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by Debette, Stéphanie|Ibrahim Verbaas, Carla A.|Bressler, Jan|Schuur, Maaike|Smith, Albert|Bis, Joshua C.|Davies, Gail|Wolf, Christiane|Gudnason, Vilmundur|Chibnik, Lori B.|Yang, Qiong|deStefano, Anita L.|de Quervain, Dominique J.F.|Srikanth, Velandai|Lahti, Jari|Grabe, Hans J.|Smith, Jennifer A.|Priebe, Lutz|Yu, Lei|Karbalai, Nazanin|Hayward, Caroline|Wilson, James F.|Campbell, Harry|Petrovic, Katja|Fornage, Myriam|Chauhan, Ganesh|Yeo, Robin|Boxall, Ruth|Becker, James|Stegle, Oliver|Mather, Karen A.|Chouraki, Vincent|Sun, Qi|Rose, Lynda M.|Resnick, Susan|Oldmeadow, Christopher|Kirin, Mirna|Wright, Alan F.|Jonsdottir, Maria K.|Au, Rhoda|Becker, Albert|Amin, Najaf|Nalls, Mike A.|Turner, Stephen T.|Kardia, Sharon L.R.|Oostra, Ben|Windham, Gwen|Coker, Laura H.|Zhao, Wei|Knopman, David S.|Heiss, Gerardo|Griswold, Michael E.|Gottesman, Rebecca F.|Vitart, Veronique|Hastie, Nicholas D.|Zgaga, Lina|Rudan, Igor|Polasek, Ozren|Holliday, Elizabeth G.|Schofield, Peter|Choi, Seung Hoan|Tanaka, Toshiko|An, Yang|Perry, Rodney T.|Kennedy, Richard E.|Sale, Michèle M.|Wang, Jing|Wadley, Virginia G.|Liewald, David C.|Ridker, Paul M.|Gow, Alan J.|Pattie, Alison|Starr, John M.|Porteous, David|Liu, Xuan|Thomson, Russell|Armstrong, Nicola J.|Eiriksdottir, Gudny|Assareh, Arezoo A.|Kochan, Nicole A.|Widen, Elisabeth|Palotie, Aarno|Hsieh, Yi-Chen|Eriksson, Johan G.|Vogler, Christian|van Swieten, John C.|Shulman, Joshua M.|Beiser, Alexa|Rotter, Jerome|Schmidt, Carsten O.|Hoffmann, Wolfgang|Nöthen, Markus M.|Ferrucci, Luigi|Attia, John|Uitterlinden, Andre G.|Amouyel, Philippe|Dartigues, Jean-François|Amieva, Hélène|Räikkönen, Katri|Garcia, Melissa|Wolf, Philip A.|Hofman, Albert|Longstreth, W.T.|Psaty, Bruce M.|Boerwinkle, Eric|DeJager, Philip L.|Sachdev, Perminder S.|Schmidt, Reinhold|Breteler, Monique M.B.|Teumer, Alexander|Lopez, Oscar L.|Cichon, Sven|Chasman, Daniel I.|Grodstein, Francine|Müller-Myhsok, Bertram|Tzourio, Christophe|Papassotiropoulos, Andreas|Bennett, David A.|Ikram, M. Arfan|Deary, Ian J.|van Duijn, Cornelia M.|Launer, Lenore|Fitzpatrick, Annette L.|Seshadri, Sudha|Mosley, Thomas H and Cohorts Heart & Aging Res in Genom and Cohorts for Heart and Aging Research in Genomic Epidemiology Consortium
Biological Psychiatry, ISSN 0006-3223, 2015, Volume 77, Issue 8, pp. 749 - 763
Journal Article
Brain, ISSN 0006-8950, 2005, Volume 128, Issue 8, pp. 1887 - 1896
Journal Article
by Chae, H and Kim, M and Kim, Y and Kim, J and Kwon, A and Choi, H and Park, J and Jang, W and Lee, Y.S and Park, S.H and Kim, M.S
Clinical Genetics, ISSN 0009-9163, 06/2016, Volume 89, Issue 6, pp. 678 - 689
Corneal dystrophy typically refers to a group of rare hereditary disorders with a heterogeneous genetic background. A comprehensive molecular genetic analysis... 
