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PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53343
Kunitz-type serine protease inhibitors are involved in various physiological processes, such as ion channel blocking, blood coagulation, fibrinolysis, and inflammation... 
VENOM | KING COBRA | MULTIDISCIPLINARY SCIENCES | PSEUDONAJA-TEXTILIS | SILK PROTEIN | CHYMOTRYPSIN INHIBITOR | MOLECULAR-CLONING | BOOPHILUS-MICROPLUS | EXPRESSION | ARANEUS-VENTRICOSUS | PEPTIDE | Factor Xa - chemistry | Chymotrypsin - metabolism | Molecular Sequence Data | Baculoviridae - genetics | Tissue Plasminogen Activator - chemistry | Pancreatic Elastase - antagonists & inhibitors | Antifibrinolytic Agents - metabolism | Trypsin Inhibitors - chemistry | Serine Proteinase Inhibitors - chemistry | Spiders - metabolism | Aprotinin - genetics | Conserved Sequence | Antifibrinolytic Agents - chemistry | Spiders - chemistry | Protein Structure, Tertiary | Amino Acid Sequence | Gene Expression | Fibrinolysin - antagonists & inhibitors | Recombinant Proteins - chemistry | Arthropod Proteins - genetics | Recombinant Proteins - genetics | Trypsin - metabolism | Arthropod Proteins - chemistry | Fibrinolysin - chemistry | Sequence Alignment | Animals | Thrombin - chemistry | Trypsin Inhibitors - genetics | Chymotrypsin - antagonists & inhibitors | Pancreatic Elastase - chemistry | Aprotinin - chemistry | Serine Proteinase Inhibitors - genetics | Enzymes | Care and treatment | Protease inhibitors | Physiological aspects | Hydrolases | Inflammation | Research | Health aspects | Risk factors | Natural resources | Blood coagulation | Serine | Thrombin | Amino acids | Proteinase inhibitors | Potassium channels | Baculovirus | Fibrinolysis | Plasmin | t-Plasminogen activator | Proteins | Chymotrypsin | Trypsin | Insects | Proteases | Lysine | Protease | Venom | Insect cells | Ion channels | Elastase | Serine proteinase | Recombinant
Journal Article
Cell stem cell, ISSN 1934-5909, 2009, Volume 5, Issue 5, pp. 491 - 503
The combined activity of three transcription factors can reprogram adult cells into induced pluripotent stem cells (iPSCs). However, the transgenic methods... 
STEMCELL | PLURIPOTENT STEM-CELLS | OCT4 | POTENT | MOUSE | HUMAN FIBROBLASTS | GENERATION | INDUCTION | DIFFERENTIATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY
Journal Article
Expert Opinion on Drug Discovery, ISSN 1746-0441, 09/2013, Volume 8, Issue 9, pp. 1095 - 1116
Introduction: Methicillin-resistant Staphylococcus aureus (MRSA) have been on the increase during the past decade, due to the steady growth of the elderly and... 
acyl t-RNA synthase inhibitor | Clp protease inhibitor | methicillin-resistant Staphylococcus aureus | two-component system inhibitor | elongation factor G inhibitor | linezolid | antibacterial agents | combination therapy | daptomycin | vancomycin | β-lactamase inhibitor | antibacterial vaccine | dihydropteroate synthase inhibitor | peptidoglycan biosynthesis inhibitor | Pol IIIC inhibitor | electron transport inhibitor | RNA-polymerase inhibitor | phage therapy | peptide deformylase inhibitor | menaquinone biosynthesis inhibitor | Gram-positive pathogens | antibiotics | antimicrobial peptides | dihydrofolate reductase inhibitor | FAS II inhibitor | Linezolid | Peptide deformylase inhibitor | Daptomycin | Dihydropteroate synthase inhibitor | Vancomycin | Combination therapy | Antimicrobial peptides | Menaquinone biosynthesis inhibitor | Antibacterial vaccine | Dihydrofolate reductase inhibitor | Acyl t-RNA synthase inhibitor | Elongation factor G inhibitor | Antibacterial agents | Electron transport inhibitor | Peptidoglycan biosynthesis inhibitor | Antibiotics | Methicillin-resistant Staphylococcus aureus | Phage therapy | Two-component system inhibitor | SOFT-TISSUE INFECTIONS | INTENSIVE-CARE UNITS | beta-lactamase inhibitor | RESISTANT STAPHYLOCOCCUS-AUREUS | PHARMACOLOGY & PHARMACY | IN-VITRO ACTIVITY | TOREZOLID PHOSPHATE TR-701 | BRIDGED BICYCLIC MACROLIDE | DIHYDROFOLATE-REDUCTASE INHIBITOR | QUALITY-CONTROL RANGES | Anti-Bacterial Agents - therapeutic use | Animals | Bacterial Infections - drug therapy | Drug Resistance, Multiple, Bacterial | Humans | Drug Discovery | Methicillin-Resistant Staphylococcus aureus | elongation factor G (EF-G) inhibitor | peptide deformylase (PDF) inhibitor | methicillin-resistant Staphylococcus aureus (MRSA) | PolIIC inhibitor | β5 -lactamase inhibitor
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2004, Volume 172, Issue 1, pp. 567 - 576
.... Overexpression of STAT3 DN completely inhibited IL-10-induced suppressor of cytokine signaling 3, tissue inhibitor of MMP-1, TNF receptor expression, and the recently identified IL-10-inducible genes... 
