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Protein Expression and Purification, ISSN 1046-5928, 08/2019, Volume 160, pp. 73 - 83
Survivin is a well-known inhibitor-of-apoptosis proteins family member and a promising molecular target for anti-cancer treatment. However, it is widely... 
Inhibitor-of-apoptosis proteins | Survivin C84A mutant | Survivin T34A-C84A mutant | Cancer | Survivin T34A mutant
Journal Article
Experimental and Molecular Pathology, ISSN 0014-4800, 06/2019, Volume 108, pp. 24 - 31
Survivin is abundantly expressed during fetal development but absent in most differentiated adult tissues; an exception being components of the immune system,... 
Survivin splice variants | Survivin | Hodgkin lymphoma
Journal Article
by Wu, Yudong and Wang, G and Wei, J and Wen, X
Survivin protein expression positively correlated with proliferative activity of cancer cells in bladder cancer, 12/2005
CONTEXT: Survivin is an inhibitor of apoptosis that is selectively over-expressed in common human cancers, but not in normal tissues, and that correlates with... 
Apoptosis, bladder cancer, proliferative activity, survivin
Journal
International Journal of Molecular Sciences, ISSN 1661-6596, 09/2018, Volume 19, Issue 9, p. 2664
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 06/2016, Volume 95, pp. 278 - 292
Microvascular endothelial cells (CMECs) oxidative damage resulting from hypoxia/reoxygenation (H/R) injury is responsible for microcirculation perfusion... 
PI3K/Akt pathway | ROS | Survivin | Liraglutide | Calcium homeostasis | CMECs | Apoptosis
Journal Article
Cancer Letters, ISSN 0304-3835, 04/2018, Volume 420, pp. 97 - 108
Elevated expression of HER3, which interacts with HER2 in breast cancer cells, confers chemoresistance via phosphoinositide 3-kinase (PI-3K)/Akt-dependent... 
miRNA | Breast cancer | Survivin | Paclitaxel resistance | HER3 | THERAPEUTICS | APOPTOSIS | PROLIFERATION | MICRORNAS | CELL LUNG-CANCER | HEPATOCELLULAR-CARCINOMA | ERBB3 | ONCOLOGY | GROWTH | UP-REGULATION | EXPRESSION | Neoplasm Transplantation | Up-Regulation | MicroRNAs - antagonists & inhibitors | Paclitaxel - pharmacology | Receptor, ErbB-2 - genetics | Humans | Gene Expression Regulation, Neoplastic | Breast Neoplasms - metabolism | Biomimetic Materials - administration & dosage | Survivin - genetics | Female | Survivin - chemistry | Protein Stability | Survivin - metabolism | Receptor, ErbB-3 - genetics | Breast Neoplasms - drug therapy | Paclitaxel - therapeutic use | Biomimetic Materials - pharmacology | Animals | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | MicroRNAs - genetics | Drug Resistance, Neoplasm - drug effects | Antimitotic agents | RNA | Antineoplastic agents | Cell culture | Colorectal cancer | Chemoresistance | AKT protein | Kinases | Drug resistance | Cancer therapies | Anticancer properties | Liver cancer | Cell cycle | Paclitaxel | DNA methylation | Inhibition | Deoxyribonucleic acid--DNA | Immunoglobulins | ErbB-2 protein | Signaling | Gene amplification | Chemotherapy | MicroRNAs | Epigenetics | Breast | Antitumor activity | Prostate cancer | Binding sites | Cancer | Apoptosis | Index Medicus
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 04/2018, Volume 150, pp. 72 - 85
DHA downregulated Mcl-1 and Survivin by targeting the JAK2-STAT3 signaling pathway and upregulating Bim, thereby synergizing with ABT-263 to trigger apoptosis... 
Dihydroartemisinin | Mcl-1 | Survivin | ABT-263 | STAT3 | Bim | ACTIVATION | PHOSPHORYLATION | KINASE | DOWN-REGULATION | SENSITIVITY | MECHANISMS | RESISTANCE | PHARMACOLOGY & PHARMACY | BCL-2 FAMILY | DERIVATIVES | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Antineoplastic Agents - administration & dosage | Dose-Response Relationship, Drug | Mutation - physiology | Survivin - genetics | Female | Aniline Compounds - administration & dosage | STAT3 Transcription Factor - metabolism | Genes, ras - genetics | Lung Neoplasms - genetics | A549 Cells | Myeloid Cell Leukemia Sequence 1 Protein - antagonists & inhibitors | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Artemisinins - administration & dosage | Survivin - antagonists & inhibitors | Xenograft Model Antitumor Assays - methods | Myeloid Cell Leukemia Sequence 1 Protein - genetics | Survivin - biosynthesis | Drug Synergism | Gene Expression Regulation, Enzymologic | Animals | Mice, Nude | Myeloid Cell Leukemia Sequence 1 Protein - biosynthesis | Mice | Mice, Inbred BALB C | Apoptosis - physiology | STAT3 Transcription Factor - antagonists & inhibitors | Sulfonamides - administration & dosage | Lung cancer, Small cell | Development and progression | Genetic aspects | Epidermal growth factor | Lung cancer, Non-small cell | Apoptosis | Index Medicus
Journal Article
Indian Journal of Medical Research, ISSN 0971-5916, 2015, Volume 142, Issue April, pp. 389 - 397
Journal Article
Journal of Experimental & Clinical Cancer Research, ISSN 0392-9078, 08/2019, Volume 38, Issue 1, pp. 1 - 22
Survivin (also named BIRC5) is a well-known cancer therapeutic target. Since its discovery more than two decades ago, the use of survivin as a target for... 
Usage | Care and treatment | Oncology, Experimental | Survivin | Drug discovery | Research | Licensing, certification and accreditation | Cancer | cIAP2 | survivin | Mcl-1 | BIRC5 | XIAP | Survivin cancer therapeutics
Journal Article
Asian Pacific Journal of Cancer Prevention, ISSN 1513-7368, 2015, Volume 16, Issue 15, pp. 6187 - 6191
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 11383 - 11
Journal Article