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1. Ulcer disease
: new aspects of pathogenesis and pharmacology
by Szabo, Sandor and Pfeiffer, Carl J
1989, CRC series in gastrointestinal disease., ISBN 0849362164, 461
Stomach | Evaluation | Animal models | Chemotherapy | Duodenum | Pathogenesis | Gastrointestinal system | Ulcers | Rats | Antiulcer drugs | Testing | Diseases
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2. Full Text Comorbidity in severe asthma requiring systemic corticosteroid therapy: Cross-sectional data from the optimum patient care research database and the british thoracic difficult asthma registry
by Sweeney, Joan and Patterson, Chris C and Menzies-Gow, Andrew and Niven, Rob M and Mansur, Adel H and Bucknall, Christine and Chaudhuri, Rekha and Price, David and Brightling, Chris E and Heaney, Liam G and British Thoracic Soc Difficult Ast and British Thoracic Society Difficult Asthma Network
Thorax, ISSN 0040-6376, 04/2016, Volume 71, Issue 4, pp. 339 - 346
Objective To determine the prevalence of systemic corticosteroid-induced morbidity in severe asthma. Design Cross-sectional observational study. Setting The... 
TERM STEROID-THERAPY | RESPIRATORY SYSTEM | CUMULATIVE BURDEN | ECONOMIC-IMPLICATIONS | DISEASE | FREQUENCY | ADULTS | RISK | COMPLICATIONS | QUALITY-OF-LIFE | ADVERSE EVENTS | Glucocorticoids - administration & dosage | Prevalence | Cataract - chemically induced | Humans | Middle Aged | Duodenal Ulcer - chemically induced | Male | Diabetes Mellitus, Type 2 - epidemiology | Adult | Female | Registries | Sleep Apnea, Obstructive - chemically induced | Glucocorticoids - adverse effects | Osteoporosis - epidemiology | Asthma - diagnosis | Body Mass Index | Severity of Illness Index | Stomach Ulcer - chemically induced | Obesity - chemically induced | Osteoporosis - chemically induced | Asthma - physiopathology | Cross-Sectional Studies | Administration, Oral | Risk Factors | Asthma - drug therapy | Obesity - epidemiology | Quality of Life | Sex Distribution | Aged | Diabetes Mellitus, Type 2 - chemically induced | United Kingdom - epidemiology | Care and treatment | Dosage and administration | Corticosteroids | Management | Patients | Asthma
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3. Full Text The role of endoscopy in the management of patients with peptic ulcer disease
by Banerjee, Subhas and Cash, Brooks D and Dominitz, Jason A and Baron, Todd H and Anderson, Michelle A and Ben-Menachem, Tamir and Fisher, Laurel and Fukami, Norio and Harrison, M. Edwyn and Ikenberry, Steven O and Khan, Khalid and Krinsky, Mary Lee and Maple, John and Fanelli, Robert D and Strohmeyer, Laura and ASGE STAND PRACTICE COMM and ASGE Standards of Practice Committee
Gastrointestinal Endoscopy, ISSN 0016-5107, 2010, Volume 71, Issue 4, pp. 663 - 668
Gastroenterology and Hepatology | GIANT DUODENAL-ULCER | UNITED-STATES | DIAGNOSIS | RISK-FACTORS | GASTRIC OUTLET OBSTRUCTION | GASTROINTESTINAL-TRACT | PYLORIC-STENOSIS | DOUBLE-BLIND | TERM-FOLLOW-UP | BALLOON DILATION | GASTROENTEROLOGY & HEPATOLOGY | Humans | Peptic Ulcer Perforation - chemically induced | Stomach Ulcer - pathology | Duodenal Ulcer - chemically induced | Duodenal Ulcer - diagnosis | Gastric Mucosa - pathology | Helicobacter Infections - complications | Stomach Neoplasms - pathology | Peptic Ulcer Perforation - diagnosis | Helicobacter Infections - pathology | Helicobacter Infections - therapy | Peptic Ulcer Hemorrhage - pathology | Peptic Ulcer Perforation - therapy | Helicobacter pylori | Gastric Outlet Obstruction - diagnosis | Gastric Outlet Obstruction - therapy | Gastric Outlet Obstruction - pathology | Helicobacter Infections - diagnosis | Endoscopy, Digestive System | Duodenal Neoplasms - pathology | Stomach Ulcer - diagnosis | Stomach Ulcer - chemically induced | Diagnosis, Differential | Peptic Ulcer Perforation - pathology | Duodenal Ulcer - pathology | Stomach Neoplasms - diagnosis | Peptic Ulcer Hemorrhage - chemically induced | Stomach Neoplasms - therapy | Duodenal Neoplasms - therapy | Duodenal Neoplasms - diagnosis | Evidence-Based Medicine | Peptic Ulcer Hemorrhage - therapy | Duodenal Ulcer - therapy | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Biopsy | Duodenum - pathology | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Gastric Outlet Obstruction - chemically induced | Stomach Ulcer - therapy | Peptic Ulcer Hemorrhage - diagnosis | Intestinal Mucosa - pathology | Peptic ulcer | Care and treatment
