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1. Full Text Coordinated regulation of hepatic phase I and II drug-metabolizing genes and transporters using AhR-, CAR-, PXR-, PPARα-, and Nrf2-null mice
by Aleksunes, Lauren M and Klaassen, Curtis D
Drug Metabolism and Disposition, ISSN 0090-9556, 07/2012, Volume 40, Issue 7, pp. 1366 - 1379
The transcription factors aryl hydrocarbon receptor (AhR), constitutive androstane receptor (CAR), pregnane X receptor (PXR), peroxisome proliferator-activated... 
TRANSCRIPTION FACTORS | PRIMARY HUMAN HEPATOCYTES | ANTIOXIDANT RESPONSE ELEMENT | MESSENGER-RNA EXPRESSION | CONSTITUTIVE ANDROSTANE RECEPTOR | BILIARY-EXCRETION | PROLIFERATOR-ACTIVATED RECEPTOR | MOUSE-LIVER | PHARMACOLOGY & PHARMACY | MICROSOMAL-ENZYME INDUCERS | PREGNANE-X-RECEPTOR | Inactivation, Metabolic | Receptors, Steroid - metabolism | Cytochrome P-450 Enzyme System - metabolism | Male | NAD(P)H Dehydrogenase (Quinone) - genetics | Organic Anion Transporters - metabolism | Glucuronosyltransferase - genetics | Organic Anion Transporters - genetics | Glutathione Transferase - genetics | Receptors, Aryl Hydrocarbon - metabolism | Female | NF-E2-Related Factor 2 - genetics | Sulfotransferases - metabolism | Aldehyde Dehydrogenase - metabolism | Sulfotransferases - genetics | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Receptors, Aryl Hydrocarbon - genetics | Glutathione Transferase - metabolism | Aldehyde Dehydrogenase - genetics | PPAR alpha - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | Mice, Knockout | Gene Expression Regulation, Enzymologic | Transcription Factors - metabolism | Animals | Glucuronosyltransferase - metabolism | Receptors, Steroid - genetics | NF-E2-Related Factor 2 - metabolism | Cytochrome P-450 Enzyme System - genetics | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | PPAR alpha - metabolism | Multidrug Resistance-Associated Proteins | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
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2. Full Text A genome-wide CRISPR screen identifies a restricted set of HIV host dependency factors
by Park, Ryan J and Wang, Tim and Koundakjian, Dylan and Hultquist, Judd F and Lamothe-Molina, Pedro and Monel, Blandine and Schumann, Kathrin and Yu, Haiyan and Krupzcak, Kevin M and Garcia-Beltran, Wilfredo and Piechocka-Trocha, Alicja and Krogan, Nevan J and Marson, Alexander and Sabatini, David M and Lander, Eric S and Hacohen, Nir and Walker, Bruce D
Nature Genetics, ISSN 1061-4036, 01/2017, Volume 49, Issue 2, pp. 193 - 203
Host proteins are essential for HIV entry and replication and can be important nonviral therapeutic targets. Large-scale RNA interference (RNAi)-based screens... 
GP120 BINDING | REPLICATION | ADHESION MOLECULE | TYROSINE SULFATION | TYROSYLPROTEIN SULFOTRANSFERASES | GENETICS & HEREDITY | EARLY-STAGE | CELL-ADHESION | ANTIRETROVIRAL THERAPY | HUMAN-IMMUNODEFICIENCY-VIRUS | T-CELLS | Sulfotransferases - genetics | Cell Line | HIV-1 - pathogenicity | Membrane Proteins - genetics | Virus Replication - genetics | HIV Infections - genetics | Humans | Genome - genetics | Receptors, CCR5 - genetics | Host-Pathogen Interactions - genetics | Clustered Regularly Interspaced Short Palindromic Repeats - genetics | RNA Interference - physiology | Activated-Leukocyte Cell Adhesion Molecule - genetics | Innovations | Genotype | Genetic aspects | Genetic engineering | Virulence (Microbiology) | Host-virus relationships | HIV infection | Health aspects | Risk factors | Pathogens | Flow cytometry | Pandemics | Viruses | Infections | Identification | Genomes | Grants | Drug resistance | Proteins | Acquired immune deficiency syndrome--AIDS | Lymphocytes | Human immunodeficiency virus--HIV | Population | Gene therapy | Viral infections | Index Medicus
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3. Full Text Association of testicular germ cell tumor with polymorphisms in estrogen receptor and steroid metabolism genes
by Alberto Ferlin and Francesco Ganz and Manuel Pengo and Riccardo Selice and Anna Chiara Frigo and Carlo Foresta
Endocrine-Related Cancer, ISSN 1351-0088, 03/2010, Volume 17, Issue 1, pp. 17 - 25
It is generally assumed that the development of testicular germ cell tumor (TGCT) is under endocrine control. In particular, unbalanced androgen/estrogen... 
