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Cancer Research, ISSN 0008-5472, 06/2018, Volume 78, Issue 11, pp. 2852 - 2863
Although prostate cancer is clinically manageable during several stages of progression, survival is severely compromised once cells invade and metastasize to... 
INSULIN | STAT3 ACTIVATION | TUMOR INVASION | IGF-IR | ONCOLOGY | RECEPTOR | KINASE INHIBITOR | SYNTENIN | EXPRESSION | GROWTH-FACTOR-I | MELANOMA METASTASIS | Interleukin-8 - genetics | Prostatic Neoplasms - pathology | Interleukin-6 - genetics | Humans | Carcinogenesis - genetics | Male | Signal Transduction - genetics | Melanoma - pathology | Vascular Endothelial Growth Factor A - genetics | Cell Movement - genetics | Carcinogenesis - pathology | Syntenins - genetics | Cell Differentiation - genetics | Matrix Metalloproteinase 2 - genetics | Animals | Prostatic Neoplasms - genetics | Matrix Metalloproteinase 9 - genetics | Melanoma - genetics | Neoplasm Invasiveness - pathology | Cell Line, Tumor | Receptors, Somatomedin - genetics | Neoplasm Invasiveness - genetics | Gene Expression Regulation, Neoplastic - genetics | STAT3 Transcription Factor - genetics | Biotechnology | Phosphorylation | Insulin-like growth factor I | Insulin-like growth factor-binding protein 2 | Insulin-like growth factors | Metastasis | Metastases | Interleukin 6 | Proteins | Dependence | Bioinformatics | Interleukin 8 | CRISPR | Cell survival | Melanoma | Organs | Stat3 protein | Data processing | Tumor cell lines | Insulin | Molecular chains | Gelatinase B | Gelatinase A | Cell lines | Prostate cancer | Prostate | Cell migration | Syndecan | Cancer
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2010, Volume 285, Issue 51, pp. 40212 - 40229
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 11/2017, Volume 21, Issue 11, pp. 3023 - 3043
Hepatitis B virus (HBV) infection plays a crucial role and is a major cause of hepatocellular carcinoma (HCC) in China. microRNAs (miRNAs) have emerged as key... 
pleiotrophin | lipogenesis | metastasis | hepatitis B virus | hepatocellular carcinoma | miR‐384 | miR-384 | MEDICINE, RESEARCH & EXPERIMENTAL | FATTY-ACID SYNTHESIS | PERITONEAL MESOTHELIAL CELLS | CHRONIC HEPATITIS-B | VEGF EXPRESSION | CELL BIOLOGY | LIVER-REGENERATION | INSULIN-RESISTANCE | COLORECTAL-CANCER | HIGH-GLUCOSE | UP-REGULATION | GROWTH-ASSOCIATED MOLECULE | Cell Proliferation | Hepatitis B - metabolism | Hepatitis B - genetics | Humans | Middle Aged | Male | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | fas Receptor - metabolism | Hepatitis B - complications | Mechanistic Target of Rapamycin Complex 1 - genetics | Lipogenesis - genetics | fas Receptor - genetics | Liver Neoplasms - pathology | Trans-Activators - pharmacology | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Cytokines - genetics | Syndecan-3 - metabolism | Liver Neoplasms - genetics | Signal Transduction | Lymphatic Metastasis | Mechanistic Target of Rapamycin Complex 1 - antagonists & inhibitors | Hepatitis B virus - physiology | Mechanistic Target of Rapamycin Complex 1 - metabolism | Host-Pathogen Interactions | Liver Neoplasms - metabolism | MicroRNAs - genetics | Carcinoma, Hepatocellular - metabolism | Hepatitis B virus - pathogenicity | Gene Expression Regulation, Neoplastic | Hepatocytes - pathology | Phosphatidylinositol 3-Kinases - metabolism | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | Liver Neoplasms - etiology | Carcinoma, Hepatocellular - genetics | Trans-Activators - genetics | Adult | Female | Carcinoma, Hepatocellular - etiology | Hepatocytes - drug effects | Sterol Regulatory Element Binding Protein 1 - metabolism | Cytokines - metabolism | Hepatitis B - pathology | Syndecan-3 - genetics | Morpholines - pharmacology | Sirolimus - pharmacology | Hep G2 Cells | Phosphatidylinositol 3-Kinases - genetics | Carrier Proteins - genetics | Carrier Proteins - metabolism | Sterol Regulatory Element Binding Protein 1 - genetics | Lipogenesis - drug effects | Carcinoma, Hepatocellular - pathology | Trans-Activators - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | MicroRNA | Synthesis | Development and progression | Hepatoma | Metastasis | Fatty acids | Health aspects | Protein binding | Hepatitis B | Cell proliferation | Chronic infection | Viruses | Hepatocellular carcinoma | AKT protein | Infections | Carcinogenesis | Metastases | Hepatitis | Liver cancer | Carcinogens | Fatty liver | Down-regulation | Sterol regulatory element-binding protein | Hepatitis B virus | Lipogenesis | Pleiotrophin | MiRNA | Gene expression | 1-Phosphatidylinositol 3-kinase | Steatosis | Hepatocytes | Syndecan | Sterol regulatory element-binding protein 1c | Index Medicus | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2008, Volume 3, Issue 12, pp. e3907 - e3907
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2008, Volume 283, Issue 4, pp. 2373 - 2384
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 6, pp. e11286 - e11286
Syndecans are a family of type-I transmembrane proteins that are involved in cell-matrix adhesion, migration, neuronal development, and inflammation. Previous... 
