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1. Intraventricular ganglioglioma with bleed: A rare case report
by Bhat, Dhananjaya I and Mahadevan, Anita and Manish, Ranjan and Sampath, Somanna and Chandramouli, B A and Shankar, S K
01/2012
Gangliogliomas are benign lesions that are predominantly temporal in location and present with chronic epilepsy. Intraventricular gangliogliomas are extremely... 
synaptophysin | Angioma | ganglioglioma | intraventricular | hemorrhage
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2. Full Text Altered expression and localization of synaptophysin in developing cerebellar cortex of neonatal rats due to maternal diabetes mellitus
by Hami, Javad and Vafaei-Nezhad, Saeed and Ivar, Ghasem and Sadeghi, Akram and Ghaemi, Kazem and Mostafavizadeh, Mostafa and Hosseini, Mehran
Metabolic Brain Disease, ISSN 0885-7490, 12/2016, Volume 31, Issue 6, pp. 1369 - 1380
There is sufficient evidence that diabetes during pregnancy is associated with a higher risk of neurodevelopmental anomalies including learning deficits,... 
Biochemistry, general | Cerebellum | Neurology | Synaptophysin | Neurosciences | Biomedicine | Synaptogenesis | Maternal Diabetes | Metabolic Diseases | Oncology | Rat Newborn | ALZHEIMER-DISEASE | HIPPOCAMPAL-FORMATION | SYNAPTIC PLASTICITY | NEUROSCIENCES | AGE-CHILDREN BORN | VESICLE PROTEIN SYNAPTOPHYSIN | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | POSTNATAL-DEVELOPMENT | LONG-TERM POTENTIATION | GROWTH-FACTOR-I | INSULIN THERAPY | Gene Expression | Rats, Wistar | Cerebellar Cortex - growth & development | Cerebellar Cortex - metabolism | Diabetes Mellitus, Experimental - genetics | Rats | Male | Random Allocation | Cerebellar Cortex - chemistry | Pregnancy | Synaptophysin - genetics | Animals | Synaptophysin - biosynthesis | Female | Blood Glucose - metabolism | Diabetes Mellitus, Experimental - metabolism | Synaptophysin - analysis | Brain | Medical colleges | Diabetes | Blood sugar | Aquaporins
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3. Full Text Synaptic number changes in the medial prefrontal cortex across adolescence in male and female rats: A role for pubertal onset
by Drzewiecki, Carly M and Willing, Jari and Juraska, Janice M
Synapse, ISSN 0887-4476, 09/2016, Volume 70, Issue 9, pp. 361 - 368
Adolescence is a unique period of development, marked by maturation of the prefrontal cortex (PFC), a region important for executive functioning. During this... 
synaptophysin | stereology | pruning | puberty | synapse | PRIMARY VISUAL-CORTEX | NEURON NUMBER | SYNAPTOGENESIS | BRAIN-DEVELOPMENT | NEUROSCIENCES | DENDRITIC SPINES | DENSITY | MONKEY | EARLY ADULTHOOD | IN-VIVO | Rats, Long-Evans | Dendritic Spines - physiology | Rats | Male | Dendritic Spines - metabolism | Presynaptic Terminals - physiology | Prefrontal Cortex - growth & development | Prefrontal Cortex - cytology | Neurogenesis | Synaptophysin - genetics | Animals | Sex Factors | Presynaptic Terminals - metabolism | Female | Synaptophysin - metabolism | Adolescence | Puberty
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4. Full Text Enriched environment combined with fluoxetine ameliorates depression-like behaviors and hippocampal SYP expression in a rat CUS model
by Liu, Cong and Gu, Jing-Yang and Han, Jin-Hong and Yan, Fu-Lin and Li, Yan and Lv, Ting-Ting and Zhao, Li-Qin and Shao, Qiu-Jing and Feng, Yan-Yan and Zhang, Xiang-Yang and Wang, Chang-Hong
Brain Research Bulletin, ISSN 0361-9230, 10/2017, Volume 135, pp. 33 - 39
Effects of enriched environment (EE) combined with fluoxetine in a chronic unpredictable stress (CUS) rat model were examined in our study. Thirty male... 
