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-induced immunotoxicity (12) 12
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Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3540 - 3551
Endothelial injury or dysfunction is an early event in the pathogenesis of atherosclerosis. Epidemiological and animal studies have shown that 2, 3, 7,... 
apoptosis | EP3 | Bcl‐2 | dioxin | p38 MAPK | endothelial cell | Bcl-2 | MEDICINE, RESEARCH & EXPERIMENTAL | TCDD-INDUCED APOPTOSIS | ATHEROSCLEROSIS | RECEPTOR | PROLIFERATION | INCREASES APOPTOSIS | 2,3,7,8-TETRACHLORODIBENZO-P-DIOXIN | CELL BIOLOGY | SIGNALING PATHWAY | GENE-EXPRESSION | DYSFUNCTION | E-DEFICIENT MICE | RNA, Small Interfering - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Endothelium, Vascular - drug effects | Re-Epithelialization - physiology | Dose-Response Relationship, Drug | Femoral Artery - injuries | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cyclooxygenase 2 - genetics | Caspase 3 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Environmental Pollutants - toxicity | Dinoprostone - agonists | Receptors, Prostaglandin E, EP3 Subtype - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Receptors, Prostaglandin E, EP3 Subtype - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Mice, Knockout | Dinoprostone - metabolism | Animals | Endothelium, Vascular - metabolism | Receptors, Prostaglandin E, EP3 Subtype - antagonists & inhibitors | Cyclooxygenase 2 - metabolism | Polychlorinated Dibenzodioxins - toxicity | Endothelium, Vascular - pathology | Human Umbilical Vein Endothelial Cells - pathology | Mice | Proto-Oncogene Proteins c-bcl-2 - genetics | RNA, Small Interfering - metabolism | Herbicides | COX-2 inhibitors | Enzymes | Dioxin | Prostaglandins E | Atherosclerosis | Genetic engineering | Epidemiology | Apoptosis | Endothelium | Phosphorylation | Femur | Femoral artery | Bcl-2 protein | Pathogenesis | Prostaglandin E2 | TCDD | Dioxins | Activation | Caspase-3 | Cell activation | Rodents | Inhibition | Heart diseases | Injuries | Caspase | MAP kinase | Pharmacology | Exposure | Morbidity | Endothelial cells | Gene silencing | Cyclooxygenase-1 | Arteriosclerosis | Cyclooxygenase-2 | Cardiovascular diseases | Umbilical vein | Original
Journal Article
Archives of Toxicology, ISSN 0340-5761, 10/2017, Volume 91, Issue 10, pp. 3365 - 3372
The maintenance of cellular homeostasis is a complex process that is governed by the receipt of prototypical growth and death signals. The endogenous functions... 
Biomedicine, general | Cell growth | Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | 6-Formylindolo[3,2- b ]carbazole (FICZ) | Pharmacology/Toxicology | Mitochondrial-dependent pathway | Apoptosis | 6-Formylindolo[3,2-b]carbazole (FICZ) | OXIDATIVE STRESS | AH RECEPTOR | HEPATOMA-CELLS | 6-Formylindolo[3,2-b] carbazole ( FICZ) | CYTOCHROME-C | 2,3,7,8-TETRACHLORODIBENZO-P-DIOXIN (TCDD)-INDUCED IMMUNOTOXICITY | CYCLE CONTROL | IMMUNE-SYSTEM | RETINOBLASTOMA PROTEIN | ARYL-HYDROCARBON RECEPTOR | TOXICOLOGY | T-CELLS | Cell Survival - drug effects | Reactive Oxygen Species - metabolism | Oxidative Stress | Apoptosis - drug effects | Glutathione - metabolism | Cytochromes c - metabolism | Humans | Carbazoles - administration & dosage | Cytochrome P-450 CYP1A1 - antagonists & inhibitors | Cytochrome P-450 CYP1A1 - metabolism | Membrane Potential, Mitochondrial - drug effects | Hep G2 Cells | Dose-Response Relationship, Drug | Cell Proliferation - drug effects | Flavonoids - pharmacology | Carbazoles - pharmacology | Carbazoles - metabolism | Flavonoids | Flavones | Growth | Bioflavonoids | Analysis | Cytochrome P-450 | Cytochrome | Oxidative stress | Enzyme activity | Low concentrations | Toxicity | Cytochrome P450 | Homeostasis | Hepatoma | Carbazole | Mitochondria | Enzymatic activity | Aromatic compounds | Cell death | Trace levels | Biocompatibility | Kinetics | Viability | Growth kinetics
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2005, Volume 175, Issue 1, pp. 90 - 103
We investigated the role of aryl hydrocarbon receptor (AhR) in the regulation of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)induced apoptosis in thymic T cells.... 
TRANSCRIPTION FACTORS | (TCDD)-INDUCED IMMUNOTOXICITY | ACTIVATION | MURINE HOST | IN-VIVO | ARYLHYDROCARBON RECEPTOR | GENE-EXPRESSION | AH RECEPTOR | DEFICIENT MICE | IMMUNOLOGY | TCDD-INDUCED TOXICITY
Journal Article
The FEBS Journal, ISSN 1742-464X, 02/2005, Volume 272, Issue 4, pp. 903 - 915
The aromatic hydrocarbon receptor (AhR)‐dependent pathway involved in 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin (TCDD)‐induced immunotoxicity has been studied... 
apoptosis | lymphoblastic T cell | dioxin | rottlerin | PKCθ | Rottlerin | Lymphoblastic T cell | Dioxin | Apoptosis | ACTIVATION | LIGAND EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TCDD | DEATH | INDUCTION | PKC theta | (TCDD)-INDUCED IMMUNOTOXICITY | ARYL-HYDROCARBON RECEPTOR | AH-RECEPTOR | CALCINEURIN | PKC-THETA
Journal Article
Journal Article
Journal Article
Archives of Toxicology, ISSN 0340-5761, 02/2019, Volume 93, Issue 2, pp. 355 - 368
Journal Article