MALE-INFERTILITY | ETIOLOGY | ONCOLOGY | DYSGENESIS SYNDROME | SUSCEPTIBILITY | ENDOCRINOLOGY & METABOLISM | RISK | ENVIRONMENTAL ASPECTS | CRYPTORCHIDISM | LINKAGE | CANCER | BREAST | Cytochrome P-450 CYP1A1 - genetics | Humans | Male | Neoplasms, Germ Cell and Embryonal - genetics | Catechol O-Methyltransferase - genetics | Seminoma - metabolism | Young Adult | Aromatase - genetics | Estrogen Receptor beta - genetics | Cytochrome P-450 CYP1A2 - genetics | Adult | Hydro-Lyases - genetics | Estrogens - metabolism | Neoplasm Proteins - genetics | Seminoma - genetics | Sulfotransferases - genetics | Genetic Predisposition to Disease | Testicular Neoplasms - genetics | Genetic Association Studies | Testicular Neoplasms - metabolism | Genotype | Neoplasms, Germ Cell and Embryonal - metabolism | Arylsulfotransferase - genetics | Peroxisomal Multifunctional Protein-2 | Estrogen Receptor alpha - genetics | Estradiol Dehydrogenases - genetics | 17-Hydroxysteroid Dehydrogenases - genetics | Cytochrome P-450 CYP1B1 | Cytochrome P-450 Enzyme System - genetics | Polymorphism, Single Nucleotide | Aryl Hydrocarbon Hydroxylases | Cryptorchidism - genetics | Index Medicus
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4. Full Text Sulfotransferase SULT1A1 Arg213His polymorphism with cancer risk: A meta-analysis of 53 case-control studies
by Xiao, Juanjuan and Zheng, Yabiao and Zhou, Yinghui and Zhang, Ping and Wang, Jianguo and Shen, Fangyuan and Fan, Lixia and Kolluri, Vijay Kumar and Wang, Weiping and Yan, Xiaolong and Wang, Minghua
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, pp. e106774 - e106774
Background: The SULT1A1 Arg213His (rs9282861) polymorphism is reported to be associated with many kinds of cancer risk. However, the findings are conflicting.... 
BREAST-CANCER | LUNG-CANCER | GENETIC-POLYMORPHISM | COLORECTAL-CANCER | JAPANESE POPULATION | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | UROTHELIAL-CANCER | ARGHIS POLYMORPHISM | CARCINOGEN-METABOLIZING ENZYMES | PHENOL SULFOTRANSFERASES | Genetic Predisposition to Disease | Gastrointestinal Neoplasms - genetics | Humans | Genotype | Male | Arylsulfotransferase - genetics | Case-Control Studies | Polymorphism, Genetic | Breast Neoplasms - genetics | Codon | Neoplasms - genetics | Alleles | Publication Bias | Female | Odds Ratio | Amino Acid Substitution | Thoracic surgery | Lung cancer | Colorectal cancer | Single-nucleotide polymorphism | Gene polymorphism | Carcinogenesis | Stomach cancer | Subgroups | Ovarian cancer | Confidence intervals | Carcinogens | Software reviews | Digestive tract | Sensitivity analysis | Statistical analysis | Health risks | Data processing | Breast cancer | Regression analysis | Risk analysis | Minority & ethnic groups | Meta-analysis | Studies | Ethnicity | Sulfotransferase | Breast | Molecular biology | Health risk assessment | Prostate cancer | Esophageal cancer | Cancer | Polymorphism | Index Medicus
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5. Full Text Heparan sulfate 6-O-sulfotransferase 1, a gene involved in extracellular sugar modifications, is mutated in patients with idiopathic hypogonadotrophic hypogonadism
by Janne Tornberg and Gerasimos P. Sykiotis and Kimberly Keefe and Lacey Plummer and Xuan Hoang and Janet E. Hall and Richard Quinton and Stephanie B. Seminara and Virginia Hughes and Guy Van Vliet and Stan Van Uum and William F. Crowley and Hiroko Habuchi and Koji Kimata and Nelly Pitteloud and Hannes E. Bülow
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2011, Volume 108, Issue 28, pp. 11524 - 11529
Neuronal development is the result of a multitude of neural migrations, which require extensive cell-cell communication. These processes are modulated by... 