Enrich environment | Chronic unpredictable stress | Rat | Fluoxetine | Hippocampus | CELLS | ADULT RATS | CHRONIC MILD STRESS | SYNAPTIC PLASTICITY | NEUROSCIENCES | SYNAPTOPHYSIN | NEUROGENESIS | DYSFUNCTION | DEFICITS | PREFERENCE | Fluoxetine - pharmacology | Depression - physiopathology | Gene Expression Regulation | Rats | Male | Hippocampus - drug effects | Depressive Disorder - metabolism | Rats, Sprague-Dawley | Synaptophysin - genetics | Depression - drug therapy | Hippocampus - metabolism | Depression - metabolism | Animals | Stress, Psychological - metabolism | Fluoxetine - metabolism | Behavior, Animal - drug effects | Environment | Disease Models, Animal | Synaptophysin - drug effects | Synaptophysin - metabolism | Depression, Mental
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5. Full Text Detection of behavioral alterations and learning deficits in mice lacking synaptophysin
by Schmitt, U and Tanimoto, N and Seeliger, M and Schaeffel, F and Leube, R.E
Neuroscience, ISSN 0306-4522, 2009, Volume 162, Issue 2, pp. 234 - 243
Abstract The integral membrane protein synaptophysin is one of the most abundant polypeptide components of synaptic vesicles. It is not essential for... 
Neurology | synaptophysin | electroretinogram | optokinetics | learning | transgenic mice | behavior | ENRICHED ENVIRONMENT | TYROSINE PHOSPHORYLATION | ELEVATED PLUS-MAZE | NEUROSCIENCES | RESTRAINT STRESS | MOUSE MODEL | RAT HIPPOCAMPUS | SYNAPTIC VESICLE EXOCYTOSIS | OPEN-FIELD | EXPRESSION | Behavior, Animal | Electroretinography | Exploratory Behavior | Learning | Synaptophysin - genetics | Animals | Memory | Synaptophysin - physiology | Mice | Recognition (Psychology) | Visual Acuity | Mice, Knockout | Psychotherapy | Neurosciences | Polypeptides | Aquaporins | Analysis
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6. Full Text Gulf War agent exposure causes impairment of long-term memory formation and neuropathological changes in a mouse model of Gulf War Illness
by Zakirova, Zuchra and Tweed, Miles and Crynen, Gogce and Reed, Jon and Abdullah, Laila and Nissanka, Nadee and Mullan, Myles and Mullan, Michael J and Mathura, Venkatarajan and Crawford, Fiona and Ait-Ghezala, Ghania
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, p. e0119579
Gulf War Illness (GWI) is a chronic multisymptom illness with a central nervous system component such as memory deficits, neurological, and musculoskeletal... 
ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | VETERANS | ADULT HIPPOCAMPAL NEUROGENESIS | DISTANCE TRANSIT | CHEMICAL WEAPONS FALLOUT | PYRIDOSTIGMINE BROMIDE | PERMETHRIN | MILD COGNITIVE IMPAIRMENT | NEONATAL-RATS | BRAIN | Permethrin - toxicity | Humans | Astrocytes - pathology | Cerebral Cortex - pathology | Male | Hippocampus - drug effects | Cerebral Cortex - metabolism | Persian Gulf Syndrome - metabolism | Cerebral Cortex - physiopathology | Synaptophysin - genetics | Time Factors | Gulf War | Persian Gulf Syndrome - physiopathology | Synaptophysin - antagonists & inhibitors | Cerebral Cortex - drug effects | Persian Gulf Syndrome - pathology | Disease Models, Animal | Synaptophysin - metabolism | Astrocytes - drug effects | Gene Expression | Mice, Inbred C57BL | Memory, Long-Term - drug effects | Memory, Short-Term - drug effects | Hippocampus - pathology | Cognition - drug effects | Hippocampus - metabolism | Animals | Pyridostigmine Bromide - toxicity | Persian Gulf Syndrome - chemically induced | Mice | Hippocampus - physiopathology | Astrocytes - metabolism | Brain | Pyridostigmine | Research | Analysis | Memory | Neurosciences | Permethrin | Veterans | Neuropathology | Change detection | Central nervous system | Cognitive ability | War | Neurogenesis | Learning | Illnesses | Etiology | Rodents | Long term memory | Military personnel | Persian Gulf War | Synaptophysin | Phenotypes | Gulf War syndrome | Pesticides | Exposure | Neurological diseases | Studies | Computer memory | Brain research | Gliosis | Hospitals | Pyridostigmine bromide | Laboratory animals | Hippocampus
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7. Full Text Neonatal Exposure to PFOS and PFOA in Mice Results in Changes in Proteins which are Important for Neuronal Growth and Synaptogenesis in the Developing Brain
by Johansson, Niclas and Eriksson, Per and Viberg, Henrik and Teknisk-naturvetenskapliga vetenskapsområdet and Biologiska sektionen and Uppsala universitet and Institutionen för fysiologi och utvecklingsbiologi and Ekotoxikologi
Toxicological Sciences, ISSN 1096-6080, 4/2009, Volume 108, Issue 2, pp. 412 - 418
Perfluorooctane sulfonate (PFOS) and perfluorooctanoic acid (PFOA) belong to the family of perfluorinated compounds. They are used in industrial and consumer... 
Synaptophysin | Tau | Neurotoxicity | CaMKII | PFCs | GAP-43 | synaptophysin | DIPHENYL ETHER PBDE-209 | MUSCARINIC RECEPTORS | ALZHEIMERS-DISEASE | DOSE-RESPONSE CHANGES | tau | ADULT MICE | PERFLUOROOCTANE SULFONATE | SPONTANEOUS BEHAVIOR | neurotoxicity | TOXICOLOGY | LONG-TERM POTENTIATION | RAT-BRAIN | BROMINATED FLAME-RETARDANT | Animals, Newborn | Fluorocarbons - toxicity | Synapses - drug effects | Brain - cytology | Caprylates - toxicity | Body Weight - drug effects | Male | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - biosynthesis | Brain - growth & development | GAP-43 Protein - genetics | GAP-43 Protein - biosynthesis | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Nerve Tissue Proteins - metabolism | Pregnancy | Synaptophysin - genetics | tau Proteins - biosynthesis | Nerve Tissue Proteins - chemistry | Animals | tau Proteins - genetics | Synaptophysin - biosynthesis | Female | Alkanesulfonic Acids - toxicity | Mice | Neurons - drug effects | Index Medicus | Biological Sciences | Naturvetenskap | Biologiska vetenskaper | Biology | Biologi | Natural Sciences
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8. Full Text Synaptic proteins, neuropathology and cognitive status in the oldest-old
by Head, Elizabeth and Corrada, Maria M and Kahle-Wrobleski, Kristin and Kim, Ronald C and Sarsoza, Floyd and Goodus, Matthew and Kawas, Claudia H
Neurobiology of Aging, ISSN 0197-4580, 2007, Volume 30, Issue 7, pp. 1125 - 1134
Abstract An increasing number of individuals in our population are surviving to over 90 years and a subset is at risk for developing dementia. However, senile... 
