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index medicus (85) 85
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American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2012, Volume 47, Issue 2, pp. 196 - 202
Proteoglycans (PGs) and their associated glycosaminoglycan side chains are effectors of inflammation, but little is known about changes to the composition of... 
Proteoglycan | Innate immunity | Macrophages | Syndecan | Neutrophils | PROTEOGLYCANS | BIOCHEMISTRY & MOLECULAR BIOLOGY | LUNG INJURY | MECHANISMS | IDENTIFICATION | proteoglycan | CELL BIOLOGY | macrophages | TOLL-LIKE RECEPTOR-4 | innate immunity | RESPIRATORY SYSTEM | syndecan | HEPARAN-SULFATE | neutrophils | PATTERN-RECOGNITION RECEPTORS | EXPRESSION | BINDING | Interleukin-8 - genetics | Syndecan-4 - genetics | Tumor Necrosis Factor-alpha - genetics | Immunity, Innate - genetics | Bronchoalveolar Lavage Fluid - immunology | Pneumonia, Bacterial - metabolism | Lipopolysaccharides - immunology | Lung Injury - metabolism | Tumor Necrosis Factor-alpha - immunology | Pneumonia - immunology | Interleukin-8 - metabolism | Neutrophils - metabolism | Pneumonia - genetics | Syndecan-4 - immunology | Syndecan-4 - metabolism | Neutrophils - drug effects | Myeloid Differentiation Factor 88 - genetics | Macrophages - metabolism | Up-Regulation - immunology | Lung - drug effects | Lipopolysaccharides - pharmacology | Mice | Interleukin-8 - immunology | Pneumonia - metabolism | Lung Injury - immunology | Heparin, Low-Molecular-Weight - immunology | Tumor Necrosis Factor-alpha - metabolism | RNA, Messenger - immunology | Epithelial Cells - metabolism | Heparan Sulfate Proteoglycans - genetics | Myeloid Differentiation Factor 88 - immunology | Pneumonia, Bacterial - genetics | Lung - metabolism | Macrophages - immunology | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Neutrophils - immunology | Up-Regulation - genetics | Heparan Sulfate Proteoglycans - immunology | Immunity, Innate - immunology | Lung Injury - genetics | Animals | Heparan Sulfate Proteoglycans - metabolism | Epithelial Cells - immunology | Pneumonia, Bacterial - immunology | Syndecan-4 - deficiency | Myeloid Differentiation Factor 88 - metabolism | Lung - immunology
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2576 - 2584
Having discovered that the dendritic cell (DC)-associated heparan sulfate proteoglycan-dependent integrin ligand (DC-HIL) receptor on APCs inhibits T cell... 
ADHESION | DC-HIL | ACTIVATION | PROTEIN | SYNDECAN-4 | TOLERANCE | DISEASE | IMMUNOLOGY | CANCER | T-CELLS | NEGATIVE REGULATOR | Eye Proteins - immunology | Reactive Oxygen Species - metabolism | Spleen - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Membrane Glycoproteins - metabolism | Syndecan-4 - genetics | Autoimmunity - genetics | Encephalomyelitis, Autoimmune, Experimental - immunology | Adoptive Transfer | Interferon-gamma - metabolism | DNA-Binding Proteins - metabolism | Flow Cytometry | T-Lymphocytes - metabolism | Autoimmunity - immunology | Myeloid Cells - immunology | Reactive Oxygen Species - immunology | Female | Encephalomyelitis, Autoimmune, Experimental - genetics | Eye Proteins - genetics | Membrane Glycoproteins - immunology | Spleen - pathology | Syndecan-4 - immunology | Nitric Oxide - immunology | DNA-Binding Proteins - immunology | CD11b Antigen - immunology | Syndecan-4 - metabolism | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Cells, Cultured | Receptors, Chemokine - immunology | DNA-Binding Proteins - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Eye Proteins - metabolism | Spleen - metabolism | Interferon-gamma - immunology | Myeloid Cells - metabolism | Myeloid Cells - transplantation | T-Lymphocytes - immunology | Mice | CD11b Antigen - metabolism | Nitric Oxide - metabolism
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 12/2017, Volume 313, Issue 6, pp. L1069 - L1086
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 88, pp. 133 - 144
Abstract Inflammation is central to heart failure progression. Innate immune signaling increases expression of the transmembrane proteoglycan syndecan-4 in... 
