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Immunity, ISSN 1074-7613, 08/2009, Volume 31, Issue 2, pp. 245 - 258
Effective clearance of apoptotic cells by macrophages is essential for immune homeostasis. The transcriptional pathways that allow macrophages to sense and... 
MOLIMMUNO | CELLIMMUNO | SIGNALING PATHWAYS | IN-VITRO | PHAGOCYTOSIS | PHOSPHATIDYLSERINE RECEPTOR | LIPID-METABOLISM | MER TYROSINE KINASE | CHOLESTEROL EFFLUX | TAM RECEPTORS | IMMUNOLOGY | LIVER-X-RECEPTORS | MICE LACKING | Spleen - immunology | Receptor Protein-Tyrosine Kinases - immunology | Signal Transduction - immunology | Receptors, Cytoplasmic and Nuclear - immunology | DNA-Binding Proteins - agonists | Autoimmunity - immunology | Immune Tolerance - immunology | Liver X Receptors | c-Mer Tyrosine Kinase | Proto-Oncogene Proteins - immunology | Orphan Nuclear Receptors | Autoimmune Diseases - metabolism | Autoimmune Diseases - pathology | Macrophages - immunology | Proto-Oncogene Proteins - metabolism | DNA-Binding Proteins - immunology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Autoimmune Diseases - immunology | Phagocytosis - immunology | Receptors, Cytoplasmic and Nuclear - agonists | Receptors, Cytoplasmic and Nuclear - genetics | Spleen - cytology | Macrophages - cytology | DNA-Binding Proteins - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Mice, Knockout | Animals | Apoptosis - immunology | Spleen - metabolism | Mice | Tyrosine | Autoimmunity | Autoantibodies | Analysis | Development and progression | Macrophages | Apoptosis | Studies | Flow cytometry | Statistical analysis | Rodents | Software | Gene expression | Variance analysis | Cholesterol | Immune system
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 09/2019, Volume 106, Issue 3, pp. 695 - 701
Arboviruses have been a huge threat for human health since the discovery of yellow fever virus in 1901. Arboviruses are arthropod born viruses, mainly... 
TAM | TIM | integrins | viral entry receptors | Flavivirus | PHOSPHATIDYLSERINE RECEPTORS | ENTRY | PROTEIN | TAM RECEPTORS | IMMUNOLOGY | ENDOCYTOSIS | CELL BIOLOGY | DENGUE-VIRUS | AXL | ENDOTHELIAL-CELLS | HEMATOLOGY | ZIKA VIRUS-INFECTION
Journal Article
CARDIOVASCULAR RESEARCH, ISSN 0008-6363, 07/2019, Volume 115, Issue 8, pp. 1286 - 1295
The TAM receptors are a distinct family of three receptor tyrosine kinases, namely Tyro3, Axl, and MerTK. Since their discovery in the early 1990s, they have... 
NATURAL-KILLER-CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | DENDRITIC CELLS | Tyro3 | Cardiovascular disease | PHOSPHATE-INDUCED CALCIFICATION | VASCULAR SMOOTH-MUSCLE | TAM receptors | Axl | MERTK RECEPTOR | TYROSINE KINASE RECEPTOR | APOPTOTIC CELL ACCUMULATION | PROTEIN-S | INFLAMMATORY RESPONSE | MerTK | ARREST-SPECIFIC GENE | Invited Reviews | Editor's Choice
Journal Article
Annals of the New York Academy of Sciences, ISSN 0077-8923, 10/2010, Volume 1209, Issue 1, pp. 23 - 29
Journal Article
Circulation Research, ISSN 0009-7330, 09/2017, Volume 121, Issue 8, pp. 930 - 940
RATIONALE:Clinical benefits of reperfusion after myocardial infarction are offset by maladaptive innate immune cell function, and therapeutic interventions are... 
