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toll-like receptors (272) 272
tlr1 protein (251) 251
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tlr5 protein (41) 41
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health aspects (39) 39
lipopolysaccharides (39) 39
recognition (39) 39
single-nucleotide polymorphism (39) 39
bacteria (38) 38
biology (38) 38
middle aged (38) 38
pattern recognition (38) 38
tlr7 protein (38) 38
toll-like receptors - genetics (38) 38
polymorphism, single nucleotide (37) 37
pathogens (36) 36
leukocytes (35) 35
polymorphism (35) 35
cutting edge (33) 33
polymerase chain reaction (33) 33
signaling (33) 33
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toll-like receptor 2 - metabolism (31) 31
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myd88 protein (30) 30
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toll-like receptor (30) 30
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lipoproteins (26) 26
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deoxyribonucleic acid--dna (25) 25
nf-κb protein (25) 25
single nucleotide polymorphisms (25) 25
toll-like receptor 1 - metabolism (25) 25
toll-like receptor 2 - genetics (25) 25
bacterial infections (24) 24
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genetic predisposition to disease (24) 24
interleukin 8 (24) 24
medicine and health sciences (24) 24
risk factors (24) 24
rna (24) 24
tumor necrosis factor-α (24) 24
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1. Full Text In Silico approach to inhibition of signaling pathways of toll-like receptors 2 and 4 by ST2L
by Basith, Shaherin and Manavalan, Balachandran and Govindaraj, Rajiv Gandhi and Choi, Sangdun
PLoS ONE, ISSN 1932-6203, 08/2011, Volume 6, Issue 8, p. e23989
Toll-like receptors (TLRs) activate a potent immunostimulatory response. There is clear evidence that overactivation of TLRs leads to infectious and... 
STRUCTURAL BASIS | CRYSTAL-STRUCTURE | PROTEIN-DOCKING | MULTIDISCIPLINARY SCIENCES | ENDOTOXIN TOLERANCE | TIR-DOMAIN | INTERLEUKIN-1 RECEPTOR | IL-1 RECEPTOR | MYD88 ADAPTER-LIKE | QUALITY ASSESSMENT | INNATE IMMUNITY | Interleukin-1 Receptor-Like 1 Protein | Humans | Protein Multimerization | Proteolipids - metabolism | Molecular Sequence Data | Myeloid Differentiation Factor 88 - chemistry | Myelin and Lymphocyte-Associated Proteolipid Proteins | Protein Structure, Quaternary | Membrane Transport Proteins - metabolism | Receptors, Cell Surface - chemistry | Protein Stability | Protein Structure, Tertiary | Toll-Like Receptor 6 - metabolism | Amino Acid Sequence | Toll-Like Receptor 6 - chemistry | Signal Transduction | Computational Biology | Receptors, Cell Surface - metabolism | Toll-Like Receptor 4 - chemistry | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Molecular Dynamics Simulation | Membrane Transport Proteins - chemistry | Myelin Proteins - chemistry | Sequence Alignment | Myelin Proteins - metabolism | Myeloid Differentiation Factor 88 - metabolism | Toll-Like Receptor 2 - chemistry | Proteolipids - chemistry | Molecular dynamics | Medicine, Experimental | Medical research | Structure | Analysis | Crystals | Nuclear magnetic resonance--NMR | Genes | Science | Homology | Innate immunity | Interleukin 1 receptors | Kinases | Immunity | Inflammatory diseases | Molecular docking | Proteins | Signal transduction | Receptors | Pathways | Hydrogen bonds | Rodents | Interleukin 1 | Toll-like receptors | Trends | Inhibition | Crystal structure | Immune system | Immunostimulation | Cytokines | Computer simulation | Adapters | TLR4 protein | Signaling | Molecular modelling | TLR1 protein | TLR2 protein | MyD88 protein | Ligands | In vivo methods and tests | Regulation | Mutation | Three dimensional models | Nuclear magnetic resonance | NMR
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2. Full Text Isolated Toll-like Receptor Transmembrane Domains Are Capable of Oligomerization
by Godfroy III, James I and Roostan, Mohammad and Moroz, Yurii S and Korendovych, Ivan V and Yin, Hang
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e48875
Toll-like receptors (TLRs) act as the first line of defense against bacterial and viral pathogens by initiating critical defense signals upon dimer activation.... 
