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Nature, ISSN 0028-0836, 10/2018, Volume 562, Issue 7725, pp. 69 - 75
Primary liver cancer represents a major health problem. It comprises hepatocellular carcinoma (HCC) and intrahepatic cholangiocarcinoma (ICC), which differ... 
PATHOGENESIS | HEPATOCELLULAR-CARCINOMA | INTRAHEPATIC CHOLANGIOCARCINOMA | READ ALIGNMENT | HEPATOCYTES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | SEQUENCING DATA | EXPRESSION | DISCOVERY | CELL-DEATH | Humans | Tumor Microenvironment | Apoptosis - genetics | Hepatocytes - pathology | Male | Gene Expression Profiling | Genes, myc | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | DNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - genetics | DNA Transposable Elements - genetics | Female | Liver Neoplasms - pathology | Cell Differentiation | Cell Lineage - genetics | Disease Models, Animal | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | Cytokines - metabolism | Liver Neoplasms - genetics | Carcinogenesis - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Transcription Factors - metabolism | Cholangiocarcinoma - pathology | Animals | Epigenesis, Genetic - genetics | Carcinoma, Hepatocellular - pathology | Cholangiocarcinoma - genetics | Mosaicism | Mice | Necrosis - genetics | Genes, ras | Liver cancer | Research | Carcinogenesis | Oncology, Experimental | Apoptosis | Cancer | Animal models | Cytokines | Liver | Hepatocellular carcinoma | Genomes | Metastasis | Risk analysis | Gene expression | Risk factors | Metastases | Hepatocytes | Morphology | DNA methylation | Tumorigenesis | Bioinformatics | Cholangiocarcinoma | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2015, Volume 212, Issue 12, pp. 2041 - 2056
The transcription factor T-bet is critical for cytotoxic T lymphocyte (CTL) differentiation, but it is unclear how it operates in a graded manner in the... 
EFFECTOR | MEDICINE, RESEARCH & EXPERIMENTAL | PROTECTIVE IMMUNITY | STEM-CELLS | SELECTIVE EXPRESSION | MEMORY | GENE | IN-VIVO | SUBSETS | GENERATION | IMMUNOLOGY | CUTTING EDGE | Lymphocytic choriomeningitis virus - immunology | Oligonucleotide Array Sequence Analysis | Homeodomain Proteins - immunology | T-Box Domain Proteins - immunology | Transcriptome - immunology | Lymphocytic Choriomeningitis - immunology | Zinc Finger E-box Binding Homeobox 2 | Host-Pathogen Interactions - immunology | Cell Differentiation - genetics | Flow Cytometry | Repressor Proteins - deficiency | CD8-Positive T-Lymphocytes - metabolism | Lymphocytic choriomeningitis virus - physiology | Receptors, Immunologic - immunology | Lymphocytic Choriomeningitis - genetics | T-Lymphocytes, Cytotoxic - immunology | Lymphocytic Choriomeningitis - virology | Mice, Inbred C57BL | Repressor Proteins - genetics | Mice, Transgenic | Transcriptome - genetics | Protein Binding - immunology | Reverse Transcriptase Polymerase Chain Reaction | T-Box Domain Proteins - genetics | Homeodomain Proteins - genetics | T-Box Domain Proteins - metabolism | Mice, Knockout | Cell Differentiation - immunology | Animals | T-Lymphocytes, Cytotoxic - metabolism | Repressor Proteins - immunology | CD8-Positive T-Lymphocytes - immunology | Receptors, Immunologic - metabolism | Cluster Analysis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, pp. e46082 - e46082
MicroRNA (miR)-155 is a critical player in both innate and adaptive immune responses. It can influence CD4(+) T cell lineage choice. To clarify the role of... 
