X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (134) 134
t-h-17 cells (131) 131
immunology (98) 98
humans (94) 94
animals (79) 79
mice (64) 64
growth-factor-beta (63) 63
differentiation (60) 60
tgf-beta (52) 52
female (46) 46
th17 cells (46) 46
th17 cells - immunology (44) 44
ror-gamma-t (43) 43
inflammation (42) 42
cytokines (36) 36
male (35) 35
t cells (35) 35
dendritic cells (30) 30
il-17 (30) 30
interleukin-17 - metabolism (30) 30
mice, inbred c57bl (30) 30
abridged index medicus (29) 29
cd4-positive t-lymphocytes - immunology (29) 29
expression (29) 29
cell differentiation - immunology (28) 28
helper-cells (28) 28
t-lymphocytes, helper-inducer - immunology (28) 28
t17 cells (28) 28
flow cytometry (27) 27
autoimmunity (26) 26
cells, cultured (26) 26
th17 cells - metabolism (26) 26
central-nervous-system (25) 25
t-cells (25) 25
interleukin-17 - immunology (24) 24
mice, knockout (24) 24
article (23) 23
cell biology (23) 23
cell differentiation (23) 23
experimental autoimmune encephalomyelitis (23) 23
human t-h-17 cells (23) 23
th17 cells - cytology (23) 23
adult (22) 22
encephalomyelitis, autoimmune, experimental - immunology (22) 22
multiple-sclerosis (22) 22
th1 cells - immunology (22) 22
hemic and immune systems (20) 20
lymphocytes (20) 20
multiple sclerosis (20) 20
regulatory t-cells (20) 20
th17 (20) 20
ifn-gamma (19) 19
biochemistry & molecular biology (18) 18
chemical and pharmacologic phenomena (18) 18
cytokines - metabolism (18) 18
gene expression (18) 18
interleukin-17 - biosynthesis (18) 18
middle aged (18) 18
t-lymphocytes, regulatory - immunology (18) 18
cd4-positive t-lymphocytes - metabolism (17) 17
in-vivo (17) 17
induction (17) 17
rheumatoid-arthritis (17) 17
transcription factor (17) 17
interleukin 22 (16) 16
transcription factors (16) 16
cutting edge (15) 15
cytokine (15) 15
dendritic cells - immunology (15) 15
lymphocytes t (15) 15
medicine, research & experimental (15) 15
activation (14) 14
encephalomyelitis, autoimmune, experimental - pathology (14) 14
interleukin-17 - genetics (14) 14
cd4-positive t-lymphocytes - cytology (13) 13
cytokines - immunology (13) 13
experimental allergic encephalomyelitis (13) 13
gene-expression (13) 13
helper cells (13) 13
il-21 (13) 13
inflammation - immunology (13) 13
interleukin 17 (13) 13
interleukins (13) 13
receptor (13) 13
t-lymphocytes, helper-inducer - metabolism (13) 13
aryl-hydrocarbon receptor (12) 12
autoimmune diseases (12) 12
t-h-17 (12) 12
cell differentiation - drug effects (11) 11
encephalomyelitis (11) 11
immune system (11) 11
interleukin-17 (11) 11
interleukins - immunology (11) 11
lineage (11) 11
multidisciplinary sciences (11) 11
research (11) 11
rodents (11) 11
signal transduction (11) 11
t-lymphocyte subsets - immunology (11) 11
adoptive transfer (10) 10
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e45680
Accumulating evidence suggests a contribution of T cell-derived IL-17, IL-21 and IL-22 cytokines in skin immune homeostasis as well as inflammatory disorders.... 