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4. Full Text Gastric Mucosal Defense and Cytoprotection: Bench to Bedside
by Laine, Loren and Takeuchi, Koji and Tarnawski, Andrzej
Gastroenterology, ISSN 0016-5085, 2008, Volume 135, Issue 1, pp. 41 - 60
The gastric mucosa maintains structural integrity and function despite continuous exposure to noxious factors, including 0.1 mol/L HCl and pepsin, that are... 
Gastroenterology and Hepatology | PROSTAGLANDIN EP1 RECEPTORS | UPPER GASTROINTESTINAL HEMORRHAGE | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | DOUBLE-BLIND TRIAL | DUODENAL BICARBONATE SECRETION | STRESS-ULCER PROPHYLAXIS | RANDOMIZED CONTROLLED-TRIAL | GASTROENTEROLOGY & HEPATOLOGY | CRITICALLY-ILL PATIENTS | INTENSIVE-CARE UNIT | REQUIRING MECHANICAL VENTILATION | Stomach Ulcer - chemically induced | Anti-Ulcer Agents - therapeutic use | Gastritis - drug therapy | Humans | Stomach Ulcer - drug therapy | Gastric Mucosa - metabolism | Gastritis - physiopathology | Stomach Ulcer - physiopathology | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Gastritis - chemically induced | Gastric Mucosa - drug effects | Gastric Mucosa - physiopathology | Cyclooxygenase Inhibitors - therapeutic use | Prostanoids | Index Medicus | Abridged Index Medicus
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5. Full Text Risk of Upper Gastrointestinal Complications Among Users of Traditional NSAIDs and COXIBs in the General Population
by García Rodríguez, Luis A and Barreales Tolosa, Laura
Gastroenterology, ISSN 0016-5085, 2007, Volume 132, Issue 2, pp. 498 - 506
Background & Aims: Traditional nonaspirin, nonsteroidal anti-inflammatory drugs (tNSAIDs) have been associated with a 3- to 5-fold increased risk in upper... 
Gastroenterology and Hepatology | SELECTIVE CYCLO-OXYGENASE-2 INHIBITORS | VARIABILITY | TRIALS | METAANALYSIS | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | HEMORRHAGE | ULCER | ROFECOXIB | NAPROXEN | ARTHRITIS | GASTROENTEROLOGY & HEPATOLOGY | Humans | Peptic Ulcer Perforation - chemically induced | Half-Life | Warfarin - adverse effects | Duodenal Ulcer - chemically induced | Case-Control Studies | Dose-Response Relationship, Drug | Cyclooxygenase 2 Inhibitors - adverse effects | Peptic Ulcer Perforation - epidemiology | Drug Interactions | Time Factors | Aspirin - adverse effects | Duodenal Ulcer - epidemiology | Retrospective Studies | Steroids - adverse effects | Stomach Ulcer - chemically induced | Risk Assessment | Cyclooxygenase 2 Inhibitors - administration & dosage | Risk Factors | Anti-Ulcer Agents - adverse effects | Peptic Ulcer Hemorrhage - chemically induced | Anticoagulants - adverse effects | Cyclooxygenase 2 Inhibitors - pharmacokinetics | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Delayed-Action Preparations - adverse effects | Stomach Ulcer - epidemiology | Upper Gastrointestinal Tract - drug effects | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Anti-Inflammatory Agents, Non-Steroidal - pharmacokinetics | Peptic Ulcer Hemorrhage - epidemiology | United Kingdom - epidemiology | Gastrointestinal Diseases - chemically induced | Cohort Studies | Complications and side effects | Nonsteroidal anti-inflammatory drugs
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6. Full Text Diosmin protects against ethanol-induced gastric injury in rats: Novel anti-ulcer actions
by Arab, Hany H and Salama, Samir A and Omar, Hany A and Arafa, El-Shaimaa A and Maghrabi, Ibrahim A
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0122417 - e0122417
Alcohol consumption has been commonly associated with gastric mucosal lesions including gastric ulcer. Diosmin (DIO) is a natural citrus flavone with... 