PREVENTS DEVELOPMENT | SUPPRESSOR | ACTIVATION | MULTIDISCIPLINARY SCIENCES | STAT3 | T(H)17 | CD4(+) T-CELLS | RECEPTOR | ABSENCE | INDUCTION | EXPRESSION | GATA3 Transcription Factor - genetics | T-Lymphocytes, Regulatory - metabolism | Transforming Growth Factor beta1 - metabolism | STAT Transcription Factors - metabolism | Suppressor of Cytokine Signaling 1 Protein | Janus Kinases - metabolism | Interferon-gamma - metabolism | Cell Differentiation - genetics | Th17 Cells - metabolism | Base Sequence | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Th17 Cells - cytology | Interferon-gamma - genetics | Interleukin-4 - genetics | GATA3 Transcription Factor - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Signal Transduction | Interleukin-4 - metabolism | Gene Expression Regulation | Suppressor of Cytokine Signaling Proteins - genetics | Forkhead Transcription Factors - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Interleukin-17 - metabolism | Animals | Mice | MicroRNAs - genetics | Smad Proteins - metabolism | Physiological aspects | Genetic aspects | MicroRNA | Research | Cell differentiation | T cells | Smad5 protein | Smad protein | Transcription factors | Transcription | Laboratories | Interleukin | Differentiation (biology) | Helper cells | Science | Homology | Lymphocytes T | Inflammatory diseases | Signal transduction | Immunology | Transfection | Lymphocytes | Smad2 protein | Atherosclerosis | CD25 antigen | Janus kinase | Cardiology | Growth factors | Immune system | Function analysis | Lupus | Immune response | Cytokines | MiRNA | Cell lineage | T cell receptors | Gene expression | Ribonucleic acid--RNA | Suppressors | CD4 antigen | Signaling | MicroRNAs | Interleukin 10 | Index Medicus | RNA | Ribonucleic acid
Journal Article
Cell, ISSN 0092-8674, 02/2016, Volume 164, Issue 5, pp. 999 - 1014
Journal Article
ISME Journal, ISSN 1751-7362, 2014, Volume 8, Issue 7, pp. 1403 - 1417
Journal Article
Circulation, ISSN 0009-7322, 03/2016, Volume 133, Issue 11, pp. 1081 - 1092
BACKGROUND—Adult mammalian cardiomyocytes (CMs) have the potential to proliferate, but this is not sufficient to generate adequate CMs after myocardial... 
Myocardial infarction | Regeneration | Myocytes, cardiac | Molecular biology | Cell cycle | molecular biology | RENEWAL | CARDIAC & CARDIOVASCULAR SYSTEMS | cell cycle | regeneration | STAGE | myocardial infarction | GENES | YAP | PERIPHERAL VASCULAR DISEASE | myocytes | cardiac | Single-Blind Method | T-Box Domain Proteins - physiology | RNA, Small Interfering - genetics | T-Box Domain Proteins - biosynthesis | Muscle Proteins - biosynthesis | RNA, Messenger - biosynthesis | RNA Interference | Cell Division | Myocardial Infarction - pathology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Electrocardiography | Female | Myocardial Infarction - physiopathology | Genes, Reporter | Heart - physiopathology | Immediate-Early Proteins - physiology | Cell Size - drug effects | Immediate-Early Proteins - biosynthesis | RNA, Messenger - genetics | Genes, cdc - drug effects | Mice, Transgenic | Myocardium - pathology | Cell Cycle Proteins - biosynthesis | Myocardial Infarction - metabolism | Random Allocation | Fetal Proteins - biosynthesis | T-Box Domain Proteins - genetics | Gene Expression Regulation - drug effects | Muscle Proteins - genetics | Myocytes, Cardiac - pathology | Tumor Suppressor Proteins - physiology | Organ Size - drug effects | Animals | Immediate-Early Proteins - genetics | Signal Transduction - drug effects | Tamoxifen - pharmacology | Myocytes, Cardiac - metabolism | Fetal Proteins - genetics | Mice | Tumor Suppressor Proteins - biosynthesis | Cell proliferation | Care and treatment | Transcription factors | Research | Heart attack | Heart cells | Index Medicus | Abridged Index Medicus | remodeling
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 08/2015, Volume 6, Issue 1, pp. 8146 - 8146
The vertebrate heart muscle (myocardium) develops from the first heart field (FHF) and expands by adding second heart field (SHF) cells. While both lineages... 
