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Oncogene, ISSN 0950-9232, 07/2012, Volume 31, Issue 27, pp. 3223 - 3234
The role of estrogen receptor-alpha (ER) in breast cancer development, and as a primary clinical marker for breast cancer prognosis, has been well documented.... 
Twist | breast cancer | histone deacetylation | hormone resistance | ER transcriptional regulation | methylation | ACTIVATION | PROTEIN | DNA METHYLATION | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | EPITHELIAL-CELL LINES | CHROMOSOMAL INSTABILITY | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | MUTATIONS | TRANSCRIPTIONAL REGULATION | AROMATASE INHIBITORS | EXPRESSION | Chromatin - metabolism | RNA, Small Interfering - genetics | Humans | Capillary Permeability - drug effects | Protein Transport - drug effects | Twist-Related Protein 1 - deficiency | Promoter Regions, Genetic - drug effects | Promoter Regions, Genetic - genetics | Valproic Acid - pharmacology | Breast Neoplasms - metabolism | DNA (Cytosine-5-)-Methyltransferases - metabolism | Nuclear Proteins - deficiency | Female | Histone Deacetylase 1 - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Estrogens - metabolism | Nuclear Proteins - genetics | Chromatin - drug effects | E-Box Elements - drug effects | Nuclear Proteins - metabolism | Breast Neoplasms - blood supply | Down-Regulation - drug effects | E-Box Elements - genetics | Breast Neoplasms - drug therapy | Down-Regulation - genetics | Azacitidine - pharmacology | Acetylation - drug effects | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Breast Neoplasms - pathology | Tamoxifen - pharmacology | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Mice | Histones - metabolism | Twist-Related Protein 1 - metabolism | Chromatin - genetics | DNA Methylation - drug effects | Index Medicus | Cell proliferation | selective estrogen receptor modulators | Azacytidine | Histone deacetylase | Chromatin | Immunoprecipitation | Prognosis | Transcription | Estrogen receptors | Breast cancer | Hormones | Permeability | Tamoxifen | Valproic acid | Condensation | Promoters | Fulvestrant | Magnetic resonance imaging | Deacetylation | DNA methylation | DNA methyltransferase | Surface area | Tumors
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2001, Volume 276, Issue 13, pp. 9817 - 9824
Estrogen receptors (ERs) mediate most of the biological effects of estrogen in mammary and uterine epithelial cells by binding to estrogen response elements in... 
SIGNAL-TRANSDUCTION | BREAST-CANCER CELLS | TRANSCRIPTIONAL ACTIVATION | PHOSPHORYLATION | 3-KINASE | TAMOXIFEN | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | KINASE-B | BINDING PROTEIN | BCL-2 EXPRESSION | Up-Regulation | Phosphorylation | Receptors, Estrogen - metabolism | Humans | Protein-Serine-Threonine Kinases | Chloramphenicol O-Acetyltransferase - metabolism | Drug Resistance, Neoplasm | Estrogen Antagonists - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Growth Differentiation Factor 15 | Breast Neoplasms - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Estrogen Receptor alpha | Transfection | PTEN Phosphohydrolase | Time Factors | Phosphoric Monoester Hydrolases - pharmacology | Trefoil Factor-1 | Tumor Cells, Cultured | Chromones - pharmacology | Estrogens - metabolism | Proto-Oncogene Proteins - metabolism | Antineoplastic Agents, Hormonal - pharmacology | Cytokines - metabolism | Signal Transduction | Cell Survival | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | Breast Neoplasms - drug therapy | Plasmids - metabolism | Serine - metabolism | Blotting, Western | Cell Division - drug effects | Estrogen Receptor Modulators - pharmacology | Precipitin Tests | Proto-Oncogene Proteins c-akt | Phosphates - metabolism | Animals | Proteins - metabolism | Breast Neoplasms - pathology | Models, Biological | Tamoxifen - pharmacology | Enzyme Activation | COS Cells | Tumor Suppressor Proteins | Apoptosis | AKT protein | ApS2 gene | Bcl-2 protein | macrophage inhibitory cytokine 1 | Index Medicus
Journal Article
The Journal of Pathology, ISSN 0022-3417, 08/2014, Volume 233, Issue 4, pp. 368 - 379
Journal Article
Oncogene, ISSN 0950-9232, 03/2011, Volume 30, Issue 9, pp. 1082 - 1097
Fulvestrant is a selective estrogen receptor downregulator (SERD) and highly effective antagonist to hormone-sensitive breast cancers following failure of... 
