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PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 1, p. e29597
Human embryonic stem cells (hESC) and induced pluripotent stem cells (iPSC) provide new prospects for studying human neurodevelopment and modeling neurological disease... 
HUMAN ES | INHIBITION | FGF | MULTIDISCIPLINARY SCIENCES | CENTRAL-NERVOUS-SYSTEM | DIFFERENTIATION CAPACITY | PRECURSORS | Oligonucleotide Array Sequence Analysis | Humans | Fibroblast Growth Factor 2 - pharmacology | Neurons - cytology | Gene Expression Profiling | Neural Stem Cells - cytology | Neuroepithelial Cells - cytology | Neuroglia - cytology | Neurons - metabolism | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | Cell Line | Pluripotent Stem Cells - cytology | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Pluripotent Stem Cells - metabolism | Transcription Factors - metabolism | Cell Differentiation - drug effects | Fluorescent Antibody Technique | Neuroglia - metabolism | Cell Proliferation - drug effects | Neuroepithelial Cells - metabolism | Epidermal Growth Factor - pharmacology | Neural Stem Cells - metabolism | Cluster Analysis | Medical research | Nervous system diseases | Epidermal growth factor | Neurons | Medicine, Experimental | Fibroblast growth factors | Comparative analysis | Embryonic stem cells | Neurophysiology | Neurosciences | Laboratories | Neurobiology | Embryo cells | Radial glial cells | Nervous system | Biochemistry | Neurodevelopmental disorders | Neuronal-glial interactions | Genotype & phenotype | Fibroblasts | Physiology | Growth factors | Fibroblast growth factor 2 | Fetuses | Embryos | Brain research | Stem cells | Comparative studies | Hindbrain | Bayesian analysis | Pluripotency
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 10, p. e110324
Human motor neurons derived from embryonic and induced pluripotent stem cells (hESCs and hiPSCs) are a potentially important tool for studying motor neuron survival and pathological cell death... 
SURVIVAL | IN-VITRO | ROCK INHIBITION | MULTIDISCIPLINARY SCIENCES | AXONAL REGENERATION | RHO | SPINAL-CORD | AMYOTROPHIC-LATERAL-SCLEROSIS | KINASE INHIBITOR | PHARMACOLOGICAL INHIBITION | MOTONEURONS | Embryonic Stem Cells - cytology | Humans | rho-Associated Kinases - antagonists & inhibitors | Brain-Derived Neurotrophic Factor - pharmacology | rho-Associated Kinases - metabolism | Glial Cell Line-Derived Neurotrophic Factor - pharmacology | Motor Neurons - cytology | Induced Pluripotent Stem Cells - cytology | Ciliary Neurotrophic Factor - pharmacology | Cyclic AMP - metabolism | Motor Neurons - drug effects | Amides - pharmacology | Cell Survival - drug effects | Gene Expression | Induced Pluripotent Stem Cells - enzymology | Induced Pluripotent Stem Cells - drug effects | Colforsin - pharmacology | Cells, Cultured | rho-Associated Kinases - genetics | Embryonic Stem Cells - enzymology | Motor Neurons - enzymology | 1-Methyl-3-isobutylxanthine - pharmacology | Embryonic Stem Cells - drug effects | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Cyclic AMP - agonists | Ciliary neurotrophic factor | Neurons | Stem cells | Flow cytometry | Motor neurons | Glial cell line-derived neurotrophic factor | Cell survival | Physicians | Embryo cells | Stem cell transplantation | Amyotrophic lateral sclerosis | Kinases | Neurotrophic factors | Survival | Rho-associated kinase | Brain-derived neurotrophic factor | Cell death | Rodents | Forskolin | Pluripotency
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2009, Volume 119, Issue 5, pp. 1109 - 1123
...), but the frequency of resistance increases in advancing stages of disease. Elimination of BCR/ABL-dependent intracellular signals triggers apoptosis, but it is unclear whether this activates additional cell survival and/or death pathways... 
