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Blood, ISSN 0006-4971, 02/2017, Volume 129, Issue 6, pp. 759 - 770
Kinases downstream of B-cell antigen receptor (BCR) represent attractive targets for therapy in non-Hodgkin lymphoma (NHL). As clinical responses vary,... 
SURFACE IGM | BCR | CHRONIC-LYMPHOCYTIC-LEUKEMIA | BRUTONS TYROSINE KINASE | HUMAN FOLLICULAR LYMPHOMA | GENE-EXPRESSION | KINASE INHIBITOR IBRUTINIB | MUTATIONS | TARGETING BTK | HEMATOLOGY | NF-KAPPA-B | Lymphoma, Follicular - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Lymphoma, Mantle-Cell - pathology | Leukemia, Lymphocytic, Chronic, B-Cell - genetics | Receptors, Antigen, B-Cell - metabolism | STAT1 Transcription Factor - metabolism | Lymphoma, Follicular - genetics | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Single-Cell Analysis | Immunoglobulin M - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | STAT1 Transcription Factor - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Lymphoma, Large B-Cell, Diffuse - diagnosis | Mitogen-Activated Protein Kinase 3 - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Receptors, Antigen, B-Cell - genetics | CD79 Antigens - genetics | src-Family Kinases - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Gene Expression Regulation, Neoplastic | Lymphoma, Follicular - diagnosis | Lymphoma, Large B-Cell, Diffuse - metabolism | Phosphoproteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Lymphoma, Mantle-Cell - diagnosis | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Protein-Tyrosine Kinases - genetics | Flow Cytometry | Syk Kinase - genetics | Phospholipase C gamma - genetics | src-Family Kinases - metabolism | Lymphoma, Mantle-Cell - genetics | Lymphoma, Mantle-Cell - metabolism | Diagnosis, Differential | Phospholipase C gamma - metabolism | Lymphoma, Large B-Cell, Diffuse - pathology | Lymphoma, Follicular - pathology | p38 Mitogen-Activated Protein Kinases - genetics | Phosphoproteins - genetics | CD79 Antigens - metabolism | Syk Kinase - metabolism | Lymphoma, Large B-Cell, Diffuse - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - diagnosis | Life Sciences | Human health and pathology | Hematology | Cancer | Lymphoid Neoplasia
Journal Article
Immunity, ISSN 1074-7613, 04/2017, Volume 46, Issue 4, pp. 635 - 648
Mice carrying a hypomorphic point mutation in the gene ( mice) develop an inflammatory skin disease that resembles neutrophilic dermatosis in humans. Here, we... 
myeloid cells | SYK | RIPK3 | MLKL | autoinflammation | Ptpn6 | IL-1β | IL-1α | MyD88 | SHP1 | TAK1 | necrosis | RIPK1 | MOTH-EATEN MICE | SIGNALING PATHWAYS | STERILE INFLAMMATION | GENE | APOPTOTIC CELL-DEATH | PHOSPHATASE | IN-VIVO | IMMUNOLOGY | SHP-1 | NF-KAPPA-B | AUTOINFLAMMATORY DISEASE | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - immunology | Phosphorylation | Interleukin-1alpha - immunology | Skin Diseases - genetics | Humans | Immunoblotting | Interleukin-1alpha - metabolism | Syk Kinase - immunology | Signal Transduction - immunology | Inflammation - metabolism | Flow Cytometry | Syk Kinase - genetics | HEK293 Cells | Myeloid Differentiation Factor 88 - immunology | Interleukin-1alpha - genetics | Receptor-Interacting Protein Serine-Threonine Kinases - immunology | Skin Diseases - metabolism | MAP Kinase Kinase Kinases - genetics | Receptors, Interleukin-1 - immunology | Myeloid Differentiation Factor 88 - genetics | Skin Diseases - immunology | Inflammation - immunology | MAP Kinase Kinase Kinases - metabolism | Signal Transduction - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - genetics | MAP Kinase Kinase Kinases - immunology | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Models, Immunological | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Syk Kinase - metabolism | Animals | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Inflammation - genetics | Mutation | Myeloid Differentiation Factor 88 - metabolism | Disease | SHP-1 protein | Crosstalk | Interleukin | Licenses | Interleukin 1 receptors | Kinases | Phosphatase | Inflammatory diseases | Proteins | Coding | Rodents | Interleukin 1 | Post-translation | Bone marrow | Skin diseases | Tumor necrosis factor-TNF | Protein-tyrosine kinase | Activation analysis | Immune system | Tyrosine | TAK1 protein | Neutrophils | Leukocytes (neutrophilic) | Inflammation | Roles | Scaffolding | Point mutation | MyD88 protein | Syk protein
Journal Article
Nature, ISSN 0028-0836, 01/2012, Volume 481, Issue 7381, pp. 329 - 334
Retinoblastoma is an aggressive childhood cancer of the developing retina that is initiated by the biallelic loss of RB1. Tumours progress very quickly... 
