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1. Full Text The transcriptional co-activator TAZ is a potent mediator of alveolar rhabdomyosarcoma tumorigenesis
by Deel, Michael D and Slemmons, Katherine K and Hinson, Ashley R and Genadry, Katia C and Burgess, Breanne A and Crose, Lisa E.S and Kuprasertkul, Nina and Oristian, Kristianne M and Bentley, Rex C and Linardic, Corinne M
Clinical cancer research : an official journal of the American Association for Cancer Research, ISSN 1078-0432, 6/2018, Volume 24, Issue 11, pp. 2616 - 2630
WWTR1 | PAX3-FOXO1 | Hippo | TAZ | rhabdomyosarcoma
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2. Full Text Aerobic glycolysis tunes YAP/TAZ transcriptional activity
by Enzo, Elena and Santinon, Giulia and Pocaterra, Arianna and Aragona, Mariaceleste and Bresolin, Silvia and Forcato, Mattia and Grifoni, Daniela and Pession, Annalisa and Zanconato, Francesca and Guzzo, Giulia and Bicciato, Silvio and Dupont, Sirio
The EMBO Journal, ISSN 0261-4189, 05/2015, Volume 34, Issue 10, pp. 1349 - 1370
Increased glucose metabolism and reprogramming toward aerobic glycolysis are a hallmark of cancer cells, meeting their metabolic needs for sustained cell... 
Hippo pathway | TEAD | YAP/TAZ | aerobic glycolysis | glucose metabolism | TISSUE-GROWTH | DROSOPHILA HOMOLOG | COMPLEX | EMERGING ROLES | HIPPO SIGNALING PATHWAY | BREAST-CANCER METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | YAP | CELL BIOLOGY | GLUCOSE | CONTACT INHIBITION | CELL-METABOLISM | Immunoprecipitation | Drosophila | Humans | Phosphoproteins - metabolism | Glycolysis - genetics | Transcription Factors - metabolism | Glycolysis - physiology | Animals | Chromatin Immunoprecipitation | Cell Line, Tumor | Bacteria, Aerobic - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Real-Time Polymerase Chain Reaction | Cell growth | Transcription factors | Breast cancer | Metabolism | Gene expression | Index Medicus | TAZ
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3. Full Text STARD13-correlated ceRNA network-directed inhibition on YAP/TAZ activity suppresses stemness of breast cancer via co-regulating Hippo and Rho-GTPase/F-actin signaling
by Zheng, Lufeng and Xiang, Chenxi and Li, Xiaoman and Guo, Qianqian and Gao, Lanlan and Ni, Haiwei and Xia, Yufeng and Xi, Tao
Journal of Hematology and Oncology, ISSN 1756-8722, 05/2018, Volume 11, Issue 1, pp. 72 - 72
Background: Targeting cancer stem cells is critical for suppressing cancer progression and recurrence. Finding novel markers or related pathways could help... 
STARD13 ceRNA YAP/TAZ Hippo F-actin CSC breast cancer | ceRNA | CELLS | STARD13 | METASTASIS | Hippo | CSC breast cancer | MESENCHYMAL TRANSITION | TAZ | COMPETING ENDOGENOUS RNA | CYP4Z1 | F-actin | LONG NONCODING RNA | ONCOLOGY | YAP/TAZ | GASTRIC-CANCER | HEMATOLOGY | YAP PATHWAY | EXPRESSION | Care and treatment | Actin | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Cellular signal transduction | Research | Transcription | Chemoresistance | Genomes | Metastasis | Gene expression | Kinases | Guanine nucleotide-binding protein | Ribonucleic acid--RNA | Cancer therapies | Yes-associated protein | Signal transduction | Chemotherapy | MicroRNAs | Stem cells | Binding sites | Guanosinetriphosphatase | Tumors
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4. Full Text Hyaluronic acid enhances cell migration and invasion via the YAP1/TAZ-RHAMM axis in malignant pleural mesothelioma
by Shigeeda, Wataru and Shibazaki, Masahiko and Yasuhira, Shinji and Masuda, Tomoyuki and Tanita, Tatsuo and Kaneko, Yuka and Sato, Tatsuhiro and Sekido, Yoshitaka and Maesawa, Chihaya
Oncotarget, ISSN 1949-2553, 11/2017, Volume 8, Issue 55, pp. 93729 - 93740
Most malignant mesotheliomas (MPMs) frequently show activated forms of Yes-associated protein 1 (YAP1) and transcriptional co-activator with PDZ-binding motif... 