INTERLEUKIN 22 | T-H-17 | MULTIDISCIPLINARY SCIENCES | HUMAN LANGERHANS CELLS | DISTINCT | B7 FAMILY | SUBSETS | TH17 POLARIZATION | T-CELLS | BETA | Antigens, CD - immunology | CD8-Positive T-Lymphocytes - cytology | Coculture Techniques | Humans | Interleukin-17 - immunology | B7 Antigens - immunology | Antigens, CD - genetics | Cell Lineage - drug effects | Dermis - drug effects | CD4-Positive T-Lymphocytes - immunology | B7 Antigens - genetics | Interleukins - immunology | Langerhans Cells - immunology | Interleukins - biosynthesis | Dermis - cytology | Langerhans Cells - drug effects | Inducible T-Cell Co-Stimulator Ligand - immunology | CD4-Positive T-Lymphocytes - cytology | Epidermis - drug effects | Interleukin-17 - biosynthesis | Dermis - immunology | Gene Expression Regulation - drug effects | Transforming Growth Factor beta - pharmacology | Epidermis - immunology | Inducible T-Cell Co-Stimulator Ligand - genetics | Signal Transduction - drug effects | Cell Differentiation - drug effects | CD8-Positive T-Lymphocytes - drug effects | Cell Communication - drug effects | Immunologic Memory | CD8-Positive T-Lymphocytes - immunology | CD4-Positive T-Lymphocytes - drug effects | Langerhans Cells - cytology | Cell Lineage - immunology | Epidermis - cytology | Leukocyte migration | Pathogenesis | CD8 antigen | Disorders | Homeostasis | Interleukin 22 | Lymphocytes T | CD14 antigen | CD28 antigen | B7 antigen | Interleukin 4 | Specialization | Lymphocytes | Interleukin 21 | Immunological memory | Memory cells | Psoriasis | Cytokines | Dendritic cells | Secretion | T cell receptors | Interleukin 17 | CD4 antigen | CD1c antigen | Antibiotics | ICOS protein | γ-Interferon | Langerhans cells | Ligands | Skin | Interferon
Journal Article
Biology of Reproduction, ISSN 0006-3363, 04/2010, Volume 82, Issue 4, pp. 698 - 705
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 07/2009, Volume 39, Issue 7, pp. 1716 - 1725
Activation of naive CD8+ T cells with antigen in the absence of skewing cytokines triggers their differentiation into effector CTL, which induces death of... 
EAE | Tc17 | CTL | STAT3 | RORγt | TGF-BETA | INTERFERON-REGULATORY FACTOR-4 | ROR gamma t | IMMUNOLOGY | IL-21 | EFFECTOR | ROR-GAMMA | EOMESODERMIN | GENERATION | DIFFERENTIATION | T-H-17 CELLS | T-CELLS | CD8-Positive T-Lymphocytes - cytology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Gene Expression - drug effects | Granzymes - metabolism | Encephalomyelitis, Autoimmune, Experimental - immunology | Immunoblotting | Green Fluorescent Proteins - genetics | Interferon-gamma - metabolism | Receptors, Retinoic Acid - genetics | Cell Differentiation - genetics | Flow Cytometry | Receptors, Thyroid Hormone - genetics | T-Lymphocytes, Helper-Inducer - immunology | T-Lymphocytes, Helper-Inducer - cytology | CD8-Positive T-Lymphocytes - metabolism | Encephalomyelitis, Autoimmune, Experimental - genetics | Interleukins - pharmacology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Nuclear Receptor Subfamily 1, Group F, Member 3 | T-Lymphocytes, Cytotoxic - immunology | Green Fluorescent Proteins - metabolism | T-Lymphocytes, Helper-Inducer - metabolism | Mice, Inbred C57BL | Receptors, Retinoic Acid - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Receptors, Thyroid Hormone - metabolism | Cell Differentiation - immunology | Interleukin-17 - metabolism | Transforming Growth Factor beta - pharmacology | Animals | Interleukin-6 - pharmacology | T-Lymphocytes, Cytotoxic - metabolism | Cell Differentiation - drug effects | Cell Line, Tumor | Mice | CD8-Positive T-Lymphocytes - immunology | Cytotoxicity, Immunologic - immunology | T-Lymphocytes, Cytotoxic - cytology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 33, pp. 14751 - 14756
In several murine models of autoimmune arthritis, Th17 cells are the dominant initiators of inflammation. In human arthritis the majority of IL-17–secreting... 
T lymphocytes | Synovial fluid | Phenotypes | Cytokines | Juvenile rheumatoid arthritis | Cultured cells | Arthritis | Joint diseases | Joints | T cell antigen receptors | Juvenile | CD161 | RORC2 | RHEUMATOID-ARTHRITIS | GAMMA | MULTIDISCIPLINARY SCIENCES | JUVENILE IDIOPATHIC ARTHRITIS | CD4(+) T-CELLS | INDUCTION | MEMORY CELLS | IL-17 | juvenile | INFLAMED JOINTS | T-H-17 CELLS | LINEAGE | Receptors, CCR6 - immunology | Humans | Interleukin-17 - immunology | Molecular Sequence Data | T-Box Domain Proteins - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - immunology | Base Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Receptors, CCR6 - metabolism | Interferon-gamma - genetics | Child | Cell Lineage - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Transforming Growth Factor beta - immunology | Amino Acid Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | NK Cell Lectin-Like Receptor Subfamily B - metabolism | Interleukin-17 - genetics | Interleukin-12 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Arthritis, Juvenile - genetics | NK Cell Lectin-Like Receptor Subfamily B - immunology | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Arthritis, Juvenile - metabolism | Interleukin-17 - metabolism | NK Cell Lectin-Like Receptor Subfamily B - genetics | Arthritis, Juvenile - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Receptors, CCR6 - genetics | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | Autoimmunity | Research | Adaptation (Physiology) | Animal models | Transcription factors | Helper cells | Interleukin 12 | Interleukin 23 | Inflammation | Lymphocytes T | Transforming growth factor- beta | double prime T-cell receptor | CCR6 protein | Interleukin 17 | Plasticity | Children | Biological Sciences
Journal Article
European Journal of Immunology, ISSN 0014-2980, 11/2010, Volume 40, Issue 11, pp. 3028 - 3042
T‐cell function is dependent on store‐operated Ca2+ influx that is activated by the stromal interaction molecules (STIM) 1 and 2. We show that mice with... 
