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Science (American Association for the Advancement of Science), ISSN 1095-9203, 2010, Volume 328, Issue 5985, pp. 1570 - 1573
Cholesterol metabolism is tightly regulated at the cellular level. Here we show that miR-33, an intronic microRNA (miRNA... 
MicroRNA | Hepatocytes | HDL lipoproteins | Liver | Genes | REPORTS | Homeostasis | Gene expression regulation | Macrophages | Cholesterols | Endothelial cells | TANGIER-DISEASE | HDL | LIPID-METABOLISM | CASSETTE TRANSPORTER 1 | CELLULAR CHOLESTEROL | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MUTATIONS | ATP Binding Cassette Transporter, Sub-Family G, Member 1 | Membrane Glycoproteins - metabolism | Cholesterol, Dietary - administration & dosage | Lipoproteins - genetics | Lipoproteins, HDL - blood | Humans | Lipoproteins, HDL - metabolism | MicroRNAs - metabolism | Transfection | ATP-Binding Cassette Transporters - genetics | Dietary Fats - administration & dosage | Lipoproteins - metabolism | ATP-Binding Cassette Transporters - metabolism | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | ATP Binding Cassette Transporter 1 | Cell Line | Introns | Liver - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Apolipoprotein A-I - metabolism | Cholesterol - metabolism | Membrane Glycoproteins - genetics | Proteins - genetics | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Proteins - metabolism | Mice | MicroRNAs - genetics | Hypercholesterolemia - genetics | Macrophages, Peritoneal - metabolism | Hypercholesterolemia - metabolism | Physiological aspects | Control | Research | High density lipoproteins | Cholesterol metabolism | Lipoproteins | Cellular biology | Ribonucleic acid--RNA | Gene expression | Cholesterol
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2004, Volume 279, Issue 19, pp. 20314 - 20326
...), a step which is essential for glucose metabolism in liver as well as for the induction of glycolytic and lipogenic genes... 
FATTY-ACID-SYNTHASE | GLUCOSE-HOMEOSTASIS | CARBOHYDRATE RESPONSE ELEMENT | RAT HEPATOCYTES | STEROL REGULATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ELEMENT-BINDING PROTEIN-1C | PYRUVATE-KINASE GENE | TRANSCRIPTION FACTOR | CARNITINE-PALMITOYLTRANSFERASE-I | TRANSGENIC MICE | Glucokinase - physiology | Liver - enzymology | Fatty Acid Synthases - metabolism | Immunoblotting | Hepatocytes - metabolism | RNA, Messenger - metabolism | DNA-Binding Proteins - metabolism | Cell Nucleus - metabolism | Glycogen - metabolism | Time Factors | CCAAT-Enhancer-Binding Proteins - physiology | Adenoviridae - genetics | Transcription, Genetic | Pentosephosphates - metabolism | RNA - metabolism | CCAAT-Enhancer-Binding Proteins - metabolism | DNA-Binding Proteins - physiology | Pyruvate Kinase - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Carbohydrate Metabolism | Cells, Cultured | Gene Expression Regulation | Lipid Metabolism | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Nuclear Proteins | Transcription Factors - metabolism | Blotting, Northern | Animals | Proteins - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors | Glucose - metabolism | Mice | Kinetics | Sterol Regulatory Element Binding Protein 1 | Glucose-6-Phosphate - metabolism | Acetyl-CoA Carboxylase - metabolism | Microscopy, Fluorescence | RNA, Small Interfering - metabolism
Journal Article
Cell metabolism, ISSN 1550-4131, 07/2014, Volume 20, Issue 1, pp. 172 - 182
Oxysterols are cholesterol metabolites that serve multiple functions in lipid metabolism, including as liver X receptor (LXR) ligands... 
