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Head & Neck, ISSN 1043-3074, 10/2011, Volume 33, Issue 10, pp. 1458 - 1466
Background. We evaluated the predictive significance of 14 reported markers using immunohistochemical study in nasopharyngeal carcinoma. Methods.... 
prognostic biomolecular markers | radiotherapy | combined score | nasopharyngeal carcinoma | immunohistochemical staining | SURVIVAL | SURGERY | SYNDECAN-1 EXPRESSION | CHEMORADIOTHERAPY | TUMOR ANGIOGENESIS | CANCER | CHEMOTHERAPY | GROWTH-FACTOR RECEPTOR | ADVANCED-STAGE | OTORHINOLARYNGOLOGY | PROTEIN EXPRESSION | DIFFERENTIATION | Immunohistochemistry | Proto-Oncogene Proteins c-met - metabolism | Nasopharyngeal Neoplasms - metabolism | Nasopharyngeal Neoplasms - mortality | Prognosis | Follow-Up Studies | Humans | Middle Aged | Receptor, ErbB-2 - metabolism | Carcinoma - mortality | Male | NM23 Nucleoside Diphosphate Kinases - metabolism | STAT5 Transcription Factor - metabolism | Receptor, Epidermal Growth Factor - metabolism | Biomarkers, Tumor - metabolism | Adult | Female | Chemotherapy, Adjuvant | STAT3 Transcription Factor - metabolism | Cellular Apoptosis Susceptibility Protein - metabolism | Tumor Suppressor Proteins - metabolism | Syndecan-1 - metabolism | Nasopharyngeal Neoplasms - therapy | Tumor Suppressor Protein p53 - metabolism | Cathepsin D - metabolism | Transcription Factors - metabolism | Disease-Free Survival | Cyclooxygenase 2 - metabolism | Carcinoma - therapy | Aged | Carcinoma - metabolism | Early Growth Response Protein 1 - metabolism | RNA-Binding Proteins - metabolism | Radiotherapy, Intensity-Modulated | Index Medicus
Journal Article
Journal Article
Development, ISSN 0950-1991, 07/2009, Volume 136, Issue 13, pp. 2153 - 2164
A key initial event in hair follicle morphogenesis is the localised thickening of the skin epithelium to form a placode, partitioning future hair follicle... 
Hair follicle | KGF | Mouse | EGF | Skin organ culture | ORGAN-CULTURE | DEVELOPMENTAL BIOLOGY | FACTOR RECEPTOR | MORPHOGENESIS | KERATINOCYTE GROWTH-FACTOR | GENE | LIGANDS | CELL-DIFFERENTIATION | EXPRESSION | SONIC-HEDGEHOG | TRANSGENIC MICE | Receptor, Epidermal Growth Factor - genetics | Cadherins - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Glycoproteins - metabolism | Hedgehog Proteins - metabolism | Quinazolines | Amphiregulin | Wnt Proteins - metabolism | Vibrissae - embryology | Intercellular Signaling Peptides and Proteins - metabolism | Fibroblast Growth Factor 7 - metabolism | Receptor, Epidermal Growth Factor - metabolism | Wnt Proteins - genetics | EGF Family of Proteins | Hedgehog Proteins - genetics | Hyaluronan Receptors - metabolism | Vibrissae - anatomy & histology | Hair Follicle - embryology | Cell Differentiation - physiology | Skin - embryology | Fibroblast Growth Factor 10 - metabolism | Skin - anatomy & histology | Syndecan-1 - metabolism | Enzyme Inhibitors - metabolism | Epidermal Growth Factor - genetics | Tissue Culture Techniques | Mice, Inbred C57BL | Oncogene Proteins - metabolism | Epidermal Growth Factor - metabolism | beta Catenin - metabolism | Fibroblast Growth Factor 7 - genetics | Heparin-binding EGF-like Growth Factor | Hair Follicle - cytology | Animals | Hair Follicle - metabolism | Tyrphostins - metabolism | Epidermis - embryology | Signal Transduction - physiology | Trans-Activators - metabolism | Mice | Zinc Finger Protein GLI1 | Morphogenesis - physiology | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Epidermis - cytology | Index Medicus | s
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2012, Volume 287, Issue 39, pp. 32578 - 32587
Staphylococcal superantigens (SAgs), such as toxic shock syndrome toxin-1 (TSST-1), are the main cause of toxic shock syndrome (TSS). SAgs deregulate the host... 