Enzymes | Phenotypes | Neurons | Medical genetics | Ligands | Sulfates | Genetic mutation | Kallmann syndrome | Human genetics | Hypogonadism | Heparan sulfotransferase | SPECIFICITY | GONADOTROPIN-RELEASING-HORMONE | MULTIDISCIPLINARY SCIENCES | DEFICIENCY | FGF RECEPTOR | FAMILY | heparan sulfotransferase | CAENORHABDITIS-ELEGANS | KALLMANN-SYNDROME | MUTATIONS | EXPRESSION | HEPARAN-SULFATE 6-O-SULFOTRANSFERASE | Species Specificity | Humans | Middle Aged | Genes, Helminth | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Caenorhabditis elegans Proteins - metabolism | Molecular Sequence Data | Kallmann Syndrome - enzymology | Male | Mutation, Missense | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Sulfotransferases - deficiency | Intercellular Signaling Peptides and Proteins - metabolism | Receptors, Fibroblast Growth Factor - genetics | Adult | Female | Sulfotransferases - metabolism | Child | Extracellular Matrix Proteins - metabolism | Sulfotransferases - genetics | Amino Acid Sequence | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Kallmann Syndrome - genetics | Extracellular Matrix Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Models, Molecular | Nerve Tissue Proteins - genetics | Hypogonadism - enzymology | Hypogonadism - genetics | Nerve Tissue Proteins - metabolism | Sequence Homology, Amino Acid | Animals | Pedigree | Receptors, Fibroblast Growth Factor - metabolism | Mutation | In Vitro Techniques | Caenorhabditis elegans Proteins - genetics | Sulfotransferases - chemistry | Amino Acid Substitution | Index Medicus | Heparan sulfate | Fibroblast growth factor | Puberty | Intercellular signalling | Polysaccharides | Hedgehog protein | Infertility | Extracellular matrix | Gonadotropin-releasing hormone | Cell migration | Sugar | Biological Sciences
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6. Full Text NDST4 Is a Novel Candidate Tumor Suppressor Gene at Chromosome 4q26 and Its Genetic Loss Predicts Adverse Prognosis in Colorectal Cancer
by Tzeng, Sheng-Tai and Tsai, Ming-Hong and Chen, Chi-Long and Lee, Jing-Xing and Jao, Tzu-Ming and Yu, Sung-Liang and Yen, Sou-Jhy and Yang, Ya-Chien
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, pp. e67040 - e67040
Background: Genomic deletion at tumor suppressor loci is a common genetic aberration in human cancers. The study aimed to explore candidate tumor suppressor... 