ADHESION | SIGNALING PATHWAYS | HEART-DISEASE | DNA-BINDING | LIVER X RECEPTORS | INFLAMMATION | TRANSCRIPTION | ENDOCRINOLOGY & METABOLISM | NF-KAPPA-B | INTERLEUKIN-6 GENE-EXPRESSION | STEROL 27-HYDROXYLASE | CELL BIOLOGY | Steroid Hydroxylases - metabolism | Apolipoproteins E - deficiency | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Apolipoproteins E - metabolism | Cytochrome P450 Family 7 | RNA Interference | Estrogen Receptor alpha - metabolism | Female | Cytokines - genetics | Steroid Hydroxylases - genetics | Macrophages - immunology | Cell Line | Atherosclerosis - pathology | NF-KappaB Inhibitor alpha | Cytokines - metabolism | Endothelial Cells - metabolism | Inflammation | Cell Adhesion - drug effects | Cholesterol - metabolism | Hydroxycholesterols - pharmacology | Atherosclerosis - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Estrogen Receptor alpha - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Endothelial Cells - cytology | Apolipoproteins E - genetics | Steroid Hydroxylases - deficiency | Hydroxycholesterols - metabolism | Macrophages - drug effects | Mice | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Metabolites | Atherosclerosis | Estrogen | Cytochrome P-450 | Physiological aspects | Phenols | Cholesterol | Prevention | Blood circulation disorders | Liver | Endothelium
Journal Article
Reproduction, Fertility and Development, ISSN 1031-3613, 08/2009, Volume 21, Issue 7, pp. 909 - 922
Peroxisome proliferator-activated receptor (PPAR) γ belongs to the PPAR family of nuclear transcription factors whose ligands, such as eicosanoids, fatty acids... 
granulosa | Leydig | SIGNALING PATHWAYS | TISSUE | DEVELOPMENTAL BIOLOGY | ARACHIDONIC-ACID | LEYDIG TUMOR-CELLS | MESSENGER-RNAS | REPRODUCTIVE BIOLOGY | ZOOLOGY | GENE-EXPRESSION | PPAR-GAMMA | STAR GENE | TRANSCRIPTION FACTOR | STIMULATED STEROIDOGENESIS | Cercopithecus aethiops | Male | Leydig Cells - drug effects | Cyclic AMP Response Element Modulator - metabolism | Cyclic CMP - analogs & derivatives | PPAR gamma - metabolism | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Granulosa Cells - drug effects | Granulosa Cells - metabolism | Dose-Response Relationship, Drug | Transfection | Cyclic CMP - pharmacology | Leydig Cells - metabolism | Female | Progesterone - biosynthesis | Steroidogenic Factor 1 - metabolism | Thiazolidinediones - pharmacology | Binding Sites | Sterol Regulatory Element Binding Protein 1 - metabolism | PPAR gamma - genetics | CCAAT-Enhancer-Binding Proteins - metabolism | Promoter Regions, Genetic | GATA4 Transcription Factor - metabolism | Signal Transduction | Proto-Oncogene Proteins c-jun - genetics | Phosphoproteins - genetics | CCAAT-Enhancer-Binding Protein-beta - metabolism | Animals | Proto-Oncogene Proteins c-jun - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | PPAR gamma - agonists | Cell Line, Tumor | Mice | COS Cells | Mitogen-Activated Protein Kinases - metabolism | Phosphoproteins, metabolism | Proto-Oncogene Proteins c-jun, metabolism | Progesterone, biosynthesis | Proto-Oncogene Proteins c-jun, genetics | Steroidogenic Factor 1, metabolism | CCAAT-Enhancer-Binding Proteins, metabolism | PPAR gamma, metabolism | PPAR gamma, genetics | Phosphoproteins, genetics | RNA, Messenger, metabolism | CCAAT-Enhancer-Binding Protein-beta, metabolism | Leydig Cells, metabolism | GATA4 Transcription Factor, metabolism | Leydig Cells, drug effects | Mitogen-Activated Protein Kinases, metabolism | PPAR gamma, agonists | Granulosa Cells, metabolism | Sterol Regulatory Element Binding Protein 1, metabolism | Cyclic CMP, pharmacology | Cyclic CMP, analogs and derivatives | Cyclic AMP Response Element Modulator, metabolism | Granulosa Cells, drug effects | Cyclic AMP Response Element-Binding Protein, metabolism | Thiazolidinediones, pharmacology
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 2014, Volume 111, Issue 3, pp. 518 - 530
Apart from transporting lipids through the body, the human plasma lipoproteins very low-density lipoprotein (VLDL) and low-density lipoprotein (LDL) are also... 