SIGNAL-TRANSDUCTION | SYSTEM | TOXR | PATTERN-RECOGNITION | STRUCTURAL BASIS | MEMBRANE | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | PROTEIN-PROTEIN INTERACTIONS | DIMERIZATION | INNATE IMMUNITY | Protein Structure, Tertiary | Toll-Like Receptors - metabolism | DNA Mutational Analysis | Circular Dichroism | Escherichia coli | Protein Multimerization | Oligomers | Peptides | Research | Membranes | Kinases | Proteins | Signal transduction | Receptors | E coli | Toll-like receptors | Bacteria | Dimerization | Protein families | Immune system | Oligomerization | Cytokines | TLR5 protein | Medical treatment | TLR4 protein | Signaling | Chemistry | TLR1 protein | Neural networks | TLR2 protein | Ligands | Transmembrane domains | Molecular biology | Adapter proteins
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3. Full Text Human Keratinocytes Express Functional Toll-Like Receptor 3, 4, 5, and 9
by Lebre, Maria C and van der Aar, Angelic M.G and van Baarsen, Lisa and van Capel, Toni M.M and Schuitemaker, Joost H.N and Kapsenberg, Martien L and de Jong, Esther C
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2007, Volume 127, Issue 2, pp. 331 - 341
Keratinocytes are continuously in contact with external stimuli and have the capacity to produce several soluble mediators. Pathogen-associated molecular... 
TOLL-LIKE-RECEPTORS | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | SKIN-ASSOCIATED CHEMOKINE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INNATE IMMUNE-RESPONSE | INFLAMMATORY PROTEIN 3-ALPHA | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | HUMAN EPIDERMAL-KERATINOCYTES | DERMATOLOGY | Oligonucleotides - genetics | Phosphorylation | Toll-Like Receptor 5 - genetics | Humans | Toll-Like Receptor 9 - genetics | I-kappa B Proteins - metabolism | RNA, Messenger - metabolism | Flagellin - pharmacology | Tissue Distribution | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Biological Transport | Oligonucleotides - pharmacology | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Chemokine CXCL9 | Toll-Like Receptor 5 - metabolism | Poly I-C - pharmacology | Cytokines - metabolism | Cells, Cultured | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Chemokine CXCL10 | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Toll-Like Receptors - genetics | Chemokines, CXC - metabolism | Lipopolysaccharides - pharmacology | CpG Islands | Ligands | Chemokines - metabolism | Toll-Like Receptor 9 - metabolism | CD40 antigen | Oligonucleotides | Flagellin | mRNA | intercellular adhesion molecule 1 | Immunity | Lipopolysaccharides | CXC chemokines | CCL20 protein | Toll-like receptors | CpG islands | TLR7 protein | Dermatology | Keratinocytes | Nuclear transport | Stress | TLR3 protein | TLR9 protein | TLR1 protein | External stimuli | CXCL10 protein | Histocompatibility antigen HLA | Interferon | Internet | Monocyte chemoattractant protein 1 | Tumor necrosis factor- alpha
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4. Phagocytosis-mediated M1 activation by chitin but not by chitosan
by Spring Davis and Aiko M Cirone and Janet Menzie and Floyd Russell and C Kathleen Dorey and Yoshimi Shibata and Jianning Wei and Changlong Nan
American Journal of Physiology, ISSN 0363-6143, 07/2018, Volume 315, Issue 1, p. C62
Chitin particles have been used to understand host response to chitin-containing pathogens and allergens and are known to induce a wide range of polarized... 
