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Journal of Cell Science, ISSN 0021-9533, 01/2014, Volume 127, Issue 1, pp. 158 - 171
Here, we show that epithelial-mesenchymal status influences how cells deposit extracellular matrix. Retinal pigmented epithelial (RPE) cells that expressed... 
Fibrillin-1 | Cell-cell junction | Integrin | Perlecan | Epithelial cell | Mesenchymal cell | Fibronectin | Syndecan | FOCAL ADHESION FORMATION | ACTIVATION | SYNDECAN-4 | FIBRONECTIN EXTRACELLULAR-MATRIX | LATENT TGF-BETA | CELL BIOLOGY | TRANSITION | EVOLUTION | HEPARAN-SULFATE | BINDING-SITES | Retinal Pigment Epithelium - metabolism | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Syndecan-4 - genetics | Actins - metabolism | Integrins - genetics | Epithelial-Mesenchymal Transition - genetics | Mammary Glands, Human - metabolism | Fibroblasts - ultrastructure | Heparitin Sulfate - metabolism | Integrins - metabolism | Actins - genetics | Actomyosin - genetics | Retinal Pigment Epithelium - ultrastructure | Female | Microfilament Proteins - metabolism | Zonula Occludens-1 Protein - metabolism | Cadherins - genetics | Microfilament Proteins - genetics | Fibroblasts - metabolism | Microfibrils - ultrastructure | Zonula Occludens-1 Protein - genetics | Cell Line | Podocytes - metabolism | Syndecan-4 - metabolism | Gene Expression Regulation | Intercellular Junctions - metabolism | Epithelial Cells - ultrastructure | Fibrillins | Organ Specificity | Actomyosin - metabolism | beta Catenin - metabolism | Fibronectins - metabolism | beta Catenin - genetics | Transforming Growth Factor beta - pharmacology | Podocytes - ultrastructure | Mammary Glands, Human - ultrastructure | Fibronectins - genetics | Intercellular Junctions - ultrastructure | Microfibrils - metabolism | Cell–cell junction
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2014, Volume 192, Issue 6, pp. 2576 - 2584
Having discovered that the dendritic cell (DC)-associated heparan sulfate proteoglycan-dependent integrin ligand (DC-HIL) receptor on APCs inhibits T cell... 
ADHESION | DC-HIL | ACTIVATION | PROTEIN | SYNDECAN-4 | TOLERANCE | DISEASE | IMMUNOLOGY | CANCER | T-CELLS | NEGATIVE REGULATOR | Eye Proteins - immunology | Reactive Oxygen Species - metabolism | Spleen - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Membrane Glycoproteins - metabolism | Syndecan-4 - genetics | Autoimmunity - genetics | Encephalomyelitis, Autoimmune, Experimental - immunology | Adoptive Transfer | Interferon-gamma - metabolism | DNA-Binding Proteins - metabolism | Flow Cytometry | T-Lymphocytes - metabolism | Autoimmunity - immunology | Myeloid Cells - immunology | Reactive Oxygen Species - immunology | Female | Encephalomyelitis, Autoimmune, Experimental - genetics | Eye Proteins - genetics | Membrane Glycoproteins - immunology | Spleen - pathology | Syndecan-4 - immunology | Nitric Oxide - immunology | DNA-Binding Proteins - immunology | CD11b Antigen - immunology | Syndecan-4 - metabolism | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Cells, Cultured | Receptors, Chemokine - immunology | DNA-Binding Proteins - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Eye Proteins - metabolism | Spleen - metabolism | Interferon-gamma - immunology | Myeloid Cells - metabolism | Myeloid Cells - transplantation | T-Lymphocytes - immunology | Mice | CD11b Antigen - metabolism | Nitric Oxide - metabolism
Journal Article
The FEBS Journal, ISSN 1742-464X, 05/2013, Volume 280, Issue 10, pp. 2228 - 2247
Sustained pressure overload induces heart failure, the main cause of mortality in the Western world. Increased understanding of the underlying molecular... 
