transport Proteins - metabolism | Endoplasmic Reticulum - metabolism | Autophagy | Arabidopsis Proteins - metabolism | Plants, Genetically Modified | Salt-Tolerance - genetics | Plastids - metabolism | Mesophyll Cells - metabolism | Carbon - metabolism | Salt-Tolerant Plants - metabolism | Arabidopsis Proteins - genetics | Vesicular Transport Proteins - genetics | Microscopy, Electron | Plant Leaves - cytology | Arabidopsis - metabolism | Protein Transport | Arabidopsis - genetics | Plant Leaves - genetics | Plant Leaves - metabolism | Plastids - ultrastructure | Transport Vesicles - metabolism | Protein Binding | Vacuoles - metabolism | Luminescent Proteins - genetics | Mutation | Chloroplast Proteins - metabolism | Luminescent Proteins - metabolism | Salt-Tolerant Plants - genetics | Arabidopsis thaliana | Botanical research | Protein research | Physiological aspects | Cellular signal transduction | Research | Binding proteins
Journal Article
Diabetes, ISSN 0012-1797, 10/2010, Volume 59, Issue 10, pp. 2426 - 2434
OBJECTIVE Branched-chain amino acids, such as leucine and glucose, stimulate protein synthesis and increase the phosphorylation and activity of the mammalian... 
MAMMALIAN TARGET | ENERGY | AMINO-ACID METABOLISM | MALONYL-COA | ENDOCRINOLOGY & METABOLISM | STARVATION | ACETYL-COA CARBOXYLASE | VEIN ENDOTHELIAL-CELLS | KINASE ACTIVATION | SIRT1 | EXERCISE | AMP-Activated Protein Kinases - metabolism | Leucine - pharmacology | Phosphoproteins - drug effects | Phosphorylation | Intracellular Signaling Peptides and Proteins - drug effects | Intracellular Signaling Peptides and Proteins - metabolism | Ribosomal Protein S6 Kinases, 70-kDa - drug effects | Carrier Proteins - drug effects | Phosphoproteins - metabolism | Dose-Response Relationship, Drug | Insulin Resistance - physiology | Adenylate Kinase - metabolism | Muscle, Skeletal - drug effects | Aminoimidazole Carboxamide - metabolism | Protein-Serine-Threonine Kinases - metabolism | Pyruvates - metabolism | Muscle, Skeletal - enzymology | Ribonucleotides - metabolism | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Rats | Glucose - pharmacology | Down-Regulation - drug effects | Animals | Carrier Proteins - metabolism | Lactates - metabolism | Aminoimidazole Carboxamide - analogs & derivatives | Protein-Serine-Threonine Kinases - drug effects | TOR Serine-Threonine Kinases | Kinetics | Branched chain amino acids | Analysis | Physiological aspects | Insulin resistance | Protein biosynthesis | Research | Diabetes | Protein kinases | Metabolism
Journal Article
Science, ISSN 0036-8075, 6/2003, Volume 300, Issue 5626, pp. 1718 - 1722
We modeled how the charged levels of different transfer RNAs (tRNAs) that carry the same amino acid (isoacceptors) respond when this amino acid becomes... 
Starvation | Transcriptional regulatory elements | Messenger RNA | Protein synthesis | Ribosomes | Amino acids | Operons | Research Articles | Kinetics | Codons | Transfer RNA | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | SEQUENCE | ADAPTATION | PPGPP | TRANSLATION | TRANSFER-RNA ABUNDANCE | ACCURACY | Protein Biosynthesis | Pyrophosphatases - genetics | Ribosomes - metabolism | Mathematics | RNA, Messenger - metabolism | Amino Acids - metabolism | Codon | Frameshifting, Ribosomal | Escherichia coli Proteins - biosynthesis | Escherichia coli - metabolism | RNA, Transfer, Amino Acyl - metabolism | RNA, Bacterial - genetics | RNA, Transfer - genetics | Escherichia coli - growth & development | Amino Acyl-tRNA Synthetases - metabolism | RNA, Bacterial - metabolism | Operon | RNA, Transfer - metabolism | RNA, Messenger - genetics | RNA, Transfer, Amino Acyl - genetics | Pyrophosphatases - metabolism | Escherichia coli - genetics | Escherichia coli Proteins - genetics | Models, Genetic | Gene Expression Regulation, Bacterial | Research | Neurotransmitters | Ribonucleic acid--RNA | Ribonucleic acid | RNA; Transfer; Amino Acyl/genetics/metabolism | Amino Acids/metabolism | Escherichia coli Proteins/biosynthesis/genetics | RNA; Bacterial/genetics/metabolism | Frameshifting; Ribosomal | Gene Expression Regulation; Bacterial | Pyrophosphatases/genetics/metabolism | Models; Genetic | Escherichia coli/genetics/growth & development/metabolism | Amino Acyl-tRNA Synthetases/metabolism | RNA; Messenger/genetics/metabolism | RNA; Transfer/genetics/metabolism | Ribosomes/metabolism
Journal Article
Journal Article
Annual review of nutrition, ISSN 0199-9885, 2006, Volume 26, Issue 1, pp. 1 - 22
Journal Article
Endocrine Journal, ISSN 0918-8959, 2017, Volume 64, Issue 3, pp. 255 - 268
In skeletal muscle, sortilin plays a predominant role in the sorting of glucose transporter 4 (Glut4), thereby controlling glucose uptake. Moreover, our... 