Chitinase | TOR protein | Allergens | Genes | Interleukin 1 receptors | Macrophages | CD14 antigen | Microparticles | Proteins | Tissue | Cell growth | Cell activation | Actin | Interleukin 1 | Toll-like receptors | Protein-tyrosine kinase | Adaptor proteins | Tyrosine | Pathogens | MAP kinase | Chitin | Rapamycin | TLR1 protein | TLR2 protein | Lck protein | MyD88 protein | Lyn protein | Chitosan | Phagocytosis
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5. Full Text IκBζ regulates human monocyte pro-inflammatory responses induced by Streptococcus pneumoniae
by Sundaram, Kruthika and Rahman, Mohd. Akhlakur and Mitra, Srabani and Knoell, Daren L and Woodiga, Shireen A and King, Samantha J and Wewers, Mark D
PLoS ONE, ISSN 1932-6203, 09/2016, Volume 11, Issue 9, p. e0161931
Pneumococcal lung infections represent a major cause of death worldwide. Single nucleotide polymorphisms (SNPs) in the NFKBIZ gene, encoding the transcription... 
Bronchi - microbiology | RNA, Small Interfering - genetics | Toll-Like Receptor 2 - antagonists & inhibitors | Toll-Like Receptor 2 - genetics | I-kappa B Proteins - immunology | Epithelial Cells - drug effects | Humans | NF-kappa B - immunology | Monocytes - immunology | Bronchi - immunology | I-kappa B Proteins - genetics | Bronchi - drug effects | RNA, Small Interfering - immunology | I-kappa B Proteins - antagonists & inhibitors | Toll-Like Receptor 4 - antagonists & inhibitors | Nuclear Proteins - genetics | Streptococcus pneumoniae - physiology | Monocytes - microbiology | Benzocycloheptenes - pharmacology | Streptococcus pneumoniae - drug effects | Interleukin-6 - genetics | Signal Transduction | Gene Expression Regulation | Toll-Like Receptor 4 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Toll-Like Receptor 4 - immunology | p38 Mitogen-Activated Protein Kinases - immunology | Nuclear Proteins - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Host-Pathogen Interactions | Monocytes - drug effects | Toll-Like Receptor 1 - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Toll-Like Receptor 1 - antagonists & inhibitors | NF-kappa B - genetics | Epithelial Cells - immunology | Epithelial Cells - microbiology | Nuclear Proteins - antagonists & inhibitors | Interleukin-6 - immunology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Primary Cell Culture | Toll-Like Receptor 1 - immunology | Pneumonia | Epithelial cells | Pathogenesis | Genes | Critical care | Biology | Infections | Single-nucleotide polymorphism | Streptococcus infections | Interleukin 6 | Proteins | Toll-like receptors | Inhibition | Cytokines | Internal medicine | MAP kinase | Inflammation | TLR4 protein | Children & youth | Medicine | Pneumolysin | Monocytes | Sleep | Streptococcus | TLR1 protein | Lungs | Pharmacy | TLR2 protein | Human behavior
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6. Full Text TLR1-10, NF-κB and p53 expression is increased in oral lichenoid disease
by Rusanen, Peter and Marttila, Emilia and Uittamo, Johanna and Hagström, Jaana and Salo, Tuula and Rautemaa-Richardson, Riina
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0181361
Toll-like receptors (TLRs) and nuclear factor-kappa B (NF-kappa B) in keratinocytes play an important role in dermatological autoimmune diseases. Tumour... 
