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Journal of Biological Chemistry, ISSN 0021-9258, 12/2011, Volume 286, Issue 52, pp. 44557 - 44568
Journal Article
Oncogene, ISSN 0950-9232, 08/2015, Volume 34, Issue 32, pp. 4229 - 4237
Manganese superoxide dismutase (MnSOD) is a mitochondrially localized primary antioxidant enzyme, known to be essential for the survival of aerobic life and to... 
OXIDATIVE STRESS | LIPID-PEROXIDATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALPHA | CELL BIOLOGY | OVEREXPRESSION | SKIN CARCINOGENESIS MODEL | EPITHELIAL-CELLS | ONCOLOGY | GLUCOSE | GENETICS & HEREDITY | CANCER METABOLISM | PROTEIN-2 | EXPRESSION | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Ion Channels - genetics | Mitochondrial Proteins - genetics | Phosphatidylinositol 3-Kinases - metabolism | TOR Serine-Threonine Kinases - genetics | Mitochondria - genetics | RNA Interference | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Cells, Cultured | PPAR alpha - genetics | Superoxide Dismutase - deficiency | Mitochondria - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Animals | Lactates - metabolism | Ion Channels - metabolism | Glycolysis | Uncoupling Protein 2 | PPAR alpha - metabolism | Uncoupling Protein 1 | Physiological aspects | Superoxide dismutase | Genetic aspects | Mitochondrial diseases | Research | Carcinogenesis | Risk factors | Antioxidants | Enzymes | Signal transduction | Protein expression | Mitochondria | Cancer | Energetic metabolism | UCPs | Redox homeostasis | MnSOD | glycolysis
Journal Article
PLoS Neglected Tropical Diseases, ISSN 1935-2727, 07/2018, Volume 12, Issue 7, p. e0006687
In this study, we have investigated the effects of manganese superoxide dismutase (SOD2 or MnSOD) deficiency on mitochondrial function and oxidative stress... 
INHIBITION | PATHWAY | MNSOD | CARDIOMYOPATHY | MYOCARDITIS | MICE | DYSFUNCTION | STRESS | EXPRESSION | PARASITOLOGY | TROPICAL MEDICINE | TERT-BUTYL-NITRONE | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | Chagas Cardiomyopathy - parasitology | Oxidative Stress | Chagas Cardiomyopathy - genetics | Humans | Mice, Inbred C57BL | Oxidative Phosphorylation | Male | Superoxide Dismutase - deficiency | Mitochondria - metabolism | Chagas Cardiomyopathy - metabolism | Hydrogen Peroxide - metabolism | Mice, Knockout | Trypanosoma cruzi - physiology | Animals | Chagas Cardiomyopathy - enzymology | Mitochondria - genetics | Myocardium - metabolism | Female | Mice | Complications and side effects | Mitochondria | Cardiomyopathy | Genetic aspects | Superoxide | Research | Gene expression | Health aspects | Heart diseases | Risk factors | Trypanosoma cruzi | Heart | Additives | 4-Hydroxynonenal | Disease | Lactate | Chronic infection | ADP | Proteins | Electron transport chain | Synthesis | Capacity | Damage | Deoxyribonucleic acid--DNA | Enzymes | Malondialdehyde | Diseases | Myocardium | Chagas' disease | Oxidative stress | Carbonyl compounds | Reactive oxygen species | Phosphorylation | Hydrogen peroxide | Inflammatory response | Superoxide dismutase | Infections | Parasites | Interleukin 6 | Immunology | Clonal deletion | Deletion | Peroxidase | Carbonyls | Chronic illnesses | Vector-borne diseases | Protozoa | NADH-ubiquinone oxidoreductase | Cardiomyocytes | Lactate dehydrogenase | Inflammation | L-Lactate dehydrogenase | Oxidative phosphorylation | Lactic acid | Genetic testing | ATP | Manganese | Deoxyribonucleic acid | DNA
Journal Article
ONCOGENE, ISSN 0950-9232, 07/2017, Volume 36, Issue 28, pp. 4087 - 4087
Journal Article
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 08/2005, Volume 172, Issue 3, pp. 306 - 313
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2011, Volume 251, Issue 3, pp. 226 - 233
Acetaminophen (APAP) hepatotoxicity is the most frequent cause of acute liver failure in many countries. The mechanism of cell death is initiated by formation... 
Manganese-SOD | Bax | Oxidative stress | c-jun-N-terminal kinase | Acetaminophen | Apoptosis-inducing factor | C-jun-N-terminal kinase | HETEROZYGOUS SOD2(+/-) MICE | OXIDATIVE DAMAGE | COVALENT BINDING | N-ACETYLCYSTEINE | KNOCKOUT MICE | TERMINAL KINASE | CELL-DEATH | REACTIVE NITROGEN | INDUCED HEPATIC-NECROSIS | IN-VIVO | PHARMACOLOGY & PHARMACY | TOXICOLOGY | In Situ Nick-End Labeling | Superoxide Dismutase - genetics | Acetaminophen - toxicity | Drug Overdose | Glutathione - metabolism | Mitochondria, Liver - metabolism | Mice, Inbred C57BL | Acetaminophen - administration & dosage | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Analgesics, Non-Narcotic - administration & dosage | Glutathione Disulfide - metabolism | Necrosis - chemically induced | Mitochondria, Liver - drug effects | Animals | Analgesics, Non-Narcotic - toxicity | Signal Transduction - drug effects | Mice | Alanine Transaminase - metabolism | DNA Fragmentation - drug effects | Oxidative Stress - drug effects | Chemical and Drug Induced Liver Injury - etiology | Drugs | Proteins | Liver diseases | Liver | Genetic research | Overdose | Mitochondrial DNA | Superoxide | Protein binding | Index Medicus | APOPTOSIS | SUPEROXIDE DISMUTASE | DIGESTIVE SYSTEM | MITOCHONDRIA | TRANSITION ELEMENTS | 60 APPLIED LIFE SCIENCES | NUCLEIC ACIDS | STRESSES | ELEMENTS | NECROSIS | GLANDS | METALS | MANGANESE | OXIDIZERS | ENZYMES | DRUGS | VERTEBRATES | MICE | RADIOPROTECTIVE SUBSTANCES | MAMMALS | ANIMALS | INJURIES | RODENTS | RESPONSE MODIFYING FACTORS | ORGANIC COMPOUNDS | FRAGMENTATION | POLYPEPTIDES | ORGANS | DISEASES | PEPTIDES | GLUTATHIONE | DNA | LIVER | PROTEINS | BODY | PATHOLOGICAL CHANGES | CELL CONSTITUENTS | OXIDOREDUCTASES | manganese-SOD | apoptosis-inducing factor | oxidative stress
Journal Article
Reproduction, ISSN 1470-1626, 10/2013, Volume 146, Issue 4, pp. 297 - 304
Oxidative stress, overproduction of reactive oxygen species (ROS) in relation to defence mechanisms, is considered to be a major cause of male infertility. For... 
MALE-INFERTILITY | OXIDATIVE STRESS | REPRODUCTIVE BIOLOGY | TESTES SIZE | LIPID-PEROXIDATION |