AMPK | Glucose | Skeletal muscle | Sortilin | 3T3-L1 ADIPOCYTES | ACTIVATED PROTEIN-KINASE | RATS | PLASMA-MEMBRANE | INSULIN | TRANSPORT | PATHWAY | GLUT4 STORAGE-VESICLES | ENDOCRINOLOGY & METABOLISM | HYPOGLYCEMIA | C2C12 MYOTUBES | Hindlimb | Rats, Wistar | TOR Serine-Threonine Kinases - metabolism | Muscle Cells - drug effects | Adaptor Proteins, Vesicular Transport - genetics | Male | Muscle, Skeletal - metabolism | Muscle, Skeletal - cytology | RNA, Messenger - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | Diabetes Mellitus, Experimental - blood | TOR Serine-Threonine Kinases - antagonists & inhibitors | Multiprotein Complexes - metabolism | Muscle, Skeletal - drug effects | Food Deprivation | Cell Differentiation | Fasting - metabolism | Diabetes Mellitus, Experimental - metabolism | Adaptor Proteins, Vesicular Transport - agonists | Cell Line | Blood Glucose - analysis | Muscle Cells - pathology | Muscle Cells - metabolism | Down-Regulation - drug effects | Sirolimus - pharmacology | Muscle Cells - cytology | Up-Regulation - drug effects | Animals | Diabetes Mellitus, Experimental - pathology | Glucose - metabolism | Protein Kinase Inhibitors - pharmacology | Muscle, Skeletal - pathology | Glucose transporter | Proteins | Post-transcription | Musculoskeletal system | Starvation | Rodents | Rapamycin | Myocytes | Gene expression | Myogenesis
Journal Article
Developmental Biology, ISSN 0012-1606, 07/2013, Volume 379, Issue 1, pp. 64 - 75
dMyc (dMyc) is known for its role in cell-autonomous regulation of growth. Here we address its role in the fat body (FB), a metabolic tissue that functions as... 
Desat1 | Trehalose | Myc | Drosophila | Glycolysis | DILP2 | Fatty acid synthesis | DIETARY MANIPULATION | C-MYC | STEAROYL-COA DESATURASE-1 | NUTRITIONAL REGULATION | CELL-PROLIFERATION | DEVELOPMENTAL BIOLOGY | AMINO-ACIDS | GENE-EXPRESSION | INSULIN-LIKE PEPTIDES | DROSOPHILA INSULIN | TRANSGENIC MICE | Drosophila Proteins - metabolism | RNA, Messenger - metabolism | Drosophila - enzymology | Brain - metabolism | DNA-Binding Proteins - metabolism | Wings, Animal - growth & development | RNA Interference | Time Factors | Gene Expression Regulation, Developmental | Larva - growth & development | Fat Body - cytology | Female | Food Deprivation | Larva - enzymology | Larva - genetics | Fat Body - metabolism | Fatty Acid Desaturases - genetics | Fat Body - enzymology | Body Size | Fatty Acid Desaturases - metabolism | Larva - metabolism | RNA, Messenger - genetics | Hemolymph - metabolism | Lipid Metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Triglycerides - metabolism | Gene Expression Regulation, Enzymologic | Transcription Factors - metabolism | Wings, Animal - metabolism | Insulin - metabolism | Trehalose - metabolism | Animals | Survival Analysis | Glucose - metabolism | Drosophila - growth & development | Drosophila - metabolism | Drosophila Proteins - genetics | Glucose metabolism | Enzymes | Starvation | Synthesis | Peptides | RNA | Growth | Physiological aspects | Triglycerides | Glucose | Fatty acids | Dextrose | Fatty Acid Synthesis
Journal Article
Journal of Comparative Physiology B, ISSN 0174-1578, 8/2016, Volume 186, Issue 6, pp. 711 - 725
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2270 - 14
Drugs that mirror the cellular effects of starvation mimics are considered promising therapeutics for common metabolic disorders, such as obesity, liver... 
