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Journal of Infectious Diseases, ISSN 0022-1899, 10/2015, Volume 212, Issue 7, pp. 1129 - 1139
Journal Article
PloS one, ISSN 1932-6203, 2019, Volume 14, Issue 5, p. e0217550
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2012, Volume 287, Issue 39, pp. 32578 - 32587
Staphylococcal superantigens (SAgs), such as toxic shock syndrome toxin-1 (TSST-1), are the main cause of toxic shock syndrome (TSS). SAgs deregulate the host... 
CELLS | ACTIVATION | ALPHA-CONVERTING ENZYME | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENTEROTOXINS | PENETRATION | NECROSIS-FACTOR-ALPHA | CLEAVAGE | EXPRESSION | SYNDECANS | ADAM17 Protein | Interleukin-8 - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Syndecan-1 - genetics | ErbB Receptors - genetics | Glycoproteins - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Amphiregulin | Superantigens - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | EGF Family of Proteins | Bacterial Toxins - genetics | Dipeptides - metabolism | Interleukin-8 - metabolism | Transforming Growth Factor alpha - metabolism | Staphylococcus aureus - metabolism | Glycoproteins - genetics | Staphylococcus aureus - genetics | ErbB Receptors - metabolism | Syndecan-1 - metabolism | Signal Transduction | Intercellular Signaling Peptides and Proteins - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Transforming Growth Factor alpha - genetics | Hydroxamic Acids - metabolism | ADAM Proteins - metabolism | Bacterial Toxins - metabolism | Enterotoxins - genetics | Models, Biological | Superantigens - genetics | Enterotoxins - metabolism | Human Umbilical Vein Endothelial Cells - pathology | ADAM Proteins - genetics | Dipeptides - genetics | Molecular Bases of Disease | Shedding | Toxic Shock Syndrome | ADAM ADAMTS | Epidermal Growth Factor Receptor (EGFR) | Epithelial Cell | Mucosal Immunology | Superantigen | Staphylococcus aureus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2012, Volume 287, Issue 2, pp. 1478 - 1488
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 12/2015, Volume 54, Issue 12, pp. 1796 - 1806
Journal Article
Cell Death and Disease, ISSN 2041-4889, 11/2018, Volume 9, Issue 11, pp. 1119 - 13
Syndecans, a family of cell surface heparan sulfate proteoglycans, regulate cell differentiation via binding of their heparan sulfate chains to growth factors... 
SULFATE PROTEOGLYCANS | ACTIVATION | SOLUBLE SYNDECAN-1 | FINE-STRUCTURE | INTEGRIN | DISEASE | CELL-SURFACE | FIBROBLAST-GROWTH-FACTOR | DIFFERENTIATION | BONE-RESORPTION | CELL BIOLOGY | Vacuolar Proton-Translocating ATPases - genetics | Humans | Syndecan-1 - genetics | Syndecan-4 - genetics | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Osteoclasts - cytology | Extracellular Signal-Regulated MAP Kinases - genetics | Vacuolar Proton-Translocating ATPases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Syndecan-4 - pharmacology | Escherichia coli - metabolism | Protein Domains | Bone Marrow Cells - drug effects | Macrophage Colony-Stimulating Factor - genetics | Proto-Oncogene Proteins c-akt - metabolism | Heparin - pharmacology | Osteogenesis - genetics | Recombinant Proteins - metabolism | Syndecan-1 - metabolism | Bone Marrow Cells - cytology | Femur - metabolism | Syndecan-4 - metabolism | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Osteogenesis - drug effects | Proto-Oncogene Proteins c-fos - metabolism | Glycosylation | Macrophage Colony-Stimulating Factor - metabolism | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Osteoclasts - metabolism | Animals | Heparin - analogs & derivatives | Cell Differentiation - drug effects | Escherichia coli - genetics | Protein Binding | Proto-Oncogene Proteins c-fos - genetics | Cell Proliferation - drug effects | Femur - cytology | Mice | Protein Processing, Post-Translational | Syndecan-1 - pharmacology | Bone Marrow Cells - metabolism | Heparin - chemistry | Osteoclasts - drug effects | NFATC Transcription Factors - genetics | Transcription factors | Mucosa | AKT protein | Macrophages | Cell surface | Bone resorption | Intestine | Colony-stimulating factor | Growth factors | c-Fos protein | Heparan sulfate | Proteoglycans | Wound healing | Cytokines | Colonies | Extracellular signal-regulated kinase | c-Jun protein | JNK protein | Bone turnover | Osteoclastogenesis | Calvaria | Heparan sulfate proteoglycans | Acid phosphatase (tartrate-resistant) | Heparin
Journal Article
BMC Cancer, ISSN 1471-2407, 01/2013, Volume 13, Issue 1, pp. 24 - 24
Journal Article
Molecular Oncology, ISSN 1574-7891, 03/2014, Volume 8, Issue 2, pp. 297 - 310
Reprogramming of NK cells with a chimeric antigen receptor (CAR) proved an effective strategy to increase NK cell reactivity and recognition specificity toward... 