CETP | proteomics | atherothrombosis | LCAT | protein S | Apolipoproteins | coagulation | prenylcysteine oxidase | prothrombin | anti-microbial peptides | cathepcin D | nLC-MS/MS | dermicidin | TFPI-1 | lysozyme C | CD14 | PLTP | lipopolysaccharide binding protein | apoAV | Cardiovascular Biology and Cell Signalling | Prenylcysteine oxidase | Coagulation | Atherothrombosis | Cathepcin D | Lysozyme C | Lipopolysaccharide binding protein | Dermicidin | Antimicrobial peptides | Proteomics | Prothrombin | Protein S | ApoAV | PATTERNS | MASS-SPECTROMETRY | PARAOXONASE-1 | PERIPHERAL VASCULAR DISEASE | DYSLIPIDEMIA | HEMATOLOGY | OXIDATION | apolipoproteins | PROTHROMBINASE | 2-DIMENSIONAL GEL-ELECTROPHORESIS | REVERSE CHOLESTEROL TRANSPORT | ENHANCEMENT | CHYLOMICRONS | Apolipoproteins A - metabolism | Humans | Antimicrobial Cationic Peptides - metabolism | Lipopolysaccharide Receptors - metabolism | Phospholipid Transfer Proteins - metabolism | Protein S - metabolism | Dyslipidemias - blood | Plasma - metabolism | Mass Spectrometry | Blood Coagulation | Lipoproteins - metabolism | Lipoproteins, LDL - metabolism | Prothrombin - metabolism | Apolipoprotein A-V | Computational Biology | Lipid Metabolism | Muramidase - metabolism | Cathepsin D - metabolism | Carbon-Sulfur Lyases - metabolism | Atherosclerosis - blood | Lipoproteins, VLDL - metabolism | Blood Coagulation Disorders - blood | Cholesterol Ester Transfer Proteins - metabolism | Proteome - metabolism | Phosphatidylcholine-Sterol O-Acyltransferase - metabolism
Journal Article
British journal of nutrition, ISSN 0007-1145, 02/2014, Volume 111, Issue 3, pp. 432 - 444
Altered lipid metabolism has been shown in fish fed plant protein sources. The present study aimed to gain further insights into how intestinal and hepatic... 
Full Papers | Metabolism and Metabolic Studies | Plant meal | Quantitative real-time PCR | Soya-saponins | Lipoproteins | Sterols | MOLECULAR CHARACTERIZATION | RAINBOW-TROUT | X-RECEPTOR LXR | NUTRITION & DIETETICS | SOYBEAN-MEAL | FISH-MEAL | PLASMA-CHOLESTEROL CONCENTRATION | GENE-EXPRESSION | FECAL BILE-ACIDS | TROUT ONCORHYNCHUS-MYKISS | DISTAL INTESTINE | Sterols - antagonists & inhibitors | Liver - pathology | Sterols - metabolism | Diet - adverse effects | Fatty Liver - pathology | Salmo salar - metabolism | Intestines - metabolism | Salmo salar - growth & development | Sterols - blood | Liver - growth & development | Lipoproteins - antagonists & inhibitors | Dietary Proteins - metabolism | Fish Proteins - genetics | Anticholesteremic Agents - metabolism | Saponins - adverse effects | Gene Expression Regulation, Developmental | Lipoproteins - metabolism | Bile Acids and Salts - antagonists & inhibitors | Plant Proteins - metabolism | Salmo salar - blood | Weight Gain | Diet - veterinary | Fish Proteins - biosynthesis | Fatty Liver - metabolism | Fatty Liver - veterinary | Intestines - pathology | Liver - metabolism | Intestines - growth & development | Fish Proteins - metabolism | Bile Acids and Salts - metabolism | Lipid Metabolism | Lipoproteins - blood | Energy Intake | Anticholesteremic Agents - adverse effects | Plant Proteins - adverse effects | Soybeans - chemistry | Animals | Glutens - metabolism | Glutens - adverse effects | Lupinus - chemistry | Saponins - metabolism | Dietary Proteins - adverse effects | Triticum - chemistry | Fatty Liver - etiology | Lipids | Diet | Metabolism | Salmon
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2016, Volume 291, Issue 7, pp. 3254 - 3267
Hepatitis C virus (HCV) relies on host lipids and lipid droplets for replication and morphogenesis. The accumulation of lipid droplets in infected hepatocytes... 