Neurology | Internal Medicine | Oldest-old | Cognitively impaired not demented | Postsynaptic density (PSD) | MMSE | Synaptophysin (SYN) | Growth-associated protein-43 (GAP-43) | Compensatory response | Clinico-pathology correlation | Dementia | SYNAPTOPHYSIN-LIKE IMMUNOREACTIVITY | HUMAN BRAIN | ALZHEIMERS-DISEASE | PATHOLOGY | PREVALENCE | NEUROSCIENCES | TANGLE FORMATION | GERIATRICS & GERONTOLOGY | CORTEX | AMYLOID-BETA | TRANSGENIC MICE | Disks Large Homolog 4 Protein | Prospective Studies | Dementia - psychology | Frontal Lobe - metabolism | Humans | Cognition Disorders - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - analysis | Dementia - metabolism | Aged, 80 and over | Female | Membrane Proteins - metabolism | Cognition Disorders - psychology | Synaptophysin - metabolism | Biomarkers - metabolism | Dementia - pathology | GAP-43 Protein - metabolism | Biomarkers - analysis | Cognition Disorders - pathology | Intracellular Signaling Peptides and Proteins - analysis | Aging - psychology | GAP-43 Protein - analysis | Disease Progression | Aging - pathology | Neuropsychological Tests | Synaptic Membranes - metabolism | Synaptophysin - analysis | Frontal Lobe - pathology | Aging - metabolism | Cohort Studies
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9. Full Text Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients
by Kim, Hyung-Wook and Rapoport, Stanley I and Rao, Jagadeesh S
Neurobiology of Disease, ISSN 0969-9961, 2009, Volume 37, Issue 3, pp. 596 - 603
Abstract Bipolar disorder (BD) is a progressive psychiatric disorder characterized by recurrent changes of mood and is associated with cognitive decline. There... 
Neurology | Caspase | Synaptophysin | Drebrin | Bipolar disorder | BDNF | Apoptosis | CYTOSOLIC PHOSPHOLIPASE A | ALZHEIMERS-DISEASE | ARACHIDONIC-ACID CASCADE | BINDING PROTEIN | NEUROSCIENCES | POSITRON-EMISSION-TOMOGRAPHY | RAT FRONTAL-CORTEX | GENE-EXPRESSION | MOOD STABILIZERS | LITHIUM | ADMINISTRATION INCREASES | Bipolar Disorder - metabolism | Arachidonic Acid - metabolism | Humans | Middle Aged | Prefrontal Cortex - physiopathology | Bipolar Disorder - physiopathology | Male | RNA, Messenger - metabolism | Brain - metabolism | Synaptophysin - genetics | Encephalitis - physiopathology | Synapses - metabolism | Encephalitis - metabolism | Up-Regulation - physiology | Atrophy - genetics | Aged, 80 and over | Apoptosis Regulatory Proteins - genetics | Adult | Female | Neuropeptides - genetics | Synaptophysin - metabolism | Biomarkers - metabolism | Atrophy - physiopathology | Encephalitis - genetics | Brain - physiopathology | Biomarkers - analysis | Neuropeptides - metabolism | Bipolar Disorder - genetics | Nerve Tissue Proteins - genetics | Disease Progression | Apoptosis Regulatory Proteins - metabolism | Down-Regulation - physiology | Nerve Tissue Proteins - metabolism | Prefrontal Cortex - metabolism | Aged | Apoptosis - physiology | Atrophy - metabolism | Unsaturated fatty acids | Messenger RNA | Lymphomas | Peptide hormones | Arachidonic acid | Growth factors
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10. Full Text DHA-PC and PSD-95 decrease after loss of synaptophysin and before neuronal loss in patients with Alzheimer's disease
by Yuki, Dai and Sugiura, Yuki and Zaima, Nobuhiro and Akatsu, Hiroyasu and Takei, Shiro and Yao, Ikuko and Maesako, Masato and Kinoshita, Ayae and Yamamoto, Takayuki and Kon, Ryo and Sugiyama, Keikichi and Setou, Mitsutoshi
Scientific Reports, ISSN 2045-2322, 2014, Volume 4, Issue 1, p. 7130
Alzheimer's disease (AD) is a progressive neurodegenerative disease that is characterized by senile plaques, neurofibrillary tangles, synaptic disruption, and... 