microRNAs | breast cancer | antiestrogen | drug resistance | fulvestrant | ACTIVATED PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TAMOXIFEN RESISTANCE | DOWN-REGULATION | CELL-LINES | CELL BIOLOGY | ENDOCRINE RESISTANCE | GROWTH-FACTOR-BETA | MESSENGER-RNA | ONCOLOGY | ESTROGEN-RECEPTOR-ALPHA | GENETICS & HEREDITY | GENE-EXPRESSION | TUMOR-SUPPRESSOR | Up-Regulation | Estradiol - analogs & derivatives | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Humans | Drug Resistance, Neoplasm | Estrogen Antagonists - pharmacology | MicroRNAs - metabolism | Signal Transduction - genetics | Breast Neoplasms - drug therapy | beta Catenin - genetics | Breast Neoplasms - metabolism | Breast Neoplasms - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Estrogen Receptor alpha - metabolism | Female | Antineoplastic Agents - pharmacology | MicroRNAs - genetics | Gene Expression Regulation, Neoplastic - drug effects | Selective Estrogen Receptor Modulators - pharmacology | Transforming Growth Factor beta - metabolism | Estradiol - pharmacology | MicroRNA | Physiological aspects | Breast cancer | Genetic aspects | Research | Drug therapy | Drug resistance | Health aspects | Fulvestrant | Proteins | Signal transduction | Cellular biology | Antagonist drugs | Ribonucleic acid--RNA | Gene expression | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 497, Issue 7447, pp. 108 - 112
Cancers acquire resistance to systemic treatment as a result of clonal evolution and selection(1,2). Repeat biopsies to study genomic evolution as a result of... 
HETEROGENEITY | LUNG-CANCER | ACTIVATION | EVOLUTION | VARIANTS | AXL | MULTIDISCIPLINARY SCIENCES | TAMOXIFEN RESISTANCE | TUMOR-CELLS | MUTATIONS | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Genomics | Humans | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Antineoplastic Agents - therapeutic use | Ovarian Neoplasms - genetics | Neoplasms - genetics | DNA Mutational Analysis | Female | Antineoplastic Agents - pharmacology | DNA, Neoplasm - analysis | Ovarian Neoplasms - drug therapy | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Intercellular Signaling Peptides and Proteins - genetics | Breast Neoplasms - drug therapy | Genome, Human - genetics | Neoplasms - drug therapy | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Exome - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Breast Neoplasms - pathology | Retinoblastoma Protein - genetics | Plasma - chemistry | Alleles | Carcinoma, Non-Small-Cell Lung - drug therapy | DNA, Neoplasm - genetics | Neoplasms - pathology | Mediator Complex Subunit 1 - genetics | Drug Resistance, Neoplasm - drug effects | Evolution, Molecular | Antimitotic agents | Care and treatment | Physiological aspects | Dosage and administration | Genetic aspects | Research | Nucleotide sequencing | Drug resistance | Antineoplastic agents | DNA sequencing | Blood plasma | Cancer | Plasma | Biopsy | Breast cancer | Genomes | Mutation | Patients | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article
细胞研究:英文版, ISSN 1001-0602, 2016, Volume 26, Issue 6, pp. 655 - 673
Journal Article
Cell Cycle, ISSN 1538-4101, 08/2011, Volume 10, Issue 15, pp. 2521 - 2528
Journal Article
Journal Article