CHRONIC MYELOGENOUS LEUKEMIA | MEDICINE, RESEARCH & EXPERIMENTAL | MALIGNANT GLIOMA-CELLS | BLAST CRISIS | CLINICAL RESISTANCE | BCR-ABL MUTATIONS | ENDOPLASMIC-RETICULUM | CYTOCHROME-C RELEASE | CASPASE ACTIVATION | IMATINIB RESISTANCE | CHRONIC MYELOID-LEUKEMIA | Transcription Factor CHOP - genetics | Neoplastic Stem Cells - cytology | Gene Expression - drug effects | Calcium - metabolism | Gene Expression - genetics | Microtubule-Associated Proteins - metabolism | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Endoplasmic Reticulum - metabolism | Antineoplastic Agents - therapeutic use | Autophagy - physiology | Thiazoles - therapeutic use | Autophagy - drug effects | Chloroquine - pharmacology | Neoplastic Stem Cells - metabolism | RNA Interference | Endoplasmic Reticulum - drug effects | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Macrolides - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Dasatinib | Chloroquine - therapeutic use | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Mice, Inbred C3H | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Cell Death - physiology | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Macrolides - therapeutic use | Protein-Tyrosine Kinases - antagonists & inhibitors | Causes of | Physiological aspects | Genetic aspects | Chronic myeloid leukemia | Research | Drug therapy | Phagocytosis
Journal Article
Pharmacology & therapeutics (Oxford), ISSN 0163-7258, 2017, Volume 171, pp. 30 - 42
Pericytes are a heterogeneous population of cells located in the blood vessel wall... 
Pericytes | Perivascular stem cells | Diabetic retinopathy | Diabetic nephropathy | Pericyte fibrosis | Cancer stem cells | DIABETIC-RETINOPATHY | Cancer stern cells | VEGF-A | BLOOD-BRAIN-BARRIER | MESENCHYMAL STEM-CELLS | EPITHELIUM-DERIVED FACTOR | HIGH GLUCOSE | PHARMACOLOGY & PHARMACY | STROMAL CELLS | TUMOR-GROWTH | GLYCATION END-PRODUCTS | ENDOTHELIAL GROWTH-FACTOR | Neoplasms - therapy | Animals | Diabetes Mellitus - physiopathology | Vascular Diseases - therapy | Diabetes Mellitus - therapy | Humans | Pericytes - cytology | Ischemia - therapy | Vascular Diseases - physiopathology | Ischemia - physiopathology | Molecular Targeted Therapy | Neoplasms - pathology | Blood circulation disorders | Therapeutics | Homeopathy | Materia medica and therapeutics | Impotence | Diabetic nephropathies | T cells | Muscle proteins | Cardiovascular agents | Blood coagulation factor VIII | Interleukins | Chronic kidney failure | Type 1 diabetes | Stem cells | Vascular endothelial growth factor | Mitogens | Protein kinases | Growth factors | UUO, unilateral ureteric obstruction | EGFR, EGF receptor | EMT, epithelial-mesenchymal transition | SFT, solitary fibrous tumour | DAN, diabetic autonomous neuropathy | ECM, extracellular matrix | NRF2, nuclear factor (erythroid-derived 2)-like 2 | IL-6, interleukin 6 | PSC, perivascular stem cell | BBB, blood-brain barrier | SOD, super oxide dismutase | HIF, hypoxia inducible factor | ED, erectile dysfunction | SDF-1, stromal derived factor 1 | RAGE, receptors of AGEs | CKD, chronic kidney disease | MMP, matrix metalloproteinases | MDSC, myeloid-derived suppressor cells | DN, diabetic nephropathy | TGF β, transforming growth factor β | PDL-1, programmed death-ligand 1 | ANG2, angiopoietin-2 | Olmfl3, Olfactomedin-like 3 | VEGF, vascular endothelial growth factor | AGE, Advanced Glycation End-Products | EC, endothelial cells | NG2, neural | R, ischemia-reperfusion | MSC, mesenchymal stromal cell | MF-EGF8, milk fat globule epidermal growth factor VIII | IL-8, interleukin 8 | glial antigen 2 | T1D, type 1 diabetic | PDGFb, platelet-derived growth factor B | MAPK, mitogen-activated protein kinase | PDGFRβ, platelet derived growth factor receptor β | DR, diabetic retinopathy | CNS, blood-retinal barrier | HB-EGF, heparin-binding EGF-like growth factor | HPC, hemangiopericytoma | PDR, proliferative diabetic retinopathy | DME, diabetic macular oedema | GFR, glomerular filtration rate | MI, myocardial infarction | SMA, smooth muscle actin | CSC, cancer stem cell | NPDR, non-proliferative diabetic retinopathy | T2D, type 2 diabetic | ROS, reactive oxygen species | Treg, regulatory T cells | BRB, blood-retina barrier | PKC, protein kinase C | DPN, diabetic peripheral neuropathy | TME, tumour microenvironment | ANG1, angiopoietin-1 | GSC, glioblastoma CSC | iPS, induced pluripotent stem cells | GSI, g-secretase inhibitor | FGF-9, fibroblastic growth factor 9 | PEDF, Pigment Epithelium-Derived Factor
Journal Article
Cancer cell, ISSN 1535-6108, 05/2017, Volume 31, Issue 5, pp. 635 - 652.e6
.... Targeting oncogenic transcription through either of these methods synergizes with HDAC inhibition, and DIPG cells resistant to HDAC inhibitor therapy retain sensitivity to CDK7 blockade... 
