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Nature (London), ISSN 1476-4687, 2018, Volume 562, Issue 7725, pp. 69 - 75
Primary liver cancer represents a major health problem. It comprises hepatocellular carcinoma (HCC) and intrahepatic cholangiocarcinoma (ICC), which differ... 
PATHOGENESIS | HEPATOCELLULAR-CARCINOMA | READ ALIGNMENT | HEPATOCYTES | DNA | INFLAMMATION | PACKAGE | MULTIDISCIPLINARY SCIENCES | FRAMEWORK | BINDING | DISCOVERY | Humans | Tumor Microenvironment | Apoptosis - genetics | Hepatocytes - pathology | Male | Gene Expression Profiling | Genes, myc | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | DNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - genetics | DNA Transposable Elements - genetics | Female | Liver Neoplasms - pathology | Cell Differentiation | Cell Lineage - genetics | Disease Models, Animal | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | Cytokines - metabolism | Liver Neoplasms - genetics | Carcinogenesis - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Transcription Factors - metabolism | Cholangiocarcinoma - pathology | Animals | Epigenesis, Genetic - genetics | Carcinoma, Hepatocellular - pathology | Cholangiocarcinoma - genetics | Mosaicism | Mice | Necrosis - genetics | Genes, ras | Liver cancer | Research | Carcinogenesis | Oncology, Experimental | Apoptosis | Cancer | Animal models | Cytokines | Liver | Hepatocellular carcinoma | Genomes | Metastasis | Risk analysis | Gene expression | Risk factors | Metastases | Hepatocytes | Morphology | DNA methylation | Tumorigenesis | Bioinformatics | Cholangiocarcinoma | Deoxyribonucleic acid--DNA | Tumors | T-Box Domain Proteins/metabolism | Hepatocytes/pathology | DNA-Binding Proteins/metabolism | Life Sciences | Cholangiocarcinoma/pathology | Transcription Factors/metabolism | Necrosis/genetics | Cytokines/metabolism | Epigenesis, Genetic/genetics | Cyclin-Dependent Kinase Inhibitor p16/deficiency | Carcinoma, Hepatocellular/pathology | DNA Transposable Elements/genetics | Cell Lineage/genetics | T-Box Domain Proteins/genetics | Transcription Factors/genetics | Apoptosis/genetics | Proto-Oncogene Proteins c-akt/genetics | Liver Neoplasms/genetics | Cholangiocarcinoma/genetics | Liver Neoplasms/pathology | DNA-Binding Proteins/genetics | Hepatocytes/metabolism | Carcinoma, Hepatocellular/genetics | Carcinogenesis/genetics
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2012, Volume 109, Issue 45, pp. 18273 - 18280
During cardiogenesis, Fibroblast Growth Factor (Fgf10) is expressed in the anterior second heart field. Together with Fibroblast growth factor 8 (Fgf8), Fgf10... 