heart failure | shedding | heparan sulfate | inflammation | proteoglycan | MYOCARDIAL-INFARCTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | EUROPEAN-SOCIETY | FAILURE | HYPERTROPHY | DISEASE | MICE | HEALTH | T-LYMPHOCYTES | CELL-LINE | EPIDEMIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Interleukin-1beta - pharmacology | Rats, Wistar | Myocytes, Cardiac - immunology | Humans | Middle Aged | Syndecan-4 - genetics | Extracellular Matrix - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Ventricular Remodeling | T-Lymphocytes - metabolism | Interleukin-1beta - metabolism | Myocardium - metabolism | HEK293 Cells | Adult | Female | Benzamides - pharmacology | Fibroblasts - metabolism | Syndecan-4 - immunology | Animals, Newborn | NF-kappa B - antagonists & inhibitors | Syndecan-4 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Rats | Myocardium - pathology | Heart Failure - metabolism | Heart Failure - pathology | Fibroblasts - pathology | Immunity, Innate | Cell Adhesion | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Focal Adhesions - drug effects | Myocytes, Cardiac - pathology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Myocytes, Cardiac - drug effects | Lipopolysaccharides - pharmacology | Myocytes, Cardiac - metabolism | Fibroblasts - immunology | Mice | Thiazoles - pharmacology | Primary Cell Culture | Heart failure | Immunohistochemistry | Enzymes | RNA | Analysis | Inflammation | Sulfates | Mitogens | Proteins | Signal transduction | Gene expression | Immune system
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2010, Volume 285, Issue 51, pp. 40212 - 40229
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2012, Volume 47, Issue 2, pp. 196 - 202
Proteoglycans (PGs) and their associated glycosaminoglycan side chains are effectors of inflammation, but little is known about changes to the composition of... 
Proteoglycan | Innate immunity | Macrophages | Syndecan | Neutrophils | PROTEOGLYCANS | BIOCHEMISTRY & MOLECULAR BIOLOGY | LUNG INJURY | MECHANISMS | IDENTIFICATION | proteoglycan | CELL BIOLOGY | macrophages | TOLL-LIKE RECEPTOR-4 | innate immunity | RESPIRATORY SYSTEM | syndecan | HEPARAN-SULFATE | neutrophils | PATTERN-RECOGNITION RECEPTORS | EXPRESSION | BINDING | Interleukin-8 - genetics | Syndecan-4 - genetics | Tumor Necrosis Factor-alpha - genetics | Immunity, Innate - genetics | Bronchoalveolar Lavage Fluid - immunology | Pneumonia, Bacterial - metabolism | Lipopolysaccharides - immunology | Lung Injury - metabolism | Tumor Necrosis Factor-alpha - immunology | Pneumonia - immunology | Interleukin-8 - metabolism | Neutrophils - metabolism | Pneumonia - genetics | Syndecan-4 - immunology | Syndecan-4 - metabolism | Neutrophils - drug effects | Myeloid Differentiation Factor 88 - genetics | Macrophages - metabolism | Up-Regulation - immunology | Lung - drug effects | Lipopolysaccharides - pharmacology | Mice | Interleukin-8 - immunology | Pneumonia - metabolism | Lung Injury - immunology | Heparin, Low-Molecular-Weight - immunology | Tumor Necrosis Factor-alpha - metabolism | RNA, Messenger - immunology | Epithelial Cells - metabolism | Heparan Sulfate Proteoglycans - genetics | Myeloid Differentiation Factor 88 - immunology | Pneumonia, Bacterial - genetics | Lung - metabolism | Macrophages - immunology | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Neutrophils - immunology | Up-Regulation - genetics | Heparan Sulfate Proteoglycans - immunology | Immunity, Innate - immunology | Lung Injury - genetics | Animals | Heparan Sulfate Proteoglycans - metabolism | Epithelial Cells - immunology | Pneumonia, Bacterial - immunology | Syndecan-4 - deficiency | Myeloid Differentiation Factor 88 - metabolism | Lung - immunology
Journal Article
Journal Article
Molecular Cancer Research, ISSN 1541-7786, 01/2007, Volume 5, Issue 1, pp. 21 - 33
Journal Article
Journal Article
Journal of cell science, ISSN 0021-9533, 2004, Volume 117, Issue Part 15, pp. 3189 - 3199
Journal Article
Journal Article