oligodendrocyte precursor cell | CDK7 | potassium channel | DIPG | EPH | super-enhancer | BRD4 | Receptors, Eph Family | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Male | Brain Stem Neoplasms | Azepines | Journal Article | Research Support, N.I.H., Extramural | Dose-Response Relationship, Drug | Research Support, U.S. Gov't, Non-P.H.S | Pyrimidines | Transfection | RNA Interference | Time Factors | Triazoles | Histones | Female | Transcription, Genetic | Cyclin-Dependent Kinases | Indoles | Tumor Cells, Cultured | Signal Transduction | Antineoplastic Combined Chemotherapy Protocols | Chromatin Assembly and Disassembly | Mice, SCID | Hydroxamic Acids | Nuclear Proteins | Drug Synergism | Xenograft Model Antitumor Assays | Protein Kinase Inhibitors | Animals | Glioma | Phenylenediamines | Research Support, Non-U.S. Gov't | Mice, Inbred NOD | Histone Deacetylase Inhibitors | Transcription Factors | Mutation | Primary Cell Culture | THERAPEUTIC STRATEGY | SUPER-ENHANCERS | GENE | ONCOLOGY | CELL IDENTITY | K27M MUTATION | C-MYC | HISTONE H3.3 | BET BROMODOMAIN INHIBITION | MYC EXPRESSION | TUMOR-GROWTH | CELL BIOLOGY | Transcription, Genetic - drug effects | Cyclin-Dependent Kinases - metabolism | Brain Stem Neoplasms - drug therapy | Glioma - genetics | Glioma - pathology | Receptors, Eph Family - metabolism | Cyclin-Dependent Kinases - antagonists & inhibitors | Receptors, Eph Family - genetics | Pyrimidines - pharmacology | Signal Transduction - drug effects | Histone Deacetylase Inhibitors - pharmacology | Phenylenediamines - pharmacology | Histones - metabolism | Glioma - drug therapy | Brain Stem Neoplasms - pathology | Brain Stem Neoplasms - metabolism | Chromatin Assembly and Disassembly - drug effects | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Glioma - metabolism | Indoles - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Cyclin-Dependent Kinases - genetics | Nuclear Proteins - genetics | Hydroxamic Acids - pharmacology | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Transcription Factors - genetics | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Brain Stem Neoplasms - genetics | Histones - genetics | Nuclear Proteins - antagonists & inhibitors | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Medical colleges | Gliomas | Developmental biology | Stem cells | Genetic aspects | Genetic transcription | Cell differentiation | Chromatin | Analysis | Potassium channels | Cells
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 10, p. e46956
We previously demonstrated that cells isolated from the mesenchymal region of the human amniotic membrane... 
DENDRITIC CELLS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | PLACENTA | PERIPHERAL-BLOOD | DIFFERENTIATION | STROMAL CELLS | MESENCHYMAL STEM-CELLS | T-CELLS | INHIBIT | ADIPOSE-TISSUE | Bone Marrow Cells - chemistry | Temperature | Molecular Weight | Immunologic Factors - isolation & purification | Prostaglandins - pharmacology | Humans | Culture Media, Conditioned - chemistry | Culture Media, Conditioned - pharmacology | Mesoderm - cytology | Dose-Response Relationship, Drug | Antibodies, Neutralizing - immunology | Mesenchymal Stromal Cells - cytology | Cytokines - isolation & purification | Prostaglandins - immunology | Female | Prostaglandins - isolation & purification | Biological Factors - isolation & purification | Cytokines - immunology | Leukocytes, Mononuclear - drug effects | Bone Marrow Cells - cytology | Amnion - cytology | Cells, Cultured | Immunologic Factors - immunology | Biological Factors - immunology | Mesenchymal Stromal Cells - chemistry | Mesoderm - chemistry | Antibodies, Neutralizing - pharmacology | Lymphocytes - cytology | Amnion - chemistry | Lymphocytes - drug effects | Leukocytes, Mononuclear - cytology | Cell Proliferation - drug effects | Biological Factors - pharmacology | Cytokines - pharmacology | Immunologic Factors - pharmacology | COX-2 inhibitors | Prostaglandins | Cytokines | Dendritic cells | Stem cells | Bone morphogenetic proteins | Hostages | B cells | Transforming growth factors | Cell proliferation | Cell culture | Transplants & implants | Mesenchyme | Thermal stability | Molecular weight | Prostaglandin endoperoxide synthase | Lymphocytes | Rodents | Cell cycle | Bone marrow | Inhibition | Conditioning | Growth factors | Immunoglobulins | Immunoregulation | Immunomodulation | Studies | Amniotic membrane | Monocytes | Nitric oxide | Skin & tissue grafts | Umbilical cord
Journal Article
STEM CELLS, ISSN 1066-5099, 09/2007, Volume 25, Issue 9, pp. 2206 - 2214
We have utilized a serum‐ and stromal cell‐free “spin embryoid body (EB)” differentiation system to investigate the roles of four growth factors, bone morphogenetic protein 4 (BMP4... 