Heart | Transgenic animals | Heart ventricles | Genes | Myocardium | Promoter regions | Transgenes | Embryos | Progenitor cells | Binding sites | Transcriptional regulation | Mouse embryo | TRANSCRIPTION FACTORS | CELLS | ARTERIAL POLE | MOUSE HEART | transcriptional regulation | MULTIDISCIPLINARY SCIENCES | OUTFLOW TRACT | mouse embryo | ENHANCER | ISL1 | DIFFERENTIATION | EXPRESSION | FGF10 | Protein Binding - genetics | LIM-Homeodomain Proteins - metabolism | Heart - embryology | Homeodomain Proteins - metabolism | Molecular Sequence Data | Chromosomes, Artificial, Bacterial - genetics | Gene Expression Regulation, Developmental | Base Pairing - genetics | Base Sequence | Myocardium - metabolism | Transcription, Genetic | Fibroblast Growth Factor 10 - genetics | Binding Sites | Fibroblast Growth Factor 10 - metabolism | Transgenes - genetics | Mice, Transgenic | Transcription Factors - genetics | T-Box Domain Proteins - genetics | Down-Regulation - genetics | Homeodomain Proteins - genetics | Regulatory Sequences, Nucleic Acid - genetics | T-Box Domain Proteins - metabolism | Transcription Factors - metabolism | Homeobox Protein Nkx-2.5 | LIM-Homeodomain Proteins - genetics | Animals | Genes, Switch - genetics | Models, Biological | Mice | Physiological aspects | Fibroblast growth factors | Genetic aspects | Research | Genetic transcription | Genetic regulation | Health aspects | Heart muscle | Regulatory Sequences, Nucleic Acid | Cellular Biology | Life Sciences | Chromosomes, Artificial, Bacterial | Genes, Switch | Down-Regulation | Base Pairing | Fibroblast Growth Factor 10 | Homeodomain Proteins | Protein Binding | LIM-Homeodomain Proteins | Transcription Factors | T-Box Domain Proteins | Biological Sciences
Journal Article
by Jones, David T. W and Jäger, Natalie and Kool, Marcel and Zichner, Thomas and Hutter, Barbara and Sultan, Marc and Cho, Yoon-Jae and Pugh, Trevor J and Hovestadt, Volker and Stütz, Adrian M and Rausch, Tobias and Warnatz, Hans-Jörg and Ryzhova, Marina and Bender, Sebastian and Sturm, Dominik and Pleier, Sabrina and Cin, Huriye and Pfaff, Elke and Sieber, Laura and Wittmann, Andrea and Remke, Marc and Witt, Hendrik and Hutter, Sonja and Tzaridis, Theophilos and Weischenfeldt, Joachim and Raeder, Benjamin and Avci, Meryem and Amstislavskiy, Vyacheslav and Zapatka, Marc and Weber, Ursula D and Wang, Qi and Lasitschka, Bärbel and Bartholomae, Cynthia C and Schmidt, Manfred and von Kalle, Christof and Ast, Volker and Lawerenz, Chris and Eils, Jürgen and Kabbe, Rolf and Benes, Vladimir and van Sluis, Peter and Koster, Jan and Volckmann, Richard and Shih, David and Betts, Matthew J and Russell, Robert B and Coco, Simona and Paolo Tonini, Gian and Schüller, Ulrich and Hans, Volkmar and Graf, Norbert and Kim, Yoo-Jin and Monoranu, Camelia and Roggendorf, Wolfgang and Unterberg, Andreas and Herold-Mende, Christel and Milde, Till and Kulozik, Andreas E and von Deimling, Andreas and Witt, Olaf and Maass, Eberhard and Rössler, Jochen and Ebinger, Martin and Schuhmann, Martin U and Frühwald, Michael C and Hasselblatt, Martin and Jabado, Nada and Rutkowski, Stefan and von Bueren, André O and Williamson, Dan and Clifford, Steven C and McCabe, Martin G and Peter Collins, V and Wolf, Stephan and Wiemann, Stefan and Lehrach, Hans and Brors, Benedikt and Scheurlen, Wolfram and Felsberg, Jörg and Reifenberger, Guido and Northcott, Paul A and Taylor, Michael D and Meyerson, Matthew and Pomeroy, Scott L and Yaspo, Marie-Laure and Korbel, Jan O and Korshunov, Andrey and Eils, Roland and Pfister, Stefan M and Lichter, Peter
Nature (London), ISSN 1476-4687, 2012, Volume 488, Issue 7409, pp. 100 - 105
Journal Article
Nature neuroscience, ISSN 1546-1726, 2010, Volume 13, Issue 5, pp. 551 - 558
... by both alterations in the NSC mitotic division plane and asymmetric inheritance of proteins that regulate cell fate (4-6). The exact mechanisms that define NSC divisions remain, how... 