ANEUPLOIDY | CELLS | GENE | TYROSINE KINASE | MCL-1 | MULTIDISCIPLINARY SCIENCES | GROWTH | TUMOR | SYK INHIBITOR | CANCER | Retinoblastoma - genetics | Protein-Tyrosine Kinases - metabolism | Genomics | Humans | Retinoblastoma - drug therapy | Gene Expression Regulation, Neoplastic | Aneuploidy | Intracellular Signaling Peptides and Proteins - metabolism | Genes, Retinoblastoma - genetics | Molecular Targeted Therapy | Retinoblastoma - pathology | Protein-Tyrosine Kinases - genetics | Cell Death - drug effects | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Cell Line | Cell Survival - drug effects | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Retinoblastoma Protein - deficiency | Mutation - genetics | Sequence Analysis, DNA | Xenograft Model Antitumor Assays | Chromosomal Instability - genetics | Animals | Epigenesis, Genetic - genetics | Protein Kinase Inhibitors - therapeutic use | Retinoblastoma Protein - genetics | Mice | Protein Kinase Inhibitors - pharmacology | Protein-Tyrosine Kinases - antagonists & inhibitors | Gene mutations | Development and progression | Genetic aspects | Diagnosis | Research | Retinoblastoma | Gene therapy | Health aspects | Risk factors | Studies | Human subjects | Epigenetics | DNA methylation | Genomes | Mutation | Kinases | Experiments | Chromosomes | Cancer | Children & youth | Apoptosis
Journal Article
Circulation Research, ISSN 0009-7330, 06/2009, Volume 104, Issue 12, pp. 1355 - 1363
Toll-like receptor (TLR)4 recognizes microbial pathogens, such as lipopolysaccharide, and mediates lipopolysaccharide-induced proinflammatory cytokine... 
Oxidized lipoprotein | Toll-like receptor-4 | Macrophage | Oxidized cholesteryl ester | toll-like receptor-4 | CARDIAC & CARDIOVASCULAR SYSTEMS | CHOLESTERYL ESTER HYDROPEROXIDES | oxidized cholesteryl ester | LOW-DENSITY-LIPOPROTEIN | macrophage | APOLIPOPROTEIN-E | PATTERN-RECOGNITION | FOAM-CELL-FORMATION | SCAVENGER-RECEPTOR-A | APOPTOTIC CELLS | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | HEMATOLOGY | E-DEFICIENT MICE | NF-KAPPA-B | oxidized lipoprotein | Cholesterol Esters - metabolism | Inflammation - pathology | Protein-Tyrosine Kinases - metabolism | Humans | Cercopithecus aethiops | rac GTP-Binding Proteins - metabolism | Pinocytosis | cdc42 GTP-Binding Protein - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Lipoproteins, LDL - genetics | MAP Kinase Signaling System | Inflammation - metabolism | Phosphorylation - genetics | Protein-Tyrosine Kinases - genetics | Mice, Mutant Strains | rac GTP-Binding Proteins - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Lipoproteins, LDL - metabolism | Cholesterol Esters - genetics | Foam Cells - pathology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Foam Cells - metabolism | Cytoskeleton - pathology | Mitogen-Activated Protein Kinase 3 - genetics | rho GTP-Binding Proteins - genetics | Cytoskeleton - genetics | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | cdc42 GTP-Binding Protein - genetics | rho GTP-Binding Proteins - metabolism | Cytoskeleton - metabolism | Inflammation - genetics | Ligands | Mice | COS Cells | Chronic Disease | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Immunity, ISSN 1074-7613, 03/2013, Volume 38, Issue 3, pp. 475 - 488
Follicular B cell survival requires signaling from BAFFR, a receptor for BAFF and the B cell antigen receptor (BCR). This “tonic” BCR survival signal is... 