EAE | CRAC channel | Stromal interaction molecule 1 | Calcium | Th17 | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | OPERATED CA2+ ENTRY | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | HELPER-CELLS | IMMUNOLOGY | T(H)17 CELLS | MULTIPLE-SCLEROSIS | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | T-H-17 CELLS | KEY REGULATORS | Th1 Cells - pathology | Inflammation - pathology | T-Lymphocytes, Regulatory - metabolism | Receptors, Interleukin - biosynthesis | Encephalomyelitis, Autoimmune, Experimental - metabolism | Membrane Glycoproteins - metabolism | Calcium - metabolism | Calcium - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Th1 Cells - immunology | Cell Movement - genetics | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Cell Movement - immunology | Th1 Cells - metabolism | Inflammation - metabolism | Th17 Cells - metabolism | Gene Deletion | Encephalomyelitis, Autoimmune, Experimental - genetics | Membrane Glycoproteins - immunology | Multiple Sclerosis - metabolism | Th17 Cells - pathology | Stromal Interaction Molecule 1 | Encephalomyelitis, Autoimmune, Experimental - pathology | Calcium Channels | Receptors, Interleukin - immunology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Stromal Interaction Molecule 2 | Multiple Sclerosis - genetics | Inflammation - immunology | Receptors, Interleukin - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Inflammation - genetics | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Th17 Cells - immunology | Mice | Homeostasis | T cell receptors | Lymphocytes | Chemokines | calcium | STIM1 | autoimmunity | STIM2 | Ca2 | T cells | CRAC
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 04/2012, Volume 168, Issue 1, pp. 68 - 74
Journal Article
The EMBO Journal, ISSN 0261-4189, 01/2016, Volume 35, Issue 1, pp. 89 - 101
Journal Article
Clinical science (London, England : 1979), ISSN 0143-5221, 08/2015, Volume 129, Issue 4, pp. 291 - 303
Multiple sclerosis (MS) is a chronic disease of the central nervous system (CNS) characterized by persistent inflammation orchestrated by cluster of... 
T helper cells | Multiple sclerosis | Interleukin-9 | Cerebrospinal fluid | Interleukin-17 | Dendritic cells | MEDICINE, RESEARCH & EXPERIMENTAL | OPTICAL COHERENCE TOMOGRAPHY | multiple sclerosis | IFN-GAMMA | interleukin-9 | interleukin-17 | GROWTH-FACTOR-BETA | INTERFERON | dendritic cells | T(H)17 CELLS | IMMUNE-RESPONSES | cerebrospinal fluid | DIFFERENTIATION | T-H-17 CELLS | STATUS SCALE EDSS | TH17 CELLS | Phosphorylation | Dendritic Cells - immunology | Humans | Middle Aged | Interferon Regulatory Factors - metabolism | Male | Multiple Sclerosis, Relapsing-Remitting - diagnosis | Case-Control Studies | STAT5 Transcription Factor - metabolism | STAT1 Transcription Factor - metabolism | Time Factors | T-Lymphocytes, Helper-Inducer - immunology | Inflammation Mediators - metabolism | Interleukin-9 - metabolism | Adult | Dendritic Cells - drug effects | Female | Dendritic Cells - metabolism | Multiple Sclerosis, Relapsing-Remitting - metabolism | Disability Evaluation | Severity of Illness Index | Multiple Sclerosis, Relapsing-Remitting - immunology | T-Lymphocytes, Helper-Inducer - metabolism | Signal Transduction | T-Lymphocytes, Helper-Inducer - classification | Tomography, Optical Coherence | Cells, Cultured | Cell Communication | Imidazoles - pharmacology | Disease Progression | Interleukin-17 - metabolism | Magnetic Resonance Imaging | Phenotype | Interleukin-9 - cerebrospinal fluid | Multiple Sclerosis, Relapsing-Remitting - cerebrospinal fluid
Journal Article
Journal Article