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Journal Article
Nature, ISSN 0028-0836, 05/2015, Volume 521, Issue 7552, pp. 322 - 327
Na+/Cl--coupled biogenic amine transporters are the primary targets of therapeutic and abused drugs, ranging from antidepressants to the psychostimulants... 
BIOGENIC-AMINE TRANSPORTERS | COCAINE BINDING | SEROTONIN TRANSPORTER | SUBSTRATE TRANSPORT | BACTERIAL HOMOLOG | MULTIDISCIPLINARY SCIENCES | RAT-BRAIN SYNAPTOSOMES | CONFORMATIONAL DYNAMICS | ALTERNATING-ACCESS | TRANSMITTER UPTAKE | MAMMALIAN-CELLS | Dopamine - chemistry | Dopamine Plasma Membrane Transport Proteins - metabolism | Cocaine - metabolism | Molecular Conformation | Crystallography, X-Ray | Dopamine Plasma Membrane Transport Proteins - chemistry | Methamphetamine - chemistry | Sodium - metabolism | Antidepressive Agents - metabolism | Antidepressive Agents - chemistry | Chlorides - metabolism | Central Nervous System Stimulants - metabolism | Dextroamphetamine - chemistry | Protein Stability | Binding Sites | Dopamine - metabolism | Cocaine - chemistry | Dextroamphetamine - metabolism | Neurotransmitter Agents - metabolism | Models, Molecular | Phenethylamines - metabolism | Cocaine - analogs & derivatives | Methamphetamine - metabolism | Neurotransmitter Agents - chemistry | Animals | Dopamine - analogs & derivatives | Drosophila melanogaster - chemistry | Central Nervous System Stimulants - chemistry | Ligands | Physiological aspects | Neurotransmitters | Dopamine | Research | Neurological research | Biological transport | Methamphetamines | Mutagenesis | Antidepressants | Nervous system | Cocaine | Binding sites
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 02/2006, Volume 96, Issue 3, pp. 656 - 668
Glial subcellular re‐sealed particles (referred to as gliosomes here) were purified from rat cerebral cortex and investigated for their ability to release... 
exocytosis | gliosomes | glutamate release | SNARE proteins | Glutamate release | Exocytosis | Gliosomes | NEURONAL-ACTIVITY | SYNAPTIC VESICLES | HIPPOCAMPAL ASTROCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLASMALEMMAL VESICLES | SYNAPTOSOMES | CALCIUM WAVES | NEUROSCIENCES | AMINO-ACIDS | CULTURED ASTROCYTES | H+-ATPASE | IN-SITU | Disks Large Homolog 4 Protein | L-Lactate Dehydrogenase - metabolism | Propionates - metabolism | Neuroglia - ultrastructure | Microscopy, Electron, Transmission - methods | Microtubule-Associated Proteins - metabolism | Vesicular Transport Proteins - metabolism | Male | Intracellular Signaling Peptides and Proteins - metabolism | Synaptosomes - metabolism | Glial Fibrillary Acidic Protein - metabolism | Cerebral Cortex - cytology | Exocytosis - physiology | Dose-Response Relationship, Drug | Tubulin - metabolism | Tritium - metabolism | Time Factors | Macrolides - pharmacology | Membrane Proteins - metabolism | Ionophores - pharmacology | Calcium - pharmacology | Myelin Basic Protein - metabolism | Vesicular Glutamate Transport Protein 1 - metabolism | Fluorescent Antibody Technique - methods | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | S100 Proteins - metabolism | D-Aspartic Acid - metabolism | Microscopy, Confocal - methods | Rats, Sprague-Dawley | Synaptosomes - ultrastructure | Animals | Ionomycin - pharmacology | Neuroglia - metabolism | Glutamic Acid - metabolism | Synaptosomal-Associated Protein 25 - metabolism | CD11b Antigen - metabolism | Vesicle-Associated Membrane Protein 2 - metabolism | Proteins | Brain | Amino acids | Neurotransmitters | Neurons | Rodents
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2003, Volume 5, Issue 8, pp. 701 - 710
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 2014, Volume 23, Issue 16, pp. 4383 - 4395
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 12/2018, Volume 55, Issue 12, pp. 9220 - 9233
Journal Article
Free Radical Research, ISSN 1071-5762, 02/2014, Volume 48, Issue 2, pp. 146 - 158
Abstract The underlying mechanisms of Alzheimer's Disease (AD) are still unclear. It is suggested that poly(ADP-ribose) polymerase-1 (PARP-1) overactivation... 