Efferocytosis | Inflammation | Ischemia reperfusion injury | Phagocytosis | Macrophage | phagocytosis | CLEARANCE | CARDIAC & CARDIOVASCULAR SYSTEMS | efferocytosis | macrophage | ischemia reperfusion injury | MECHANISMS | TAM RECEPTORS | ACUTE-INFLAMMATION | INFARCTION | inflammation | APOPTOTIC CELLS | DISEASE | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | HEMATOLOGY | RECEPTOR TYROSINE KINASE | Receptors, CCR2 - genetics | Receptors, CCR2 - immunology | Humans | Middle Aged | Receptor Protein-Tyrosine Kinases - immunology | Male | Monocytes - immunology | Myocardial Reperfusion Injury - pathology | Time Factors | Proteolysis | Disease Models, Animal | Myocardial Reperfusion Injury - immunology | Genetic Predisposition to Disease | Macrophages - pathology | Signal Transduction | Proto-Oncogene Proteins - deficiency | Receptor Protein-Tyrosine Kinases - metabolism | Myocardial Reperfusion Injury - physiopathology | Macrophages - enzymology | Mice, Knockout | Monocytes - enzymology | Phenotype | Receptors, CCR2 - metabolism | Histocompatibility Antigens Class II - immunology | ST Elevation Myocardial Infarction - enzymology | Mice | Myocardium - immunology | Myocardial Reperfusion Injury - enzymology | c-Mer Tyrosine Kinase | Proto-Oncogene Proteins - immunology | Female | Histocompatibility Antigens Class II - metabolism | Macrophages - immunology | Cytokines - immunology | Proto-Oncogene Proteins - metabolism | ST Elevation Myocardial Infarction - immunology | Receptor Protein-Tyrosine Kinases - deficiency | Cytokines - metabolism | Mice, Inbred C57BL | Myocardium - pathology | Proto-Oncogene Proteins - genetics | Immunity, Innate | ST Elevation Myocardial Infarction - physiopathology | Myocardium - enzymology | Animals | Receptor Protein-Tyrosine Kinases - genetics | ST Elevation Myocardial Infarction - pathology | Apoptosis | Heart | Myocardial infarction | Tyrosine | CC chemokine receptors | Heart attacks | Cytokines | Therapeutic applications | Transforming growth factor | Immune clearance | Wounds | Myocardial ischemia | Macrophages | Phagocytes | Reperfusion | Major histocompatibility complex | Ischemia | CCR2 protein | Rodents | Histocompatibility | Heart diseases | Protein-tyrosine kinase | Monocyte chemoattractant protein 1
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 5/2012, Volume 69, Issue 9, pp. 1391 - 1414
Ample clinical and preclinical evidence indicates that macrophages interact with tumor cells as well as with virtually all populations of host cells present in... 
Life Sciences | Biochemistry, general | Life Sciences, general | Tumor-macrophage crosstalk | TAMR (Tyro3, Axl, Mer receptors) | Inflammation | Gas6 | Biomedicine general | TAM (Tumor-associated macrophages) | Cell Biology | Cancer | EXPRESSION ANALYSIS | CANCER-RELATED INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ARREST-SPECIFIC GENE-6 | NECROSIS-FACTOR-ALPHA | ACUTE MYELOID-LEUKEMIA | CELL BIOLOGY | ACUTE LYMPHOBLASTIC-LEUKEMIA | GROWTH-ARREST | REGULATORY T-CELLS | PROTEIN-S | NF-KAPPA-B | Macrophages - physiology | Leukocytes - pathology | Macrophages - classification | Prognosis | Macrophages - pathology | Fibroblasts - physiology | Humans | Receptor Protein-Tyrosine Kinases - physiology | Male | Leukocytes - physiology | Tumor Microenvironment - physiology | Fibroblasts - pathology | Intercellular Signaling Peptides and Proteins - physiology | Protein S - physiology | Models, Biological | Female | Ligands | Neoplasms - physiopathology | Endothelial Cells - pathology | Neoplasms - pathology | Endothelial Cells - physiology | Macrophages - immunology | Tyrosine | Care and treatment | Genes | Macrophages | Endothelium | Tumors | Kinases | Molecular biology | Cell proliferation | Tumor cells | Malignancy | Leukocytes | Endothelial cells | Axl protein | Reviews | Fibroblasts | Protein-tyrosine kinase receptors | Microenvironments
Journal Article