PATHOGENESIS | ISOZYMES | MOLECULAR MARKERS | INSTABILITY | BIOSYNTHESIS | MULTIDISCIPLINARY SCIENCES | PATTERNS | HEPARAN-SULFATE PROTEOGLYCANS | CARCINOMA | DELETIONS | SULFOTRANSFERASE | Sulfotransferases - genetics | Intestinal Mucosa - metabolism | Prognosis | Membrane Proteins - genetics | Colorectal Neoplasms - genetics | Down-Regulation | Humans | Middle Aged | Genetic Loci - genetics | Male | Chromosomes, Human, Pair 4 - genetics | Colorectal Neoplasms - diagnosis | Sulfotransferases - deficiency | Membrane Proteins - deficiency | Colonic Polyps - genetics | Alleles | Aged, 80 and over | Adult | Female | Aged | Colorectal Neoplasms - pathology | Neoplasm Staging | Genes, Tumor Suppressor | Gene Expression Regulation, Neoplastic - genetics | RNA | Analysis | Oncology, Experimental | Genes | Colorectal cancer | Genetic aspects | Genetic transcription | Research | Tumors | Cancer | Biotechnology | DNA polymerase | Laboratories | Epithelial cells | Colorectal carcinoma | Reverse transcription | Biosynthesis | Metastasis | Tissues | Clonal deletion | Surgery | Deletion | Bioindicators | Chromosomes | Deoxyribonucleic acid--DNA | Heparan sulfate | Microsatellites | Tumor cells | Chromosome deletion | Chromosome 4 | Gene expression | Ribonucleic acid--RNA | Loci | Heterozygosity | Medicine | Polymerase chain reaction | Genetic markers | Hospitals | Medical prognosis | Loss of heterozygosity | Biomarkers | Tumor suppressor genes | Mutation | Aberration | Gene mapping | Index Medicus | Deoxyribonucleic acid | Ribonucleic acid | DNA
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7. Full Text Identification of Targets of Twist1 Transcription Factor in Thyroid Cancer Cells
by Di Maro, Gennaro and Orlandella, Francesca Maria and Bencivenga, Tammaro Claudio and Salerno, Paolo and Ugolini, Clara and Basolo, Fulvio and Maestro, Roberta and Salvatore, Giuliana
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 09/2014, Volume 99, Issue 9, pp. E1617 - E1626
Context: Anaplastic thyroid carcinoma (ATC) is one of the most aggressive human tumors. Twist1 is a basic helix-loop-helix transcription factor involved in... 
COLLAGEN | GROWTH | ENDOCRINOLOGY & METABOLISM | MECHANISMS | PROMOTES | EMT | SIGNATURE | CARCINOMA | P53 | RNA, Small Interfering - genetics | Carcinoma, Papillary - genetics | Humans | Gene Expression Regulation, Neoplastic | Cell Survival - genetics | Inhibitor of Differentiation Proteins - genetics | Promoter Regions, Genetic - genetics | Carcinoma, Papillary - metabolism | Thyroid Hormone Receptors alpha - genetics | rhoB GTP-Binding Protein - genetics | Carcinoma - pathology | Nuclear Proteins - genetics | Sulfotransferases - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Membrane Proteins - genetics | Nuclear Proteins - metabolism | Carcinoma, Papillary - pathology | Thyroid Cancer, Papillary | Thyroid Neoplasms - genetics | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Carcinoma - genetics | Carcinoma - metabolism | Twist-Related Protein 1 - metabolism | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Index Medicus | Abridged Index Medicus
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8. Full Text The endometrial cancer cell lines Ishikawa and HEC-1A, and the control cell line HIEEC, differ in expression of estrogen biosynthetic and metabolic genes, and in androstenedione and estrone-sulfate metabolism
by Hevir-Kene, Neli and Rižner, Tea Lanišnik
Chemico-Biological Interactions, ISSN 0009-2797, 06/2015, Volume 234, pp. 309 - 319
Estrogens have important roles in the pathogenesis of endometrial cancer. They can have carcinogenic effects through stimulation of cell proliferation or... 