OXIDATIVE STRESS | MUCOSAL INJURY | ANTIOXIDANT | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | ULCER | SUCRALFATE | NF-KAPPA-B | EXPRESSION | PROSTAGLANDIN E-2 | MODULATION | Stomach Ulcer - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Rats, Wistar | Sucralfate - administration & dosage | Diosmin - administration & dosage | Rats | Stomach Ulcer - pathology | Male | Diosmin - pharmacology | Anti-Ulcer Agents - administration & dosage | Gene Expression Regulation - drug effects | Animals | Ethanol - adverse effects | Anti-Ulcer Agents - pharmacology | Oxidative Stress - drug effects | Stomach Ulcer - prevention & control | Disease Models, Animal | Peroxidase - metabolism | Sucralfate | Antioxidants | Alcohol | Alcohol, Denatured | Cytochrome | Oxidative stress | Bcl-2 protein | Pathogenesis | Mucosa | Interleukin | Liver | Prostaglandin E2 | Biochemistry | Caspase-3 | Toxicology | Rodents | Tumor necrosis factor-TNF | Peroxides | Peroxidase | Pretreatment | Lesions | Heart diseases | Injuries | Glutathione | Glutathione peroxidase | NF-κB protein | Cell survival | Cytokines | Ethanol | Neutrophils | Caspase | Pharmacology | Inflammation | Tumor necrosis factor-α | Lymphoma | Cytochrome c | White blood cells | Lymphocytes B | Nitric oxide | Ulcers | Pharmacy | Interleukin 10 | Laboratory animals | Apoptosis | Index Medicus
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7. Full Text Protective activity of salidroside against ethanol-induced gastric ulcer via the MAPK/NF-κB pathway in vivo and in vitro
by Chang, Xiayun and Luo, Fen and Jiang, Wenjiao and Zhu, Lingpeng and Gao, Jin and He, He and Wei, Tingting and Gong, Shilin and Yan, Tianhua
International Immunopharmacology, ISSN 1567-5769, 09/2015, Volume 28, Issue 1, pp. 604 - 615
Salidroside (Sal) is a traditional Chinese medicine with various pharmacological effects. The present study aimed to investigate the protective effect of Sal... 
Salidroside | Inflammation | Ethanol | MAPKs/NF-κB | Gastric ulcer | ACTIVATION | OXIDATIVE DAMAGE | INVOLVEMENT | GASTROPROTECTIVE ACTIVITY | IMMUNOLOGY | SYNTHETIC SALIDROSIDE | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | CELL | MAPKs/NF-kappa B | KINASE C-ZETA | MUCOSAL DAMAGE | EXTRACT | Humans | Stomach Ulcer - pathology | Male | NF-kappa B - metabolism | Leukotriene B4 - metabolism | Anti-Ulcer Agents - pharmacology | Glucosides - therapeutic use | Stomach - drug effects | Cytokines - blood | Phenols - pharmacology | Stomach Ulcer - chemically induced | Anti-Ulcer Agents - therapeutic use | Cell Line | Cell Survival - drug effects | Glucosides - pharmacology | Stomach Ulcer - metabolism | Stomach - pathology | Arachidonate 5-Lipoxygenase - metabolism | Cytokines - metabolism | Hydrogen Peroxide - pharmacology | Stomach Ulcer - drug therapy | Animals | Signal Transduction - drug effects | Phenols - therapeutic use | Cyclooxygenase 2 - metabolism | Mice, Inbred BALB C | Mitogen-Activated Protein Kinases - metabolism
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8. Full Text Prevalence and incidence of gastroduodenal ulcers during treatment with vascular protective doses of aspirin
by Yeomans, N.D and Lanas, A.I and Talley, N.J and Thomson, A.B.R and Daneshjoo, R and Eriksson, B and Appelman-Eszczuk, S and Långström, G and Naesdal, J and Serrano, P and Singh, M and Skelly, M.M and Hawkey, C.J
Alimentary Pharmacology and Therapeutics, ISSN 0269-2813, 11/2005, Volume 22, Issue 9, pp. 795 - 801
Background: Aspirin is valuable for preventing vascular events, but information about ulcer frequency is necessary to inform risk-benefit decisions in... 