Cardiovascular | Proteoglycan | Heart failure | Innate immunity | Sepsis | Fibrosis | APOPTOSIS | PROTEOGLYCANS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | CELL BIOLOGY | HEART | ADHESION | INFLAMMATION | HEPARAN-SULFATE | EXPRESSION | LYMPHOCYTES | Antigens, CD - immunology | ADAM Proteins - immunology | Sepsis - prevention & control | Rats, Wistar | Myocytes, Cardiac - immunology | Humans | Syndecan-4 - genetics | Matrix Metalloproteinase 9 - immunology | Procollagen N-Endopeptidase - immunology | Male | Antigens, CD - genetics | Lipopolysaccharides | Heart Failure - prevention & control | Sepsis - pathology | Syndecan-4 - secretion | Matrix Metalloproteinase 9 - genetics | HEK293 Cells | Heart Failure - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Neutrophil Infiltration - drug effects | ADAMTS1 Protein | Syndecan-4 - immunology | Vascular Cell Adhesion Molecule-1 - immunology | Sepsis - immunology | Signal Transduction | Intercellular Adhesion Molecule-1 - immunology | Mice, Inbred C57BL | Procollagen N-Endopeptidase - genetics | Gene Expression Regulation | Injections, Intraperitoneal | Rats | Heart Failure - pathology | Fibroblasts - pathology | ADAMTS4 Protein | Stroke Volume | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Sepsis - chemically induced | Fibroblasts - drug effects | Intercellular Adhesion Molecule-1 - genetics | Fibroblasts - immunology | Antigens, Differentiation - genetics | Mice | ADAM Proteins - genetics | Heart Failure - chemically induced | Antigens, Differentiation - immunology | RNA | Sulfates | Mitogens | Collagen | Cardiac patients
Journal Article
Journal Article
Journal Article
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2012, Volume 71, Issue 6, pp. 1004 - 1011
Objective To elucidate the mechanisms involved in cartilage damage in an experimental model of rheumatoid arthritis (RA) by specifically addressing the time... 
RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | MURINE MODEL | TUMOR-NECROSIS-FACTOR | OSTEOCLAST FORMATION | JOINT DAMAGE | GENE-TRANSFER | TNF-ALPHA | RHEUMATOLOGY | PANNUS FORMATION | FACTOR-ALPHA | Bone and Bones - pathology | Cell Communication - immunology | Synovitis - metabolism | Cartilage - pathology | Humans | Tumor Necrosis Factor-alpha - genetics | Extracellular Matrix - immunology | Extracellular Matrix - metabolism | Synovial Membrane - immunology | Synovial Membrane - pathology | Arthritis, Rheumatoid - metabolism | Bone and Bones - metabolism | Interleukin-1 - immunology | Synovitis - immunology | Tumor Necrosis Factor-alpha - immunology | Disease Models, Animal | Fibroblasts - metabolism | Cartilage - immunology | Interleukin-1 - metabolism | Syndecan-4 - metabolism | Mice, Inbred C57BL | Proteoglycans - metabolism | Mice, Transgenic | Cartilage - metabolism | Fibroblasts - pathology | Synovial Membrane - metabolism | Arthritis, Rheumatoid - pathology | Synovitis - pathology | Animals | Fibroblasts - immunology | Mice | Bone and Bones - immunology | Arthritis, Rheumatoid - immunology | Extracellular Matrix - pathology | Synovitis | Control | Tumor necrosis factor | Causes of | Arthritis | Inflammation | Diagnosis | Research | Health aspects | Risk factors | Tumor necrosis factor-TNF | Cytokines
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2011, Volume 286, Issue 22, pp. 19541 - 19548
HIV-1 p17 contains C-and N-terminal sequences with positively charged residues and a consensus cluster for heparin binding. We have previously demonstrated by... 