TACI | AKT | BCMA | IMMUNOLOGY | MATURATION | ACTIVATING FACTOR | AUTOIMMUNE-DISEASE | SYK TYROSINE KINASE | MICE LACKING | FAMILY | ANTIGEN | Oligonucleotide Array Sequence Analysis | Protein-Tyrosine Kinases - metabolism | Cell Survival - genetics | Immunoblotting | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Receptors, Antigen, B-Cell - metabolism | Extracellular Signal-Regulated MAP Kinases - immunology | Phosphatidylinositol 3-Kinases - immunology | Phosphorylation - drug effects | B-Cell Activation Factor Receptor - genetics | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | 3-Phosphoinositide-Dependent Protein Kinases | B-Lymphocytes - metabolism | Protein-Serine-Threonine Kinases - metabolism | B-Cell Activation Factor Receptor - immunology | Cell Survival - drug effects | RNA, Untranslated | Mice, Transgenic | Mice, Knockout | B-Cell Activation Factor Receptor - metabolism | Models, Immunological | B-Lymphocytes - immunology | Signal Transduction - drug effects | Mice | CD79 Antigens - immunology | Intracellular Signaling Peptides and Proteins - immunology | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Protein-Tyrosine Kinases - immunology | Signal Transduction - immunology | Protein-Tyrosine Kinases - genetics | Flow Cytometry | B-Cell Activating Factor - immunology | Protein-Serine-Threonine Kinases - genetics | Cell Survival - immunology | Proteins - immunology | Receptors, Antigen, B-Cell - immunology | CD79 Antigens - metabolism | Proteins - genetics | Receptor Cross-Talk - immunology | B-Lymphocytes - drug effects | Animals | Proteins - metabolism | Tamoxifen - pharmacology | Protein-Serine-Threonine Kinases - immunology | B-Cell Activating Factor - pharmacology | Proteins | Genetic research | Cells | Bone marrow | Antigens | Kinases | Rodents | Immune system
Journal Article
Immunity, ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 443 - 456
The mechanisms underlying human natural killer (NK) cell phenotypic and functional heterogeneity are unknown. Here, we describe the emergence of diverse... 
NATURAL-KILLER-CELLS | ACTIVATION | CMV REACTIVATION | INNATE | RECEPTOR | FCR-GAMMA | DIFFERENTIATION | IMMUNOLOGY | PLZF CONTROLS | EXPRESSION | T-CELLS | Cell Proliferation | Humans | Transcription Factors - deficiency | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | T-Lymphocytes, Cytotoxic - pathology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Kruppel-Like Transcription Factors - deficiency | Transcription Factors - immunology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Adaptive Immunity | T-Lymphocytes, Cytotoxic - immunology | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | T-Lymphocytes, Cytotoxic - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Transcription Factors - genetics | Killer Cells, Natural - classification | Promyelocytic Leukemia Zinc Finger Protein | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Kruppel-Like Transcription Factors - genetics | Kruppel-Like Transcription Factors - immunology | GPI-Linked Proteins - genetics | Virus diseases | Proteins | Epigenetic inheritance | Killer cells | Immunotherapy | Genomics | Medical genetics | Cytomegalovirus infections | T cells | Methylation | Health aspects | Antigens | Flow cytometry | Cytomegalovirus | Data analysis | Biomedical research | Transplants & implants | Cytokines | Leukemia | Cytotoxicity | Infections | Kinases | Diversification | Gene expression | Experiments | Scholarships & fellowships | Lymphocytes | DNA methylation | Epigenetics | Viral infections | Immune system | Cancer
Journal Article
Nature Immunology, ISSN 1529-2908, 12/2013, Volume 14, Issue 12, pp. 1247 - 1255
The inflammasome adaptor ASC contributes to innate immunity through the activation of caspase-1. Here we found that signaling pathways dependent on the kinases... 