CELLS | ACTIVATION | ALPHA-CONVERTING ENZYME | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENTEROTOXINS | PENETRATION | NECROSIS-FACTOR-ALPHA | CLEAVAGE | EXPRESSION | SYNDECANS | ADAM17 Protein | Interleukin-8 - genetics | Receptor, Epidermal Growth Factor - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Syndecan-1 - genetics | Glycoproteins - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Amphiregulin | Superantigens - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | EGF Family of Proteins | Bacterial Toxins - genetics | Dipeptides - metabolism | Interleukin-8 - metabolism | Transforming Growth Factor alpha - metabolism | Staphylococcus aureus - metabolism | Glycoproteins - genetics | Staphylococcus aureus - genetics | Syndecan-1 - metabolism | Signal Transduction | Intercellular Signaling Peptides and Proteins - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Transforming Growth Factor alpha - genetics | Hydroxamic Acids - metabolism | ADAM Proteins - metabolism | Bacterial Toxins - metabolism | Enterotoxins - genetics | Models, Biological | Superantigens - genetics | Enterotoxins - metabolism | Human Umbilical Vein Endothelial Cells - pathology | ADAM Proteins - genetics | Dipeptides - genetics | Index Medicus | Molecular Bases of Disease | Shedding | Toxic Shock Syndrome | ADAM ADAMTS | Epidermal Growth Factor Receptor (EGFR) | Epithelial Cell | Mucosal Immunology | Superantigen | Staphylococcus aureus
Journal Article
Clinical Immunology, ISSN 1521-6616, 2014, Volume 153, Issue 2, pp. 264 - 276
Abstract As vaccine-elicited antibodies have now been associated with HIV protective efficacy, a thorough understanding of mucosal and systemic B-cell... 
Allergy and Immunology | SIV/SHIV rhesus macaque model | Plasmablasts/plasma cells | Homing markers | Mucosal memory B cell phenotypes and distribution | DENDRITIC CELLS | GASTROINTESTINAL-TRACT | IGA-SECRETING CELLS | PERIPHERAL-BLOOD | IMMUNOLOGY | IMMUNE-RESPONSES | HUMAN PLASMA-CELLS | ANIMAL-MODELS | IMMUNODEFICIENCY VIRUS-INFECTION | CHEMOKINE RECEPTORS | SIV INFECTION | Simian Acquired Immunodeficiency Syndrome - metabolism | Intestinal Mucosa - metabolism | Humans | Interferon Regulatory Factors - metabolism | Ki-67 Antigen - metabolism | Macaca mulatta | Rectum - virology | Host-Pathogen Interactions - immunology | Jejunum - metabolism | Simian Acquired Immunodeficiency Syndrome - virology | Flow Cytometry | Ki-67 Antigen - immunology | Syndecan-1 - immunology | Intestinal Mucosa - immunology | Duodenum - metabolism | B-Lymphocytes - virology | Histocompatibility Antigens Class II - metabolism | Plasma Cells - metabolism | B-Lymphocytes - metabolism | Immunologic Memory - immunology | Antigens, CD19 - immunology | Disease Models, Animal | Simian Immunodeficiency Virus - immunology | Duodenum - immunology | Antigens, CD20 - immunology | Duodenum - virology | Immunoglobulin A - metabolism | Syndecan-1 - metabolism | Plasma Cells - virology | Receptors, Chemokine - metabolism | Rectum - metabolism | Immunophenotyping | Intestinal Mucosa - virology | Jejunum - immunology | Antigens, CD20 - metabolism | Receptors, Chemokine - immunology | Rectum - immunology | Simian Immunodeficiency Virus - physiology | Interferon Regulatory Factors - immunology | Animals | B-Lymphocytes - immunology | Histocompatibility Antigens Class II - immunology | Jejunum - virology | Simian Acquired Immunodeficiency Syndrome - immunology | Immunoglobulin A - immunology | Antigens, CD19 - metabolism | Plasma Cells - immunology | Immunoglobulin A | Analysis | Index Medicus | homing markers | SIV | plasmablasts | mucosal memory B cell phenotypes and distribution | plasma cells | SHIV rhesus macaque model
Journal Article
Molecular Cancer, ISSN 1476-4598, 03/2017, Volume 16, Issue 1, pp. 57 - 57
Background: Inflammatory breast cancer (IBC), a particularly aggressive form of breast cancer, is characterized by cancer stem cell (CSC) phenotype. Due to a... 