Aromatase pathway | Sulfatase pathway | Phase I and phase II estrogen metabolism | Aldo–keto reductase 1C3 (AKR1C3) | 17β-Hydroxysteroid dehydrogenases (17β-HSDs, HSD17B) | Catechol-O-methyl transferase (COMT) | 17b-Hydroxysteroid dehydrogenases | (17b-HSDs HSD17B)Aldoketo reductase 1C3 (AKR1C3) | 17 beta-Hydroxysteroid dehydrogenases (17 beta-HSDs, HSD17B) | DEHYDROGENASES | QUINONES | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROGESTERONE ACTION | 17-BETA-ESTRADIOL | CARCINOGENESIS | OVARIAN ENDOMETRIOSIS | Aldo-keto reductase 1C3 (AKR1C3) | ENZYMES | PHARMACOLOGY & PHARMACY | TOXICOLOGY | PHASE-I | CARCINOMA | PRE-RECEPTOR REGULATION | Sulfatases - genetics | Progesterone Reductase - metabolism | Humans | Endometrial Neoplasms - metabolism | Aromatase - metabolism | Catechol O-Methyltransferase - genetics | Hydroxyprostaglandin Dehydrogenases - metabolism | Androstenedione - metabolism | Catechol O-Methyltransferase - metabolism | Aromatase - genetics | Endometrial Neoplasms - genetics | Sulfatases - metabolism | Estrogens - genetics | 3-Hydroxysteroid Dehydrogenases - metabolism | Female | Sulfotransferases - metabolism | Estrogens - metabolism | Quinones - pharmacology | Glutathione S-Transferase pi - genetics | Sulfotransferases - genetics | Estrone - genetics | Quinone Reductases - metabolism | Androstenedione - genetics | Glutathione S-Transferase pi - metabolism | Estrogens - biosynthesis | RNA, Messenger - genetics | Transcriptome - genetics | Progesterone Reductase - genetics | Arylsulfotransferase - genetics | Hydroxyprostaglandin Dehydrogenases - genetics | Estrone - analogs & derivatives | Aldo-Keto Reductase Family 1 Member C3 | Estrone - metabolism | Cell Line, Tumor | Quinone Reductases - genetics | 3-Hydroxysteroid Dehydrogenases - genetics | Arylsulfotransferase - metabolism | Physiological aspects | Endometrial cancer | Sulfates | Genes | Estrogen | Steroids | Index Medicus
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9. Full Text Eight common genetic variants associated with serum dheas levels suggest a key role in ageing mechanisms
by Zhai, Guangju and Teumer, Alexander and Stolk, Lisette and Perry, John and Vandenput, Liesbeth and Coviello, Anea and Koster, Annemarie and Bell, Jordana and Bhasin, Shalender and Eriksson, Joel and Ernst, Florian and Ferrucci, Luigi and Frayling, Timothy and Glass, Daniel and Grundberg, Elin and Haring, Robin and HedmanÅ, A.K and Hofman, Albert and Kiel, Douglas and Kroemer, Heyo and Liu, Yongmei and Lunetta, Kathryn and Maggio, Marcello and Lorentzon, Mattias and Mangino, Massimo and Melzer, David and Miljkovic, Iva and Nica, Alexana and Penninx, Brenda and Vasan, Ramachanan Srini and Rivadeneira Ramirez, Fernando and Small, Kerrin and Soranzo, Nicole and Uitterlinden, Ané and Völzke, Henry and Wilson, Scott and Xi, Li and Zhuang, W.V and Jong, Frank and Harris, Tamara and Murabito, Joanne and Ohlsson, Claes and Murray, Anna and Spector, Timothy and Wallaschofski, Henri and MuTHER Consortium
P L o S Genetics (Print), ISSN 1553-7390, 04/2011, Volume 7, Issue 4, pp. e1002025 - e1002025
textabstractDehydroepiandrosterone sulphate (DHEAS) is the most abundant circulating steroid secreted by adrenal glands-yet its function is unknown. Its serum... 
POLYCYSTIC-OVARY-SYNDROME | DEHYDROEPIANDROSTERONE-SULFATE LEVELS | RISK LOCI | BONE-MINERAL DENSITY | XENOBIOTIC METABOLISM | INSULIN-RESISTANCE | ACID-METABOLISM | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | CARDIOVASCULAR-DISEASE | GENOME-WIDE ASSOCIATION | Cytochrome P-450 CYP2C9 | Homeodomain Proteins - metabolism | Humans | Middle Aged | Aryl Hydrocarbon Hydroxylases - genetics | Actin-Related Protein 2-3 Complex - genetics | Male | Young Adult | Actin-Related Protein 2-3 Complex - metabolism | Aging - genetics | Bcl-2-Like Protein 11 | Kruppel-Like Transcription Factors - metabolism | Apoptosis Regulatory Proteins - genetics | Adult | Female | Sulfotransferases - metabolism | Membrane Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Sulfotransferases - genetics | European Continental Ancestry Group - genetics | Gene Expression | Genome-Wide Association Study | Membrane Proteins - genetics | Liver - metabolism | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | Dehydroepiandrosterone Sulfate - blood | Homeodomain Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Aryl Hydrocarbon Hydroxylases - metabolism | Linkage Disequilibrium | Aging - blood | Transcription Factors - metabolism | Adaptor Proteins, Signal Transducing - genetics | Aged | Polymorphism, Single Nucleotide | Adaptor Proteins, Signal Transducing - metabolism | Kruppel-Like Transcription Factors - genetics | Physiological aspects | Aging | Dehydroepiandrosterone | Research | Single nucleotide polymorphisms | Gene expression | Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap | Mens health | Womens health | Mortality | Men | Technological change | Genomes | Lymphomas | Diabetes | Metabolism | Age
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10. Full Text A Shared Genetic Basis for Self-Limited Delayed Puberty and Idiopathic Hypogonadotropic Hypogonadism
by Zhu, Jia and Choa, Ruth E.-Y and Guo, Michael H and Plummer, Lacey and Buck, Cassandra and Palmert, Mark R and Hirschhorn, Joel N and Seminara, Stephanie B and Chan, Yee-Ming
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 04/2015, Volume 100, Issue 4, pp. E646 - E654
Context: Delayed puberty (DP) is a common issue and, in the absence of an underlying condition, is typically self limited. Alhough DP seems to be heritable, no... 
