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Journal Article
Journal Article
Cell (Cambridge), ISSN 0092-8674, 10/2016, Volume 167, Issue 2, pp. 457 - 470.e13
Activated macrophages undergo metabolic reprogramming, which drives their pro-inflammatory phenotype, but the mechanistic basis for this remains obscure. Here,... 
succinate | reverse electron transport | toll-like receptors | innate immunity | immunometabolism | macrophage | succinate dehydrogenase | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Mitochondria - enzymology | Reactive Oxygen Species - metabolism | Transcriptome | Malonates - pharmacology | Lipopolysaccharides - immunology | Oxidative Phosphorylation - drug effects | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Membrane Potential, Mitochondrial | Oxidation-Reduction - drug effects | Interleukin-10 - metabolism | Plant Proteins - metabolism | Macrophages - immunology | Oxidoreductases - metabolism | Mice, Inbred C57BL | Inflammation - immunology | Mitochondria - drug effects | Citric Acid Cycle | Macrophage Activation | Macrophages - metabolism | Succinate Dehydrogenase - genetics | Animals | Sequence Analysis, RNA | Succinic Acid - metabolism | Glycolysis | Inflammation - genetics | Succinate Dehydrogenase - metabolism | Mice | Oxidases | RNA sequencing | Glucose metabolism | Medical colleges | RNA | Gastrointestinal diseases | Genes | Research institutes | Macrophages | Gene expression | Mitogens | Index Medicus
Journal Article
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 09/2008, Volume 295, Issue 3, pp. 678 - 685
Journal Article
Nature (London), ISSN 1476-4687, 04/2018, Volume 556, Issue 7702, pp. 501 - 504
.... The major effects of itaconate on cellular metabolism during macrophage activation have been attributed to the inhibition of succinate dehydrogenase , yet this inhibition alone is not sufficient... 
Succinates - chemistry | Glutathione - metabolism | Humans | I-kappa B Proteins - metabolism | Inflammation - metabolism | Succinates - administration & dosage | Psoriasis - pathology | Inflammation - drug therapy | Female | Succinates - metabolism | Stress, Physiological - drug effects | Interleukin-6 - metabolism | Cytokines - immunology | Psoriasis - drug therapy | Cytokines - metabolism | Mice, Inbred C57BL | Toll-Like Receptors - immunology | Cells, Cultured | Activating Transcription Factor 3 - metabolism | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Succinates - therapeutic use | Keratinocytes - drug effects | Keratinocytes - metabolism | NF-E2-Related Factor 2 - metabolism | Macrophages - drug effects | Mice | Succinates - pharmacology | Phosphorylation | Transcription factors | Transcription | Macrophages | Interleukin 6 | Proteins | Cell activation | Metabolites | Rodents | Toll-like receptors | Inhibition | Stress response | Glutathione | Immune system | Immune response | Psoriasis | Immunoregulation | Activating transcription factor 3 | Medical treatment | Interleukin 12 | Inflammation | Metabolism | Gene expression | Interleukin 17 | Chromatography | Dimethylitaconate | Tumor necrosis factor | Proteomics | Skin | Autoimmune diseases | Index Medicus
Journal Article
Developmental cell, ISSN 1534-5807, 03/2017, Volume 40, Issue 6, pp. 583 - 594.e6
Mitochondrial fission mediated by the GTPase dynamin-related protein 1 (Drp1) is an attractive drug target in numerous maladies that range from heart disease... 
succinate | bioenergetics | mitochondria | fission | neuron | reverse electron transfer | respiration | superoxide | brain | fragmentation | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Biology | Dynamins - metabolism | Reactive Oxygen Species - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Cercopithecus aethiops | Saccharomyces cerevisiae - drug effects | GTP Phosphohydrolases - antagonists & inhibitors | Electron Transport Complex I - metabolism | Fibroblasts - ultrastructure | Saccharomyces cerevisiae - metabolism | Mitochondrial Proteins - metabolism | Cell Respiration - drug effects | Oxidation-Reduction - drug effects | Neurons - metabolism | NAD - metabolism | Quinazolinones - pharmacology | Fibroblasts - metabolism | Dynamins - antagonists & inhibitors | Electron Transport Complex I - antagonists & inhibitors | Mitochondrial Proteins - antagonists & inhibitors | Mitochondria - metabolism | Microtubule-Associated Proteins - antagonists & inhibitors | Mitochondria - drug effects | Rats, Sprague-Dawley | Mice, Knockout | Animals | GTP Phosphohydrolases - metabolism | Oxygen Consumption - drug effects | Saccharomyces cerevisiae Proteins - metabolism | Mice | COS Cells | Brain | Medical colleges | Nervous system diseases | Heart diseases | Injuries | Biomedical engineering | Neurons | Superoxide | Index Medicus
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 03/2009, Volume 284, Issue 11, pp. 7201 - 7213
Journal Article