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Journal of Immunology, ISSN 0022-1767, 11/2010, Volume 185, Issue 10, pp. 6226 - 6233
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2006, Volume 7, Issue 2, pp. 148 - 155
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 136, Issue 1, pp. 177 - 188.e11
Background Rhinovirus infections are the dominant cause of asthma exacerbations, and deficient virus induction of IFN-α/β/λ in asthmatic patients is important... 
Allergy and Immunology | cytokine | suppressor of cytokine signaling | innate immunity | atopy | asthma exacerbation | asthma | interferon | TH2 inflammation | Rhinovirus | Cytokine | Innate immunity | Suppressor of cytokine signaling | Interferon | Asthma exacerbation | Asthma | Atopy | 2 inflammation | CELLS | RECEPTOR | IMMUNOLOGY | T(H)2 inflammation | I INTERFERON | BETA | ANTIVIRAL RESPONSES | ALLERGY | IMMUNE-RESPONSES | DEFICIENT | DOUBLE-BLIND | EXPRESSION | IFN-ALPHA | Humans | Middle Aged | Immunity, Innate - genetics | Child, Preschool | Suppressor of Cytokine Signaling 1 Protein | Male | Respiratory Mucosa - virology | Interferon-gamma - metabolism | Rhinovirus - physiology | Picornaviridae Infections - virology | Young Adult | Cell Nucleus - metabolism | Respiratory Mucosa - immunology | Asthma - immunology | Adult | Female | Asthma - virology | Interferon-gamma - genetics | Child | Picornaviridae Infections - immunology | Mice, Inbred C57BL | Cells, Cultured | Suppressor of Cytokine Signaling Proteins - genetics | Up-Regulation - genetics | Mutation - genetics | Mice, Knockout | Protein Transport | Animals | Virus Replication | Asthma - complications | Adolescent | Mice | Picornaviridae Infections - complications | Suppressor of Cytokine Signaling Proteins - metabolism | Cytokines | Mortality | Families & family life | Infections | Kinases | Gene expression | Investigations | Molecular weight | Proteins | Human subjects | Rodents | Protein expression | Viral infections | Index Medicus | Abridged Index Medicus | ISRE, Interferon-stimulated response element | NF-κB, Nuclear factor κB | BEC, Bronchial epithelial cell | ISG, Interferon-stimulated gene | GFP, Green fluorescent protein | SOCS, Suppressor of cytokine signaling | BAL, Bronchoalveolar lavage | NANA, Nonatopic nonasthmatic | LIX, LPS-induced CXC chemokine | NLS, Nuclear localization sequence | AA, Atopic asthma | CISH, Cytokine-inducible SH2-containing protein | STRA, Severe therapy-resistant atopic asthma | polyI:C, Polyinosinic-polycytidylic acid | STAT, Signal transducer and activator of transcription | SOCS1wt, Full-length wild-type human SOCS1 | KC, Keratinocyte-derived chemokine | Mechanisms of Allergy and Clinical Immunology
Journal Article
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 02/2019, Volume 21, Issue 1, pp. 50 - 50
BackgroundAcute gouty arthritis currently is the most common form of inflammatory arthritis in developed countries. Treatment is still suboptimal. Dosage of... 
Inflammation | Cytokines | Gout | HDAC | HDAC INHIBITORS | POPULATION | HISTONE DEACETYLASE INHIBITOR | PREVALENCE | RHEUMATOLOGY | TRICHOSTATIN-A | PATHWAY | INDUCED ARTHRITIS | NF-KAPPA-B | Usage | Care and treatment | Analysis | Development and progression | Romidepsin | Patient compliance | Enzymes | Industrialized nations | Cell cycle | Rheumatism | Arthritis | Gene expression | Kinases
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2008, Volume 180, Issue 6, pp. 3746 - 3756
Suppressor of cytokine signaling 1 (SOCS1) is an important negative regulator for cytokines; however, the role of SOCS1 in Th17 differentiation has not been... 
SUPPRESSOR | GROWTH-FACTOR-BETA | TGF-BETA | SOCS3 GENE | INTERFERON-GAMMA | DENDRITIC CELLS | IMMUNE-RESPONSES | CYTOKINE SIGNALING-1 | GENE-EXPRESSION | IMMUNOLOGY | AUTOIMMUNE ENCEPHALOMYELITIS | Th1 Cells - pathology | Smad Proteins - antagonists & inhibitors | Encephalomyelitis, Autoimmune, Experimental - metabolism | Interleukin-17 - physiology | Encephalomyelitis, Autoimmune, Experimental - immunology | Molecular Sequence Data | Suppressor of Cytokine Signaling 1 Protein | Th1 Cells - immunology | Suppressor of Cytokine Signaling Proteins - deficiency | Th1 Cells - metabolism | Cell Differentiation - genetics | STAT1 Transcription Factor - metabolism | T-Lymphocytes - metabolism | Interferon-gamma - antagonists & inhibitors | T-Lymphocytes - pathology | STAT3 Transcription Factor - metabolism | Amino Acid Sequence | Encephalomyelitis, Autoimmune, Experimental - pathology | Mice, Inbred C57BL | Cells, Cultured | Interferon-gamma - physiology | Suppressor of Cytokine Signaling Proteins - genetics | Mice, Transgenic | Mice, Knockout | Cell Differentiation - immunology | Encephalomyelitis, Autoimmune, Experimental - therapy | Animals | Suppressor of Cytokine Signaling Proteins - physiology | T-Lymphocytes - immunology | Mice | STAT3 Transcription Factor - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 23, pp. 9554 - 9559
Endogenous and pharmacologic glucocorticoids (GCs) limit inflammatory cascades initiated by Toll-like receptor (TLR) activation. A long-standing clinical... 
Phosphorylation | Cytokines | Glucocorticoids | Secretion | Interferons | Ligands | Mice | Physiological regulation | Pretreatment | Macrophages | JAK | Knockout mice | CELLS | knockout mice | INFLAMMATORY RESPONSES | PHOSPHORYLATION | IFN-GAMMA | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | COOPERATION | INHIBITION | GENE | MODULATION | ACUTE ASTHMA | Suppressor of Cytokine Signaling 1 Protein | Mice, 129 Strain | STAT1 Transcription Factor - metabolism | Dexamethasone - pharmacology | Janus Kinase 2 - metabolism | Phosphorylation - drug effects | Toll-Like Receptors - metabolism | Interleukin-6 - metabolism | Interferon-alpha - metabolism | Interleukin-12 Subunit p40 - metabolism | Poly I-C - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Suppressor of Cytokine Signaling Proteins - genetics | Macrophages - cytology | STAT1 Transcription Factor - genetics | Blotting, Western | Mice, Knockout | Receptors, Interferon - metabolism | Macrophages - metabolism | Animals | Interferon-beta - pharmacology | Lipopolysaccharides - pharmacology | Glucocorticoids - pharmacology | Interferon Regulatory Factor-3 - metabolism | Macrophages - drug effects | Suppressor of Cytokine Signaling Proteins - metabolism | Signal transduction | T cell receptors | Effects | Rodents | Index Medicus | Cell activation | Transcription | Toll-like receptors | Data processing | Inflammation | Interferon | SOCS-1 protein | Stat1 protein | TLR3 protein | Biological Sciences
Journal Article
Journal Article
Journal Article