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Glia, ISSN 0894-1491, 04/2007, Volume 55, Issue 5, pp. 453 - 462
Inflammation is implicated in the progressive nature of neurodegenerative diseases, such as Parkinson's disease, but the mechanisms are poorly understood. A... 
neurodegeneration | TNFα | substantia nigra | neuroinflammation | microglia | LPS | Neuroinflammation | Neurodegeneration | Substantia nigra | Microglia | TNF alpha | TNF-ALPHA TRANSPORT | BACTERIAL-ENDOTOXIN EXPOSURE | INFLAMMATION-MEDIATED NEURODEGENERATION | INNATE IMMUNE-SYSTEM | AMYOTROPHIC-LATERAL-SCLEROSIS | DOPAMINE NEURON LOSS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | TUMOR-NECROSIS-FACTOR | CORTICOTROPIN-RELEASING FACTOR | LIPOPOLYSACCHARIDE-INDUCED NEUROTOXICITY | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Neurodegenerative Diseases - etiology | Lipopolysaccharides - administration & dosage | Tumor Necrosis Factor-alpha - genetics | Cell Death - immunology | RNA, Messenger - analysis | Substantia Nigra - immunology | Male | Neurons - cytology | Substantia Nigra - metabolism | Neurodegenerative Diseases - immunology | Lipopolysaccharides - immunology | Substantia Nigra - cytology | Brain - metabolism | Inflammation - complications | Microglia - immunology | Time Factors | Inflammation Mediators - metabolism | Neurons - metabolism | Parkinson Disease - metabolism | Dopamine - metabolism | Disease Models, Animal | Inflammation Mediators - immunology | Brain - cytology | Parkinson Disease - immunology | Liver - metabolism | Mice, Inbred C57BL | Injections, Intraperitoneal | Neurodegenerative Diseases - metabolism | Mice, Inbred Strains | Mice, Knockout | Animals | Mice | Brain - immunology
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2009, Volume 37, Issue 3, pp. 630 - 640
Abstract Inflammation, and in particular microglia activation, is regarded as a constant component of brain pathology in Parkinson's disease (PD). Microglial... 
Neurology | Inflammation | Akinesia | Adenovector | Parkinson | Tumor necrosis factor α | Microglia | MICROGLIAL ACTIVATION | SINGLE INTRANIGRAL INJECTION | NITRIC-OXIDE SYNTHASE | DOPAMINERGIC-NEURONS | INFLAMMATORY PROCESS | NEUROSCIENCES | Tumor necrosis factor alpha | GLIAL REACTION | TNF-ALPHA | INDUCED NEUROTOXICITY | MPTP-MOUSE MODEL | PARKINSONS-DISEASE | Substantia Nigra - physiopathology | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Neurons - pathology | Rats, Wistar | Tumor Necrosis Factor-alpha - genetics | Nerve Degeneration - genetics | Substantia Nigra - immunology | Male | Time | Substantia Nigra - metabolism | Nerve Degeneration - metabolism | Microglia - immunology | Transfection - methods | Cell Death - genetics | Time Factors | Interleukin-1beta - metabolism | Encephalitis - metabolism | Neurons - metabolism | Parkinson Disease - metabolism | Dopamine - metabolism | Dyskinesias - immunology | Encephalitis - genetics | Cytokines - metabolism | Gene Expression Regulation - immunology | Parkinson Disease - immunology | Neurons - immunology | Dyskinesias - physiopathology | Rats | Encephalitis - immunology | Nerve Degeneration - immunology | Parkinson Disease - physiopathology | Dyskinesias - metabolism | Animals | Genetic Vectors - pharmacology | Chemotaxis, Leukocyte - immunology | Index Medicus
Journal Article
Movement Disorders, ISSN 0885-3185, 03/2008, Volume 23, Issue 4, pp. 474 - 483
Dopaminergic neurons of the substantia nigra are particularly vulnerable to oxidative and inflammatory attack. Such processes may play a crucial role in the... 