apoptosis | PARP-1 inhibition | amyloid β peptide | oxidative stress | nicotinamide | Amyloid β peptide | Oxidative stress | Nicotinamide | Apoptosis | NERVOUS-SYSTEM | NEURODEGENERATIVE DISORDERS | LIPID-PEROXIDATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | SULFHYDRYL-GROUPS | CELL-DEATH | amyloid beta peptide | POLY(ADP-RIBOSE) POLYMERASE-1 | NF-KAPPA-B | BRAIN | TRANSGENIC MICE | Neuroprotective Agents - therapeutic use | Synaptosomes - drug effects | Peptide Fragments | Reactive Oxygen Species - metabolism | Gene Expression - drug effects | Glutathione - metabolism | Antioxidants - metabolism | Humans | Male | NF-kappa B - metabolism | Hippocampus - drug effects | Synaptosomes - metabolism | Neuroprotective Agents - pharmacology | Amyloid beta-Peptides | Hippocampus - enzymology | Drug Evaluation, Preclinical | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Glutathione Peroxidase - metabolism | Alzheimer Disease - drug therapy | Niacinamide - therapeutic use | Rats | Poly(ADP-ribose) Polymerase Inhibitors | Rats, Sprague-Dawley | Apoptosis Regulatory Proteins - metabolism | Catalase - metabolism | Poly(ADP-ribose) Polymerases - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | NF-kappa B - genetics | Alzheimer Disease - metabolism | Niacinamide - pharmacology | Oxidative Stress - drug effects | Poly (ADP-Ribose) Polymerase-1 | Lipid Peroxidation
Journal Article
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 10/2018, Volume 55, Issue 10, pp. 8084 - 8102
The striatal-enriched protein tyrosine phosphatase (STEP) is a brain-specific phosphatase involved in synaptic transmission. The current hypothesis on STEP... 
Synapsin I | Neurology | Neurosciences | Glutamate release | Biomedicine | Ca 2+ homeostasis | CaMKII | Synaptic transmission | Neurobiology | Synaptosomes | Striatal-enriched tyrosine phosphatase | Cell Biology | homeostasis | MOLECULAR CHARACTERIZATION | SYNAPTIC PLASTICITY | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | INOSITOL 1,4,5-TRISPHOSPHATE RECEPTOR | Ca2+ homeostasis | NERVE-TERMINALS | ENDOPLASMIC-RETICULUM | VESICLE POOLS | LONG-TERM POTENTIATION | PROTEIN-KINASE-II | SYNAPSIN-I | Protein Tyrosine Phosphatases, Non-Receptor - deficiency | Phosphorylation | Calcium - metabolism | Homeostasis | Hippocampus - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | Synaptosomes - metabolism | Mutation - genetics | Synapsins - metabolism | Synaptic Transmission | Mice, Knockout | Hippocampus - metabolism | Animals | Synapses - metabolism | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Models, Biological | Intracellular Space - metabolism | Protein Tyrosine Phosphatases, Non-Receptor - metabolism | Presynaptic Terminals - metabolism | Cytosol - metabolism | Neostriatum - metabolism | Glutamic Acid - metabolism | Calcineurin - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Tyrosine | Medical colleges | Phosphatases | Neurons | Phenols | Glutamate | Protein kinases | Calmodulin | Brain | Synaptic strength | Calcium | Intracellular signalling | Kinases | Phosphatase | Proteins | Rodents | Calcium-binding protein | Neostriatum | Protein-tyrosine kinase | Calcium homeostasis | Fyn protein | Nerve endings | Neurotransmitter release | Calcium (intracellular) | Extracellular signal-regulated kinase | Plasticity (synaptic) | Synaptic vesicles | Mice | Glutamatergic transmission | Intracellular | Hippocampus | Protein-tyrosine-phosphatase | Ca2+/calmodulin-dependent protein kinase II
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e57380
Background: Autism spectrum disorders (ASDs) are caused by both genetic and environmental factors. Mitochondria act to connect genes and environment by... 
RESPONSES | ACTIVATION | SURAMIN | PROTEIN | NEUROTRANSMISSION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | RECEPTOR | STRESS | BRAIN | CHILDREN | Autistic Disorder - chemically induced | Electron Transport | Autistic Disorder - metabolism | Immunoglobulins - immunology | Autistic Disorder - immunology | Male | Fragile X Mental Retardation Protein - metabolism | Synaptosomes - metabolism | Suramin - administration & dosage | Behavior, Animal | Brain - metabolism | Hypothermia - metabolism | Social Behavior | Immunoglobulins - blood | Female | Poly I-C - adverse effects | Corticosterone - blood | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Psychomotor Performance | Receptors, Nicotinic - metabolism | Purkinje Cells - metabolism | Signal Transduction | Mitochondria - metabolism | Synaptosomes - ultrastructure | Animals | Energy Metabolism | Hypothermia - drug therapy | Mitogen-Activated Protein Kinase 3 - metabolism | Mice | Autistic Disorder - drug therapy | Mitogen-Activated Protein Kinase 1 - metabolism | Autism | Cellular signal transduction | Metabolites | Analysis | Genes | Cerebellum | Suramin | Therapy | Pediatrics | Neurosciences | Pathogenesis | Disorders | Metabolic networks | Drug development | Kinases | Proteins | Signal transduction | Mitochondria | Rodents | Sensorimotor system | Immune response | Abnormalities | Extracellular signal-regulated kinase | Hazards | Environmental factors | Permeability | Medicine |