TONGUE | ACTIVATION | IMMUNE-RESPONSES | PLANUS | MULTIDISCIPLINARY SCIENCES | SQUAMOUS-CELL CARCINOMA | TOLL-LIKE RECEPTORS | CANCER | SOLUBLE FORMS | LESIONS | Immunohistochemistry | Gene Expression | Lichen Planus, Oral - metabolism | Humans | Middle Aged | Tumor Suppressor Protein p53 - metabolism | Male | NF-kappa B - metabolism | Young Adult | Mouth Mucosa - metabolism | Lichen Planus, Oral - pathology | Biopsy | Adolescent | Adult | Female | Aged | Toll-Like Receptors - metabolism | Mouth Mucosa - pathology | Mucosal immunity | Disease | Pathogenesis | p53 Protein | Infections | Proteins | Receptors | Cell cycle | Toll-like receptors | Localization | Deoxyribonucleic acid--DNA | Immune system | NF-κB protein | TLR7 protein | Polypeptides | Cytokines | TLR5 protein | Keratinocytes | TLR4 protein | Gene expression | Patients | TLR3 protein | Hospitals | TLR1 protein | TLR2 protein | Autoimmune diseases | Cancer | Tumors | Apoptosis | Deoxyribonucleic acid | DNA
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7. Full Text The Herpes Simplex Virus 1-Encoded Envelope Glycoprotein B Activates NF-κB through the Toll-Like Receptor 2 and MyD88/TRAF6-Dependent Signaling Pathway
by Cai, Mingsheng and Li, Meili and Wang, Kezhen and Wang, Shuai and Lu, Qiong and Yan, Jinghua and Mossman, Karen L and Lin, Rongtuan and Zheng, Chunfu
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e54586
The innate immune response plays a critical role in the host defense against invading pathogens, and TLR2, a member of the Toll-like receptor (TLR) family, has... 
PROTEIN | RECOGNITION | CELL ACTIVATION | MULTIDISCIPLINARY SCIENCES | INNATE | HEPARAN-SULFATE | CYTOKINE | CD14 | BINDING | CUTTING EDGE | TLR2 | Toll-Like Receptor 6 - metabolism | Cell Line | Cytokines - metabolism | Herpesvirus 1, Human - metabolism | Signal Transduction | Humans | Solubility | Monocytes - metabolism | NF-kappa B - metabolism | Monocytes - immunology | Recombinant Proteins | Toll-Like Receptor 2 - metabolism | Immunity, Innate | Lipopolysaccharide Receptors - metabolism | Viral Envelope Proteins - chemistry | Inflammation Mediators - metabolism | TNF Receptor-Associated Factor 6 - metabolism | Viral Envelope Proteins - metabolism | Protein Binding | Herpesvirus 1, Human - genetics | Herpesvirus 1, Human - immunology | Myeloid Differentiation Factor 88 - metabolism | Cell culture | Target recognition | Blocking antibodies | Herpes simplex | CD14 antigen | Recruitment | Proteins | Signal transduction | Coding | Virions | Pathogens | NF-κB protein | Glycoprotein B | Cytokines | Glycoprotein | Glycoproteins | Pattern recognition | TLR2 protein | Herpes viruses | MyD88 protein | Ligands | Kinetics | Kidney transplantation | Biotechnology | Laboratories | Interleukin | Antibodies | Viruses | Biology | Infections | Activation | Biological effects | Immunology | Toll-like receptors | Interleukin 8 | Immune system | Encephalitis | Cytomegalovirus | Immune response | TRAF6 protein | Incubation | Secretion | Inflammation | Virology | Signaling | Monocytes | TLR1 protein | Plasmids | Phenols | Chemokines
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8. Full Text Innate immune receptors over expression correlate with chronic chagasic cardiomyopathy and digestive damage in patients
by Pereira, Nathalie de Sena and Queiroga, Tamyres Bernadete Dantas and Nunes, Daniela Ferreira and Andrade, Cléber de Mesquita and Nascimento, Manuela Sales Lima and Do-Valle-Matta, Maria Adelaide and Câmara, Antônia Cláudia Jácome da and Galvão, Lúcia Maria da Cunha and Guedes, Paulo Marcos Matta and Chiari, Egler
PLoS Neglected Tropical Diseases, ISSN 1935-2727, 07/2018, Volume 12, Issue 7, p. e0006589
Chronic chagasic cardiomyopathy (CCC) is observed in 30% to 50% of the individuals infected by Trypanosoma cruzi and heart failure is the important cause of... 