HUMAN-CHROMOSOMES | PROTEIN | LIS1 EXPRESSION | LISSENCEPHALY GENE | SECKEL-SYNDROME | NASHI | SPINDLE ORIENTATION | NEUROGENESIS | NEUROSCIENCES | MESSENGER-RNA LOCALIZATION | CEREBRAL CORTICAL SIZE | Brain - embryology | Microcephaly - genetics | Embryo, Mammalian | Homeodomain Proteins - metabolism | Humans | Apoptosis - genetics | Green Fluorescent Proteins - genetics | RNA, Messenger - metabolism | Neurogenesis - genetics | DNA Mutational Analysis | Organ Size - genetics | Repressor Proteins - metabolism | Animals, Newborn | Oligonucleotide Array Sequence Analysis - methods | Repressor Proteins - genetics | Genotype | Mice, Transgenic | In Situ Nick-End Labeling - methods | Mutation - genetics | T-Box Domain Proteins - metabolism | Eye Proteins - metabolism | Brain - pathology | Mice | HeLa Cells | Paired Box Transcription Factors - genetics | Neurons - pathology | Age Factors | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Gene Expression Regulation, Developmental - genetics | Brain - growth & development | Cell Differentiation - genetics | 1-Alkyl-2-acetylglycerophosphocholine Esterase - genetics | Transfection | Microcephaly - pathology | Microcephaly - physiopathology | Bromodeoxyuridine - metabolism | Eye Proteins - genetics | Nuclear Proteins - genetics | RNA Interference - physiology | Cell Division - genetics | Mice, Inbred C57BL | Gene Expression Profiling - methods | Nuclear Proteins - metabolism | PAX6 Transcription Factor | Nerve Tissue Proteins - genetics | T-Box Domain Proteins - genetics | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | 1-Alkyl-2-acetylglycerophosphocholine Esterase - metabolism | Animals | Stem Cells - physiology | Paired Box Transcription Factors - metabolism | Animal experimentation | Axons | Usage | Brain research | Cell division | Physiological aspects | Causes of | Research | Microcephaly | Mental retardation | Risk factors
Journal Article
Nature genetics, ISSN 1546-1718, 2000, Volume 26, Issue 3, pp. 291 - 299
To identify new immortalizing genes with potential roles in tumorigenesis, we performed a genetic screen aimed to bypass the rapid and tight senescence arrest of primary fibroblasts deficient for the oncogene Bmi1... 
LYMPHOMAGENESIS | APOPTOSIS | STABILIZES P53 | AMPLIFICATION | INK4A LOCUS | GENETICS & HEREDITY | C-MYC | MDM2 | GENE FAMILY | CELL-CYCLE ARREST | ARF TUMOR-SUPPRESSOR | T-Box Domain Proteins - physiology | Protein Biosynthesis | Humans | E2F Transcription Factors | Cyclin-Dependent Kinase Inhibitor p16 | T-Box Domain Proteins - isolation & purification | Adenocarcinoma - metabolism | Cell Transformation, Neoplastic - genetics | Gene Deletion | Repressor Proteins - isolation & purification | Neoplastic Syndromes, Hereditary - genetics | Genes, BRCA1 | Neoplasm Proteins - genetics | Tumor Suppressor Protein p14ARF | Oncogenes | DNA-Binding Proteins | Fibroblasts - metabolism | Cell Cycle Proteins - isolation & purification | Repressor Proteins - genetics | Transcription Factor DP1 | Breast Neoplasms - genetics | Polycomb Repressive Complex 1 | Neoplasm Proteins - isolation & purification | Proto-Oncogene Proteins p21(ras) - antagonists & inhibitors | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Fibroblasts - cytology | Mice | COS Cells | Cell Cycle Proteins - physiology | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Chromosomes, Human, Pair 17 - genetics | Breast Neoplasms - metabolism | Repressor Proteins - physiology | Transfection | Cell Cycle Proteins - genetics | Female | Adenocarcinoma - genetics | Tumor Cells, Cultured | Nuclear Proteins - genetics | Cellular Senescence - genetics | Promoter Regions, Genetic | E2F1 Transcription Factor | Neoplasm Proteins - biosynthesis | Cells, Cultured | Proto-Oncogene Proteins - genetics | Transcription Factors - antagonists & inhibitors | T-Box Domain Proteins - genetics | Proteins - genetics | Carrier Proteins - genetics | Animals | Gene Amplification | Retinoblastoma-Binding Protein 1 | Fibroblasts | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Research | Risk factors
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 2016, Volume 133, Issue 11, pp. 1081 - 1092
BACKGROUND—Adult mammalian cardiomyocytes (CMs) have the potential to proliferate, but this is not sufficient to generate adequate CMs after myocardial... 