Mesothelioma | YAP1/TAZ | Migration | Invasion | RHAMM | TRANSCRIPTION | HIPPO PATHWAY | YAP | PROLIFERATION | RECEPTORS CD44 | TAZ | CELL BIOLOGY | BREAST-CANCER CELLS | ORGAN SIZE | invasion | YAP/TAZ | migration | mesothelioma
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5. Full Text Lipophilic statins inhibit YAP nuclear localization, co-activator activity and colony formation in pancreatic cancer cells and prevent the initial stages of pancreatic ductal adenocarcinoma in Kras(G12D) mice
by Hao, F and Xu, QH and Wang, J and Yu, S and Chang, HH and Sinnett-Smith, J and Eibl, G and Rozengurt, E
PLOS ONE, ISSN 1932-6203, 05/2019, Volume 14, Issue 5
We examined the impact of statins on Yes-associated Protein (YAP) localization, phosphorylation and transcriptional activity in human and mouse pancreatic... 
HIGH-FAT | INSULIN-RECEPTOR | PROTEIN-COUPLED RECEPTOR | PATHWAY | YAP/TAZ | MULTIDISCIPLINARY SCIENCES | HIPPO | MUTANT P53 | MEVALONATE | TAZ | SIGNALING SYSTEMS
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6. Full Text Restoration of miR-26b expression partially reverses the cisplatin resistance of NSCLC by targeting tafazzin
by Zang S and Zhao S and Gao X and Li Y and Zhong C and Gao J
OncoTargets and Therapy, ISSN 1178-6930, 09/2019, Volume 12, pp. 7551 - 7560
Shuzhi Zang,1 Shasha Zhao,2 Xinyuan Gao,1 Yunxia Li,3 Chunlei Zhong,3 Jianlian Gao4 1Respiratory Ward 1, The First Affiliated Hospital of Xinxiang Medical... 
NSCLC | miR-26b | cisplatin | TAZ | resistance
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7. Full Text POPX2 is a novel LATS phosphatase that regulates the Hippo pathway
by Rahmat, Muhammad Bakhait and Zhang, Songjing and Koh, Cheng-Gee
Oncotarget, ISSN 1949-2553, 02/2019, Volume 10, Issue 15, pp. 1525 - 1538
The Hippo pathway regulates cell proliferation, survival, apoptosis and differentiation. During carcinogenesis, members of the Hippo pathway are mutated to... 
Hippo pathway | Anchorage-independent growth | LATS1 kinase | YAP/TAZ | POPX2 phosphatase
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8. Full Text Novel insights into endothelial cell malignancies
by Ha, Taekyu and Ohnuki, Hidetaka and Tosato, Giovanna
Oncotarget, ISSN 1949-2553, 12/2018, Volume 9, Issue 101, pp. 37468 - 37470
Angiosarcoma | YAP/TAZ | DLC1 | Endothelial cells | Endothelial cell tumors
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9. Full Text YAP/TAZ-Dependent Reprogramming of Colonic Epithelium Links ECM Remodeling to Tissue Regeneration
by Yui, Shiro and Azzolin, Luca and Maimets, Martti and Pedersen, Marianne Terndrup and Fordham, Robert P and Hansen, Stine L and Larsen, Hjalte L and Guiu, Jordi and Alves, Mariana R.P and Rundsten, Carsten F and Johansen, Jens V and Li, Yuan and Madsen, Chris D and Nakamura, Tetsuya and Watanabe, Mamoru and Nielsen, Ole H and Schweiger, Pawel J and Piccolo, Stefano and Jensen, Kim B
Cell Stem Cell, ISSN 1934-5909, 01/2018, Volume 22, Issue 1, pp. 35 - 49.e7
Tissue regeneration requires dynamic cellular adaptation to the wound environment. It is currently unclear how this is orchestrated at the cellular level and... 