C-REACTIVE PROTEIN | CLINICAL FORMS | VENTRICULAR-ARRHYTHMIAS | HEART-DISEASE | NUCLEAR-FACTOR | IFN-GAMMA | TRYPANOSOMA-CRUZI INFECTION | T-CELLS | PARASITOLOGY | TROPICAL MEDICINE | NALP3 INFLAMMASOME | CUTTING EDGE | Chagas Cardiomyopathy - immunology | Chagas Cardiomyopathy - parasitology | Chagas Cardiomyopathy - genetics | Interleukin-12 - genetics | Humans | Middle Aged | NLR Proteins - genetics | Interleukin-1beta - immunology | Male | Caspase 1 - immunology | Interleukin-1beta - genetics | NLR Proteins - immunology | Digestive System Diseases - genetics | Trypanosoma cruzi - physiology | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Caspase 1 - genetics | Interleukin-12 - immunology | Digestive System Diseases - immunology | Adult | Female | Aged | Digestive System Diseases - parasitology | Immune response | Cardiomyopathy | Cardiac patients | Patient outcomes | Genetic aspects | Research | Gene expression | Health aspects | Heart diseases | Risk factors | Protein binding | Heart | Pathogenesis | Funding | Leukocytes (mononuclear) | Interleukin 23 | Nucleotides | Immunity | Caspase-1 | Proteins | Signal transduction | Receptors | Pathogens | Antigens | Parasitology | Cytokines | Adapters | Interleukin 12 | Tumor necrosis factor-α | Patients | Interleukin 18 | TLR2 protein | Rectum | MyD88 protein | Chagas' disease | Ejection | Correlation | β-Interferon | Innate immunity | Lymphocytes T | Infections | Parasites | Interleukin 6 | Molecules | Lymphocytes | Toll-like receptors | Peripheral blood mononuclear cells | Supervision | Immune system | Vector-borne diseases | Protozoa | Caspase | Population decline | TLR1 protein | Lymphocytes B | Levels | Ventricle
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9. Full Text Toll-Like Receptor 1/2 and 5 Ligands Enhance the Expression of Cyclin D1 and D3 and Induce Proliferation in Mantle Cell Lymphoma
by Mastorci, Katy and Muraro, Elena and Pasini, Elisa and Furlan, Chiara and Sigalotti, Luca and Cinco, Marina and Dolcetti, Riccardo and Fratta, Elisabetta
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 4, p. e0153823
Mantle cell lymphoma (MCL) is an aggressive B-cell non-Hodgkin's lymphoma with a still undefined etiology. Several lines of evidence are consistent with the... 
PATHWAYS | BORRELIA-BURGDORFERI | ACTIVATION | CD40 | MICROENVIRONMENT | MULTIDISCIPLINARY SCIENCES | TUMOR | NON-HODGKIN-LYMPHOMA | INFECTION | NF-KAPPA-B | HUMAN MONOCYTES | Cyclin D1 - metabolism | Toll-Like Receptor 5 - metabolism | Humans | Interleukin-4 - metabolism | Toll-Like Receptor 1 - metabolism | Cell Proliferation - physiology | NF-kappa B - metabolism | Glycogen Synthase Kinase 3 - metabolism | Toll-Like Receptor 4 - metabolism | CD40 Ligand - metabolism | Lymphoma, Non-Hodgkin - metabolism | Cell Line, Tumor | Ligands | Signal Transduction - physiology | Proto-Oncogene Proteins c-akt - metabolism | Lymphoma, Mantle-Cell - metabolism | Cyclin D3 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Toll-like receptors | Cyclin D proteins | Research | Analysis | Mantle cell lymphoma | Immunoglobulin M | Biotechnology | CD40 antigen | Authorship | Pathogenesis | AKT protein | Infections | Activation | Kinases | Cyclin D1 | Drug resistance | CD40L protein | Cyclin D3 | Interleukin 6 | Proteins | Receptors | Interleukin 4 | Cell growth | Lymphocytes | Etiology | Immunotherapy | Cascades | Cell cycle | Post-translation | Mantle | Immune system | NF-κB protein | Cell survival | TLR5 protein | MAP kinase | Cultures | Gene expression | Lymphoma | Signaling | Overexpression | TLR1 protein | Lymphocytes B | Medical prognosis | TLR2 protein | Cell lines | Lymphomas | Stimuli | Cancer | Apoptosis
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10. Full Text Molecular modeling-based evaluation of hTLR10 and identification of potential ligands in toll-like receptor signaling
by Govindaraj, Rajiv Gandhi and Manavalan, Balachandran and Lee, Gwang and Choi, Sangdun
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 9, pp. 1 - 13
Toll-like receptors (TLRs) are pattern recognition receptors that recognize pathogens based on distinct molecular signatures. The human (h) TLR1, 2, 6 and 10... 