NONSTEROIDAL ANTIINFLAMMATORY DRUGS | PREVENTION | RISK | PHARMACOLOGY & PHARMACY | COMPLICATIONS | GASTROENTEROLOGY & HEPATOLOGY | PEPTIC-ULCER | ASSOCIATION | Stomach Ulcer - chemically induced | Risk Assessment - methods | Prevalence | Humans | Middle Aged | Risk Factors | Duodenal Ulcer - chemically induced | Male | Duodenal Ulcer - physiopathology | Stomach Ulcer - physiopathology | Incidence | Aspirin - adverse effects | Stomach Ulcer - epidemiology | Duodenal Ulcer - epidemiology | Female | Aged | Endoscopy, Gastrointestinal - methods | Platelet Aggregation Inhibitors - adverse effects
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9. Full Text Use of single and combined antithrombotic therapy and risk of serious upper gastrointestinal bleeding: population based case-control study
by Hallas, Jesper and Dall, Michael and Andries, Alin and Andersen, Birthe Søgaard and Aalykke, Claus and Hansen, Jane Møller and Andersen, Morten and Lassen, Annmarie Touborg
BMJ, ISSN 0959-8138, 10/2006, Volume 333, Issue 7571, pp. 726 - 728
Abstract Objectives To assess the risk of serious upper gastrointestinal bleeding associated with the newer antithrombotic agents used alone or in combination... 
Case control studies | Nonsteroidal antiinflammatory agents | Gastrointestinal agents | Serotonin agents | Antiulcer agents | Peptic ulcer | Research | Drug prescriptions | Dosage | Bleeding | Vitamin K | MEDICINE, GENERAL & INTERNAL | PERCUTANEOUS CORONARY INTERVENTION | CONTROLLED TRIAL | CLOPIDOGREL | EVENTS | MYOCARDIAL-INFARCTION | PRETREATMENT | PREVENTION | ELEVATION | ORAL ANTIPLATELET THERAPY | ASPIRIN | Stomach Ulcer - chemically induced | Fibrinolytic Agents - adverse effects | Gastrointestinal Hemorrhage - chemically induced | Aspirin - adverse effects | Humans | Peptic Ulcer Hemorrhage - chemically induced | Female | Male | Aged | Drug Therapy, Combination | Case-Control Studies | Platelet Aggregation Inhibitors - adverse effects
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10. Full Text Celecoxib versus Diclofenac and Omeprazole in Reducing the Risk of Recurrent Ulcer Bleeding in Patients with Arthritis
by Chan, Francis K.L and Hung, Lawrence C.T and Suen, Bing Y and Wu, Justin C.Y and Lee, Kenneth C and Leung, Vincent K.S and Leung, Wai K and Hui, Aric J and To, Ka F and Wong, Vincent W.S and Chung, S.C. Sydney and Sung, Joseph J.Y
The New England Journal of Medicine, ISSN 0028-4793, 12/2002, Volume 347, Issue 26, pp. 2104 - 2110
This randomized, controlled trial compared treatment with celecoxib and treatment with diclofenac plus omeprazole in patients with arthritis and ulcer... 