reprogramming | regeneration | YAP/TAZ | intestinal stem cells | mechano-sensing | PROGENITORS | IN-VITRO | REPAIR | INFLAMMATORY-BOWEL-DISEASE | COLORECTAL CANCERS | COLITIS | PATHWAY | STEM-CELL | YAP | DRIVES INTESTINAL REGENERATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Biomarkers - metabolism | Intestinal Mucosa - metabolism | Transcriptional Activation - genetics | Signal Transduction | Humans | Mice, Inbred C57BL | Fetus - metabolism | Extracellular Matrix - metabolism | Phosphoproteins - metabolism | Cellular Reprogramming | Mechanotransduction, Cellular | Regeneration | Animals | Transcription, Genetic | Adaptor Proteins, Signal Transducing - metabolism | Intestinal Mucosa - pathology | Index Medicus | TAZ
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10. Full Text yap1b, a divergent Yap/Taz family member, cooperates with yap1 in survival and morphogenesis via common transcriptional targets
by Vazquez-Marin, J and Gutierrez-Triana, JA and Almuedo-Castillo, M and Buono, L and Gomez-Skarmeta, JL and Mateo, JL and Wittbrodt, J and Martinez-Morales, JR
DEVELOPMENT, ISSN 0950-1991, 07/2019, Volume 146, Issue 13, p. dev173286
Yap1/Taz are well-known Hippo effectors triggering complex transcriptional programs controlling growth, survival and cancer progression. Here, we describe... 
YES-ASSOCIATED PROTEIN | CHROMATIN-BINDING | Chromatin binding | CELL-PROLIFERATION | DEVELOPMENTAL BIOLOGY | TAZ | DROSOPHILA | GENOME | Morphogenesis | DamID-seq | Yap/Taz | Transcriptional activation domain | HIPPO | GROWTH | PROMOTES | Apoptosis
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11. Full Text N-cadherin adhesive interactions modulate matrix mechanosensing and fate commitment of mesenchymal stem cells
by Cosgrove, Brian D and Mui, Keeley L and Driscoll, Tristan P and Caliari, Steven R and Mehta, Kush D and Assoian, Richard K and Burdick, Jason A and Mauck, Robert L
Nature Materials, ISSN 1476-1122, 12/2016, Volume 15, Issue 12, pp. 1297 - 1306
During mesenchymal development, the microenvironment gradually transitions from one that is rich in cell-cell interactions to one that is dominated by cell-ECM... 
PHYSICS, CONDENSED MATTER | HYDROGELS | PHYSICS, APPLIED | MATERIALS SCIENCE, MULTIDISCIPLINARY | CHEMISTRY, PHYSICAL | TENSION | CYTOSKELETAL | FRAGMENTS | STIFFNESS | YAP/TAZ | RAC | ALPHA-CATENIN | RHO GTPASES | FIBRONECTIN | Cadherins - metabolism | Extracellular Matrix - drug effects | Hyaluronic Acid - chemistry | Extracellular Matrix - metabolism | Hyaluronic Acid - pharmacology | Cell Adhesion - drug effects | Biomechanical Phenomena | Animals | Cattle | Mesenchymal Stem Cells - cytology | Mesenchymal Stem Cells - drug effects | Mechanical Phenomena | Methylation | Signal transduction | Materials science | Biomedical materials | Biophysics | Stem cells | Cell adhesion & migration | Index Medicus | Cues | Adhesives | Electrochemical machining | Perception | Evolution | Stiffness | Fibronectin | YAP | Mechanotransduction | N-Cadherin | TAZ | Mechanosensing
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12. Full Text A novel HMGA1-CCNE2-YAP axis regulates breast cancer aggressiveness
by Pegoraro, Silvia and Ros, Gloria and Ciani, Yari and Sgarra, Riccardo and Piazza, Silvano and Manfioletti, Guidalberto
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 22, pp. 19087 - 19101
High Mobility Group A1 (HMGA1) is an architectural chromatin factor that promotes neoplastic transformation and progression. However, the mechanism by which... 