Fibroblast growth factor 2 | Bone morphogenetic protein 4 | Embryonic stem cells | Hematopoiesis | Vascular endothelial growth factor | Stem cell factor | stem cell factor | embryonic stem cells | MOUSE | COCULTURE | SPECIFICATION | MURINE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | hematopoiesis | COMMITMENT | LETHALITY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | vascular endothelial growth factor | fibroblast growth factor 2 | MOVEMENT | HEMANGIOBLAST | STROMAL CELLS | HEMATOLOGY | bone morphogenetic protein 4 | MESODERM INDUCTION | Bone Morphogenetic Protein 4 | Embryonic Stem Cells - cytology | Bone Morphogenetic Proteins - physiology | Homeodomain Proteins - metabolism | Humans | Cells, Cultured | Fibroblast Growth Factor 2 - pharmacology | Primitive Streak - metabolism | Homeodomain Proteins - genetics | Hematopoiesis - drug effects | Drug Synergism | Animals | Cell Differentiation - drug effects | Gene Expression Regulation, Developmental | Stem Cell Factor - pharmacology | Hematopoiesis - physiology | Fibroblast Growth Factor 2 - physiology | Mice | Culture Media, Serum-Free - pharmacology | Bone Morphogenetic Proteins - pharmacology | Stem Cell Factor - physiology | Vascular Endothelial Growth Factor A - pharmacology | Vascular Endothelial Growth Factor A - physiology | Drug Combinations
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 10, p. e0185775
Limb muscles derive from pax3 expressing precursor cells that migrate from the hypaxial somite into the developing limb bud... 
SKELETAL-MUSCLE | ORIGIN | RETINOIC ACID | SONIC HEDGEHOG | SOMITE | MULTIDISCIPLINARY SCIENCES | SF/HGF | CXCR4 | MUSCLE PROGENITOR CELLS | DIFFERENTIATION | EXPRESSION | Insulin-Like Growth Factor I - pharmacology | Podophyllotoxin - pharmacology | Receptor, IGF Type 1 - antagonists & inhibitors | Nitriles - pharmacology | MyoD Protein - genetics | Fibroblast Growth Factors - genetics | Muscle, Skeletal - metabolism | Myogenin - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Fibroblast Growth Factors - metabolism | Chick Embryo - metabolism | Gene Expression Regulation, Developmental | Muscle, Skeletal - drug effects | PAX3 Transcription Factor - genetics | Insulin-Like Growth Factor II - pharmacology | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Chromones - pharmacology | Butadienes - pharmacology | Muscle Development - physiology | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | PAX3 Transcription Factor - metabolism | MyoD Protein - metabolism | Pyrroles - pharmacology | Animals | Muscle Development - drug effects | Hindlimb - drug effects | Chick Embryo - drug effects | Hindlimb - metabolism | Myogenin - metabolism | Podophyllotoxin - analogs & derivatives | Genetic aspects | Research | Insulin-like growth factor 1 | Myogenesis | Fibroblast growth factor | Insulin-like growth factor I | mRNA | Insulin-like growth factors | Kinases | Limb buds | Signal transduction | Embryology | Developmental stages | Fibroblast growth factor 18 | Pax3 protein | Insulin-like growth factor II | Vascular endothelial growth factor | Growth factors | Myogenin | MyoD protein | Muscles | Gene expression | Insulin | Embryos | 1-Phosphatidylinositol 3-kinase | Myoblasts | Vascular endothelial growth factor receptors | Musculoskeletal system | Inhibitors | Transduction | Differentiation | Beads | Retinoic acid | Cell migration | Fibroblast growth factor receptors
Journal Article