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Free Radical Biology and Medicine, ISSN 0891-5849, 06/2014, Volume 71, pp. 390 - 401
β-Amyloid (Aβ)-induced toxicity and oxidative stress have been postulated to play critical roles in the pathogenic mechanism of Alzheimer disease (AD). We... 
Cytochrome c oxidase | Oxidative stress | Free radicals | Alzheimer disease | Mitochondria-targeted antioxidants | β-Amyloid peptides | Complex IV | Electron transport chain | Caenorhabditis elegans | MitoQ | Healthspan | Cardiolipin | Lifespan | PROTEIN | MOUSE MODEL | ENDOCRINOLOGY & METABOLISM | beta-Amyloid peptides | DYSFUNCTION | STRESS | BRAIN | OXIDATIVE DNA-DAMAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | BETA-AMYLOID PEPTIDE | CYTOCHROME-C-OXIDASE | REACTIVE OXYGEN | IN-VIVO | Mitochondria - enzymology | Protein Carbonylation | Reactive Oxygen Species - metabolism | Oxidative Stress | Longevity - drug effects | Humans | Alzheimer Disease - pathology | Ubiquinone - pharmacology | Mitochondrial Membranes - drug effects | Amyloid beta-Peptides - genetics | Adenosine Triphosphate - metabolism | Organophosphorus Compounds - pharmacology | Transgenes | Disease Models, Animal | Gene Expression | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Amyloid beta-Peptides - biosynthesis | Ubiquinone - analogs & derivatives | Oxygen Consumption | Electron Transport Chain Complex Proteins - agonists | Antioxidants - pharmacology | Mitochondria - drug effects | Mitochondria - pathology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Caenorhabditis elegans - drug effects | Electron Transport Chain Complex Proteins - metabolism | Alzheimer Disease - metabolism | Alzheimer Disease - genetics | Antioxidants | Membrane lipids | Enzymes | Developmental biology | Analysis | Fluorescence | Genetic engineering | Mitochondrial DNA | Paralysis | Alzheimer's disease | Index Medicus
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2010, Volume 49, Issue 5, pp. 748 - 756
The goal of this study was to assess mitochondrial function and ROS production in an experimental model of cocaine-induced cardiac dysfunction. We hypothesized... 
Heart | Interfibrillar mitochondria | Reactive oxygen species | Mitochondria | Free radicals | MitoQ | Cocaine | Cardiac dysfunction | OXIDATIVE STRESS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | MUSCLE | PATHOPHYSIOLOGY | ISCHEMIA-REPERFUSION INJURY | HYPERTROPHY | OXIDASE | ENDOCRINOLOGY & METABOLISM | RAT-HEART | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Heart Diseases - metabolism | Rats, Wistar | Mitochondria, Heart - pathology | Mitochondrial Diseases - metabolism | Heart Diseases - prevention & control | Male | Mitochondria, Heart - drug effects | Molecular Targeted Therapy | Oxygen Consumption - physiology | Ubiquinone - pharmacology | Mitochondrial Diseases - complications | Cocaine-Related Disorders - metabolism | Organophosphorus Compounds - pharmacology | Ubiquinone - therapeutic use | Drug Evaluation, Preclinical | Heart Diseases - chemically induced | Cocaine-Related Disorders - etiology | Disease Susceptibility | Ubiquinone - analogs & derivatives | Organophosphorus Compounds - therapeutic use | Rats | Antioxidants - therapeutic use | Heart Diseases - etiology | Animals | Cocaine-Related Disorders - prevention & control | Antioxidants | Cytochrome b | Analysis | Albumin | Mitochondrial DNA | Free radicals (Chemistry) | Organic chemistry | Chemical Sciences
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 05/2019, Volume 370, pp. 78 - 92
Mitochondrial dysfunctions induced by oxidative stress could play a pivotal role in the development of testicular damage and degeneration, leading to impaired... 
Blood-Testis Barrier | MitoQ | Oxidative Stress | Nrf2/Keap1 | Pharmacokinetics | Mitochondrial Targeting | SPERMATOGENESIS | BARRIER | MALE-INFERTILITY | TARGETED ANTIOXIDANT MITOQ | REACTIVE OXYGEN | SIGNALING PATHWAY | PHARMACOLOGY & PHARMACY | TOXICOLOGY | DYSFUNCTION | STRESS | TRIPTOLIDE | MOLECULAR-MECHANISMS | Proteins | Antioxidants | Testosterone | Analysis | Genes
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 09/2018, Volume 1864, Issue 9, pp. 2859 - 2870
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 6631 - 10
To investigate the effect of maternal MitoQ treatment on renal disorders caused by maternal cigarette smoke exposure (SE). We have demonstrated that maternal... 
PATHOGENESIS | TARGETED ANTIOXIDANT MITOQ | MALONDIALDEHYDE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DISEASE | CIGARETTE-SMOKE | DYSFUNCTION | KIDNEY | EXPOSURE | AGE | Oxidative stress | Reactive oxygen species | Tobacco smoke | Kidneys | Lactation | Body weight | Gestation | Pregnancy | Antioxidants | Progeny | Mitochondria | Supplementation | Cigarette smoke | Smoking
Journal Article
Journal Article
Nature Reviews Neuroscience, ISSN 1471-003X, 02/2018, Volume 19, Issue 2, pp. 63 - 80
Synapses enable neurons to communicate with each other and are therefore a prerequisite for normal brain function. Presynaptically, this communication requires... 
MOTOR-NERVE TERMINALS | PRIMARY CORTICAL-NEURONS | SYSTEM IN-VIVO | AMYLOID BETA-PEPTIDE | TARGETED ANTIOXIDANT MITOQ | SYNAPTIC VESICLE ENDOCYTOSIS | FAST AXONAL-TRANSPORT | DYNAMIN-RELATED PROTEIN-1 | AMYOTROPHIC-LATERAL-SCLEROSIS | ACTIVITY-DEPENDENT REGULATION | NEUROSCIENCES | Presynapse | Homeostasis | Calcium buffering | Mitochondria
Journal Article