Hippo pathway | Oncogene/tumour suppressor | CDK inhibitors | Cyclin E2 | YAP/TAZ | HMGA1a Protein - genetics | Phosphorylation | Signal Transduction | Neoplasm Invasiveness | Humans | Cyclins - genetics | Nuclear Proteins - metabolism | Transcription Factors - genetics | HMGA1a Protein - metabolism | Cell Movement - physiology | Breast Neoplasms - metabolism | Transcription Factors - metabolism | Breast Neoplasms - genetics | Transfection | Cyclins - metabolism | Breast Neoplasms - pathology | HEK293 Cells | Cell Line, Tumor | Female | Nuclear Proteins - genetics | Index Medicus
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13. Full Text Suppression of YAP/TAZ-Notch1-NICD axis by bromodomain and extraterminal protein inhibition impairs liver regeneration
by Liu, Chen and Cheng, Xiawei and Chen, Juntao and Wang, Yao and Wu, Xiaoying and Tian, Rui and Liu, Baoqing and Ding, Xianfeng and Sun, Qiming and Gong, Weihua
Theranostics, ISSN 1838-7640, 2019, Volume 9, Issue 13, pp. 3840 - 3852
: Biological mechanisms that control liver regeneration remain poorly defined. However, these mechanisms are remarkable issues in the clinic that affect... 
Notch signaling pathway | BET proteins | YAP/TAZ | Liver regeneration
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14. Full Text Stretch-activated ion channel Piezo1 directs lineage choice in human neural stem cells
by Medha M. Pathak and Jamison L. Nourse and Truc Tran and Jennifer Hwe and Janahan Arulmoli and Dai Trang T. Le and Elena Bernardis and Lisa A. Flanagan and Francesco Tombola
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2014, Volume 111, Issue 45, pp. 16148 - 16153
Neural stem cells are multipotent cells with the ability to differentiate into neurons, astrocytes, and oligodendrocytes. Lineage specification is strongly... 
Neurons | Stem cells | Cell lines | Small interfering RNA | Neural stem cells | Stiffness | Ion channels | Cellular differentiation | Electric current | Mesenchymal stem cells | Calcium signaling | Blebbistatin | Myosin II | YAP/TAZ | Matrix mechanics | PROGENITOR CELLS | MATRIX | MULTIDISCIPLINARY SCIENCES | calcium signaling | TENSION | MECHANISMS | matrix mechanics | IDENTIFICATION | CULTURE | COMMITMENT | PATHWAY | SUBSTRATE | myosin II | Yap/Taz | INHIBITOR | blebbistatin | Calcium Signaling - physiology | Humans | Multipotent Stem Cells - metabolism | Cells, Cultured | Ion Channels - genetics | Male | Mechanotransduction, Cellular - physiology | Neural Stem Cells - cytology | Gene Knockdown Techniques | Ion Channel Gating - physiology | Ion Channels - metabolism | Multipotent Stem Cells - cytology | Neurogenesis - physiology | Female | Cell Differentiation - physiology | Neural Stem Cells - metabolism | Stem cell research | Research | Cell differentiation | Health aspects | Proteins | Calcium | Ribonucleic acid--RNA | Index Medicus | Biological Sciences | Yap | Taz
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15. Full Text YAP regulates cell mechanics by controlling focal adhesion assembly
by Nardone, Giorgia and Oliver-De La Cruz, Jorge and Vrbsky, Jan and Martini, Cecilia and Pribyl, Jan and Skládal, Petr and Pešl, Martin and Caluori, Guido and Pagliari, Stefania and Martino, Fabiana and Maceckova, Zuzana and Hajduch, Marian and Sanz-Garcia, Andres and Pugno, Nicola Maria and Stokin, Gorazd Bernard and Forte, Giancarlo
Nature Communications, ISSN 2041-1723, 05/2017, Volume 8, Issue 1, pp. 15321 - 15321
Hippo effectors YAP/TAZ act as on-off mechanosensing switches by sensing modifications in extracellular matrix (ECM) composition and mechanics. The regulation... 
