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Cell metabolism, ISSN 1550-4131, 09/2017, Volume 26, Issue 3, pp. 493 - 508.e4
Type 2 cytokines are important signals triggering biogenesis of thermogenic beige adipocytes in white adipose tissue (WAT) during cold acclimation. However,... 
adipose remodeling | macrophages | adipocyte precursor cells | beiging | adaptive thermogenesis | eotaxin | eosinophils | type 2 immunity | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Eosinophils - metabolism | Eosinophils - drug effects | Sympathetic Nervous System - drug effects | Adaptation, Physiological - drug effects | Glucuronidase - metabolism | Adipose Tissue, White - metabolism | Adipocytes - cytology | Male | Autocrine Communication - drug effects | Stem Cells - cytology | Adipocytes - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | Stem Cells - metabolism | Sympathetic Nervous System - metabolism | Immunity - drug effects | Fibroblast Growth Factors - metabolism | Cold Temperature | Chemokine CCL11 - metabolism | Mice, Inbred C57BL | Fibroblast Growth Factors - deficiency | Recombinant Proteins - pharmacology | Mice, Knockout | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Adipocytes - metabolism | Stem Cells - drug effects | Adipose Tissue, Beige - drug effects | Macrophages - drug effects | Cell Proliferation - drug effects | Thermogenesis - drug effects | Adipose Tissue, Beige - metabolism | Adipose Tissue, White - drug effects | Adipose tissues | Fibroblast growth factors | Immune response | Macrophages | Pharmaceutical biotechnology | Index Medicus
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 1524-4563, 03/2013, Volume 61, Issue 3, pp. 556 - 560
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 7, pp. e22682 - e22682
Oxidative stress in the central nervous system mediates the increase in sympathetic tone that precedes the development of hypertension... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Genetic Therapy | Reactive Oxygen Species - metabolism | Sympathetic Nervous System - drug effects | Autonomic Nervous System - pathology | NADPH Oxidases - metabolism | Brain - enzymology | Central Nervous System - pathology | Vagus Nerve - drug effects | Male | Sympathetic Nervous System - physiopathology | Primary Dysautonomias - physiopathology | Autonomic Nervous System - physiopathology | Primary Dysautonomias - pathology | Vagus Nerve - pathology | Gene Deletion | Peptidyl-Dipeptidase A - metabolism | Blood Pressure - drug effects | Sympathetic Nervous System - pathology | Angiotensin II - pharmacology | Autonomic Nervous System - drug effects | Brain - physiopathology | Myocardium - pathology | Aging - pathology | Vagus Nerve - physiopathology | Animals | Brain - pathology | Cell Line, Tumor | Central Nervous System - drug effects | Mice | Central Nervous System - physiopathology | Oxidative Stress - drug effects | Oxidases | Hypertension | Advertising executives | Oxidative stress | Angiotensin | Central nervous system | Genetic engineering | Negotiation, mediation and arbitration | Gene therapy | Heart | Brain | Health sciences | Reactive oxygen species | ACE2 gene | Oxidase | Smooth muscle | Nervous system | Paraventricular nucleus | Gene deletion | Kinases | Experiments | Angiotensin-converting enzyme 2--ACE2 | NAD(P)H oxidase | Infusion | Reduction | Clonal deletion | Autonomic nervous system | Rodents | Cell cycle | Aging | Physiology | Blood pressure | Angiotensin II | Heart diseases | Genotypes | Age | Enzymes | Oxygen | Transgenic mice | Cardiomyocytes | Pharmacology | Dysautonomia | Sympathetic nervous system | Medicine | Insects | Hypoxia | In vivo methods and tests | Index Medicus | Angiotensin-converting enzyme 2 | ACE2
Journal Article
Cell metabolism, ISSN 1550-4131, 06/2016, Volume 23, Issue 6, pp. 1185 - 1199
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 11/2009, Volume 54, Issue 19, pp. 1747 - 1762
.... Among these responses is the activation of the sympathetic nervous system (SNS) that provides inotropic support to the failing heart increasing stroke volume... 
adrenergic receptor | beta-blockers | heart failure | sympathomimetic inotropes | sympatholytics | sympathetic nervous system | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Receptors, Adrenergic, alpha-1 - metabolism | Sympathetic Nervous System - drug effects | Humans | Heart Failure - physiopathology | Adrenergic alpha-Antagonists - therapeutic use | Receptors, Adrenergic, beta-2 - drug effects | Sympathetic Nervous System - physiopathology | Sympatholytics - therapeutic use | Cardiotonic Agents - therapeutic use | Receptors, Adrenergic - genetics | Parasympathetic Nervous System - physiopathology | Digoxin - therapeutic use | Adrenergic beta-Antagonists - therapeutic use | Sympathomimetics - therapeutic use | Receptors, Adrenergic, alpha-1 - drug effects | Heart Failure - genetics | Heart Failure - metabolism | Receptors, Adrenergic, beta-2 - metabolism | Heart Failure - drug therapy | Polymorphism, Genetic | Randomized Controlled Trials as Topic | Parasympathetic Nervous System - drug effects | Exercise Tolerance | Renin-Angiotensin System - drug effects | Hemodynamics - drug effects | Proteins | Confidence intervals | Heart rate | Heart failure | Heart attacks | Rodents | Mortality | Hospitalization | Kinases | Beta blockers | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 04/2011, Volume 108, Issue 19, pp. 8030 - 8035
.... To balance energy intake, dietary excess and starvation trigger an increase or a decrease in energy expenditure, respectively, by regulating the activity of the sympathetic nervous system (SNS... 
