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Science, ISSN 0036-8075, 2/2003, Volume 299, Issue 5609, pp. 1057 - 1061
Regulatory T cells engage in the maintenance of immunological self-tolerance by actively suppressing self-reactive lymphocytes. Little is known, however, about... 
T lymphocytes | Molecules | Cell growth | Inflammatory bowel diseases | B lymphocytes | Antigen presenting cells | Cultured cells | Reports | Autoimmune diseases | Cells | Gastritis | MUTANT | HOMEOSTASIS | LYMPHOCYTE-ASSOCIATED ANTIGEN-4 | ENTEROPATHY | CD25(+) | MULTIDISCIPLINARY SCIENCES | IMMUNOLOGICAL SELF-TOLERANCE | INDUCTION | AUTOIMMUNE-DISEASE | SCURFY MOUSE | T-Lymphocyte Subsets - immunology | T-Lymphocytes, Regulatory - metabolism | T-Lymphocyte Subsets - cytology | Inflammatory Bowel Diseases - immunology | Gastritis - prevention & control | Recombinant Fusion Proteins - metabolism | T-Lymphocytes, Regulatory - immunology | Antigens, CD - analysis | CD4-Positive T-Lymphocytes - immunology | DNA-Binding Proteins - metabolism | Receptors, Antigen, T-Cell - immunology | Thymus Gland - metabolism | Inflammatory Bowel Diseases - prevention & control | Transduction, Genetic | Lymphocyte Activation | Autoimmune Diseases - immunology | Self Tolerance | Thymus Gland - cytology | Immune Tolerance | Autoimmune Diseases - prevention & control | Mice, Transgenic | DNA-Binding Proteins - genetics | Mice, SCID | Animals | Gastritis - immunology | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | Mutation | Forkhead Transcription Factors | Cytokines - biosynthesis | Receptors, Interleukin-2 - analysis | Genetics | Control | Genetic aspects | Research | T cells | Cellular control mechanisms | Immunology | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2013, Volume 14, Issue 2, pp. 162 - 171
Signaling through the G protein-coupled receptors for the complement fragments C3a and C5a (C3aR and C5aR, respectively) by dendritic cells and CD4(+) cells... 
Forkhead Transcription Factors - immunology | Cell Communication - immunology | T-Lymphocytes, Regulatory - metabolism | TOR Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - metabolism | Complement C5a - metabolism | Receptors, Complement - metabolism | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Transforming Growth Factor beta1 - immunology | Forkhead Transcription Factors - metabolism | Class Ib Phosphatidylinositol 3-Kinase - metabolism | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Receptor, Anaphylatoxin C5a - immunology | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Complement C3a - metabolism | Interleukin-6 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Receptors, Chemokine - metabolism | Gene Expression Regulation | Mice, Transgenic | Receptors, Chemokine - immunology | Receptor Cross-Talk - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Receptors, Complement - immunology | Cyclic AMP-Dependent Protein Kinases - immunology | Interleukin-6 - immunology | Mice | Complement C3a - immunology | Complement C5a - immunology | Class Ib Phosphatidylinositol 3-Kinase - immunology | Interleukin-10 - immunology | Receptor, Anaphylatoxin C5a - metabolism | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 10/2010, Volume 207, Issue 11, pp. 2331 - 2341
Foxp3-expressing regulatory T (T reg) cells have been implicated in parasite-driven inhibition of host immunity during chronic infection. We addressed whether... 
B-CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | TH2 RESPONSES | PROTECTIVE IMMUNITY | TRANSCRIPTION FACTOR FOXP3 | IN-VIVO | AIRWAY INFLAMMATION | NEMATODE INFECTION | POLYGYRUS INFECTION | IMMUNOLOGY | ORAL ANTIGEN | AUTOIMMUNE-DISEASE | Receptors, Transforming Growth Factor beta - genetics | Strongylida Infections - metabolism | Receptors, Transforming Growth Factor beta - immunology | Smad3 Protein - immunology | Nematospiroides dubius - metabolism | Th2 Cells - immunology | T-Lymphocytes, Regulatory - immunology | Th1 Cells - metabolism | Host-Parasite Interactions - genetics | Phosphorylation - genetics | Smad2 Protein - genetics | Dioxoles - pharmacology | Phosphorylation - immunology | Protein-Serine-Threonine Kinases - metabolism | Smad2 Protein - metabolism | Mice, Transgenic | Signal Transduction - genetics | Antigens, Helminth - immunology | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Signal Transduction - drug effects | Mice | Mice, Inbred BALB C | Chronic Disease | Forkhead Transcription Factors - immunology | T-Lymphocytes, Regulatory - metabolism | Antigens, Helminth - metabolism | Strongylida Infections - genetics | Smad3 Protein - metabolism | Th1 Cells - immunology | Nematospiroides dubius - immunology | Signal Transduction - immunology | Smad3 Protein - genetics | Strongylida Infections - immunology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Benzamides - pharmacology | Transforming Growth Factor beta - immunology | Forkhead Transcription Factors - biosynthesis | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Protein-Serine-Threonine Kinases - genetics | Host-Parasite Interactions - immunology | Forkhead Transcription Factors - genetics | Th2 Cells - metabolism | Gene Expression Regulation - drug effects | Host-Parasite Interactions - drug effects | Smad2 Protein - immunology | Animals | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Protein-Serine-Threonine Kinases - immunology | Cell Proliferation - drug effects | Transforming Growth Factor beta - metabolism | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 27, pp. 12204 - 12209
To maintain intestinal health, the immune system must faithfully respond to antigens from pathogenic microbes while limiting reactions to self-molecules. The... 
T lymphocytes | Microbiota | Inflammatory bowel diseases | Germ free animals | Animals | Germ free state | Bacteria | Mice | Inflammation | Colitis | Inflammatory bowel disease | Polysaccharide A | Bacteroides fragilis | Toll-like receptor | Interleukin 10 | Forkhead Transcription Factors - immunology | Immunity, Mucosal - immunology | T-Lymphocytes, Regulatory - metabolism | Humans | Intestines - metabolism | Adoptive Transfer | Trinitrobenzenesulfonic Acid | Lipopolysaccharides - immunology | Intestines - immunology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Forkhead Transcription Factors - metabolism | Immune Tolerance - immunology | Colitis - chemically induced | Interleukin-10 - metabolism | Colitis - immunology | Bacteroides fragilis - immunology | Bacteroides fragilis - physiology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Forkhead Transcription Factors - genetics | Mice, Knockout | Host-Pathogen Interactions | T-Lymphocytes, Regulatory - drug effects | Intestines - microbiology | CD4-Positive T-Lymphocytes - transplantation | Lipopolysaccharides - pharmacology | Mice, Inbred BALB C | Colitis - prevention & control | Interleukin-10 - immunology | Antigens | Molecules | Signal transduction | Immunology | Lymphocytes | Gram-negative bacteria | Immune system | Index Medicus | Biological Sciences | inflammatory bowel disease | interleukin 10 | polysaccharide A
Journal Article