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Seminars in immunopathology, ISSN 1863-2300, 10/2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | Pathology | Life Sciences & Biomedicine | Science & Technology | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Index Medicus | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2009, Volume 297, Issue 6, pp. 2096 - 2108
Diabetic cardiomyopathy is an important contributor to diastolic and systolic heart failure. We examined the nature and mechanism of the cardiomyopathy in... 
Sarco(endo)plasmic reticulum calcium-ATPase 2a | Insulin | Fibrosis | Hypertrophy | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiomyopathies - diagnostic imaging | Mitochondria, Heart - metabolism | Age Factors | Diastole | Natriuretic Peptide, Brain - metabolism | Diabetes Mellitus, Type 1 - metabolism | Myocardial Contraction - drug effects | Male | Diabetes Mellitus, Type 1 - diagnostic imaging | Diabetes Mellitus, Type 1 - complications | Systole | Insulin - blood | Echocardiography, Doppler, Pulsed | Myosin Heavy Chains - metabolism | Cardiomyopathies - genetics | Cardiomyopathies - physiopathology | Diglycerides - metabolism | Ventricular Dysfunction, Left - genetics | Mice, Mutant Strains | Myocardium - metabolism | Lipid Metabolism - genetics | Insulin - genetics | Fatty Acids - metabolism | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Cardiomyopathies - drug therapy | Insulin - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Ceramides - metabolism | Acyl-CoA Dehydrogenase, Long-Chain - metabolism | Diabetes Mellitus, Type 1 - physiopathology | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - genetics | Disease Progression | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Diabetes Mellitus, Type 1 - drug therapy | Hypoglycemic Agents - pharmacology | Triglycerides - metabolism | Stroke Volume | Ventricular Dysfunction, Left - metabolism | Animals | Ventricular Dysfunction, Left - drug therapy | Cardiomyopathies - metabolism | Lipid Metabolism - drug effects | Mice | Ventricular Pressure | Blood Glucose - metabolism | Heart failure | Complications and side effects | Prognosis | Cardiomyopathy | Type 1 diabetes | Heart diseases | Risk factors | Heart | Rodents | Oxidation | Glucose | Diabetes | Kinases | Index Medicus
Journal Article
Molecular neurobiology, ISSN 0893-7648, 7/2016, Volume 53, Issue 5, pp. 3154 - 3167
Ischemia/reperfusion (I/R) injury after middle cerebral artery occlusion (MCAO) induces detrimental processes such as oxidative stress, inflammation, and... 
Neurology | Neurosciences | Biomedicine | Neurobiology | ROS | Rac-1 | Fibulin-5 | Ischemia/reperfusion | Cell Biology | Apoptosis | Blood–brain barrier | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Reperfusion Injury - etiology | Phosphorylation | Reactive Oxygen Species - metabolism | Humans | Caspase 3 - metabolism | Stroke - complications | Male | Blood-Brain Barrier - ultrastructure | Occludin - metabolism | Brain - metabolism | Stroke - pathology | Matrix Metalloproteinase 9 - metabolism | Brain Edema - complications | Infarction, Middle Cerebral Artery - complications | Proteolysis | HEK293 Cells | Recombination, Genetic - genetics | Proto-Oncogene Proteins c-akt - metabolism | Reperfusion Injury - metabolism | Superoxide Dismutase - metabolism | Calcium-Binding Proteins - metabolism | Extravasation of Diagnostic and Therapeutic Materials - pathology | Brain Edema - metabolism | Reperfusion Injury - pathology | Endothelial Cells - metabolism | Brain Edema - pathology | Rats | Infarction, Middle Cerebral Artery - pathology | Extravasation of Diagnostic and Therapeutic Materials - complications | Rats, Sprague-Dawley | Blood-Brain Barrier - metabolism | Blood-Brain Barrier - pathology | Magnetic Resonance Imaging | Stroke - metabolism | Animals | Brain - pathology | Adenoviridae - metabolism | rac1 GTP-Binding Protein - metabolism | Antioxidants | Brain | Superoxide | Permeability | Injuries | Ischemia | Gene expression | Rodents | Veins & arteries | Index Medicus
Journal Article
Molecular neurobiology, ISSN 1559-1182, 01/2017, Volume 55, Issue 2, pp. 1082 - 1096
Multi-protein complexes, termed “inflammasomes,” are known to contribute to neuronal cell death and brain injury following ischemic stroke. Ischemic stroke... 
Neurology | Neurosciences | Stroke | Biomedicine | Neuronal cell death | Neurobiology | NLRP | Inflammasome | Cell Biology | Nuclear factor kappa B | Mitogen-activated protein kinases | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Inflammasomes - metabolism | Nitriles - pharmacology | Brain Ischemia - metabolism | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | NF-kappa B - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Sulfones - pharmacology | Brain - metabolism | Inflammasomes - drug effects | Neurons - metabolism | Neurons - drug effects | Butadienes - pharmacology | NF-kappa B - antagonists & inhibitors | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Imidazoles - pharmacology | Anthracenes - pharmacology | Apoptosis Regulatory Proteins - metabolism | Brain - drug effects | Stroke - metabolism | Animals | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Pyridines - pharmacology | Adaptor Proteins, Signal Transducing - metabolism | Brain | NF-κB protein | Immunoglobulins | Intravenous administration | Oligomerization | Bcl-2 protein | Therapeutic applications | Neurons | MAP kinase | Interleukin 18 | Proteins | Signal transduction | Pyrin protein | Bcl-x protein | Ischemia | Cell death | Brain injury | Apoptosis | Index Medicus
Journal Article