LISTERIA-MONOCYTOGENES | AIM2 INFLAMMASOME | PROTEIN | INNATE IMMUNE-RESPONSES | MACROPHAGES | KINASE | HOST-DEFENSE | NLRP3 INFLAMMASOME | IMMUNOLOGY | CASPASE-1 ACTIVATION | VIRAL-INFECTION | Interleukin-18 - immunology | Inflammasomes - metabolism | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Protein-Tyrosine Kinases - metabolism | Dendritic Cells - immunology | Humans | JNK Mitogen-Activated Protein Kinases - immunology | Caspase 1 - metabolism | Immunoblotting | Intracellular Signaling Peptides and Proteins - metabolism | RNA Interference | Bone Marrow Cells - immunology | Phosphorylation - immunology | JNK Mitogen-Activated Protein Kinases - genetics | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Carrier Proteins - immunology | DNA-Binding Proteins | Tyrosine - immunology | Mice, Knockout | Macrophages - metabolism | Tyrosine - metabolism | Lipopolysaccharides - pharmacology | Mice | Interleukin-18 - metabolism | Intracellular Signaling Peptides and Proteins - immunology | JNK Mitogen-Activated Protein Kinases - metabolism | Caspase 1 - immunology | Protein-Tyrosine Kinases - immunology | Protein-Tyrosine Kinases - genetics | HEK293 Cells | Cytoskeletal Proteins - metabolism | Female | Nuclear Proteins - genetics | Dendritic Cells - metabolism | Macrophages - immunology | Mice, Inbred C57BL | Cells, Cultured | Nuclear Proteins - metabolism | Nuclear Proteins - immunology | Inflammasomes - genetics | Carrier Proteins - genetics | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Cytoskeletal Proteins - immunology | CARD Signaling Adaptor Proteins | Inflammasomes - immunology | Macrophages - drug effects | Nigericin - pharmacology | Bone Marrow Cells - metabolism | Tyrosine - genetics | Cellular signal transduction | Inflammation | Genetic aspects | Research | Properties | Phosphotransferases
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, pp. e83145 - e83145
Oxidation of low-density lipoprotein (LDL) is one of the major causative mechanisms in the development of atherosclerosis. In previous studies, we showed that... 
TOLL-LIKE RECEPTOR-4 | ASP299GLY POLYMORPHISM | LIPID-PEROXIDATION | CORONARY | MULTIDISCIPLINARY SCIENCES | DEFICIENT | LOW-DENSITY-LIPOPROTEIN | ATHEROSCLEROSIS | OXIDATION-SPECIFIC EPITOPES | EXPRESSION | 12/15-LIPOXYGENASE | Inflammation - pathology | Lipoproteins, LDL - chemistry | Plaque, Atherosclerotic - genetics | Lymphocyte Antigen 96 - metabolism | Humans | Protein Multimerization | JNK Mitogen-Activated Protein Kinases - metabolism | Protein-Tyrosine Kinases - deficiency | Cholesterol - chemistry | Intracellular Signaling Peptides and Proteins - deficiency | Lymphocyte Antigen 96 - genetics | Inflammation - metabolism | Protein-Tyrosine Kinases - genetics | MAP Kinase Kinase 4 - metabolism | Biological Transport | Toll-Like Receptor 4 - deficiency | Mitogen-Activated Protein Kinase 1 - genetics | JNK Mitogen-Activated Protein Kinases - genetics | Plaque, Atherosclerotic - pathology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Cell Line | Cholesterol - analogs & derivatives | Macrophages - pathology | Mitogen-Activated Protein Kinase 3 - genetics | Oxidation-Reduction | Plaque, Atherosclerotic - metabolism | Signal Transduction | Cholesterol - isolation & purification | Gene Expression Regulation | Toll-Like Receptor 4 - genetics | Mice, Knockout | Macrophages - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Cholesterol - pharmacology | Cholesterol, LDL - metabolism | Inflammation - genetics | MAP Kinase Kinase 4 - genetics | Macrophages - drug effects | Mice | Primary Cell Culture | Mitogen-Activated Protein Kinase 1 - metabolism | Dextran | Low density lipoproteins | Atherosclerosis | Development and progression | Liquid chromatography | Blood lipids | Macrophages | Mass spectrometry | Cholesterol | Lipoproteins (low density) | Health sciences | Transcription factors | Phosphorylation | Laboratories | Esters | Biochemistry | Accumulation | Cell spreading | Blood plasma | Signal transduction | Bone growth | Rodents | Toll-like receptors | Bone marrow | Oxidation | Lesions | Dimerization | Review boards | Extracellular signal-regulated kinase | c-Jun protein | Mass spectroscopy | Pharmacology | Inflammation | TLR4 protein | Apolipoproteins | Biological activity | Medicine | Studies | Human subjects | Signaling | Arteriosclerosis | Low density lipoprotein receptors | Syk protein | Food safety | Intracellular | Cell migration | Veins & arteries | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 02/2014, Volume 25, Issue 2, pp. 226 - 242
Cooperative dependencies between mutant oncoproteins and wild-type proteins are critical in cancer pathogenesis and therapy resistance. Although spleen... 
ONCOLOGY | IN-VIVO | THERAPEUTIC TARGET | BCR-ABL | MYELOGENOUS LEUKEMIA | CHRONIC LYMPHOCYTIC-LEUKEMIA | RISK MYELODYSPLASTIC SYNDROME | TYROSINE KINASE 3 | INHIBITOR | MOUSE MODELS | MLL-REARRANGED LEUKEMIA | CELL BIOLOGY | fms-Like Tyrosine Kinase 3 - antagonists & inhibitors | Phosphorylation | Cell Proliferation | Protein-Tyrosine Kinases - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Drug Resistance, Neoplasm | Intracellular Signaling Peptides and Proteins - metabolism | Immunoenzyme Techniques | fms-Like Tyrosine Kinase 3 - genetics | Protein-Tyrosine Kinases - genetics | Antimetabolites, Antineoplastic - pharmacology | Intracellular Signaling Peptides and Proteins - genetics | Real-Time Polymerase Chain Reaction | Syk Kinase | Leukemia, Myeloid, Acute - pathology | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Cells, Cultured | Survival Rate | Mutation - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | fms-Like Tyrosine Kinase 3 - metabolism | Leukemia, Myeloid, Acute - mortality | Animals | Cell Transformation, Neoplastic | Fluorouracil - pharmacology | Mice | Mice, Inbred BALB C | Protein Kinase Inhibitors - pharmacology | Apoptosis | Protein-Tyrosine Kinases - antagonists & inhibitors | Proteins | Medical colleges | Analysis | AML | MPD | SYK | FLT3-ITD | MYC | tyrosine kinase | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
Journal Article
Immunity, ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 431 - 442
Long-lived “memory-like” NK cells have been identified in individuals infected by human cytomegalovirus (HCMV), but little is known about how the memory-like... 
ADAPTIVE IMMUNITY | CD94/NKG2C RECEPTOR | REPERTOIRE | NATURAL-KILLER-CELLS | ACTIVATION | TYROSINE KINASE | FCR-GAMMA | UMBILICAL-CORD BLOOD | DIFFERENTIATION | IMMUNOLOGY | CHILDREN | Cell Proliferation | Humans | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Killer Cells, Natural - classification | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Immunologic Memory | GPI-Linked Proteins - genetics | Proteins | Viral antibodies | Epigenetic inheritance | Killer cells | Molecular genetics | Antibodies | Gene expression | Antigens | Cytomegalovirus | Cloning | Colleges & universities | Infections | Experiments | Long term | DNA methylation | Epigenetics | Expansion | Viral infections | Deoxyribonucleic acid--DNA | Immune system
Journal Article