FORMS | HOMING RECEPTOR CD44 | REPLICATION | ANTIRETROVIRAL-THERAPY | BIOCHEMISTRY & MOLECULAR BIOLOGY | LYMPH-NODES | IMMUNODEFICIENCY-VIRUS TYPE-1 | INFECTION | GROWTH-FACTOR | BINDING | LYMPHOCYTES | Heparitin Sulfate - immunology | Humans | Syndecan-4 - genetics | Tumor Necrosis Factor-alpha - genetics | HIV Antigens - metabolism | Male | Heparitin Sulfate - metabolism | CD4-Positive T-Lymphocytes - immunology | gag Gene Products, Human Immunodeficiency Virus - metabolism | Interleukin-2 - immunology | Syndecan-2 - immunology | Hyaluronan Receptors - metabolism | Syndecan-2 - genetics | Tumor Necrosis Factor-alpha - immunology | Female | gag Gene Products, Human Immunodeficiency Virus - genetics | Syndecan-4 - immunology | Syndecan-2 - metabolism | HIV-1 - metabolism | Lymphocyte Activation | Syndecan-4 - metabolism | CD4-Positive T-Lymphocytes - metabolism | HIV-1 - genetics | Hyaluronan Receptors - genetics | Interleukin-2 - genetics | HIV-1 - immunology | HIV Antigens - genetics | HIV Antigens - immunology | Heparitin Sulfate - genetics | Protein Binding | Hyaluronan Receptors - immunology | HeLa Cells | Interleukin-2 - biosynthesis | gag Gene Products, Human Immunodeficiency Virus - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Syndecans | Viral Protein | Proteoglycans | Microbiology | HIV | Cytokine | p17 | Heparan Sulfate | AIDS | Matrix Protein | Heparin-binding Protein
Journal Article
Cellular Microbiology, ISSN 1462-5814, 12/2016, Volume 18, Issue 12, pp. 1846 - 1856
Summary Pulmonary tuberculosis (TB) is an airborne disease caused by the intracellular bacterial pathogen Mycobacterium tuberculosis (Mtb). Alveolar epithelial... 
IMMUNE-RESPONSE | PROTEOGLYCANS | HBHA | HEPARIN-BINDING HEMAGGLUTININ | TUBERCULOSIS | INFLAMMATION | MACROPHAGES | DISSEMINATION | MICROBIOLOGY | ADHESIN | MOLECULAR-MECHANISMS | CELL BIOLOGY | Lung - microbiology | RNA, Messenger - immunology | RNA, Small Interfering - genetics | Tuberculosis, Pulmonary - microbiology | Adhesins, Bacterial - genetics | Humans | Syndecan-1 - genetics | Syndecan-4 - genetics | Mycobacterium tuberculosis - pathogenicity | Adhesins, Bacterial - metabolism | Syndecan-4 - antagonists & inhibitors | Syndecan-1 - immunology | Bacterial Adhesion - drug effects | Mycobacterium tuberculosis - metabolism | Macrophages, Alveolar - immunology | Syndecan-4 - immunology | A549 Cells | Macrophages, Alveolar - microbiology | Signal Transduction | RNA, Messenger - genetics | Gene Expression Regulation | Antibodies, Neutralizing - pharmacology | Tuberculosis, Pulmonary - pathology | Syndecan-1 - deficiency | Mice, Knockout | Host-Pathogen Interactions | Tuberculosis, Pulmonary - immunology | Animals | Epithelial Cells - immunology | Epithelial Cells - microbiology | Mice | Syndecan-4 - deficiency | Lung - immunology | Mycobacterium tuberculosis - growth & development | RNA, Small Interfering - metabolism | Tuberculosis | Bacterial infections | RNA | Dendritic cells | Analysis | Lectins | Antitubercular agents | Rodents | Genes
Journal Article
Journal Article
Journal Article