HORMONE DEFICIENCY | VARIANTS | CONSTITUTIONAL DELAY | GROWTH | ENDOCRINOLOGY & METABOLISM | BOYS | PATTERNS | KALLMANN-SYNDROME | INHERITANCE | ASSOCIATION | ACTIVATING MUTATIONS | Receptors, Tachykinin - genetics | Sulfotransferases - genetics | Puberty, Delayed - genetics | Extracellular Matrix Proteins - genetics | Humans | Male | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Nerve Tissue Proteins - genetics | Receptors, Peptide - genetics | Hypogonadism - genetics | Tachykinins - genetics | DNA Mutational Analysis | Pedigree | Adolescent | Female | Receptors, G-Protein-Coupled - genetics | Retrospective Studies | Index Medicus | Abridged Index Medicus | Advances in Genetics | JCEM Online
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11. Full Text The peptide growth factor, phytosulfokine, attenuates pattern‐triggered immunity
by Igarashi, Daisuke and Tsuda, Kenichi and Katagiri, Fumiaki
The Plant Journal, ISSN 0960-7412, 07/2012, Volume 71, Issue 2, pp. 194 - 204
Pattern‐triggered immunity (PTI) is triggered by recognition of elicitors called microbe‐associated molecular patterns (MAMPs). Although immune responses may... 
Arabidopsis thaliana | pattern‐triggered immunity | microbe‐associated molecular pattern | phytosulfokine | receptor‐like protein kinase | PSKR | pattern-triggered immunity | microbe-associated molecular pattern | receptor-like protein kinase | DEFENSE RESPONSES | ARABIDOPSIS-THALIANA | SYSTEMIC ACQUIRED-RESISTANCE | PLANT SCIENCES | RICH REPEAT RECEPTOR | MOLECULAR-PATTERNS | TYROSYLPROTEIN SULFOTRANSFERASE | RECOGNITION RECEPTORS | MESOPHYLL-CELLS | DISEASE RESISTANCE | INNATE IMMUNITY | Arabidopsis - physiology | Plant Diseases - immunology | Arabidopsis - immunology | Plant Roots - genetics | Seedlings - genetics | Seedlings - microbiology | Plant Diseases - microbiology | Peptide Hormones - genetics | Arabidopsis Proteins - metabolism | Peptide Hormones - metabolism | Plant Roots - physiology | Intercellular Signaling Peptides and Proteins - metabolism | Seedlings - immunology | Plants, Genetically Modified | Flagellin - metabolism | Sulfotransferases - metabolism | Plant Proteins - metabolism | Pseudomonas syringae - physiology | Sulfotransferases - genetics | Arabidopsis Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Cell Communication | Gene Expression Regulation, Plant - genetics | Receptors, Cell Surface - metabolism | Plant Immunity - physiology | RNA, Plant - genetics | Plant Roots - microbiology | Plant Leaves - immunology | Arabidopsis - genetics | Arabidopsis - microbiology | Plant Proteins - genetics | DNA, Bacterial - genetics | Plant Leaves - genetics | Seedlings - physiology | Plant Roots - immunology | Signal Transduction - physiology | Mutagenesis, Insertional | Plant Leaves - microbiology | Plant Leaves - physiology | Receptors, Cell Surface - genetics | Enzymes | Anopheles | Messenger RNA | Chorionic gonadotropin | Plant physiology | Analysis | Cholecystokinin | Sulfates | Gene expression | Protein kinases | Plant biology | Signal transduction | Peptides | Plant growth | Index Medicus
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12. Full Text Axon growth and guidance genes identify T-dependent germinal centre B cells
by Yu, Di and Cook, Matthew C and Shin, Dong‐Mi and Silva, Diego G and Marshall, Jennifer and Toellner, Kai‐Michael and Havran, Wendy L and Caroni, Pico and Cooke, Michael P and Morse, Herbert C and MacLennan, Ian CM and Goodnow, Christopher C and Vinuesa, Carola G
Immunology and Cell Biology, ISSN 0818-9641, 01/2008, Volume 86, Issue 1, pp. 3 - 14
Selection of B cells subjected to hypermutation in germinal centres (GC) during T cell‐dependent (TD) antibody responses yields memory cells and long‐lived... 