posterior polymorphous corneal dystrophy | macular corneal dystrophy | molecular genetic analysis | Thiel–Behnke corneal dystrophy | Fuchs endothelial corneal dystrophy | granular corneal dystrophy | Schnyder corneal dystrophy | lattice corneal dystrophy | Korea | Molecular genetic analysis | Granular corneal dystrophy | Posterior polymorphous corneal dystrophy | Thiel-Behnke corneal dystrophy | Lattice corneal dystrophy | Macular corneal dystrophy | IC3D CLASSIFICATION | MISSENSE MUTATIONS | COL8A2 GENE | HEREDITARY ENDOTHELIAL DYSTROPHY | GENETICS & HEREDITY | BIGH3 GENE | L527R MUTATION | TGFBI GENE-MUTATIONS | JAPANESE PATIENTS | PROTEIN FUNCTION | LATTICE | Sulfotransferases - genetics | Genetic Predisposition to Disease - genetics | Humans | Middle Aged | Asian Continental Ancestry Group - genetics | Corneal Dystrophies, Hereditary - genetics | Family Health | Male | Corneal Dystrophies, Hereditary - ethnology | Zinc Finger E-box-Binding Homeobox 1 - genetics | Transforming Growth Factor beta - genetics | Dimethylallyltranstransferase - genetics | DNA Mutational Analysis | Pedigree | Base Sequence | Adult | Female | Genetic Predisposition to Disease - ethnology | Aged | Mutation | Republic of Korea | Collagen Type VIII - genetics | Genetic aspects | Gene mutations | Analysis | Endothelium | Eye diseases | Cornea | Genetic disorders | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e13872 - e13872
Background: BORIS/CTCFL is a paralogue of CTCF, the major epigenetic regulator of vertebrate genomes. BORIS is normally expressed only in germ cells but is... 
CELLS | METHYLATION | IMPRINTED SITES | BIOLOGY | ENHANCER-BLOCKING | TESTIS ANTIGEN | CTCF-BINDING | H19 GENE | PROMOTER | TRANSCRIPTION FACTOR | EXPRESSION | Gametogenesis - genetics | Testis - metabolism | Alternative Splicing | Humans | Molecular Sequence Data | Male | Promoter Regions, Genetic - genetics | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | DNA Methylation | Neoplasms - genetics | Base Sequence | HEK293 Cells | Female | Repressor Proteins - metabolism | Amino Acid Sequence | Zinc Fingers | Testis - cytology | HCT116 Cells | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Repressor Proteins - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Sequence Homology, Amino Acid | Animals | CCCTC-Binding Factor | K562 Cells | Luminescent Proteins - genetics | Neoplasms - pathology | Microscopy, Fluorescence | Luminescent Proteins - metabolism | Protein Isoforms - genetics | Epigenetic inheritance | Methyltransferases | Analysis | Genes | Genomics | Genetic aspects | Genomes | Genetic transcription | Zinc finger proteins | Tumor proteins | Methylation | Cancer | Germ cells | p53 Protein | Activation | Inactivation | Nuclei | Proteins | Conserved sequence | Bursting strength | Transcription activation | Imprinting | Zinc finger | DNA methylation | Primates | Evolution | Insulin-like growth factor II | Telomerase | Speciation | Deoxyribonucleic acid--DNA | CpG islands | Binding | Antigens | Deactivation | Gametogenesis | Cloning | DNMT1 protein | Gene expression | Zinc | Infectious diseases | Sulfotransferase | Isoforms | Stem cells | Epigenetics | Aberration | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Carcinogenesis, ISSN 0143-3334, 11/2002, Volume 23, Issue 11, pp. 1839 - 1849
Susceptibility to colorectal cancer, one of the most common forms of cancer in the Western world, has been associated with several environmental and dietary... 