NK cells | Chimeric antigen receptor | CD138 (syndecan-1) | Multiple myeloma | Adoptive immunotherapy | CD138 (syndecan‐1) | Humans | Middle Aged | Syndecan-1 - genetics | Neoplasm Proteins - immunology | Male | Multiple Myeloma - immunology | Killer Cells, Natural - pathology | Lymphocyte Activation - genetics | Lymphocyte Activation - immunology | Multiple Myeloma - therapy | Single-Chain Antibodies - genetics | Transfection | Syndecan-1 - immunology | Killer Cells, Natural - immunology | Adult | Female | Neoplasm Proteins - genetics | Antibodies, Neoplasm - immunology | Mice, SCID | Multiple Myeloma - pathology | Animals | Cell Line, Tumor | Mice, Inbred NOD | Antibodies, Neoplasm - genetics | Mice | Multiple Myeloma - genetics | Single-Chain Antibodies - immunology | Antigen-antibody reactions | Medical colleges | Antigens | Oncology, Experimental | Genes | Research | Biological response modifiers | Chemotherapy | Killer cells | Immunotherapy | Genetically modified organisms | Genetic research | Genetic aspects | Lymphomas | Cancer | Interferon gamma | Cell proliferation | Cytolytic activity | Plasma | Severe combined immunodeficiency | Leukemia | Radiation | Effector cells | Clinical trials | Cytotoxicity | Cell activation | Granzyme B | Xenografts | Bone marrow | Remission | Natural killer cells | Immunoglobulins | Tumor cells | Cell division | Interferon | Females
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2015, Volume 195, Issue 2, pp. 736 - 748
Genotoxic stress can promote antitumor NK cell responses by upregulating the surface expression of activating ligands on cancer cells. Moreover, a number of... 
MHC CLASS-I | NATURAL-KILLER-CELLS | ACTIVATING RECEPTOR NKG2D | DOWN-REGULATION | SOLUBLE MICA | MEDIATED LYSIS | IMMUNOLOGY | DEPENDENT MECHANISM | MALIGNANT DISEASES | T-CELLS | ADAM PROTEASES | Amyloid Precursor Protein Secretases - genetics | Amyloid Precursor Protein Secretases - immunology | ADAM Proteins - immunology | Humans | Syndecan-1 - genetics | Gene Expression Regulation, Neoplastic | NK Cell Lectin-Like Receptor Subfamily K - immunology | Multiple Myeloma - immunology | Killer Cells, Natural - pathology | Plasma Cells - pathology | Plasma Cells - drug effects | Cytotoxins - pharmacology | Syndecan-1 - immunology | Cellular Senescence | Proteolysis | Reactive Oxygen Species - immunology | Bone Marrow Cells - immunology | ADP-ribosyl Cyclase 1 - immunology | Killer Cells, Natural - immunology | Bone Marrow Cells - drug effects | Melphalan - pharmacology | Membrane Glycoproteins - immunology | ADP-ribosyl Cyclase 1 - genetics | Signal Transduction | Membrane Proteins - genetics | Bone Marrow Cells - pathology | Histocompatibility Antigens Class I - immunology | ADAM10 Protein | Membrane Proteins - immunology | Histocompatibility Antigens Class I - genetics | NK Cell Lectin-Like Receptor Subfamily K - genetics | Membrane Glycoproteins - genetics | Matrix Metalloproteinase Inhibitors - pharmacology | Multiple Myeloma - pathology | Cell Line, Tumor | Killer Cells, Natural - drug effects | DNA Damage | Primary Cell Culture | ADAM Proteins - genetics | Doxorubicin - pharmacology | Multiple Myeloma - genetics | Plasma Cells - immunology
Journal Article
BMC Cancer, ISSN 1471-2407, 11/2016, Volume 16, Issue 1, p. 903
Background: Keratin (K) 19-positive hepatocellular carcinoma (HCC) is well known to have a higher malignant potential than K19-negative HCC: However, the... 
Angiogenesis | Hepatocellular carcinoma | Senescence | Keratin 19 | Apoptosis | SYNDECAN-1 | ACTIVATION | PROTEIN | BILIARY DIFFERENTIATION | VASOHIBIN | CYTOKERATIN-19 EXPRESSION | GENE | ONCOLOGY | PROSTATE-CANCER | PROGNOSTIC-SIGNIFICANCE | CONTRIBUTES | Immunohistochemistry | Prognosis | Cadherins - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Cell Survival - genetics | Apoptosis - genetics | Male | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | RNA Interference | Carcinoma, Hepatocellular - genetics | Aged, 80 and over | Adult | Carcinoma, Hepatocellular - blood supply | Female | Cadherins - genetics | Cell Proliferation - genetics | Liver Neoplasms - genetics | Neoplasm Invasiveness | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Disease Progression | Hep G2 Cells | Keratin-19 - genetics | Liver Neoplasms - blood supply | Liver Neoplasms - metabolism | Cell Line, Tumor | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Neovascularization, Pathologic - genetics | Aged | Neovascularization, Pathologic - metabolism | Keratin-19 - metabolism | Carcinoma, Hepatocellular - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Keratin | Care and treatment | Stem cells | Fibroblast growth factors | Dosage and administration | Research | Hepatoma | Diagnosis | Gene expression | Health aspects
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2014, Volume 289, Issue 9, pp. 5499 - 5509
Journal Article
Journal Article