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1. Full Text Tumor necrosis factor‐α antagonists and neuropathy
by Stübgen, Joerg‐Patrick
Muscle & nerve, ISSN 0148-639X, 03/2008, Volume 37, Issue 3, pp. 281 - 292
TNF‐α antagonists | TNF‐α | neuropathy | TNF-α | TNF-α antagonists | Neuropathy | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cranial nerves. Spinal roots. Peripheral nerves. Autonomic nervous system. Gustation. Olfaction | Neurology | Biological and medical sciences | Multiple sclerosis and variants. Guillain barré syndrome and other inflammatory polyneuropathies. Leukoencephalitis | Medical sciences | Nervous system (semeiology, syndromes) | Tumor Necrosis Factor-alpha - metabolism | Anti-Infective Agents - therapeutic use | Polyneuropathies - drug therapy | Models, Biological | Humans | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
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2. Full Text Microglial derived tumor necrosis factor-α drives Alzheimer's disease-related neuronal cell cycle events
by Bhaskar, Kiran and Maphis, Nicole and Xu, Guixiang and Varvel, Nicholas H and Kokiko-Cochran, Olga N and Weick, Jason P and Staugaitis, Susan M and Cardona, Astrid and Ransohoff, Richard M and Herrup, Karl and Lamb, Bruce T
Neurobiology of disease, ISSN 0969-9961, 2013, Volume 62, pp. 273 - 285
Neurology | Neuronal cell cycle | Neuroinflammation | Tumor necrosis factor-α (TNFα) | Alzheimer's disease | c-Jun Kinase (JNK) | Adoptive transfer | Microglia | C-Jun Kinase (JNK) | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Microglia - metabolism | Frontal Lobe - metabolism | Microglia - drug effects | Amyloid beta-Peptides - pharmacology | Humans | Mice, Inbred C57BL | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Mice, Transgenic | Temporal Lobe - metabolism | Animals | Cell Cycle | Alzheimer Disease - metabolism | Mice | Neurons - metabolism | Index Medicus | Alzheimer’s disease | neuroinflammation | tumor necrosis factor-α (TNFα) | adoptive transfer | microglia | neuronal cell cycle
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3. Full Text Tumour necrosis factor-α as a tumour promoter
by Szlosarek, Peter and Charles, Kellie A and Balkwill, Frances R
European journal of cancer (1990), ISSN 0959-8049, 2006, Volume 42, Issue 6, pp. 745 - 750
TNF-α | Cancer | Life Sciences & Biomedicine | Oncology | Science & Technology | Inflammation - complications | Inflammation - pathology | Animals | Neoplasms - etiology | Humans | Tumor Necrosis Factor-alpha - physiology | Mice, Transgenic | Models, Animal | Mice | Neoplasms - pathology | Chronic Disease | Necrosis - pathology | Index Medicus
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4. Full Text Tumor necrosis factor α suppresses the mesenchymal stem cell osteogenesis promoter miR‐21 in estrogen deficiency–induced osteoporosis
by Yang, Nan and Wang, Guang and Hu, Chenghu and Shi, Yuanyuan and Liao, Li and Shi, Songtao and Cai, Yan and Cheng, Shuli and Wang, Xi and Liu, Yali and Tang, Liang and Ding, Yin and Jin, Yan
Journal of bone and mineral research, ISSN 0884-0431, 03/2013, Volume 28, Issue 3, pp. 559 - 573
OSTEOGENESIS | miR‐21 | TNF‐α | MESENCHYMAL STEM CELL | OSTEOPOROSIS | miR-21 | TNF-α | Enzyme-Linked Immunosorbent Assay | Ovariectomy | Down-Regulation | Humans | Mesenchymal Stromal Cells - metabolism | Mesenchymal Stromal Cells - cytology | Animals | MicroRNAs | Tumor Necrosis Factor-alpha - physiology | Female | Cell Differentiation | Mice | Osteoblasts - cytology | Osteoblasts - metabolism | Osteogenesis - genetics | Index Medicus | Cytokines | Mesenchyme | Estrogens | Inflammation | Bone diseases | Inflammatory diseases | Promoters | Osteoporosis | Osteoblastogenesis | Stem cells | miRNA | Osteogenesis | Tumor necrosis factor- alpha
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5. Full Text Behavior of Tumor Necrosis Factor-α and Tumor Necrosis Factor Receptor 1/Tumor Necrosis Factor Receptor 2 System in Mononuclear Cells Recovered From Peritoneal Fluid of Women With Endometriosis at Different Stages
by Salmeri, Francesca M and Laganà, Antonio S and Sofo, Vincenza and Triolo, Onofrio and Sturlese, Emanuele and Retto, Giovanni and Pizzo, Alfonsa and D'Ascola, Angela and Campo, Salvatore
Reproductive sciences (Thousand Oaks, Calif.), ISSN 1933-7191, 2/2015, Volume 22, Issue 2, pp. 165 - 172
TNFR2 | Endometriosis | TNF-α | PFMCs | TNFR1 | Life Sciences & Biomedicine | Obstetrics & Gynecology | Reproductive Biology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Cell Proliferation | Endometriosis - genetics | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Receptors, Tumor Necrosis Factor, Type I - metabolism | RNA, Messenger - metabolism | Case-Control Studies | Young Adult | Ascitic Fluid - metabolism | Receptors, Tumor Necrosis Factor, Type II - metabolism | Adult | Female | Transcription, Genetic | Ascitic Fluid - cytology | Severity of Illness Index | Signal Transduction | Cells, Cultured | Endometriosis - pathology | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Disease Progression | Endometriosis - metabolism | Apoptosis | Index Medicus | endometriosis | Original
