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Cancer science, ISSN 1347-9032, 07/2016, Volume 107, Issue 7, pp. 1039 - 1046
Antibody–drug conjugates deliver anticancer agents selectively and efficiently to tumor tissue and have significant antitumor efficacy with a wide therapeutic window. DS... 
topoisomerase I inhibitor | T‐DM | HER2 | bystander killing | Antibody‐drug conjugate | T-DM | Antibody-drug conjugate | Life Sciences & Biomedicine | Oncology | Science & Technology | Receptor, ErbB-2 - genetics | Breast Neoplasms - immunology | Humans | Ado-Trastuzumab Emtansine | Stomach Neoplasms - pathology | Maytansine - pharmacology | Immunoconjugates - immunology | Breast Neoplasms - enzymology | Immunoconjugates - pharmacology | Topoisomerase I Inhibitors - pharmacology | Neoplasms - genetics | Antibodies, Monoclonal, Humanized - pharmacology | Female | Camptothecin - immunology | Receptor, ErbB-2 - immunology | Camptothecin - analogs & derivatives | Stomach Neoplasms - enzymology | Stomach Neoplasms - genetics | Maytansine - analogs & derivatives | Neoplasms - enzymology | Stomach Neoplasms - immunology | Animals | Breast Neoplasms - genetics | Bystander Effect - drug effects | Breast Neoplasms - pathology | Mice, Nude | Neoplasms - immunology | Cell Membrane Permeability - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Antibodies, Monoclonal, Humanized - immunology | Neoplasms - pathology | Camptothecin - pharmacology | Maytansine - immunology | Trastuzumab | Viral antibodies | Epidermal growth factor | Imaging systems | Antibodies | Permeability | Drug therapy | Biopharmaceutics | Tumors | Antigens | Hematology | Laboratories | Toxicity | DNA topoisomerase | Polymerization | Clinical trials | Cytotoxicity | Breast cancer | ErbB-2 protein | Stomach cancer | Hypotheses | Antitumor agents | Xenografts | Antitumor activity | Lymphomas | Drug dosages | Payloads | Index Medicus | Original
Journal Article
PloS one, ISSN 1932-6203, 08/2011, Volume 6, Issue 8, p. e23888
Cervical cancer cells exhibit an increased requirement for ubiquitin-dependent protein degradation associated with an elevated metabolic turnover rate, and for... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Heat-Shock Response - drug effects | Cyclin D1 - metabolism | Papillomaviridae - drug effects | Humans | Ubiquitin - metabolism | Uterine Cervical Neoplasms - pathology | Papillomaviridae - genetics | Chloroquine - pharmacology | Proteolysis - drug effects | Ubiquitination - drug effects | Biocatalysis - drug effects | Female | Antineoplastic Agents - pharmacology | Stress, Physiological - drug effects | Cell Death - drug effects | Polyubiquitin - metabolism | Cell Survival - drug effects | Tumor Stem Cell Assay | Tumor Suppressor Protein p53 - metabolism | Antineoplastic Agents - chemistry | Cell Adhesion - drug effects | Drug Synergism | Keratinocytes - drug effects | Protein Stability - drug effects | Cell Line, Tumor | HSP90 Heat-Shock Proteins - metabolism | Uterine Cervical Neoplasms - virology | Proteasome Endopeptidase Complex - metabolism | Proteasome Inhibitors | Drug Screening Assays, Antitumor | Ubiquitin | Care and treatment | Chemical inhibitors | Heat shock proteins | Prevention | Cell death | Proteolysis | Cancer cells | Tumor proteins | Papillomavirus infections | Cervical cancer | Cancer | Isoflavones | Flow cytometry | Hsp90 protein | Triterpenoids | Chloroquine | p53 Protein | Hydrocarbons | Oncology | Biochemistry | Stomach cancer | Turnover rate | Ovarian cancer | Anticancer properties | Degradation | Proteins | Antioxidants | Genotype & phenotype | Human papillomavirus | Antitumor agents | Pathways | Protein folding | Cooperation | Cell cycle | Polyphenols | Inhibition | Catalysis | Carbonyls | Bortezomib | Gynecology | Breast cancer | Cervix | Obstetrics | Tea | Signaling | Flavonoids | Active sites | Womens health | Pancreatic cancer | Proteasomes | Molecular biology | Apoptosis
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2014, Volume 356, Issue 2, pp. 704 - 712
Journal Article
EMBO molecular medicine, ISSN 1757-4684, 09/2014, Volume 6, Issue 10, pp. 1279 - 1293
Epithelial‐mesenchymal transition (EMT) is a reversible and dynamic process hypothesized to be co‐opted by carcinoma during invasion and metastasis. Yet, there... 