CELLS | STRUCTURAL BASIS | PROTEIN-DOCKING | SERVER | LEUCINE-RICH REPEATS | BIOLOGY | SIMULATIONS | EXPRESSION | BINDING | PATHOGEN RECOGNITION | GLYCOSYLATION SITES | Protein Structure, Tertiary | Amino Acid Sequence | Toll-Like Receptor 2 - genetics | Signal Transduction | Humans | Toll-Like Receptor 1 - metabolism | Models, Molecular | Molecular Sequence Data | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 10 - metabolism | Lipopeptides - metabolism | Toll-Like Receptor 1 - genetics | Sequence Alignment | Toll-Like Receptor 10 - genetics | Protein Binding | Toll-Like Receptor 10 - chemistry | Ligands | Lipopeptides - chemistry | Protein Stability | Binding Sites | Dimerization | Toll-Like Receptor 1 - chemistry | Toll-Like Receptor 2 - chemistry | Molecular dynamics | Structure | Analysis | Crystals | Protein binding | Binding | Computer simulation | Science | Homology | Biosynthesis | Pattern recognition | Molecular docking | Proteins | Signaling | Receptors | Molecular modelling | TLR1 protein | Simulation | TLR2 protein | Toll-like receptors | Modelling | Trends | Three dimensional models | Crystal structure | Immune system
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11. Full Text Investigation of interactions between TLR2, MyD88 and TIRAP by bioluminescence resonance energy transfer is hampered by artefacts of protein overexpression
by Sampaio, Natália G and Kocan, Martina and Schofield, Louis and Pfleger, Kevin D. G and Eriksson, Emily M
PLoS ONE, ISSN 1932-6203, 08/2018, Volume 13, Issue 8, p. e0202408
Toll like receptors (TLRs) are important pattern recognition receptors that can detect pathogen and danger associated molecular patterns to initiate an innate... 
SIGNAL-TRANSDUCTION | TRANSFER BRET | ACTIVATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTORS | INDUCTION | IDENTIFICATION | ADAPTER PROTEIN | BINDING | FAMILY | Gene Expression | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Humans | Receptors, Interleukin-1 - genetics | Myeloid Differentiation Factor 88 - genetics | Signal Transduction - genetics | Toll-Like Receptor 2 - metabolism | Membrane Glycoproteins - genetics | Receptors, Interleukin-1 - metabolism | Microscopy, Confocal | Signal Transduction - drug effects | HEK293 Cells | Fluorescence Resonance Energy Transfer | Myeloid Differentiation Factor 88 - metabolism | Cytokines | Gene expression | Research | Bioluminescence | Transcription factors | Immunoprecipitation | Confocal microscopy | Biology | Activation | Kinases | Recruitment | Proteins | Signal transduction | Receptors | Immunology | Transcription activation | Toll-like receptors | Artefacts | Immune system | Binding | Adaptor proteins | CRISPR | Medical research | NF-κB protein | Immune response | Cell walls | Hazards | Pattern recognition | Adaptors | Real time | Artifacts | TLR1 protein | Microscopy | TLR2 protein | Tagging | MyD88 protein | Ligands | Resonance | Dimers | Kinetics | Receptor mechanisms | Protein interaction | Lipopeptides | Energy transfer
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