TARGET | liver injury | OXIDATIVE STRESS | SREBP | hepatitis virus | CHOLESTEROL | INTERLEUKIN-1-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | lipid metabolism | NLRP3 inflammasome | DROPLETS | inflammation | PATHWAY | inflammasome | FATTY-ACID | CASPASES | INFECTION | EXPRESSION | Hepatitis C, Chronic - pathology | Inflammasomes - metabolism | Cytoskeletal Proteins - antagonists & inhibitors | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Golgi Apparatus - drug effects | Humans | Hepatitis C, Chronic - virology | Non-alcoholic Fatty Liver Disease - etiology | Caspase 1 - metabolism | Host-Pathogen Interactions - drug effects | Intracellular Signaling Peptides and Proteins - metabolism | Protein Transport - drug effects | Sterol Regulatory Element Binding Protein 1 - agonists | RNA Interference | Hepacivirus - physiology | Sterol Regulatory Element Binding Protein 2 - metabolism | Endopeptidases - metabolism | Carrier Proteins - antagonists & inhibitors | Serine Endopeptidases - chemistry | Cysteine Proteinase Inhibitors - pharmacology | Cell Line, Tumor | Hepatocytes - virology | Sterol Regulatory Element Binding Protein 2 - agonists | Hepatitis C, Chronic - metabolism | Hepatitis C, Chronic - physiopathology | Hepatocytes - pathology | Golgi Apparatus - pathology | Hepatocytes - metabolism | Golgi Apparatus - virology | Caspase 1 - chemistry | Proteolysis - drug effects | Endopeptidases - chemistry | Inflammasomes - drug effects | Enzyme Induction - drug effects | Cytoskeletal Proteins - metabolism | Membrane Proteins - metabolism | Hepatocytes - drug effects | Sterol Regulatory Element Binding Protein 1 - metabolism | Hepacivirus - drug effects | Proprotein Convertases - metabolism | Proprotein Convertases - chemistry | Carrier Proteins - genetics | Carrier Proteins - metabolism | Caspase 1 - genetics | CARD Signaling Adaptor Proteins | Lipogenesis - drug effects | Golgi Apparatus - metabolism | Serine Endopeptidases - metabolism | Microbiology
Journal Article
PloS one, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0173676
Autophagy is a catabolic mechanism to degrade cellular components to maintain cellular energy levels during starvation, a condition where PPAR alpha may be... 
INSULIN SIGNALING TRANSDUCTION | GLUCOSE-HOMEOSTASIS | FIBROBLAST-GROWTH-FACTOR-21 | DEACETYLATION | METABOLISM | PATHWAY | STEATOSIS | MULTIDISCIPLINARY SCIENCES | RESISTANCE | RECEPTORS | FOXO1 | TOR Serine-Threonine Kinases - metabolism | Autophagy-Related Protein 7 - metabolism | Fibroblast Growth Factors - genetics | Autophagy-Related Protein 5 - genetics | fas Receptor - metabolism | Autophagy - drug effects | Fibroblast Growth Factors - metabolism | TOR Serine-Threonine Kinases - genetics | Liver - drug effects | fas Receptor - genetics | Autophagy - genetics | Proto-Oncogene Proteins c-akt - metabolism | Forkhead Box Protein O1 - metabolism | Signal Transduction | Liver - metabolism | PPAR alpha - genetics | Ubiquitin-Conjugating Enzymes - genetics | Stearoyl-CoA Desaturase - genetics | Mice, Knockout | Triglycerides - metabolism | Sequestosome-1 Protein - genetics | Ubiquitin-Conjugating Enzymes - metabolism | Cysteine Endopeptidases - genetics | Autophagy-Related Protein 5 - metabolism | Beclin-1 - genetics | Stearoyl-CoA Desaturase - metabolism | Mice | PPAR alpha - metabolism | Autophagy-Related Proteins - antagonists & inhibitors | Blood Glucose - metabolism | Autophagy-Related Proteins - metabolism | Forkhead Box Protein O1 - genetics | Autophagy-Related Protein 5 - antagonists & inhibitors | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Fenofibrate - pharmacology | Cysteine Endopeptidases - metabolism | Autophagy-Related Proteins - genetics | Sequestosome-1 Protein - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Autophagy-Related Protein 7 - antagonists & inhibitors | Mice, Inbred C57BL | Autophagy-Related Protein 7 - genetics | Gene Expression Regulation - drug effects | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | PPAR alpha - agonists | Ubiquitin-Conjugating Enzymes - antagonists & inhibitors | Beclin-1 - metabolism | Transcription factors | Adipose tissue | Liver | Body weight | AKT protein | Biochemistry | Glucose | Assaying | Proteins | Signal transduction | Temperature effects | Fibroblasts | Physiology | Acetylation | Inhibition | Growth factors | Hepatotoxicity | Activation analysis | Methanol | Starvation | Ethanol | AMP | Metabolism | Insulin | Fatty acids | Studies | Acetaminophen | Food intake | Weight reduction | Animal welfare | Circulation | Drugs | Biotechnology | Drug abuse | Laboratories | Centrifugation | Glass | Homeostasis | Activation | Biology | Kinases | AMP-activated protein kinase | Autophagy | Nutrient status | Rodents | Nutrients | Oxidation | Heart diseases | Age | Epinephrine | AKT1 protein | Fasting | Chloroform | Diabetes mellitus | Cardiomyocytes | Acclimatization | Triglycerides | Pharmacology | Nitrogen | Calories | Medicine | Nuclear fuels | Protein kinase | Insulin resistance | Diabetes
Journal Article
Cell metabolism, ISSN 1550-4131, 2012, Volume 15, Issue 5, pp. 725 - 738
.... The liver is a key organ in insulin-mediated regulation of metabolism. To assess the role of hepatic mTORC2, we generated liver-specific rictor knockout (LiRiKO) mice... 
TRANSCRIPTION FACTORS | LIPID-METABOLISM | INSULIN-RESISTANCE | ELEMENT-BINDING PROTEIN-1C | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | MOTIF PHOSPHORYLATION | DIABETIC-NEPHROPATHY | GLUCOSE-UTILIZATION | CELL-GROWTH | PHOSPHOINOSITIDE 3-KINASE | CELL BIOLOGY | Glucose Intolerance - metabolism | Phosphorylation | Liver - enzymology | TOR Serine-Threonine Kinases - metabolism | Homeostasis | Glycogen Synthase Kinase 3 beta | Male | Hepatocytes - metabolism | Mechanistic Target of Rapamycin Complex 2 | Hyperglycemia - genetics | Proto-Oncogene Proteins c-akt - genetics | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Forkhead Transcription Factors - metabolism | Trans-Activators - genetics | Hyperinsulinism - genetics | Insulin - genetics | Lipogenesis | Proto-Oncogene Proteins c-akt - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Gluconeogenesis | Hyperinsulinism - metabolism | Glucose Intolerance - genetics | Signal Transduction | Liver - metabolism | Glucokinase - genetics | Glucose - genetics | Lipid Metabolism | Forkhead Transcription Factors - genetics | Glucokinase - metabolism | Glycogen Synthase Kinase 3 - metabolism | Mice, Knockout | Hyperglycemia - metabolism | Proteins - genetics | Insulin - metabolism | Animals | Proteins - metabolism | Trans-Activators - deficiency | Glycogen Synthase Kinase 3 - genetics | Sterol Regulatory Element Binding Protein 1 - genetics | Glucose - metabolism | Glycolysis | Trans-Activators - metabolism | Forkhead Box Protein O1 | Mice | Transcription Factors | Glucose metabolism | Hyperglycemia | Glucose | Isoenzymes | Dextrose
Journal Article