Myocardial infarction | Regeneration | Myocytes, cardiac | Molecular biology | Cell cycle | molecular biology | RENEWAL | CARDIAC & CARDIOVASCULAR SYSTEMS | cell cycle | regeneration | STAGE | myocardial infarction | GENES | YAP | PERIPHERAL VASCULAR DISEASE | myocytes | cardiac | Single-Blind Method | T-Box Domain Proteins - physiology | RNA, Small Interfering - genetics | T-Box Domain Proteins - biosynthesis | Muscle Proteins - biosynthesis | RNA, Messenger - biosynthesis | RNA Interference | Cell Division | Myocardial Infarction - pathology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Electrocardiography | Female | Myocardial Infarction - physiopathology | Genes, Reporter | Heart - physiopathology | Immediate-Early Proteins - physiology | Cell Size - drug effects | Immediate-Early Proteins - biosynthesis | RNA, Messenger - genetics | Genes, cdc - drug effects | Mice, Transgenic | Myocardium - pathology | Cell Cycle Proteins - biosynthesis | Myocardial Infarction - metabolism | Random Allocation | Fetal Proteins - biosynthesis | T-Box Domain Proteins - genetics | Gene Expression Regulation - drug effects | Muscle Proteins - genetics | Myocytes, Cardiac - pathology | Tumor Suppressor Proteins - physiology | Organ Size - drug effects | Animals | Immediate-Early Proteins - genetics | Signal Transduction - drug effects | Tamoxifen - pharmacology | Myocytes, Cardiac - metabolism | Fetal Proteins - genetics | Mice | Tumor Suppressor Proteins - biosynthesis | Cell proliferation | Care and treatment | Transcription factors | Research | Heart attack | Heart cells | remodeling
Journal Article
Development (Cambridge), ISSN 1477-9129, 2014, Volume 141, Issue 6, pp. 1381 - 1391
How signaling gradients supply positional information in a field of moving cells is an unsolved question in patterning and morphogenesis. Here, we ask how a... 
Signal gradient | Wnt signaling | Embryonic elongation | Fgf signaling | Segmentation clock | Time-lapse microscopy | WAVE-FRONT ACTIVITY | SIGNALING PATHWAYS | MOUSE EMBRYOS | NOTCH | DEVELOPMENTAL BIOLOGY | BOUNDARY FORMATION | GENE-EXPRESSION | BODY FORMATION | PRESOMITIC MESODERM | VERTEBRATE SEGMENTATION | T-Box Domain Proteins - physiology | Fibroblast Growth Factors - genetics | Wnt Signaling Pathway - physiology | Zebrafish - embryology | Intercellular Signaling Peptides and Proteins - physiology | Somites - embryology | Wnt Proteins - genetics | Gene Expression Regulation, Developmental | Fibroblast Growth Factors - physiology | Somites - metabolism | Basic Helix-Loop-Helix Transcription Factors - physiology | Basic Helix-Loop-Helix Transcription Factors - genetics | Animals, Genetically Modified | Zebrafish Proteins - antagonists & inhibitors | Intercellular Signaling Peptides and Proteins - genetics | Heat-Shock Response - physiology | T-Box Domain Proteins - genetics | Zebrafish - genetics | Zebrafish Proteins - physiology | Body Patterning - physiology | Heat-Shock Response - genetics | Animals | Wnt Signaling Pathway - genetics | Models, Biological | Zebrafish - metabolism | Wnt Proteins - physiology | Zebrafish Proteins - genetics | Wnt Proteins - antagonists & inhibitors | Body Patterning - genetics | Life Sciences | Genetics
Journal Article
Journal Article
Developmental biology, ISSN 0012-1606, 09/2012, Volume 369, Issue 2, pp. 319 - 329
.... Hedgehog activates wingless anterior to the Hedgehog domain. midline/H15 are responsible in part for repressing wingless in cells posterior to the Hedgehog expressing cells... 