drug response | microarray | gene expression signature | epithelial‐mesenchymal transition | prognosis | Drug response | Epithelial-mesenchymal transition | Prognosis | Microarray | Gene expression signature | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Lung Neoplasms - drug therapy | Oligonucleotide Array Sequence Analysis | Colorectal Neoplasms - genetics | Humans | Epithelial-Mesenchymal Transition - drug effects | Antineoplastic Agents - therapeutic use | Epithelial-Mesenchymal Transition - genetics | Ovarian Neoplasms - genetics | Urinary Bladder Neoplasms - genetics | Neoplasms - genetics | Colorectal Neoplasms - drug therapy | Female | Gene Expression Regulation, Neoplastic - drug effects | Ovarian Neoplasms - drug therapy | Lung Neoplasms - genetics | Stomach Neoplasms - genetics | Treatment Outcome | Transcriptome - drug effects | Transcriptome - genetics | Stomach Neoplasms - drug therapy | Urinary Bladder Neoplasms - drug therapy | Breast Neoplasms - drug therapy | Neoplasms - drug therapy | Disease-Free Survival | Breast Neoplasms - genetics | Cell Line, Tumor | Cancer patients | Chemotherapy | Patient outcomes | Stem cells | Development and progression | Drug therapy | Gene expression | Ovarian cancer | Cancer | Mesenchyme | Colorectal carcinoma | Genomes | Breast cancer | Metastasis | Kinases | Cancer therapies | Metastases | Breast carcinoma | Genotype & phenotype | Paclitaxel | Breast | Software | Tumors | epithelial-mesenchymal transition
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 4, pp. 784 - 792.e9
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 565 - 575.e4
... neurons, and the effects of TRPA1 activation on intestinal function have not been determined. We characterized expression of TRPA1 by enteric neurons and determined its involvement in the control of intestinal contractility and transit... 
Gastroenterology and Hepatology | TRP Channels | Enteric Nervous System | Motility | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Ganglia - metabolism | Colon - drug effects | Ileum - metabolism | Colon - innervation | Male | RNA, Messenger - metabolism | Muscle, Smooth - drug effects | Ileum - drug effects | Duodenum - metabolism | Muscle, Smooth - physiology | Stomach - innervation | Transient Receptor Potential Channels - metabolism | Isothiocyanates - pharmacology | Ileum - innervation | Neurons, Afferent - drug effects | Muscle, Smooth - innervation | Duodenum - physiology | Reverse Transcriptase Polymerase Chain Reaction | Cecum - drug effects | Colon - metabolism | Mice, Knockout | Motor Neurons - metabolism | Interneurons - metabolism | Muscle Contraction - drug effects | Muscle Contraction - physiology | Mice | Neurons, Afferent - metabolism | Duodenum - innervation | Acrolein - analogs & derivatives | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Colon - physiology | Gastrointestinal Motility - drug effects | Gastrointestinal Motility - physiology | Stomach - physiology | Substance P - pharmacology | Stomach - metabolism | Cecum - physiology | Cecum - metabolism | Aldehydes - pharmacology | Cecum - innervation | Female | Transient Receptor Potential Channels - physiology | Stomach - drug effects | Motor Neurons - drug effects | Acrolein - pharmacology | Mice, Inbred C57BL | Interneurons - drug effects | Transient Receptor Potential Channels - agonists | Carbachol - pharmacology | Animals | TRPA1 Cation Channel | Fluorescent Antibody Technique | Gastric Emptying - physiology | Ileum - physiology | Duodenum - drug effects | Physiological aspects | Neurons | Gastrointestinal diseases | Nitric oxide
Journal Article
PloS one, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35196
Nonsteroidal antiand/orinflammatory drugs are among the most commonly used prescription and over-the-counter medications, but they often produce significant gastrointestinal ulceration and bleeding... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Naproxen - analogs & derivatives | Pyrazoles - therapeutic use | Obesity - drug therapy | Rats, Wistar | Hypertension - drug therapy | Male | Nitrates - adverse effects | Nitrates - therapeutic use | Aspirin - adverse effects | Gastric Mucosa - drug effects | Aspirin - therapeutic use | Drug Therapy, Combination | Pyrazoles - adverse effects | Naproxen - adverse effects | Rats, Inbred SHR | Omeprazole - therapeutic use | Rats | Gastrointestinal Tract - drug effects | Omeprazole - adverse effects | Celecoxib | Naproxen - therapeutic use | Rats, Zucker | Animals | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Sulfonamides - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Arthritis - drug therapy | Sulfonamides - adverse effects | Gastrointestinal Diseases - chemically induced | Hypertension | Hydrogen sulfide | Omeprazole | COX-2 inhibitors | Aspirin | Nitric oxide | Naproxen | Arthritis | Nonprescription drugs | Sulfide | Drugs | Animal models | Toxicity | Hydrogen | Mucosa | Bleeding | Prostaglandin endoperoxide synthase | Na+/Ca2+-exchanging ATPase | Intestine | Rodents | Older people | Gastroenterology | Bacteria | Trends | Nonsteroidal anti-inflammatory drugs | Damage | Drug dosages | Hydrogen ion concentration | Medical research | Inflammation | Medical screening | Patients | Anti-inflammatory agents | Ulcers | Health risk assessment | Geriatrics
Journal Article