Proteoglycan | Notch | Cancer stem cell | IL-6/STAT3 | Inflammatory breast cancer | Syndecan-1 | EGFR | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TARGETING NOTCH | E-CADHERIN | CHEMOTHERAPY | CARCINOMA IN-SITU | EPITHELIAL-MESENCHYMAL TRANSITION | C-TERMINAL FRAGMENT | GROWTH-FACTOR RECEPTOR | ONCOLOGY | EXPRESSION | DUCTAL CARCINOMA | Inflammatory Breast Neoplasms - pathology | Receptors, Notch - metabolism | Humans | Middle Aged | Syndecan-1 - genetics | NF-kappa B - metabolism | Gene Knockdown Techniques | Neoplasm Metastasis | Receptor, Epidermal Growth Factor - metabolism | Neoplasm Grading | Neoplastic Stem Cells - metabolism | Hyaluronan Receptors - metabolism | Inflammatory Breast Neoplasms - metabolism | Triple Negative Breast Neoplasms - pathology | Neoplastic Stem Cells - pathology | Adult | Female | Proteomics - methods | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Gene Expression | Syndecan-1 - metabolism | Signal Transduction | Gene Silencing | Proteome | Inflammatory Breast Neoplasms - genetics | Phenotype | Triple Negative Breast Neoplasms - genetics | Triple Negative Breast Neoplasms - metabolism | Biomarkers | Cell Line, Tumor | Flow cytometry | Immunoglobulins | Cytokines | Cloning | Breast cancer | Inflammation | Lymphocytes T | Metastasis | Cell adhesion & migration | Genotype & phenotype | Chemotherapy | Epidermal growth factor | Immunology | Medical prognosis | Stem cells | Notch protein | Apoptosis | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 2016, Volume 128, Issue 5, pp. 667 - 679
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2013, Volume 33, Issue 9, pp. 2065 - 2074
OBJECTIVE—Chylomicron and very low-density lipoprotein remnants are cleared from the circulation in the liver by heparan sulfate proteoglycan (HSPG) receptors... 
low-density lipoprotein receptor-related protein-1 | low-density lipoprotein receptor | cholesterol | syndecan-1 | triglycerides | heparan sulfate proteoglycans | lipoproteins | CHYLOMICRON REMNANTS | LDL RECEPTOR | TRIGLYCERIDE-RICH LIPOPROTEINS | APOLIPOPROTEIN-E | III HYPERLIPOPROTEINEMIA | PLASMA | METABOLISM | NONFASTING TRIGLYCERIDES | IN-VIVO | LIVER | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Hyperlipidemias - genetics | Heparan Sulfate Proteoglycans - blood | Cholesterol, Dietary - administration & dosage | Cholesterol, Dietary - metabolism | Hepatocytes - metabolism | Chylomicron Remnants - blood | Time Factors | Tumor Suppressor Proteins - deficiency | Diet, High-Fat | Tumor Suppressor Proteins - genetics | Postprandial Period | Receptors, LDL - deficiency | Chylomicron Remnants - metabolism | Sulfotransferases - metabolism | Dietary Sucrose - metabolism | Sulfotransferases - genetics | Receptors, LDL - genetics | Tumor Suppressor Proteins - metabolism | Hyperlipidemias - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Receptors, LDL - metabolism | Mice, Knockout | Triglycerides - metabolism | Particle Size | Animals | Heparan Sulfate Proteoglycans - metabolism | Dietary Sucrose - administration & dosage | Mice | Index Medicus | heparan sulfate | LRP1 | Syndecan-1 | Triglycerides | LDLR
Journal Article