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1. Full Text Cellular neuroadaptations to chronic opioids: tolerance, withdrawal and addiction
by Christie, M J
British journal of pharmacology, ISSN 0007-1188, 05/2008, Volume 154, Issue 2, pp. 384 - 396
opioid tolerance | downregulation | opioid | neuroadaptation | opioid withdrawal | synaptic plasticity | desensitization | morphine | opioid addiction | Neuroadaptation | Opioid addiction | Opioid tolerance | Desensitization | Synaptic plasticity | Opioid | Morphine | Downregulation | Opioid withdrawal | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Biological and medical sciences | Pharmacology. Drug treatments | Medical sciences | Adaptation, Physiological | Neural Pathways - drug effects | Humans | Brain - enzymology | Neuronal Plasticity - drug effects | Opioid-Related Disorders - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Behavior, Addictive - metabolism | Brain - metabolism | Analgesics, Opioid - adverse effects | Substance Withdrawal Syndrome - metabolism | Synaptic Transmission - drug effects | Behavior, Addictive - physiopathology | Cyclic AMP - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Opioid-Related Disorders - psychology | Receptors, Opioid, mu - drug effects | Receptors, Opioid, mu - metabolism | Brain - physiopathology | Drug Tolerance | Adenylyl Cyclases - metabolism | Brain - drug effects | Animals | Signal Transduction - drug effects | Neural Pathways - metabolism | Opioid-Related Disorders - physiopathology | Substance Withdrawal Syndrome - physiopathology | Substance Withdrawal Syndrome - psychology | Mitogen-Activated Protein Kinases - metabolism | Index Medicus | Focused Reviews
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2. Full Text Binge ethanol withdrawal: Effects on post-withdrawal ethanol intake, glutamate–glutamine cycle and monoamine tissue content in P rat model
by Das, Sujan C and Althobaiti, Yusuf S and Alshehri, Fahad S and Sari, Youssef
Behavioural brain research, ISSN 0166-4328, 04/2016, Volume 303, pp. 120 - 125
Binge ethanol | Glutamine synthetase | Dopamine | Glutamate | GLT-1 | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Behavioral Sciences | Science & Technology | Nucleus Accumbens - metabolism | Amino Acid Transport Systems, Acidic - metabolism | Ethanol - administration & dosage | Glutamine - metabolism | Rats | Male | Excitatory Amino Acid Transporter 1 - metabolism | Excitatory Amino Acid Transporter 2 - metabolism | Animals | Substance Withdrawal Syndrome - metabolism | Ethanol - adverse effects | Serotonin - metabolism | Prefrontal Cortex - metabolism | Glutamic Acid - metabolism | Biogenic Monoamines - metabolism | Alcohol Drinking - metabolism | Glutamate-Ammonia Ligase - metabolism | Dopamine - metabolism | Disease Models, Animal | Alcohol | Alcohol, Denatured | Glutamine | GABA | Ligases | Index Medicus | glutamine synthetase | dopamine | glutamate
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3. Full Text Blocking microglial pannexin-1 channels alleviates morphine withdrawal in rodents
by Burma, Nicole E and Bonin, Robert P and Leduc-Pessah, Heather and Baimel, Corey and Cairncross, Zoe F and Mousseau, Michael and Shankara, Jhenkruthi Vijaya and Stemkowski, Patrick L and Baimoukhametova, Dinara and Bains, Jaideep S and Antle, Michael C and Zamponi, Gerald W and Cahill, Catherine M and Borgland, Stephanie L and De Koninck, Yves and Trang, Tuan
Nature medicine, ISSN 1078-8956, 03/2017, Volume 23, Issue 3, pp. 355 - 360
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Connexins - antagonists & inhibitors | Microglia - metabolism | Posterior Horn Cells - drug effects | Posterior Horn Cells - metabolism | Substance Withdrawal Syndrome - etiology | Coculture Techniques | Narcotics - adverse effects | Substance Withdrawal Syndrome - metabolism | Apyrase - pharmacology | Adenosine Triphosphate - metabolism | Nociception - drug effects | Substance Withdrawal Syndrome - genetics | Behavior, Animal - drug effects | Neurons - metabolism | Cell Culture Techniques | Neurons - drug effects | Mefloquine - pharmacology | Nerve Tissue Proteins - antagonists & inhibitors | Microglia - drug effects | Probenecid - pharmacology | Connexins - genetics | Narcotic Antagonists - adverse effects | Rats | Nerve Tissue Proteins - genetics | Blotting, Western | Connexins - metabolism | Nerve Tissue Proteins - metabolism | Animals | Naloxone - pharmacology | Mice | Morphine - adverse effects | Care and treatment | Drug withdrawal symptoms | Physiological aspects | Morphine | Models | Properties | Membrane proteins | Index Medicus
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4. Full Text Reward, addiction, withdrawal to nicotine
by De Biasi, Mariella and Dani, John A
Annual review of neuroscience, ISSN 0147-006X, 07/2011, Volume 34, Issue 1, pp. 105 - 130
