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Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2013, Volume 110, Issue 9, pp. 3591 - 3596
Prostaglandin E2 is now widely recognized to play critical roles in brain inflammation and injury, although the responsible prostaglandin receptors have not... 
Encephalitis | Receptors | Prostaglandins | Mortality | Mice | Inflammation | Blood brain barrier | Status epilepticus | Seizures | Vehicles | Neuronal injury | Epileptogenesis | Electroencephalography | Gliosis | Inflammatory cytokine | electroencephalography | TEMPORAL-LOBE EPILEPSY | COX-2 | neuronal injury | MULTIDISCIPLINARY SCIENCES | INJURY | MODEL | NEUROTOXICITY | BARRIER LEAKAGE | inflammatory cytokine | epileptogenesis | DISEASE | SEIZURE | gliosis | CEREBRAL-ISCHEMIA | CYCLOOXYGENASE-2 INHIBITOR | Inflammation - pathology | Receptors, Prostaglandin E, EP2 Subtype - metabolism | Recovery of Function - drug effects | Humans | Blood-Brain Barrier - physiopathology | Nerve Degeneration - physiopathology | Status Epilepticus - drug therapy | Pilocarpine | Hippocampus - drug effects | Weight Loss - drug effects | Anticonvulsants - pharmacology | Inflammation - complications | Inflammation - metabolism | Receptors, Prostaglandin E, EP2 Subtype - antagonists & inhibitors | Status Epilepticus - complications | Indoles - pharmacology | Disease Models, Animal | Brain - physiopathology | Status Epilepticus - pathology | Anticonvulsants - therapeutic use | Hippocampus - pathology | Blood-Brain Barrier - drug effects | Nerve Degeneration - complications | Nerve Degeneration - pathology | Brain - drug effects | Blood-Brain Barrier - pathology | Animals | Survival Analysis | Brain - pathology | Status Epilepticus - physiopathology | Indoles - therapeutic use | Models, Neurological | Hippocampus - physiopathology | Nerve Degeneration - drug therapy | Dinoprostone | Care and treatment | Cell receptors | Health aspects | Neurochemistry | Brain | Neurology | Hormones | Neurodegeneration | Rodents | Index Medicus | Biological Sciences
Journal Article
Neurology, ISSN 0028-3878, 01/2017, Volume 88, Issue 4, pp. 386 - 394
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 04/2017, Volume 114, Issue 17, pp. E3536 - E3545
Status epilepticus (SE), a medical emergency that is typically terminated through antiepileptic drug treatment, leads to hippocampus dysfunction typified by... 
Neuroinflammation | Memory dysfunction | Exosomes | Status epilepticus | Adult neurogenesis | adult neurogenesis | TEMPORAL-LOBE EPILEPSY | MULTIDISCIPLINARY SCIENCES | status epilepticus | NEURAL STEM-CELLS | SPONTANEOUS SEIZURES | PATTERN SEPARATION | MESENCHYMAL STROMAL CELLS | EXTRACELLULAR VESICLES | BRAIN INFLAMMATION | neuroinflammation | exosomes | memory dysfunction | DENTATE GYRUS | GRANULE CELLS | HIPPOCAMPAL NEUROGENESIS | Exosomes - metabolism | Cell Line | Memory Disorders - physiopathology | Exosomes - transplantation | Humans | Status Epilepticus - pathology | Memory Disorders - metabolism | Mesenchymal Stromal Cells - metabolism | Exosomes - pathology | Male | Administration, Intranasal | Neurogenesis | Animals | Memory Disorders - therapy | Status Epilepticus - physiopathology | Status Epilepticus - therapy | Mice | Mesenchymal Stromal Cells - pathology | Status Epilepticus - metabolism | Memory Disorders - pathology | Brain | Neuroprotection | Mesenchyme | Epilepsy | Memory | Cognitive ability | Stem cell transplantation | Pilocarpine | Antiepileptic agents | γ-Aminobutyric acid | Neurodegeneration | Bone marrow | Emergency medical services | Receiving | Preservation | Neurons | Animal memory | Side effects | Stem cells | Glutamatergic transmission | Hippocampus | Animal cognition | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
Journal of Clinical Neurophysiology, ISSN 0736-0258, 03/2019, Volume 36, Issue 2, pp. 141 - 145
Journal Article
Journal Article
FASEB Journal, ISSN 0892-6638, 04/2012, Volume 26, Issue 4, pp. 1616 - 1628
Prolonged seizures [status epilepticus (SE)] constitute a neurological emergency that can permanently damage the brain. SE results from a failure of the normal... 
Pharmacoresistance | Temporal lobe epilepsy | Anticonvulsant | ATP | EXTRACELLULAR ATP | temporal lobe epilepsy | MICROGLIAL ACTIVATION | TEMPORAL-LOBE EPILEPSY | GLUTAMATE RELEASE | anticonvulsant | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEFICIENT MICE | CELL BIOLOGY | pharmacoresistance | P2X RECEPTOR | BIOLOGY | CENTRAL-NERVOUS-SYSTEM | RAT HIPPOCAMPUS | SYNAPTIC TERMINALS | C-FOS EXPRESSION | Lorazepam - pharmacology | Neuroprotective Agents - therapeutic use | Microglia - metabolism | Seizures - prevention & control | Seizures - drug therapy | Status Epilepticus - chemically induced | Status Epilepticus - drug therapy | Male | Neurons - cytology | Purinergic P2X Receptor Antagonists - pharmacology | Anticonvulsants - pharmacology | Receptors, Purinergic P2X7 - genetics | Seizures - chemically induced | Lorazepam - therapeutic use | Seizures - physiopathology | Neuroprotective Agents - pharmacology | Seizures - pathology | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Neurons - metabolism | Neurons - drug effects | Microglia - cytology | Glutamic Acid - pharmacology | Kainic Acid - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Status Epilepticus - pathology | Anticonvulsants - therapeutic use | Excitatory Amino Acid Agonists - pharmacology | Hippocampus - pathology | Hippocampus - cytology | Purinergic P2X Receptor Agonists - pharmacology | Mice, Knockout | Animals | Receptors, Purinergic P2X7 - metabolism | Status Epilepticus - physiopathology | Mice | Index Medicus
Journal Article
Journal Article