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Science, ISSN 0036-8075, 9/2010, Volume 329, Issue 5998, pp. 1530 - 1534
Endosomal Toll-like receptors (TLRs) 7 and 9 recognize viral pathogens and induce signals leading to the activation of nuclear factor KB (NF-KB)-dependent... 
T lymphocytes | Molecules | Cytokines | Microscopy | Genes | REPORTS | Toll like receptors | Agonists | Macrophages | Cells | Endosomes | LOCALIZATION | ACTIVATION | DENDRITIC CELLS | TRAF3 | MULTIDISCIPLINARY SCIENCES | TLR9 | TOLL-LIKE-RECEPTOR-9 | INDUCTION | INTERFERON-ALPHA PRODUCTION | I INTERFERON | AP-3 ADAPTER | Recombinant Fusion Proteins - immunology | Dendritic Cells - immunology | Transcriptional Activation | Interferon Type I - immunology | Recombinant Fusion Proteins - metabolism | Endosomes - metabolism | Toll-Like Receptor 9 - immunology | Cytoplasmic Vesicles - metabolism | Interferon Type I - metabolism | Membrane Transport Proteins - metabolism | Cytokines - genetics | Dendritic Cells - metabolism | Macrophages - immunology | Cytokines - immunology | Oligodeoxyribonucleotides - immunology | Cytokines - metabolism | Signal Transduction | Vesicle-Associated Membrane Protein 3 - metabolism | Mice, Inbred C57BL | Cells, Cultured | TNF Receptor-Associated Factor 3 - metabolism | Adaptor Protein Complex beta Subunits | Interferon Regulatory Factor-7 - metabolism | Adaptor Protein Complex 3 - metabolism | Protein Transport | Animals | Adaptor Protein Complex 3 - genetics | Interferon Type I - genetics | Mice | Toll-Like Receptor 9 - metabolism | Myeloid Differentiation Factor 88 - metabolism | Lysosomal-Associated Membrane Protein 2 - metabolism | Cellular control mechanisms | Physiological aspects | TLR7 | Research | Health aspects | Immune system | Proteins | Signal transduction | Interferon | Cellular biology | Receptors | Compartments | Bifurcations | Activation
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2016, Volume 11, Issue 4, p. e0151967
Highly purified outer membrane vesicles (OMVs) of the periodontal pathogens, Porphyromonas gingivalis, Treponema denticola and Tannerella forsythia were... 
PROTEINASE-ADHESIN COMPLEXES | BACTEROIDES-GINGIVALIS | BACTERIA | TREPONEMA-DENTICOLA | EPITHELIAL-CELLS | SOLUBLE PEPTIDOGLYCAN | PORPHYROMONAS-GINGIVALIS | DNA | MULTIDISCIPLINARY SCIENCES | RELEASE | VIRULENCE | Cell Line | Receptors, Pattern Recognition - isolation & purification | Bacterial Outer Membrane Proteins - immunology | Bacteroides - chemistry | Receptors, Pattern Recognition - immunology | Receptors, Pattern Recognition - chemistry | Bacteroides - immunology | Humans | Treponema denticola - immunology | Bacterial Outer Membrane Proteins - chemistry | Nod2 Signaling Adaptor Protein - immunology | Porphyromonas gingivalis - chemistry | Toll-Like Receptor 4 - immunology | Nod1 Signaling Adaptor Protein - immunology | Bacterial Outer Membrane Proteins - isolation & purification | Treponema denticola - chemistry | Chronic Periodontitis - microbiology | Lipopolysaccharides - analysis | Chronic Periodontitis - immunology | Toll-Like Receptor 2 - immunology | Peptidoglycan - analysis | Porphyromonas gingivalis - immunology | Virulence (Microbiology) | Periodontitis | Analysis | Risk factors | Pattern recognition receptors | Cell culture | Dental schools | Disease | Lipids | Ultracentrifugation | Nanoparticles | Proteins | Signal transduction | Receptors | Vesicles | Ultrafiltration | Rodents | Toll-like receptors | Bacteria | Nod1 protein | Deoxyribonucleic acid--DNA | NOD2 protein | Pathogens | Enzymes | Enumeration | TLR7 protein | Light scattering | Peptidoglycans | Membrane vesicles | Inflammation | Nucleic acids | Pattern recognition | TLR4 protein | Fatty acids | TLR9 protein | Cytometry | Biofilms | Oral hygiene | TLR2 protein | Deoxyribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 5, p. e19664
T-cell immunoglobulin and mucin domain-containing protein 3 (Tim-3) is a newly identified negative immunomodulator that is up-regulated on dysfunctional T... 