LOOP-HELIX PROTEIN | REGULATOR | ACTIVATED PROTEIN-KINASE | LIPID-METABOLISM | MULTIDISCIPLINARY SCIENCES | CALCIUM | CALCINEURIN | RECEPTOR | AUTOPHAGY | ENERGY-METABOLISM | LYSOSOMAL BIOGENESIS | Biguanides - pharmacology | Calcium - metabolism | Caloric Restriction | Longevity - drug effects | Humans | Metabolic Syndrome - metabolism | Autophagy - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - agonists | Autophagosomes - drug effects | Lysosomes - metabolism | Liver - drug effects | Diet, High-Fat | Lactams - pharmacology | Lysosomes - drug effects | Starvation | Caenorhabditis elegans - metabolism | Fatty Liver - metabolism | Liver - metabolism | Enzyme Inhibitors - pharmacology | Mitochondria - metabolism | Autophagosomes - metabolism | Mitochondria - drug effects | Digoxin - pharmacology | Animals | Caenorhabditis elegans - drug effects | High-Throughput Screening Assays | Lipid Metabolism - drug effects | Mice | HeLa Cells | Drugs | Animal models | Digoxin | Calcium | Biodegradability | Liver | Disorders | Autophagy | Nanoparticles | Dietary restrictions | Mitochondria | Fatty liver | Biocompatibility | Lipid metabolism | Biodegradation | Metabolic diseases | Lead compounds | Metabolism | Steatosis | Mode of action | Life span | Nematodes | Aging (natural) | Metabolic disorders | Nanotechnology
Journal Article
Nature, ISSN 0028-0836, 05/2017, Volume 545, Issue 7652, pp. 108 - 111
Nine neurodegenerative diseases are caused by expanded polyglutamine (polyQ) tracts in different proteins, such as huntingtin in Huntington's disease and... 
PATHOGENESIS | CELLS | PROTEIN | MULTIDISCIPLINARY SCIENCES | MACHADO-JOSEPH-DISEASE | MUTANT HUNTINGTIN | ATAXIN-3 | INTRANUCLEAR INCLUSIONS | LC3 | MOUSE MODELS | AGGREGATION | Humans | Ubiquitin - metabolism | Male | Neurons - cytology | Autophagy | Brain - metabolism | Peptides - metabolism | Protein Domains | Ataxin-3 - chemistry | Female | Food Deprivation | Neurons - metabolism | Protein Stability | Ataxin-3 - metabolism | Disease Models, Animal | Binding, Competitive | Cell Line | Ataxin-3 - deficiency | Huntingtin Protein - metabolism | Mice, Inbred C57BL | Phagosomes - metabolism | Cells, Cultured | Mutant Proteins - genetics | Exons - genetics | Mutant Proteins - metabolism | Huntington Disease - metabolism | Huntingtin Protein - chemistry | Animals | Mutant Proteins - chemistry | Brain - pathology | Huntington Disease - genetics | Protein Binding | Mice | Huntingtin Protein - genetics | Mutation | Proteasome Endopeptidase Complex - metabolism | Beclin-1 - metabolism | Ataxin-3 - genetics | Physiological aspects | Autophagy (Cytology) | Neural circuitry | Observations | Huntingtons disease | Competition | Biotechnology | Huntingtin | Toxicity | Pathogenesis | Impairment | Biosynthesis | Kinases | Experiments | Degradation | Proteins | Scholarships & fellowships | Ataxin | Rodents | Spinocerebellar ataxia | Ataxia | Biocompatibility | Trinucleotide repeat diseases | Medical research | Starvation | Neurodegenerative diseases | Polyglutamine diseases | Huntington's disease | Neurological diseases | Machado-Joseph disease | Cell lines | Scientific imaging | Mass spectrometry | Phagocytosis | Index Medicus
Journal Article