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Nature immunology, ISSN 1529-2916, 03/2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific... 
Life Sciences & Biomedicine | Immunology | Science & Technology | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis | Index Medicus
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 06/2013, Volume 114, Issue 12, pp. 1677 - 1688
Neutrophils serve as an intriguing model for the study of innate immune cellular activity induced by physiological stress... 
Endurance exercise | Systemic inflammatory response | Neutrophil transcriptome | Counterregulation of inflammation | Muscle-derived damage-associated molecule patterns | Physiology | Sport Sciences | Life Sciences & Biomedicine | Science & Technology | Toll-Like Receptor 2 - genetics | Gene Expression - genetics | Humans | Receptors, Interleukin-1 - genetics | Transcriptome | Complement System Proteins - immunology | Hydrocortisone - metabolism | Male | Physical Endurance - genetics | Complement System Proteins - metabolism | Leukocytes - immunology | Signal Transduction - immunology | Hydrocortisone - immunology | Gene Expression - immunology | Interleukin-10 - metabolism | Adult | Complement System Proteins - genetics | Transcription, Genetic | Hydrocortisone - blood | Cytokines - genetics | Interleukin-6 - metabolism | Neutrophils - metabolism | Cytokines - blood | Cytokines - immunology | Biomarkers - metabolism | Cytokines - metabolism | Interleukin-6 - genetics | Receptors, Interleukin-1 - immunology | Stress, Physiological - genetics | Neutrophils - immunology | Toll-Like Receptor 4 - genetics | Gene Expression Profiling - methods | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Biomarkers - blood | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Receptors, Interleukin-1 - metabolism | Hydrocortisone - genetics | Interleukin-10 - genetics | Interleukin-6 - immunology | Toll-Like Receptor 2 - immunology | Exercise - physiology | Interleukin-10 - immunology | Leukocytes - metabolism | Physical Endurance - immunology | Stress, Physiological - immunology | Index Medicus
Journal Article
Immunological reviews, ISSN 1600-065X, 01/2009, Volume 227, Issue 1, pp. 150 - 160
.... Studies of humans and mice deficient in these signaling subunits have provided surprising insights into the physiological functions of DAP10 and DAP12, demonstrating that they can either activate... 
DAP12 | immunoreceptor tyrosine‐based activation motif | innate immunity | DAP10 | Innate immunity | Immunoreceptor tyrosine-based activation motif | Life Sciences & Biomedicine | Immunology | Science & Technology | Autoantigens - metabolism | Membrane Glycoproteins - metabolism | Humans | Host-Pathogen Interactions - immunology | Signal Transduction - immunology | Feedback, Physiological - immunology | Adaptor Proteins, Signal Transducing - immunology | Membrane Proteins - metabolism | Semaphorins - immunology | Conserved Sequence - immunology | Membrane Glycoproteins - immunology | Receptors, Immunologic - immunology | Natural Cytotoxicity Triggering Receptor 2 - immunology | Infection - immunology | Membrane Proteins - genetics | Membrane Proteins - immunology | Semaphorins - metabolism | Carbohydrates - immunology | Immunity, Innate | Animals | Triggering Receptor Expressed on Myeloid Cells-1 | Autoantigens - immunology | Adaptor Proteins, Signal Transducing - genetics | Natural Cytotoxicity Triggering Receptor 2 - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Stress, Physiological - immunology | Evolution, Molecular | Physiological aspects | Research | Dendritic cells | Oncology, Experimental | Cancer | Index Medicus | ITAM
Journal Article
Acta physiologica (Oxford), ISSN 1748-1708, 07/2015, Volume 214, Issue 3, pp. 329 - 348
Journal Article