CUBITUS INTERRUPTUS | GENES MIDLINE | COREPRESSOR | PROTEIN | INTERACTS | TRANSCRIPTION | DEVELOPMENTAL BIOLOGY | DEPENDENT REPRESSION | EXPRESSION | H15 | DROSOPHILA | Wnt1 Protein - genetics | Drosophila melanogaster - embryology | T-Box Domain Proteins - chemistry | Molecular Sequence Data | DNA Primers - genetics | Wnt1 Protein - metabolism | Drosophila Proteins - metabolism | Drosophila melanogaster - genetics | Drosophila melanogaster - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Gene Expression Regulation, Developmental | Serrate-Jagged Proteins | Base Sequence | Membrane Proteins - metabolism | Protein Interaction Domains and Motifs | Repressor Proteins - metabolism | Wnt1 Protein - chemistry | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Genes, Insect | Amino Acid Sequence | Repressor Proteins - chemistry | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Animals, Genetically Modified | Membrane Proteins - genetics | Intercellular Signaling Peptides and Proteins - genetics | Repressor Proteins - genetics | Drosophila Proteins - chemistry | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Body Patterning - physiology | Sequence Homology, Amino Acid | Animals | Drosophila Proteins - genetics | Body Patterning - genetics | Basic Helix-Loop-Helix Transcription Factors - chemistry | Calcium-Binding Proteins - genetics
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2015, Volume 212, Issue 12, pp. 2041 - 2056
The transcription factor T-bet is critical for cytotoxic T lymphocyte (CTL) differentiation, but it is unclear how it operates in a graded manner in the... 
EFFECTOR | MEDICINE, RESEARCH & EXPERIMENTAL | PROTECTIVE IMMUNITY | STEM-CELLS | SELECTIVE EXPRESSION | MEMORY | GENE | IN-VIVO | SUBSETS | GENERATION | IMMUNOLOGY | CUTTING EDGE | Lymphocytic choriomeningitis virus - immunology | Oligonucleotide Array Sequence Analysis | Homeodomain Proteins - immunology | T-Box Domain Proteins - immunology | Transcriptome - immunology | Lymphocytic Choriomeningitis - immunology | Zinc Finger E-box Binding Homeobox 2 | Host-Pathogen Interactions - immunology | Cell Differentiation - genetics | Flow Cytometry | Repressor Proteins - deficiency | CD8-Positive T-Lymphocytes - metabolism | Lymphocytic choriomeningitis virus - physiology | Receptors, Immunologic - immunology | Lymphocytic Choriomeningitis - genetics | T-Lymphocytes, Cytotoxic - immunology | Lymphocytic Choriomeningitis - virology | Mice, Inbred C57BL | Repressor Proteins - genetics | Mice, Transgenic | Transcriptome - genetics | Protein Binding - immunology | Reverse Transcriptase Polymerase Chain Reaction | T-Box Domain Proteins - genetics | Homeodomain Proteins - genetics | T-Box Domain Proteins - metabolism | Mice, Knockout | Cell Differentiation - immunology | Animals | T-Lymphocytes, Cytotoxic - metabolism | Repressor Proteins - immunology | CD8-Positive T-Lymphocytes - immunology | Receptors, Immunologic - metabolism | Cluster Analysis
Journal Article