ACTIVATION | DENDRITIC CELLS | MULTIDISCIPLINARY SCIENCES | ELEVATED FREQUENCIES | HEPATITIS-C-VIRUS | CD4(+) | T-CELL IMMUNOGLOBULIN | GENE FAMILY | CORE PROTEIN | CONTAINING MOLECULE-3 TIM-3 | PD-1 EXPRESSION | Up-Regulation | Phosphorylation | Hepacivirus - immunology | Coculture Techniques | Humans | Middle Aged | Hepatitis C, Chronic - virology | Suppressor of Cytokine Signaling 1 Protein | Male | Monocytes - metabolism | Hepatocytes - metabolism | Complement C1q - metabolism | Antigens, CD - metabolism | Lipopolysaccharide Receptors - metabolism | STAT1 Transcription Factor - metabolism | Mitochondrial Proteins - metabolism | Viral Core Proteins - metabolism | Adult | Membrane Proteins - metabolism | Interleukin-12 Subunit p40 - biosynthesis | Interleukin-12 Subunit p40 - metabolism | Signal Transduction | Macrophage Activation - immunology | Down-Regulation | Gene Silencing | Suppressor of Cytokine Signaling Proteins - genetics | Hepatitis A Virus Cellular Receptor 2 | Apoptosis Regulatory Proteins - metabolism | Macrophages - metabolism | Carrier Proteins - metabolism | Hepatitis C, Chronic - immunology | Hepatocytes - virology | Protein Binding | Programmed Cell Death 1 Receptor | Infection | Medical research | Cytokines | Immunotherapy | Medicine, Experimental | T cells | Hepatitis C virus | Gene expression | Health aspects | Mitogens | Health care | Multiple sclerosis | PD-1 protein | Veterans | Crosstalk | Lymphocytes T | Arthritis | Infections | Macrophages | CD14 antigen | Lipopolysaccharides | Proteins | Hepatitis | Lymphocytes | Toll-like receptors | Inhibition | Stat1 protein | Immune system | Antigens | Immunoglobulins | Immune response | TLR7 protein | Internal medicine | Immunoregulation | Secretion | Melanoma | Blocking | Interleukin 12 | T cell receptors | Inflammation | TLR4 protein | Medicine | Complement component C1q | Signaling | Monocytes | Hospitals | Infectious diseases | Mucin | Core protein | Hepatocytes | Ligands | Human behavior | Viral infections
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e103609
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2007, Volume 127, Issue 2, pp. 331 - 341
Keratinocytes are continuously in contact with external stimuli and have the capacity to produce several soluble mediators. Pathogen-associated molecular... 
TOLL-LIKE-RECEPTORS | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | SKIN-ASSOCIATED CHEMOKINE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INNATE IMMUNE-RESPONSE | INFLAMMATORY PROTEIN 3-ALPHA | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | HUMAN EPIDERMAL-KERATINOCYTES | DERMATOLOGY | Oligonucleotides - genetics | Phosphorylation | Toll-Like Receptor 5 - genetics | Humans | Toll-Like Receptor 9 - genetics | I-kappa B Proteins - metabolism | RNA, Messenger - metabolism | Flagellin - pharmacology | Tissue Distribution | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Biological Transport | Oligonucleotides - pharmacology | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Chemokine CXCL9 | Toll-Like Receptor 5 - metabolism | Poly I-C - pharmacology | Cytokines - metabolism | Cells, Cultured | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Chemokine CXCL10 | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Toll-Like Receptors - genetics | Chemokines, CXC - metabolism | Lipopolysaccharides - pharmacology | CpG Islands | Ligands | Chemokines - metabolism | Toll-Like Receptor 9 - metabolism | CD40 antigen | Oligonucleotides | Flagellin | mRNA | intercellular adhesion molecule 1 | Immunity | Lipopolysaccharides | CXC chemokines | CCL20 protein | Toll-like receptors | CpG islands | TLR7 protein | Dermatology | Keratinocytes | Nuclear transport | Stress | TLR3 protein | TLR9 protein | TLR1 protein | External stimuli | CXCL10 protein | Histocompatibility antigen HLA | Interferon | Internet | Monocyte chemoattractant protein 1 | Tumor necrosis factor- alpha
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2011, Volume 6, Issue 1, p. e15324
textabstractChronic hepatitis B virus (HBV) infection is caused by inadequate anti-viral immunity. Activation of plasmacytoid dendritic cells (pDC) leading to... 
NATURAL-KILLER | BIOLOGY | GENE-EXPRESSION | RECEPTOR | C VIRUS | IMMUNOLOGY | NONPARENCHYMAL LIVER-CELLS | HBV | IFN-ALPHA PRODUCTION | VIRAL-INFECTION | I INTERFERON | Cell Communication - immunology | Dendritic Cells - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | Cells, Cultured | Viral Proteins - pharmacology | Monocytes - immunology | Dendritic Cells - virology | Hepatitis B virus - immunology | Immunity | Tumor Necrosis Factor-alpha - biosynthesis | Case-Control Studies | Medical research | Antiviral agents | Dendritic cells | Research | Genetic transcription | Proteins | Infection | Killer cells | Medicine, Experimental | Hepatitis B virus | Comparative analysis | Health aspects | Hepatitis B | TOR protein | Animal models | Phosphorylation | Transcription | Liver | Chronic infection | Viruses | Infections | Activation | Interleukin 6 | Hepatitis | Cell activation | Hepatology | Gastroenterology | Toll-like receptors | IP-10 protein | Natural killer cells | Active control | Deoxyribonucleic acid--DNA | CpG islands | Antigens | TLR7 protein | Hepatitis B surface antigen | Cytokines | Tumor necrosis factor-α | Gene expression | Patients | TLR9 protein | Hepatitis B e antigen | Interferon regulatory factor 7 | Immunosuppression | Monocytes | Lymphocytes B | Influenza | Ligands | Interferon | Viral infections | Deoxyribonucleic acid | DNA
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