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Free Radical Biology and Medicine, ISSN 0891-5849, 11/2017, Volume 112, pp. 336 - 349
Aberrant modulation of mitochondrial dynamic network, which shifts the balance of fusion and fission towards fission, is involved in brain damage of various... 
Mdivi-1 | Inflammation | Early brain injury | Blood-brain barrier disruption | Subarachnoid hemorrhage | Apoptosis | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | COMPLEX I INHIBITOR | HIPPOCAMPAL-NEURONS | RAT MODEL | MITOCHONDRIAL FISSION | CELL-DEATH | SIGNALING PATHWAY | MOUSE MODEL | ENDOCRINOLOGY & METABOLISM | NF-KAPPA-B | Interleukin-6 - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Proto-Oncogene Proteins c-bcl-2 - agonists | Tumor Necrosis Factor-alpha - genetics | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Subarachnoid Space - drug effects | Occludin - metabolism | Matrix Metalloproteinase 9 - metabolism | Interleukin-1beta - metabolism | Matrix Metalloproteinase 9 - genetics | Subarachnoid Hemorrhage - mortality | Zonula Occludens-1 Protein - metabolism | Interleukin-6 - metabolism | Subarachnoid Hemorrhage - drug therapy | Interleukin-1beta - antagonists & inhibitors | Zonula Occludens-1 Protein - genetics | NF-kappa B - antagonists & inhibitors | Endoplasmic Reticulum Stress - drug effects | Interleukin-6 - genetics | bcl-2-Associated X Protein - metabolism | Dynamins - genetics | Rats | Subarachnoid Hemorrhage - genetics | Rats, Sprague-Dawley | Blood-Brain Barrier - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Survival Analysis | Subarachnoid Space - metabolism | Tumor Necrosis Factor-alpha - metabolism | Dynamins - metabolism | Mitochondrial Dynamics - genetics | Claudin-5 - metabolism | Occludin - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Mitochondria - genetics | Quinazolinones - pharmacology | bcl-2-Associated X Protein - genetics | Dynamins - antagonists & inhibitors | Claudin-5 - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Subarachnoid Hemorrhage - pathology | bcl-2-Associated X Protein - antagonists & inhibitors | Blood-Brain Barrier - drug effects | Animals | Mitochondrial Dynamics - drug effects | NF-kappa B - genetics | Subarachnoid Space - pathology | Proto-Oncogene Proteins c-bcl-2 - genetics | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Stroke (Disease) | Proteins | Medical research | Brain | Neurons | Analysis | Medicine, Experimental | Brain damage | Injuries
Journal Article
American Journal of Physiology. Heart and Circulatory Physiology, ISSN 0363-6135, 11/2016, Volume 311, Issue 5, pp. H1132 - H1138
Insulin affects multiple important central nervous system (CNS) functions including memory and appetite, yet the pathway(s) by which insulin reaches brain... 
Injections, Intravenous | Iodine Radioisotopes | Microvessels/metabolism | Enzyme-Linked Immunosorbent Assay | Cerebrospinal Fluid/metabolism | Injections, Intraventricular | Extracellular Fluid/metabolism | Rats | Blood-Brain Barrier/metabolism | Male | Receptor, Insulin/antagonists & inhibitors | Rats, Sprague-Dawley | Pia Mater/blood supply | Subarachnoid Space/metabolism | Animals | Endothelial Cells/metabolism | Biological Transport | Diet, High-Fat | Insulin/pharmacokinetics | Glucose Clamp Technique | In Vitro Techniques | Hypoglycemic Agents/pharmacokinetics | Brain interstitial fluid | Blood-brain barrier | Insulin receptor | High-fat diet | Virchow-Robin space | blood-brain barrier | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | BARRIER | MUSCLE | RATS | SENSITIVITY | RECEPTOR | CEREBROSPINAL-FLUID | brain interstitial fluid | TRANSPORT | PLASMA | high-fat diet | DOGS | PERIPHERAL VASCULAR DISEASE | CENTRAL-NERVOUS-SYSTEM | insulin receptor | Hypoglycemic Agents - pharmacokinetics | Endothelial Cells - metabolism | Microvessels - metabolism | Pia Mater - blood supply | Receptor, Insulin - antagonists & inhibitors | Blood-Brain Barrier - metabolism | Extracellular Fluid - metabolism | Cerebrospinal Fluid - metabolism | Receptor, Insulin - metabolism | Subarachnoid Space - metabolism | Insulin - pharmacokinetics | Health aspects | Insulin | Neural circuitry | Call for Papers
Journal Article
Journal Article
Brain, ISSN 0006-8950, 2013, Volume 136, Issue 12, pp. 3596 - 3608
A substantial proportion of cases with secondary progressive multiple sclerosis have extensive inflammation in the leptomeninges that is associated with... 