QUANTITATIVE TRAIT LOCI | LIFE-SPAN | SKELETAL-MUSCLE | GENE DISRUPTION PROJECT | DROSOPHILA-MELANOGASTER | MULTIDISCIPLINARY SCIENCES | AFFECTING LONGEVITY | INSULIN-LIKE PEPTIDES | HEPARAN-SULFATE PROTEOGLYCAN | SUSCEPTIBILITY LOCUS | STRESS RESISTANCE | Body Weight | Humans | Male | Syndecans - genetics | Drosophila melanogaster - genetics | Genetic Complementation Test | Drosophila melanogaster - metabolism | Genetic Variation | Homozygote | Animals | Energy Metabolism | Syndecans - physiology | Female | Polymorphism, Single Nucleotide | Mutation | Blood Glucose - metabolism | Child | Brain | Energy metabolism | Peptides | Leukocyte migration | Hispanic Americans | Gallbladder diseases | Cardiovascular disease | Single-nucleotide polymorphism | Glucose | Proteins | Signal transduction | Mitochondria | Genetics | Complementation | Teenagers | Growth factors | Obesity | Starvation | Metabolism | Mammals | Insulin | Membrane proteins | Storage | Insects | Flies | Cell migration | Syndecan | Metabolic rate | Haplotypes | Genes | Families & family life | Homeostasis | African Americans | Homology | Genomes | Body composition | Glucose metabolism | Energy | Body composition (biology) | Gallbladder | Children | Nutrition | Drosophila | Energy expenditure | Mutants | Sleep | Life span | Alleles | Insulin resistance | Diabetes | Electron transport | Variation | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2012, Volume 287, Issue 39, pp. 32578 - 32587
Staphylococcal superantigens (SAgs), such as toxic shock syndrome toxin-1 (TSST-1), are the main cause of toxic shock syndrome (TSS). SAgs deregulate the host... 
CELLS | ACTIVATION | ALPHA-CONVERTING ENZYME | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENTEROTOXINS | PENETRATION | NECROSIS-FACTOR-ALPHA | CLEAVAGE | EXPRESSION | SYNDECANS | ADAM17 Protein | Interleukin-8 - genetics | Receptor, Epidermal Growth Factor - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Syndecan-1 - genetics | Glycoproteins - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Amphiregulin | Superantigens - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | EGF Family of Proteins | Bacterial Toxins - genetics | Dipeptides - metabolism | Interleukin-8 - metabolism | Transforming Growth Factor alpha - metabolism | Staphylococcus aureus - metabolism | Glycoproteins - genetics | Staphylococcus aureus - genetics | Syndecan-1 - metabolism | Signal Transduction | Intercellular Signaling Peptides and Proteins - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Transforming Growth Factor alpha - genetics | Hydroxamic Acids - metabolism | ADAM Proteins - metabolism | Bacterial Toxins - metabolism | Enterotoxins - genetics | Models, Biological | Superantigens - genetics | Enterotoxins - metabolism | Human Umbilical Vein Endothelial Cells - pathology | ADAM Proteins - genetics | Dipeptides - genetics | Index Medicus | Molecular Bases of Disease | Shedding | Toxic Shock Syndrome | ADAM ADAMTS | Epidermal Growth Factor Receptor (EGFR) | Epithelial Cell | Mucosal Immunology | Superantigen | Staphylococcus aureus
Journal Article
细胞研究:英文版, ISSN 1001-0602, 2015, Volume 25, Issue 4, pp. 412 - 428
Journal Article
Cancer Science, ISSN 1347-9032, 09/2018, Volume 109, Issue 9, pp. 2919 - 2936
Journal Article