CELLS | PROGENITORS | ZEBRAFISH | GENE | FIELD | MULTIDISCIPLINARY SCIENCES | MOUSE | MOLECULAR-BASIS | EXPRESSION | LINEAGES | CARDIAC DEVELOPMENT | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 2014, Volume 15, Issue 4, pp. 373 - 383
Journal Article
Circulation research, ISSN 0009-7330, 2007, Volume 100, Issue 3, pp. 354 - 362
The sinoatrial node, which resides at the junction of the right atrium and the superior caval vein, contains specialized myocardial cells that initiate the... 
Cx40 | Sinus node | Heart development | Sinoatrial node | Transgenic mice | Nkx2-5 | Tbx3 | Pitx2c | heart development | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIAC CONDUCTION SYSTEM | sinoatrial node | ULNAR-MAMMARY SYNDROME | PATTERNS | PACEMAKER CHANNELS | HEART | sinus node | HOMEOBOX GENE NKX2-5 | transgenic mice | Tbc3 | PERIPHERAL VASCULAR DISEASE | MICE | MUTATIONS | HEMATOLOGY | EXPRESSION | Connexins - biosynthesis | T-Box Domain Proteins - physiology | Myosin Light Chains - genetics | Heart Atria - embryology | Transcription Factors - deficiency | Recombinant Fusion Proteins - physiology | T-Box Domain Proteins - biosynthesis | Cardiac Myosins - biosynthesis | Gene Expression Regulation, Developmental - genetics | Heart Ventricles - embryology | Ion Channels - genetics | Myosin Light Chains - biosynthesis | Natriuretic Peptide, C-Type - biosynthesis | In Situ Hybridization | Troponin I - genetics | Myocardium - metabolism | Imaging, Three-Dimensional | Genes, Reporter | Gene Expression Regulation, Developmental - physiology | Transcription Factors - physiology | Ion Channels - biosynthesis | Protein Precursors - genetics | Cardiac Myosins - genetics | Troponin I - biosynthesis | Connexins - genetics | Sinoatrial Node - cytology | Mice, Transgenic | Sinoatrial Node - embryology | Transcription Factors - genetics | T-Box Domain Proteins - genetics | Homeodomain Proteins - genetics | Body Patterning - physiology | Mice, Knockout | Homeobox Protein Nkx-2.5 | Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels | Animals | Protein Precursors - biosynthesis | Biomarkers | Natriuretic Peptide, C-Type - genetics | Mice | Homeodomain Proteins - physiology | Body Patterning - genetics | Cyclic Nucleotide-Gated Cation Channels | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2012, Volume 122, Issue 9, pp. 3281 - 3294
CD4 T follicular helper (TFH) cells interact with and stimulate the generation of antigen-specific B cells. TFH cell interaction with B cells correlates with... 
BINDING PROTEIN-ALPHA | MEDICINE, RESEARCH & EXPERIMENTAL | GERMINAL CENTER B | C/EBP-ALPHA | ACTIVATION | COLONY-STIMULATING FACTOR | PROLIFERATION ARREST | IMMUNE-RESPONSES | IMMUNODEFICIENCY VIRUS-INFECTION | RECEPTOR | DIFFERENTIATION | Cell Proliferation | Lymph Nodes - pathology | Oligonucleotide Array Sequence Analysis | Transcriptome | Macaca mulatta | Antibodies, Viral - blood | T-Lymphocytes, Helper-Inducer - immunology | Cell Death | Receptors, Lysosphingolipid - genetics | Receptors, Lysosphingolipid - metabolism | Simian Acquired Immunodeficiency Syndrome - pathology | B-Lymphocytes - metabolism | Simian Immunodeficiency Virus - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | Cytokines - metabolism | Proto-Oncogene Proteins c-maf - genetics | Lymph Nodes - metabolism | Lymph Nodes - virology | Germinal Center - pathology | Transcription Factors - genetics | Simian Acquired Immunodeficiency Syndrome - blood | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Simian Immunodeficiency Virus - physiology | Host-Pathogen Interactions | Transcription Factors - metabolism | Animals | B-Lymphocytes - immunology | Simian Acquired Immunodeficiency Syndrome - immunology | Germinal Center - virology | Proto-Oncogene Proteins c-maf - metabolism | CD4 Antigens - metabolism | Genetic aspects | Research | Simian immunodeficiency virus | Gene expression | CD4 lymphocytes | Index Medicus | Abridged Index Medicus
Journal Article