IMAGING MASS-SPECTROMETRY | DOCOSAHEXAENOIC ACID | SAMPLE PREPARATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | IN-VIVO | MILD COGNITIVE IMPAIRMENT | SYNAPTIC PLASTICITY | BRAIN | TRANSGENIC MICE | BETA | Disks Large Homolog 4 Protein | Neurons - pathology | Humans | Male | Intracellular Signaling Peptides and Proteins - metabolism | Synapses - pathology | Phosphatidylcholines - metabolism | Alzheimer Disease - pathology | Synaptophysin - genetics | Synapses - metabolism | Amyloid beta-Peptides - genetics | Autopsy | Gray Matter - metabolism | Amyloid beta-Peptides - metabolism | Cell Death | Aged, 80 and over | Female | Membrane Proteins - metabolism | Neurons - metabolism | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization | Intracellular Signaling Peptides and Proteins - genetics | Synapses - chemistry | Synaptophysin - metabolism | Gene Expression | Docosahexaenoic Acids - chemistry | Membrane Proteins - genetics | Gray Matter - chemistry | Phosphatidylcholines - chemistry | Disease Progression | Alzheimer Disease - metabolism | Gray Matter - pathology | Aged | Brain Chemistry | Amyloid beta-Peptides - chemistry | Alzheimer Disease - genetics | Docosahexaenoic Acids - metabolism | Neuroimaging | Synaptophysin | Neurodegenerative diseases | Ions | Mass spectroscopy | Desorption | Docosahexaenoic acid | Substantia grisea | Neurofibrillary tangles | Senile plaques | Amyloid | Postsynaptic density proteins | Alzheimers disease | Alzheimer's disease
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11. Full Text Synaptophysin Regulates the Kinetics of Synaptic Vesicle Endocytosis in Central Neurons
by Kwon, Sung E and Chapman, Edwin R
Neuron, ISSN 0896-6273, 06/2011, Volume 70, Issue 5, pp. 847 - 854
Despite being the most abundant synaptic vesicle membrane protein, the function of synaptophysin remains enigmatic. For example, synaptic transmission was... 
SYNAPTOTAGMIN-I | TYROSINE PHOSPHORYLATION | BIOGENESIS | MECHANISM | CHANNEL PROTEIN | SYNAPSES | MEMBRANE | IDENTIFICATION | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | SYNAPTOBREVIN | Membrane Potentials - drug effects | Membrane Potentials - genetics | Synaptic Vesicles - metabolism | Electric Stimulation | Synaptic Transmission - genetics | Synaptophysin - deficiency | Enzyme Inhibitors - pharmacology | Synaptophysin - physiology | Hippocampus - cytology | Membrane Potentials - physiology | Endocytosis - physiology | Mice, Knockout | Patch-Clamp Techniques | Animals | Time Factors | Neurons - physiology | Macrolides - pharmacology | Synaptic Transmission - drug effects | Endocytosis - genetics | Mice | Neurons - drug effects | Proteins | Studies | Glucose | Neurons
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12. Full Text The impacts of diabetes in pregnancy on hippocampal synaptogenesis in rat neonates
by Vafaei-Nezhad, S and Hami, J and Sadeghi, A and Ghaemi, K and Hosseini, M and Abedini, M.R and Haghir, H
Neuroscience, ISSN 0306-4522, 2016, Volume 318, pp. 122 - 133
Highlights • A down-regulation in hippocampal SYN expression in the pups due to maternal diabetes. • A decrease in the localization of SYN in various subfields... 
Neurology | synaptophysin | rat newborn | maternal diabetes | hippocampus | synaptogenesis | Maternal diabetes | Rat newborn | Synaptophysin | Synaptogenesis | Hippocampus | CONGENITAL-MALFORMATIONS | SYNAPTIC PLASTICITY | NEUROSCIENCES | ENDOCRINE PANCREAS | VESICLE PROTEIN SYNAPTOPHYSIN | AGE-RELATED DECREASE | MATERNAL METABOLISM | SPATIAL MEMORY | GLUCOSE-CONCENTRATION | DENTATE GYRUS | GROWTH-FACTOR-I | Animals, Newborn | Insulin - pharmacology | Maze Learning - physiology | Rats, Wistar | Hippocampus - drug effects | Prenatal Exposure Delayed Effects - physiopathology | Maze Learning - drug effects | Pregnancy | Hippocampus - metabolism | Animals | Synapses - metabolism | Neurogenesis - physiology | Pregnancy in Diabetics - metabolism | Female | Blood Glucose - metabolism | Hippocampus - physiopathology | Memory - physiology | Synaptophysin - metabolism | Blood sugar
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