axon | T‐dependent | germinal centre | B cell memory | T-dependent | Germinal centre | Axon | PLASMA-CELL | IV SEMAPHORIN CD100 | IMMUNOLOGY | CELL BIOLOGY | INVASIVE GROWTH | NEURITE OUTGROWTH | IN-VIVO | NONREDUNDANT ROLES | ANTIBODY-RESPONSES | SCATTER-FACTOR | PRIMARY IMMUNE-RESPONSE | TRANSGENIC MICE | Sulfotransferases | Antigens, T-Independent - immunology | Oligonucleotide Array Sequence Analysis | Cytoskeletal Proteins - genetics | Germinal Center - immunology | Humans | B-Lymphocyte Subsets - cytology | Axons - physiology | Lymphocyte Cooperation - genetics | Lymphocyte Cooperation - immunology | Hedgehog Proteins - genetics | B-Lymphocyte Subsets - immunology | Gene Rearrangement, B-Lymphocyte | Germinal Center - cytology | Transcription Factors - immunology | Leukemia, Lymphoid - genetics | Calmodulin-Binding Proteins - genetics | Nerve Tissue Proteins - immunology | Gene Expression Regulation - immunology | Calmodulin-Binding Proteins - immunology | Nerve Growth Factors - immunology | Transcription Factors - genetics | Organ Specificity | Nerve Tissue Proteins - genetics | Animals | Cytoskeletal Proteins - immunology | T-Lymphocytes - cytology | Nerve Growth Factors - genetics | Antigens, T-Independent - genetics | Immunologic Memory | T-Lymphocytes - immunology | Mice | Hedgehog Proteins - immunology | Leukemia, Lymphoid - immunology | Cytoskeletal Proteins, genetics | Intracellular Signaling Peptides and Proteins, metabolism | Up-Regulation | Spleen, cytology | Molecular Chaperones, metabolism | Membrane Proteins, metabolism | Germinal Center, physiology | Hedgehog Proteins, metabolism | Gene Expression Profiling | Spleen, metabolism | Sulfotransferases, genetics | Gene Expression, physiology | Sulfotransferases, metabolism | Heat-Shock Proteins, metabolism | GABA Plasma Membrane Transport Proteins, genetics | B-Lymphocytes, physiology | Germinal Center, metabolism | Membrane Proteins, genetics | RNA, Messenger, metabolism | Molecular Chaperones, genetics | Down-Regulation | B-Lymphocytes, metabolism | Reverse Transcriptase Polymerase Chain Reaction | Calmodulin-Binding Proteins, genetics | Calmodulin-Binding Proteins, metabolism | Cytoskeletal Proteins, metabolism | Nerve Tissue Proteins, genetics | GABA Plasma Membrane Transport Proteins, metabolism | Hedgehog Proteins, genetics | Intracellular Signaling Peptides and Proteins, genetics | Neurites, physiology | Nerve Tissue Proteins, metabolism | Heat-Shock Proteins, genetics | Index Medicus
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