S-TRANSFERASE M1 | HUMAN CYTOCHROME P4501A1 | COLON-CANCER | GENETIC-POLYMORPHISM | ONCOLOGY | HETEROCYCLIC AMINES | MICROSOMAL EPOXIDE HYDROLASE | HUMAN LIVER | METABOLIC-ACTIVATION | NICOTINE METABOLISM | MEAT CONSUMPTION | Oxidoreductases Acting on CH-NH Group Donors - physiology | Diet - adverse effects | Epoxide Hydrolases - physiology | Adenocarcinoma - chemically induced | Carcinogens - adverse effects | Colorectal Neoplasms - genetics | Humans | Middle Aged | England | Male | Methylenetetrahydrofolate Reductase (NADPH2) | Cell Transformation, Neoplastic - chemically induced | NAD(P)H Dehydrogenase (Quinone) - genetics | Case-Control Studies | Glutathione Transferase - genetics | Polycyclic Aromatic Hydrocarbons - adverse effects | Cell Transformation, Neoplastic - genetics | Glutathione Transferase - physiology | Biotransformation | Aged, 80 and over | Female | Adenocarcinoma - genetics | Amines - adverse effects | Carcinogens - pharmacokinetics | Sulfotransferases - physiology | Cytochrome P-450 Enzyme System - physiology | Isoenzymes - physiology | Colorectal Neoplasms - epidemiology | Sulfotransferases - genetics | Polycyclic Aromatic Hydrocarbons - pharmacokinetics | Colorectal Neoplasms - enzymology | Genetic Predisposition to Disease | Isoenzymes - genetics | Adenocarcinoma - enzymology | Oxidoreductases Acting on CH-NH Group Donors - genetics | Genotype | Colorectal Neoplasms - chemically induced | Epoxide Hydrolases - genetics | Heterocyclic Compounds - adverse effects | Alleles | Amines - pharmacokinetics | Cytochrome P-450 Enzyme System - genetics | Heterocyclic Compounds - pharmacokinetics | Aged | Arylamine N-Acetyltransferase - genetics | NAD(P)H Dehydrogenase (Quinone) - physiology | Adenocarcinoma - epidemiology | Arylamine N-Acetyltransferase - physiology | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2013, Volume 123, Issue 11, pp. 4909 - 4917
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2010, Volume 107, Issue 22, pp. 10202 - 10207
The balance between anabolic and catabolic signaling pathways is critical in maintaining cartilage homeostasis and its disturbance contributes to joint... 
Cartilage | Knee joint | Phosphorylation | Receptors | Articular cartilage | Chondrocytes | Small interfering RNA | Fibroblast growth factors | Gene expression regulation | Osteoarthritis | Heparan sulfate | Smad | Erk | osteoarthritis | MULTIDISCIPLINARY SCIENCES | FIBROBLAST-GROWTH-FACTOR | HUMAN ARTICULAR CHONDROCYTES | NOGGIN | heparan sulfate | INTERVERTEBRAL DISC | HEPARAN-SULFATE | MICE | BONE MORPHOGENETIC PROTEIN-4 | EXPRESSION | SPEMANN ORGANIZER | RNA, Small Interfering - genetics | Sulfatases - genetics | Humans | Homeostasis | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Sulfotransferases - deficiency | Fibroblast Growth Factors - metabolism | Bone Morphogenetic Proteins - metabolism | Cartilage, Articular - metabolism | Sulfatases - metabolism | Fibroblast Growth Factor 2 - metabolism | Sulfotransferases - metabolism | Osteoarthritis - pathology | Chondrocytes - metabolism | Sulfatases - antagonists & inhibitors | Sulfotransferases - genetics | ADAMTS5 Protein | Signal Transduction | Matrix Metalloproteinase 13 - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Osteoarthritis - metabolism | Smad Proteins - genetics | Sulfatases - deficiency | Osteoarthritis - genetics | Mice, Knockout | Carrier Proteins - genetics | Cartilage, Articular - pathology | Animals | Bone Morphogenetic Protein 7 - metabolism | Mice | ADAM Proteins - genetics | Osteoarthritis - etiology | Smad Proteins - metabolism | Extracellular signal-regulated kinases | Bone morphogenetic proteins | Genetic aspects | Cellular signal transduction | Properties | Observations | Methods | Studies | Proteins | Rodents | Arthritis | Gene expression | Index Medicus | Biological Sciences
Journal Article
BMC Cancer, ISSN 1471-2407, 01/2013, Volume 13, Issue 1, pp. 24 - 24
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