RHEUMATOID-ARTHRITIS | TUMOR-NECROSIS-FACTOR | DNA-BINDING | HUMAN NEUTROPHILS | TYROSINE PHOSPHORYLATION | INTERLEUKIN-10 RECEPTOR | GENE-EXPRESSION | KAPPA-B-ALPHA | IMMUNOLOGY | HUMAN MONOCYTES | MONONUCLEAR PHAGOCYTES | Protein Binding - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Humans | Tumor Necrosis Factor-alpha - genetics | Immunoglobulins - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Repressor Proteins - antagonists & inhibitors | Antigens, CD - metabolism | Trans-Activators - physiology | Protein Tyrosine Phosphatases - antagonists & inhibitors | RNA, Messenger - biosynthesis | Protein Tyrosine Phosphatases - genetics | Inflammation Mediators - physiology | Glycoproteins - genetics | DNA-Binding Proteins - physiology | Protein Tyrosine Phosphatases - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction - genetics | DNA - metabolism | Down-Regulation - genetics | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 | Repressor Proteins - biosynthesis | Up-Regulation - immunology | Interleukin-10 - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Adenoviruses, Human - genetics | Interleukin-10 - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Antigens, CD - biosynthesis | Receptors, Cell Surface | Receptors, IgG - biosynthesis | Receptors, IgG - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Signaling Lymphocytic Activation Molecule Family Member 1 | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type II | Trans-Activators - genetics | Inflammation Mediators - antagonists & inhibitors | Trans-Activators - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Inflammation Mediators - immunology | Receptors, Tumor Necrosis Factor - metabolism | Immune Sera - pharmacology | Proteins - physiology | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Histocompatibility Antigens Class II - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Up-Regulation - genetics | DNA-Binding Proteins - genetics | DNA - antagonists & inhibitors | Glycoproteins - biosynthesis | Suppressor of Cytokine Signaling 3 Protein | Down-Regulation - immunology | Interleukin-6 - biosynthesis | Receptors, Tumor Necrosis Factor - biosynthesis | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Genetic Vectors | DNA-Binding Proteins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
International journal of cancer, ISSN 0020-7136, 2014, Volume 134, Issue 6, pp. 1484 - 1494
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 4, p. e0195116
Over expression of Tissue Inhibitor of Metalloproteinases-3 (TIMP-3) in vascular smooth muscle cells (VSMCs... 
MATRIX METALLOPROTEINASES | DEATH DOMAIN | OVEREXPRESSION | LEUKEMIC-CELLS | CD95 | INVASION | MECHANISM | REDISTRIBUTION | MULTIDISCIPLINARY SCIENCES | LIPID RAFTS | PROLIFERATION | Disintegrins - metabolism | Muscle, Smooth, Vascular - metabolism | Apoptosis - drug effects | Humans | Cells, Cultured | fas Receptor - antagonists & inhibitors | Membrane Potential, Mitochondrial - drug effects | Muscle, Smooth, Vascular - cytology | fas Receptor - metabolism | ADAM17 Protein - metabolism | Microscopy, Confocal | ADAM17 Protein - genetics | Caspases - metabolism | ADAM17 Protein - antagonists & inhibitors | RNA Interference | Tissue Inhibitor of Metalloproteinase-3 - metabolism | fas Receptor - genetics | Disintegrins - antagonists & inhibitors | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | Staurosporine - pharmacology | RNA, Small Interfering - metabolism | Tissue Inhibitor of Metalloproteinase-3 - genetics | Medical research | Tumor necrosis factor | Physiological aspects | Medicine, Experimental | Research | Vascular smooth muscle | Health aspects | Apoptosis | Shedding | Immunoprecipitation | Smooth muscle | Lipids | Infections | Activation | Tissue inhibitor of metalloproteinase 3 | Caspase-3 | Cell surface | Proteins | Fas-associated protein | FADD protein | Signal transduction | Mitochondria | Rodents | FLIP protein | Interleukin 1 | Inhibition | Enzymes | ADAM protein | Cytokines | Mortality | Muscles | Caspase | c-FLIP protein | Clustering | Lymphoma | Caspase-8 | Signaling | Depletion | Overexpression | Inhibitors | Lymphocytes B | Adenoviruses | Caspase-9 | Death | Position (location) | Immunofluorescence | Cancer | B-cell lymphoma
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2005, Volume 288, Issue 2, pp. 461 - 468
The balance between matrix metalloproteinases (MMPs) and their natural inhibitors, the tissue inhibitors of metalloproteinases (TIMPs... 
Matrix metalloproteinase | Proliferation | Apoptosis | Phenotypic differentiation | DEATH DOMAIN | CELLS | phenotypic differentiation | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | proliferation | RAT | apoptosis | FAILING HUMAN HEART | GROWTH-FACTOR-BETA | matrix metalloproteinase | RECOMBINANT ADENOVIRUSES | MYOFIBROBLASTS | PERIPHERAL VASCULAR DISEASE | MOLECULAR-CLONING | EXPRESSION | Humans | Actins - metabolism | Culture Media, Conditioned - pharmacology | Tissue Inhibitor of Metalloproteinase-3 - antagonists & inhibitors | Tissue Inhibitor of Metalloproteinase-2 - genetics | Tissue Inhibitor of Metalloproteinases - genetics | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Myocardium - metabolism | Tissue Inhibitor of Metalloproteinase-3 - metabolism | Adenoviridae - genetics | Fibroblasts - metabolism | Tissue Inhibitor of Metalloproteinase-3 - genetics | Recombinant Proteins - metabolism | Recombinant Proteins - antagonists & inhibitors | Mice, Inbred C57BL | Tissue Inhibitor of Metalloproteinases - antagonists & inhibitors | Cells, Cultured | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Tissue Inhibitor of Metalloproteinases - metabolism | Myocardium - cytology | Cell Division - physiology | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Fibroblasts - cytology | Mice | Apoptosis - physiology | Tissue Inhibitor of Metalloproteinase-2 - antagonists & inhibitors
Journal Article