RHEUMATOID-ARTHRITIS | RANDOMIZED CONTROLLED TRIAL | MEDICINE, GENERAL & INTERNAL | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | DOUBLE-BLIND | PREVENTION | ROFECOXIB | SERIOUS GASTROINTESTINAL COMPLICATIONS | OSTEOARTHRITIS | MISOPROSTOL | CYCLOOXYGENASE-2 INHIBITION | Prospective Studies | Cyclooxygenase 2 | Humans | Duodenal Ulcer - chemically induced | Secondary Prevention | Prostaglandin-Endoperoxide Synthases | Peptic Ulcer Hemorrhage - prevention & control | Drug Therapy, Combination | Duodenal Ulcer - prevention & control | Stomach Ulcer - prevention & control | Stomach Ulcer - chemically induced | Anti-Ulcer Agents - therapeutic use | Diclofenac - therapeutic use | Omeprazole - therapeutic use | Double-Blind Method | Diclofenac - adverse effects | Pyrazoles | Risk Factors | Peptic Ulcer Hemorrhage - chemically induced | Probability | Celecoxib | Membrane Proteins | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Cyclooxygenase 2 Inhibitors | Sulfonamides - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Arthritis - drug therapy | Sulfonamides - adverse effects | Helicobacter pylori - isolation & purification | Cyclooxygenase Inhibitors - adverse effects | Cyclooxygenase Inhibitors - therapeutic use | Isoenzymes - antagonists & inhibitors | Prevention | Omeprazole | Stomach ulcer | Evaluation | Arthritis | Drug therapy | Diclofenac | Drug dosages | Ulcers
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11. Full Text Hydrogen sulfide enhances ulcer healing in rats
by Wallace, John L and Dicay, Michael and McKnight, Webb and Martin, Gary R
FASEB Journal, ISSN 0892-6638, 12/2007, Volume 21, Issue 14, pp. 4070 - 4076
Hydrogen sulfide is an endogenous mediator that relaxes vascular smooth muscle, exhibits several antiinflammatory activities, and contributes to gastric... 
Mucosal defense | Nitric oxide | Nonsteroidal antiinflammatory drugs | Resolution | CELLS | nitric oxide | MUCOSAL LESIONS | STIMULATION | K-ATP CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GASTROINTESTINAL-TRACT | INJURY | resolution | mucosal defense | CELL BIOLOGY | INHIBITION | nonsteroidal antiinflammatory drugs | DRUGS | BIOLOGY | NITRIC-OXIDE | GROWTH-FACTOR | Hydrogen Sulfide - metabolism | Stomach Ulcer - chemically induced | Hydrogen Sulfide - pharmacology | Stomach Ulcer - metabolism | Rats, Wistar | Stomach Ulcer - drug therapy | Acetic Acid - administration & dosage | Rats | Thioamides - pharmacology | Male | Organothiophosphorus Compounds - pharmacology | Stomach Ulcer - enzymology | Animals | Acetic Acid - toxicity | Intubation, Gastrointestinal | Wound Healing - physiology | Wound Healing - drug effects
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12. Full Text Prostaglandins, NSAIDs, and Gastric Mucosal Protection: Why Doesn't the Stomach Digest Itself?
by John L. Wallace
Physiological Reviews, ISSN 0031-9333, 10/2008, Volume 88, Issue 4, pp. 1547 - 1565
Except in rare cases, the stomach can withstand exposure to highly concentrated hydrochloric acid, refluxed bile salts, alcohol, and foodstuffs with a wide... 
ISCHEMIA-REPERFUSION INJURY | PHYSIOLOGY | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | ACID BACK-DIFFUSION | MUCUS-BICARBONATE BARRIER | NITRIC-OXIDE SYNTHASE | INDUCED GASTROINTESTINAL DAMAGE | PLATELET-ACTIVATING-FACTOR | PROSTACYCLIN IP RECEPTOR | ENDOTHELIAL GROWTH-FACTOR | ASPIRIN-INDUCED ULCERATION | Stomach Ulcer - chemically induced | Prostaglandins - physiology | Animals | Anti-Inflammatory Agents, Non-Steroidal - adverse effects | Gastric Mucosa - physiology | Humans | Gastric Mucosa - drug effects | Cyclooxygenase 1 - physiology | Stomach Ulcer - pathology | Cyclooxygenase 2 - physiology | Regional Blood Flow - physiology | Gastric Mucosa - blood supply | Physiological aspects | Gastrointestinal mucosa | Prostaglandins | Nonsteroidal anti-inflammatory drugs | Health aspects | Index Medicus
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