Nucleus accumbens | Dopamine | Interpeduncular nucleus | Ventral tegmental area | Habenula | Synaptic plasticity | Limbic System - pathology | Receptors, Nicotinic - metabolism | Substance Withdrawal Syndrome - etiology | Humans | Cerebral Cortex - pathology | Neuronal Plasticity - drug effects | Nicotine - adverse effects | Substance Withdrawal Syndrome - pathology | Animals | Neuronal Plasticity - physiology | Neurons - physiology | Reward | Behavior, Addictive - physiopathology | Substance Withdrawal Syndrome - psychology | Dopamine - metabolism | Psychological aspects | Physiological aspects | Health aspects | Nicotine | Tobacco habit | Addictions | Neurons | Index Medicus | interpeduncular nucleus | synaptic plasticity | nucleus accumbens | ventral tegmental area | habenula | dopamine
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5. Full Text Non-Opioid Neurotransmitter Systems that Contribute to the Opioid Withdrawal Syndrome: A Review of Preclinical and Human Evidence
by Dunn, Kelly E and Huhn, Andrew S and Bergeria, Cecilia L and Gipson, Cassandra D and Weerts, Elise M
The Journal of pharmacology and experimental therapeutics, ISSN 0022-3565, 11/2019, Volume 371, Issue 2, pp. 422 - 452
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Drug Evaluation, Preclinical - methods | Opioid-Related Disorders - drug therapy | Humans | Neurotransmitter Agents - metabolism | Opioid-Related Disorders - metabolism | Substance Withdrawal Syndrome - drug therapy | Analgesics, Opioid - therapeutic use | Narcotic Antagonists - metabolism | Substance Withdrawal Syndrome - metabolism | Narcotic Antagonists - therapeutic use | Serotonin - metabolism | Glutamic Acid - metabolism | Analgesics, Opioid - metabolism | Clinical Trials as Topic - methods | Dopamine - metabolism | Index Medicus | Special Section on The Opioid Crisis
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6. Full Text Gut microbiota modulates alcohol withdrawal-induced anxiety in mice
by Xiao, Hui-wen and Ge, Chang and Feng, Guo-xing and Li, Yuan and Luo, Dan and Dong, Jia-li and Li, Hang and Wang, Haichao and Cui, Ming and Fan, Sai-jun
Toxicology letters, ISSN 0378-4274, 05/2018, Volume 287, pp. 23 - 30
Gut microbiota | Faecal microbiota transplantation | Alcohol withdrawal-induced anxiety | Intestinal bacterial composition | Life Sciences & Biomedicine | Toxicology | Science & Technology | Anxiety - microbiology | Brain-Derived Neurotrophic Factor - genetics | Receptors, Corticotropin-Releasing Hormone - metabolism | Receptors, Corticotropin-Releasing Hormone - genetics | Male | Motor Activity | Behavior, Animal | Feces - microbiology | Bacteria - growth & development | Substance Withdrawal Syndrome - metabolism | Alcoholism - microbiology | Bacteria - classification | Substance Withdrawal Syndrome - genetics | Brain-Derived Neurotrophic Factor - metabolism | Alcoholism - psychology | Anxiety - metabolism | Disease Models, Animal | Ethanol | Receptors, Opioid, mu - metabolism | Mice, Inbred C57BL | Anxiety - psychology | Gastrointestinal Microbiome | Bacteria - genetics | Fecal Microbiota Transplantation | Host-Pathogen Interactions | Hippocampus - metabolism | Anxiety - genetics | Animals | Intestines - microbiology | Ribotyping | Alcoholism - metabolism | Alcoholism - genetics | Receptors, Opioid, mu - genetics | Substance Withdrawal Syndrome - psychology | Substance Withdrawal Syndrome - microbiology | Index Medicus
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7. Full Text Cannabinoid Type 1 Receptor (CB1) Ligands with Therapeutic Potential for Withdrawal Syndrome in Chemical Dependents of Cannabis sativa
by Ferreira, Jaderson V and Chaves, Gisele A and Marino, Bianca L. B and Sousa, Kessia P. A and Souza, Lucilene R and Brito, Maiara F. B and Teixeira, Hueldem R. C and da Silva, Carlos H. T. P and Santos, Cleydson B. R and Hage‐Melim, Lorane I. S
ChemMedChem, ISSN 1860-7179, 08/2017, Volume 12, Issue 16, pp. 1408 - 1416
structural similarity | Cannabis sativa | virtual screening | computational chemistry | medicinal chemistry | Caco-2 Cells | Resorcinols - pharmacokinetics | Receptor, Cannabinoid, CB2 - agonists | Humans | Half-Life | Substance Withdrawal Syndrome - drug therapy | Cannabis - metabolism | Blood-Brain Barrier - drug effects | Resorcinols - therapeutic use | Blood-Brain Barrier - metabolism | Substance Withdrawal Syndrome - pathology | Resorcinols - pharmacology | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabis - chemistry | Animals | Receptor, Cannabinoid, CB1 - agonists | Substance Withdrawal Syndrome - metabolism | Resorcinols - chemistry | Cell Membrane Permeability - drug effects | Drug Design | Ligands | Mice | Marijuana | Health aspects | Index Medicus
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