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6. Full Text Baicalin prevents tumor necrosis factor‐α−induced apoptosis and dysfunction of pancreatic β‐cell line Min6 via upregulation of miR‐205
by You, Wenjun and Wang, Kun and Yu, Changhong and Song, Lijuan
Journal of cellular biochemistry, ISSN 0730-2312, 10/2018, Volume 119, Issue 10, pp. 8547 - 8554
baicalin (BAI) | Min6 cell | tumor necrosis factor‐α (TNF‐α) | miR‐205 | type 1 diabetes mellitus (T1DM) | miR-205 | tumor necrosis factor-α (TNF-α) | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Tumor Necrosis Factor-alpha - metabolism | Cell Survival - drug effects | Up-Regulation | Apoptosis - drug effects | Plant Extracts - pharmacology | Pancreatic Neoplasms - pathology | MicroRNAs - metabolism | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Insulin-Secreting Cells - metabolism | Animals | Analysis of Variance | Signal Transduction - drug effects | Cell Line, Tumor | Insulinoma - pathology | Flavonoids - pharmacology | Mice | Proto-Oncogene Proteins c-akt - metabolism | Scutellaria baicalensis - chemistry | AKT protein | Propidium iodide | Diabetes mellitus (insulin dependent) | Activation | Kinases | Inactivation | Anticancer properties | Proteins | Bcl-x protein | Radioimmunoassay | Tumor necrosis factor-TNF | miRNA | Inhibition | Pancreas | Baicalin | Deactivation | Iodides | Secretion | Diabetes mellitus | Caspase | Isothiocyanate | Inflammation | Ribonucleic acid--RNA | Insulin | Lymphoma | Nitric-oxide synthase | 1-Phosphatidylinositol 3-kinase | Beta cells | Polymerase chain reaction | Tumor necrosis factor | Nitric oxide | Annexin V | Diabetes | Viability | Apoptosis | Tumors | Index Medicus
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7. Full Text A Paracrine Loop Between Adipocytes and Macrophages Aggravates Inflammatory Changes: Role of Free Fatty Acids and Tumor Necrosis Factor α
by Suganami, Takayoshi and Nishida, Junko and Ogawa, Yoshihiro
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 10/2005, Volume 25, Issue 10, pp. 2062 - 2068
Adipocyte | Obesity | TNF-α | Fatty acids | Macrophage | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Heart | Toxicology | Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous | Atherosclerosis (general aspects, experimental research) | Biological and medical sciences | Medical sciences | Alcoholism and acute alcohol poisoning | Coronary heart disease | Blood and lymphatic vessels | Tumor Necrosis Factor-alpha - metabolism | Cell Communication - immunology | Inflammation - pathology | Obesity - immunology | Coculture Techniques | Tumor Necrosis Factor-alpha - genetics | Adipocytes - cytology | Metabolic Syndrome - metabolism | Adipose Tissue - immunology | Paracrine Communication - immunology | Adipose Tissue - metabolism | Fatty Acids, Nonesterified - metabolism | Inflammation - metabolism | Chemokine CCL2 - metabolism | Metabolic Syndrome - immunology | Adipocytes - immunology | Macrophages - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Gene Expression Regulation - immunology | Adipose Tissue - pathology | Chemokine CCL2 - genetics | Inflammation - immunology | Macrophages - cytology | 3T3-L1 Cells | Atherosclerosis - metabolism | Obesity - metabolism | Obesity - pathology | Macrophages - metabolism | Animals | Adipocytes - metabolism | Mice | Metabolic Syndrome - pathology | Hypertrophy | Index Medicus
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8. Full Text PD‐L1 expression enhancement by infiltrating macrophage‐derived tumor necrosis factor‐α leads to poor pancreatic cancer prognosis
by Tsukamoto, Masayo and Imai, Katsunori and Ishimoto, Takatsugu and Komohara, Yoshihiro and Yamashita, Yo‐ichi and Nakagawa, Shigeki and Umezaki, Naoki and Yamao, Takanobu and Kitano, Yuki and Miyata, Tatsunori and Arima, Kota and Okabe, Hirohisa and Baba, Yoshifumi and Chikamoto, Akira and Ishiko, Takatoshi and Hirota, Masahiko and Baba, Hideo
Cancer science, ISSN 1347-9032, 01/2019, Volume 110, Issue 1, pp. 310 - 320
macrophage | pancreatic cancer | tumor stroma | TNF‐α | PD‐L1 | PD-L1 | TNF-α | Life Sciences & Biomedicine | Oncology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Pancreatic Neoplasms - metabolism | Prognosis | Macrophages - pathology | Humans | Middle Aged | Pancreatic Neoplasms - pathology | Tumor Necrosis Factor-alpha - genetics | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Kaplan-Meier Estimate | Male | Pancreatic Neoplasms - genetics | B7-H1 Antigen - genetics | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - genetics | B7-H1 Antigen - metabolism | Macrophages - metabolism | Cell Line, Tumor | Female | Aged | Adenocarcinoma | Cell culture | Flow cytometry | Antigens | Immunoglobulins | Cell survival | Cytokines | Therapeutic applications | CD8 antigen | Stroma | Macrophages | Metastases | Proteins | Tumor necrosis factor | Immunotherapy | Medical prognosis | Pancreatic cancer | Tumor necrosis factor-TNF | Index Medicus | Original
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