inflammation | astrocytes | MPTP | α‐synuclein | microglia | oxidative stress | oligodendroglia | Oxidative stress | Astrocytes | Oligodendroglia | Inflammation | α-synuclein | Microglia | ACTIVATED PROTEIN-KINASE | MICROGLIAL ACTIVATION | NITRIC-OXIDE SYNTHASE | alpha-synuclein | SYNERGISTIC DOPAMINERGIC NEUROTOXICITY | RAT MESENCEPHALIC CULTURES | SUBSTANTIA-NIGRA | CLINICAL NEUROLOGY | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | IN-VIVO | ROTENONE-INDUCED DEGENERATION | Immunohistochemistry | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Neurons - pathology | Substantia Nigra - pathology | Humans | Oxidative Stress - physiology | Astrocytes - pathology | Substantia Nigra - immunology | Male | Substantia Nigra - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Microglia - immunology | Astrocytes - immunology | Microglia - pathology | Female | Neurons - metabolism | Parkinson Disease - metabolism | Dopamine - metabolism | Parkinson Disease - pathology | Intercellular Adhesion Molecule-1 - immunology | Parkinson Disease - immunology | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - adverse effects | Neurotoxins - adverse effects | Neurons - immunology | Haplorhini | Brain - drug effects | Animals | HLA-DR Antigens - immunology | alpha-Synuclein - metabolism | Astrocytes - metabolism
Journal Article
Journal of Neuropathology and Experimental Neurology, ISSN 0022-3069, 12/2008, Volume 67, Issue 12, pp. 1149 - 1158
Microglial activation and adaptive immunity have been implicated in the neurodegenerative processes in Parkinson disease. It has been proposed that these... 
Adeno-associated virus | Dopamine | Eurodegeneration | Lymphocytes | Euroinflammation | Immunoglobulin | Glia | CELL INJURY | PROTEIN-KINASE | Neuroinflammation | MEDIATED GENE-TRANSFER | DOPAMINERGIC-NEURONS | REACTIVE MICROGLIA | PATHOLOGY | SUBSTANTIA-NIGRA | NEUROSCIENCES | CLINICAL NEUROLOGY | ADENOASSOCIATED VIRUS TYPE-2 | Neurodegeneration | CENTRAL-NERVOUS-SYSTEM | ALTERNATIVE ACTIVATION | LEWY BODIES | Substantia Nigra - physiopathology | Antigens, CD - immunology | Immunoglobulins - immunology | Substantia Nigra - pathology | Humans | alpha-Synuclein - immunology | Substantia Nigra - immunology | Male | Antigens, CD - metabolism | Immunoglobulins - metabolism | Lymphocyte Activation - immunology | Microglia - immunology | Transfection | alpha-Synuclein - genetics | Antigens, Differentiation, Myelomonocytic - immunology | Dopamine - metabolism | Cytokines - immunology | Disease Models, Animal | Cytokines - metabolism | Parkinson Disease - immunology | Brain - physiopathology | Mice, Inbred C57BL | Nerve Degeneration - immunology | Parkinson Disease - genetics | Immune System Phenomena - immunology | Parkinson Disease - physiopathology | Nerve Degeneration - pathology | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Brain - pathology | Mice | Chemotaxis, Leukocyte - immunology | Genetic Vectors | Brain - immunology
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 12/2010, Volume 88, Issue 16, pp. 3530 - 3539
Journal Article
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 11/2011, Volume 8, Issue 1, pp. 166 - 166
Journal Article
Environmental Health Perspectives, ISSN 0091-6765, 6/2011, Volume 119, Issue 6, pp. 807 - 814
Background: Mechanisms whereby gene-environment interactions mediate chronic, progressive neurodegenerative processes in Parkinson's disease (PD)—the second... 
Oxidases | Pathology | Animal models | Disease models | Neurons | Environmental health | Parkinson disease | Antibodies | Inflammation | Research | Microglia | Oxidative stress | Parkinson's disease | Neurodegeneration | NADPH oxidase | Gene-environment interaction | INOS | α-synuclein | oxidase | NADPH OXIDASE | MICROGLIAL ACTIVATION | SUSCEPTIBILITY | neuro degeneration | NEUROTOXICITY | CELL-DEATH | NEURON LOSS | ENVIRONMENTAL SCIENCES | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | DOPAMINERGIC NEURODEGENERATION | MUTATION | MICE | oxidative stress | LEWY BODIES | Oxidative Stress | Substantia Nigra - pathology | Substantia Nigra - immunology | Neurodegenerative Diseases - immunology | Lipopolysaccharides - immunology | Nerve Degeneration - chemically induced | Nitric Oxide Synthase Type II - antagonists & inhibitors | alpha-Synuclein - genetics | Lewy Bodies - pathology | Disease Models, Animal | Parkinson Disease - pathology | Neurodegenerative Diseases - pathology | Animals, Genetically Modified | Parkinson Disease - immunology | NADPH Oxidases - antagonists & inhibitors | Injections, Intraperitoneal | Nerve Degeneration - immunology | Neurodegenerative Diseases - genetics | Onium Compounds - pharmacology | Parkinson Disease - genetics | Nerve Degeneration - pathology | Animals | Neurodegenerative Diseases - chemically induced | Lipopolysaccharides - pharmacology | Amidines - pharmacology | Mice | Benzylamines - pharmacology | alpha-Synuclein - metabolism | Index Medicus
Journal Article
Journal Article