Heart rate | Receptors | Neurons | Propionates | Myocardium | Ketone bodies | Diabetes | Physiological regulation | Lipid metabolism | Fatty acids | Probiotics | FFAR3 | Microbiota | Fasting | Superior cervical ganglion | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | RNA, Small Interfering - genetics | Signal Transduction | Sympathetic Nervous System - drug effects | Humans | Ketone Bodies - administration & dosage | Mice, Inbred C57BL | Propionates - administration & dosage | Gene Knockdown Techniques | Mice, Knockout | Fatty Acids, Volatile - administration & dosage | 3-Hydroxybutyric Acid - administration & dosage | Animals | Ketone Bodies - physiology | Receptors, G-Protein-Coupled - deficiency | Sympathetic Nervous System - physiology | Fatty Acids, Volatile - physiology | Base Sequence | HEK293 Cells | Receptors, G-Protein-Coupled - antagonists & inhibitors | Mice | Receptors, G-Protein-Coupled - genetics | Action Potentials - drug effects | Energy Metabolism - drug effects | Receptors, G-Protein-Coupled - physiology | Physiological aspects | Glucose metabolism | G proteins | Properties | Membrane proteins | Proteins | Bioenergetics | Homeostasis | Nervous system | Glucose | Dietary fiber | Metabolism | Index Medicus | Energy intake | Starvation | G protein-coupled receptors | Diabetes mellitus | Liver | Propionic acid | Energy expenditure | siRNA | Sympathetic ganglia | Energy requirements | Fermentation | Fuels | Sympathetic nervous system | ketones | Energy balance | sympathetic nerves | Metabolic disorders | microbiota | superior cervical ganglion | Biological Sciences | fasting | probiotics
Journal Article
Brain, behavior, and immunity, ISSN 0889-1591, 10/2014, Volume 41, Issue 1, pp. 200 - 209
.... Post-stroke immunodeficiency is believed to result from neurohormonal dysregulation of the sympathetic nervous system (SNS) and hypothalamic–pituitary–adrenal (HPA) axis... 
Psychiatry | Allergy and Immunology | Catecholamines | Stroke-induced immunosuppression | Glucocorticoids | Lymphocytes | Neurosciences | Immunology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Receptors, Adrenergic, beta-2 - analysis | Infarction, Middle Cerebral Artery - physiopathology | Receptors, Glucocorticoid - antagonists & inhibitors | Apoptosis - drug effects | Male | Neuroimmunomodulation - physiology | Infarction, Middle Cerebral Artery - immunology | Receptors, Adrenergic, beta-2 - drug effects | Sympathetic Nervous System - physiopathology | Receptors, Glucocorticoid - analysis | Lymphopenia - etiology | Metanephrine - blood | Corticosterone - blood | Hypothalamo-Hypophyseal System - physiopathology | Leukocytes - cytology | Pituitary-Adrenal System - physiopathology | Mice, Inbred C57BL | Cells, Cultured | Cortisone - blood | Prednisolone - pharmacology | Infarction, Middle Cerebral Artery - blood | Receptors, Adrenergic, beta-2 - biosynthesis | Feedback, Physiological | Animals | Leukocytes - drug effects | Mice | Mifepristone - pharmacology | Propanolamines - pharmacology | Epinephrine - pharmacology | Interferon-gamma - biosynthesis | Neuroimmunomodulation - drug effects | Stroke (Disease) | Epinephrine | Brain | Corticosterone | Analysis | Mortality | ACTH | Biological response modifiers | Prednisolone | Cortisone | Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 07/2018, Volume 9, Issue 1, pp. 2723 - 10
... (DREADDs), resulted in reduced tumor weight. This effect was mediated via the sympathetic nervous system (SNS... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Ventral Tegmental Area - immunology | Dopaminergic Neurons - pathology | Sympathetic Nervous System - drug effects | Melanoma, Experimental - drug therapy | Carcinoma, Lewis Lung - immunology | Injections, Intraventricular | Male | Myeloid-Derived Suppressor Cells - drug effects | Sympathetic Nervous System - immunology | Clozapine - pharmacology | Melanoma, Experimental - immunology | Adrenergic Neurons - pathology | Adrenergic Neurons - immunology | Bone Marrow Cells - immunology | Dopaminergic Neurons - drug effects | Bone Marrow Cells - drug effects | Sympathetic Nervous System - pathology | Adrenergic Neurons - drug effects | Dopamine - metabolism | Myeloid-Derived Suppressor Cells - pathology | Myeloid-Derived Suppressor Cells - immunology | Ventral Tegmental Area - drug effects | Dopaminergic Neurons - immunology | Immunity, Innate - drug effects | Mice, Inbred C57BL | Bone Marrow Cells - pathology | Ventral Tegmental Area - pathology | Melanoma, Experimental - pathology | Mice, Transgenic | Clozapine - analogs & derivatives | Carcinoma, Lewis Lung - drug therapy | Stereotaxic Techniques | Animals | Norepinephrine - metabolism | Tumor Burden - drug effects | Carcinoma, Lewis Lung - pathology | Mice | Reward | Immunologic Factors - pharmacology | Brain | Immune response | Circuits | Brain tumors | Melanoma | Activation | Lung carcinoma | Sympathetic nervous system | Suppressor cells | Immunity | Immunosuppression | Immunology | Weight reduction | Bone marrow | Reinforcement | Norepinephrine | Cancer | Tumors | Immune system | Index Medicus
Journal Article