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Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2013, Volume 288, Issue 3, pp. 1696 - 1705
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 1609 - 11
Bacterial degradation of organosulfonates plays an important role in sulfur recycling, and has been extensively studied. However, this process in anaerobic... 
DECARBOXYLASE | ENZYME | PROTEIN | METABOLISM | DISSIMILATION | MULTIDISCIPLINARY SCIENCES | PURIFICATION | GENE CLUSTERS | TAURINE | SULFATE-REDUCING BACTERIA | ELECTRON-ACCEPTORS | Hydrogen Sulfide - metabolism | Recombinant Proteins - metabolism | Sulfur - metabolism | Acetyltransferases - metabolism | Intestinal Mucosa - metabolism | Bilophila - metabolism | Hydrogen Sulfide - toxicity | Mercaptoethanol - analogs & derivatives | Bacterial Proteins - genetics | Bile Acids and Salts - metabolism | Enzyme Assays | Gastrointestinal Microbiome - physiology | Recombinant Proteins - genetics | Intestinal Mucosa - microbiology | Acetyltransferases - genetics | Acetyltransferases - isolation & purification | Recombinant Proteins - isolation & purification | Mercaptoethanol - metabolism | Desulfovibrio - metabolism | Bacterial Proteins - metabolism | Metabolic Networks and Pathways - physiology | Bacterial Proteins - isolation & purification | Taurine - metabolism | Biodegradation | Anaerobic bacteria | Taurine | Sulfur compounds | Anaerobic microorganisms | Transformation | Sulfite-reducing bacteria | Anaerobic respiration | Organosulfonates | Sulfonates | Metabolism | Acetaldehyde | Degradation | Hydrogen sulfide | Sulfite | Microorganisms | Pathways | Anaerobic processes | Bacteria | Cleavage | Sulfate | Sulfur | Structural analysis | Bile
Journal Article
Journal of Inherited Metabolic Disease, ISSN 0141-8955, 3/2018, Volume 41, Issue 2, pp. 187 - 196
Molybdenum cofactor deficiency is an autosomal recessive inborn error of metabolism, which results from mutations in genes involved in Moco biosynthesis. Moco... 
Human Genetics | Biochemistry, general | Pediatrics | S -sulfocysteine | Metabolic Diseases | Internal Medicine | Kozak-sequence | Urothione | Molybdenum cofactor | Medicine & Public Health | Moco deficiency | Sulfite oxidase | MOCS1 | cPMP | S-sulfocysteine | MEDICINE, RESEARCH & EXPERIMENTAL | MECHANISM | SULFITE OXIDASE DEFICIENCY | READING FRAMES | IDENTIFICATION | INBORN-ERRORS | GENE | METABOLISM | BIOSYNTHESIS | ENDOCRINOLOGY & METABOLISM | GENETICS & HEREDITY | MUTATIONS | Metal Metabolism, Inborn Errors - metabolism | Genetic Predisposition to Disease | Peptide Fragments - metabolism | Frameshift Mutation | Metal Metabolism, Inborn Errors - genetics | Humans | Child, Preschool | Male | Nuclear Proteins - metabolism | Pteridines - metabolism | Metal Metabolism, Inborn Errors - diet therapy | Diet, Protein-Restricted | Coenzymes - metabolism | Magnetic Resonance Imaging | Metalloproteins - metabolism | Phenotype | Metal Metabolism, Inborn Errors - diagnosis | Age of Onset | HEK293 Cells | Nuclear Proteins - genetics | Child | Peptide Fragments - genetics | Proteins | Physiological aspects | Sulfites | Molybdenum | Analysis | Urine | Phenotypes | Translation | Inborn errors of metabolism | Transcription | Translation initiation | Frameshift mutation | Protein deficiency | Sulfite | Neurodegeneration | Genetic analysis | Translation termination | Children | Mutation | Seizures | Index Medicus
Journal Article
Journal Article
Gene, ISSN 0378-1119, 12/2013, Volume 531, Issue 2, pp. 191 - 198
Sulfite oxidase (SO) deficiency is biochemically characterized by tissue accumulation and high urinary excretion of sulfite, thiosulfate and S-sulfocysteine.... 
Sulfite oxidase deficiency | Sulfite | Thiosulfate | Reactive species | Rat brain | Bioenergetic dysfunction | MOLYBDENUM-COFACTOR DEFICIENCY | OXIDATIVE STRESS | LIPID-PEROXIDATION | RAT CEREBRAL-CORTEX | HUMAN SKELETAL-MUSCLE | CYTOCHROME-C-OXIDASE | MITOCHONDRIAL CREATINE-KINASE | REACTIVE OXYGEN | ACID IN-VITRO | GENETICS & HEREDITY | NITRIC-OXIDE | Amino Acid Metabolism, Inborn Errors - complications | Electron Transport - drug effects | Rats, Wistar | Amino Acid Metabolism, Inborn Errors - physiopathology | Electron Transport - genetics | Male | Brain - physiology | Sulfite Oxidase - deficiency | Sulfites - pharmacology | Brain - metabolism | Amino Acid Metabolism, Inborn Errors - metabolism | Amino Acid Metabolism, Inborn Errors - genetics | Oxidation-Reduction - drug effects | Electron Transport - physiology | Homeostasis - drug effects | Energy Metabolism - physiology | Sulfite Oxidase - genetics | Brain Diseases, Metabolic - genetics | Rats | Thiosulfates - pharmacology | Brain Diseases, Metabolic - metabolism | Sulfites - metabolism | Thiosulfates - metabolism | Brain - drug effects | Animals | Sulfite Oxidase - metabolism | Brain - pathology | Brain Diseases, Metabolic - etiology | Energy Metabolism - drug effects | Oxidases | Sulfites | Metabolites | Molybdenum compounds | Creatine kinase | Nitric oxide | Physiological aspects | Homeostasis | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2017, Volume 127, Issue 12, pp. 4365 - 4378