MIGRATION | PROTEIN | PATHWAY | RAC | YAP/TAZ | MULTIDISCIPLINARY SCIENCES | HIPPO | SELF-RENEWAL | ACTIN-FILAMENTS | TAZ | BINDING | Cell Line | Actin Cytoskeleton - metabolism | Humans | Extracellular Matrix - metabolism | Nuclear Proteins - metabolism | rhoA GTP-Binding Protein - metabolism | Gene Expression Profiling | Transcription Factors - genetics | rhoA GTP-Binding Protein - genetics | Mechanotransduction, Cellular - physiology | Cell Movement - genetics | Cell Movement - physiology | Focal Adhesions - genetics | Transcription Factors - metabolism | Cell Differentiation - genetics | Cell Shape | Focal Adhesions - metabolism | HEK293 Cells | Cell Line, Tumor | Cell Membrane - metabolism | Focal Adhesions - physiology | Nuclear Proteins - genetics | Cell Differentiation - physiology | Mechanotransduction, Cellular - genetics | Index Medicus
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16. Full Text GTSE1: A novel TEAD4-E2F1 target gene involved in cell protrusions formation in triple-negative breast cancer cell models
by Stelitano, Debora and Leticia, Yamila Peche and Dalla, Emiliano and Monte, Martin and Piazza, Silvano and Schneider, Claudio
Oncotarget, ISSN 1949-2553, 09/2017, Volume 8, Issue 40, pp. 67422 - 67438
GTSE1 over-expression has been reported as a potential marker for metastasis in various types of malignancies, including breast cancer. Despite this, the... 
TEAD4 | E2F1 | Cell protrusions | YAP/TAZ | GTSE1
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17. Full Text Cellular Volume and Matrix Stiffness Direct Stem Cell Behavior in a 3D Microniche
by Bao, Min and Xie, Jing and Katoele, Nando and Hu, Xinyu and Wang, Baoxiu and Piruska, Aigars and Huck, Wilhelm T.S
ACS Applied Materials and Interfaces, ISSN 1944-8244, 01/2019, Volume 11, Issue 2, pp. 1754 - 1759
The central question addressed in this study is whether cells with different sizes have different responses to matrix stiffness. We used methacrylated... 
3D cell culture | matrix stiffness | cell volume | hydrogel | YAP/TAZ | mechanotransduction | ELASTICITY | COLLAGEN | MATERIALS SCIENCE, MULTIDISCIPLINARY | NANOSCIENCE & NANOTECHNOLOGY | Letter | YAP | TAZ
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18. Full Text Clinical and Echocardiographic Impact of Tafazzin Variants on Dilated Cardiomyopathy Phenotype in Left Ventricular Non-Compaction Patients in Early Infancy
by Hirono, Keiichi and Hata, Yukiko and Nakazawa, Makoto and Momoi, Nobuo and Tsuji, Tohru and Matsuoka, Taro and Ayusawa, Mamoru and Abe, Yuriko and Hayashi, Tamaki and Tsujii, Nobuyuki and Abe, Tadaaki and Sakaguchi, Heima and Wang, Ce and Takasaki, Asami and Takarada, Shinya and Okabe, Mako and Miyao, Nariaki and Nakaoka, Hideyuki and Ibuki, Keijiro and Saito, Kazuyoshi and Ozawa, Sayaka and Nishida, Naoki and Bowles, Neil E and Ichida, Fukiko
Circulation Journal, ISSN 1346-9843, 2018
Background: Left ventricular non-compaction (LVNC) is a cardiomyopathy morphologically characterized by 2-layered myocardium and numerous prominent... 
Cardiomyopathy | Tafazzin (TAZ) | Left ventricular non-compaction
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19. Full Text A ROR1 HER3 IncRNA signalling axis modulates the Hippo YAP pathway to regulate bone metastasis
by Li, CL and Wang, SY and Xing, Z and Lin, AF and Liang, K and Song, J and Hui, QS and Yao, J and Chen, ZY and Park, PK and Hawke, DH and Zhou, JW and Zhou, Y and Zhang, SX and Liang, H and Hung, MC and Gallick, GE and Han, L and Lin, CR and Yang, LQ
NATURE CELL BIOLOGY, ISSN 1465-7392, 02/2017, Volume 19, Issue 2, pp. 106 - 119
Bone metastases remain a serious health concern because of limited therapeutic options. Here, we report that crosstalk between ROR1-HER3 and the Hippo-YAP... 
ACTIVATION | RECEPTOR TYROSINE KINASES | LONG NONCODING RNA | BREAST-CANCER METASTASIS | BIOLOGY | LUNG ADENOCARCINOMA | TAZ | TRANSCRIPTION FACTOR | EXPRESSION | FAMILY | CELL BIOLOGY
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