Neuroinflammation | Multiple sclerosis | Immune-mediated demyelination | Demyelination | Microglia | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | multiple sclerosis | MENINGEAL INFLAMMATION | PATHOLOGY | NEUROSCIENCES | CEREBRAL-CORTEX | CLINICAL NEUROLOGY | immune-mediated demyelination | PROGRESSIVE MULTIPLE-SCLEROSIS | TUMOR-NECROSIS-FACTOR | neuroinflammation | demyelination | REMYELINATION | CENTRAL-NERVOUS-SYSTEM | microglia | B-CELL FOLLICLES | TRANSGENIC MICE | Myelin-Oligodendrocyte Glycoprotein - immunology | Microglia - metabolism | Humans | Middle Aged | Cerebral Cortex - pathology | Encephalomyelitis, Autoimmune, Experimental - immunology | Male | Subarachnoid Space - drug effects | Cerebral Cortex - metabolism | Brain - metabolism | Dose-Response Relationship, Drug | Myelin-Oligodendrocyte Glycoprotein - toxicity | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Neurons - physiology | Aged, 80 and over | Microglia - pathology | Adult | Female | Neurons - drug effects | Cytokines - genetics | Disease Models, Animal | Cytokines - metabolism | Microglia - drug effects | Multiple Sclerosis - cerebrospinal fluid | Rats | Animals | Myelin-Oligodendrocyte Glycoprotein - metabolism | Encephalomyelitis, Autoimmune, Experimental - complications | Multiple Sclerosis - pathology | Subarachnoid Space - metabolism | Aged | Postmortem Changes | Cytokines - pharmacology
Journal Article
Journal of Comparative Neurology, ISSN 0021-9967, 05/2006, Volume 496, Issue 1, pp. 13 - 26
We have evaluated a small water‐soluble molecule, biotin ethylenediamine (BED, 286 Da), as a permeability tracer across the blood‐brain barrier. This molecule... 
cerebrospinal fluid | electron microscopy | brain barrier permeability | tight junction | biotinylated tracers | claudin‐5 | choroid plexus | Brain barrier permeability | Choroid plexus | Claudin-5 | Tight junction | Biotinylated tracers | Cerebrospinal fluid | Electron microscopy | LIPID-INSOLUBLE MOLECULES | PERMEABILITY | RAT | CEREBROSPINAL-FLUID | HORSERADISH-PEROXIDASE | NEUROSCIENCES | TIGHT JUNCTION STRANDS | ZOOLOGY | EPITHELIA | ENDOTHELIAL-CELLS | CHOROID-PLEXUS | EXPRESSION | claudin-5 | Solubility - drug effects | Molecular Weight | Extracellular Space - drug effects | Cerebral Arteries - growth & development | Capillaries - drug effects | Subarachnoid Space - drug effects | Cerebral Arteries - drug effects | Endothelial Cells - ultrastructure | Extracellular Space - metabolism | Blood-Brain Barrier - growth & development | Membrane Proteins - metabolism | Cerebrospinal Fluid - drug effects | Tight Junctions - drug effects | Animals, Newborn | Tight Junctions - metabolism | Microscopy, Electron, Transmission | Subarachnoid Space - ultrastructure | Biotin - pharmacokinetics | Endothelial Cells - metabolism | Monodelphis - metabolism | Ethylenediamines - pharmacokinetics | Rats | Blood-Brain Barrier - drug effects | Capillaries - growth & development | Blood-Brain Barrier - metabolism | Tight Junctions - ultrastructure | Animals | Cerebrospinal Fluid - metabolism | Cerebral Arteries - metabolism | Subarachnoid Space - metabolism | Monodelphis - growth & development | Endothelial Cells - drug effects
Journal Article
Journal Article
Epilepsia, ISSN 0013-9580, 04/2006, Volume 47, Issue 4, pp. 681 - 694
Journal Article
Journal Article