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Journal of Neuroimmunology, ISSN 0165-5728, 2007, Volume 194, Issue 1, pp. 54 - 61
Abstract Interleukin (IL)-17-producing helper T cells may play a pivotal role in the pathogenesis of multiple sclerosis. Here, we examined the effects of IL-17... 
Neurology | Allergy and Immunology | EAE | Cytokine | IL-17 | Microglia | cytokine | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MULTIPLE-SCLEROSIS LESIONS | MESSENGER-RNA EXPRESSION | NEUROTROPHIC FACTOR | IMMUNOLOGY | NEUROSCIENCES | NERVE GROWTH-FACTOR | INTERLEUKIN-17 | NEURONAL DEATH | NITRIC-OXIDE | TNF-ALPHA | microglia | T-CELLS | Brain-Derived Neurotrophic Factor - genetics | Microglia - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Interleukin-17 - physiology | Receptors, Interleukin-17 - genetics | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | Interleukin-23 - genetics | RNA, Messenger - biosynthesis | Chemokine CXCL2 - genetics | Brain-Derived Neurotrophic Factor - biosynthesis | Interleukin-1beta - biosynthesis | Multiple Sclerosis - metabolism | Autocrine Communication | Nitric Oxide - biosynthesis | Cells, Cultured - drug effects | Interleukin-6 - genetics | Microglia - drug effects | Receptors, Interleukin-17 - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - genetics | Interleukin-17 - biosynthesis | Recombinant Proteins - pharmacology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Interleukin-23 - biosynthesis | Interleukin-6 - biosynthesis | Cells, Cultured - metabolism | Chemokine CXCL2 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Multiple sclerosis | Interleukins | T cells | Nitric oxide
Journal Article
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, ISSN 1422-0067, 07/2019, Volume 20, Issue 13, p. 3262
Chenopodium quinoa Wild is a pseudocereal grain which attracts a lot of attention in the scientific community as it has a positive effect on health. Here, we... 
nitric oxide | TNF-alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | CAT-2B | COLON | CHEMISTRY, MULTIDISCIPLINARY | IL-6 | METHYL-ESTER | PHYTOCHEMICALS | inflammation | COLORECTAL-CANCER | ENZYMES | PLANTS | l-arginine | INFLAMMATORY RESPONSE | oxidative stress | TNF-α | l -arginine
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 12/2015, Volume 52, Issue 3, pp. 1547 - 1560
In this study, we tried to explore the molecular mechanism that Corilagin protected against herpes simplex virus-1 encephalitis through inhibiting the TLR2... 
Signaling pathways | Neurology | Neurosciences | Biomedicine | Toll-like receptor 2 | Neurobiology | Corilagin | Inflammation | Herpes simplex virus-1 | Cell Biology | ACTIVATION | INNATE IMMUNE-SYSTEM | MICROGLIAL CELLS | NEUROSCIENCES | ETHANOL EXTRACT | RESPONSES | HERPES-SIMPLEX-VIRUS | TOLL-LIKE RECEPTORS | INFECTION | TNF-ALPHA | PATTERN-RECOGNITION RECEPTORS | Microglia - metabolism | RNA, Small Interfering - genetics | TNF Receptor-Associated Factor 6 - biosynthesis | Toll-Like Receptor 2 - antagonists & inhibitors | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - biosynthesis | Receptors, Interleukin-1 - genetics | Tumor Necrosis Factor-alpha - genetics | Male | Brain - metabolism | RNA, Messenger - biosynthesis | Protein Processing, Post-Translational - drug effects | RNA Interference | Nerve Tissue Proteins - biosynthesis | Hydrolyzable Tannins - pharmacology | Encephalitis, Herpes Simplex - prevention & control | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | TNF Receptor-Associated Factor 6 - genetics | Intracellular Signaling Peptides and Proteins - genetics | Lipopeptides - toxicity | Antiviral Agents - pharmacology | Glucosides - pharmacology | Interleukin-6 - genetics | Microglia - drug effects | RNA, Messenger - genetics | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Receptors, Interleukin-1 - biosynthesis | Down-Regulation - drug effects | Intracellular Signaling Peptides and Proteins - biosynthesis | Nerve Tissue Proteins - genetics | Toll-Like Receptor 2 - biosynthesis | Herpesvirus 1, Human | Membrane Glycoproteins - genetics | Animals | NF-kappa B - genetics | Signal Transduction - drug effects | NF-kappa B - biosynthesis | Brain - pathology | Interleukin-6 - biosynthesis | Mice | Mice, Inbred BALB C | Myeloid Differentiation Factor 88 - biosynthesis | Tumor Necrosis Factor-alpha - biosynthesis | RNA | Yuan (China) | Encephalitis | Signaling | Tumor necrosis factor-TNF | Molecular biology | Neurons
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2008, Volume 181, Issue 10, pp. 7420 - 7427
Journal Article
Journal Article
Journal Article
PLoS Genetics, ISSN 1553-7390, 2014, Volume 10, Issue 6, p. e1004368
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, p. e0167420
Substance P (SP) is a neuropeptide that mediates many physiological as well as inflammatory responses. Recently, SP has been implicated in the resolution of... 
HEME OXYGENASE-1 EXPRESSION | CYTOKINE PRODUCTION | INHIBITION | CARBON-MONOXIDE | PROLIFERATOR-ACTIVATED RECEPTORS | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | TNF-ALPHA | NF-KAPPA-B | HUMAN MONOCYTES | UP-REGULATION | Inflammation - chemically induced | Inflammation - pathology | Nitric Oxide Synthase Type II - biosynthesis | Humans | Periodontal Ligament - drug effects | Periodontal Ligament - metabolism | Transcription Factor RelA - genetics | Heme Oxygenase-1 - genetics | Inflammation - drug therapy | Nitric Oxide - biosynthesis | Heme Oxygenase-1 - biosynthesis | Lipopolysaccharides - toxicity | Membrane Proteins - genetics | Heme Oxygenase-1 - antagonists & inhibitors | Periodontal Ligament - pathology | Gene Expression Regulation - drug effects | Membrane Proteins - biosynthesis | Animals | Membrane Proteins - antagonists & inhibitors | Transcription Factor RelA - biosynthesis | Inflammation - genetics | Interleukin-6 - biosynthesis | Interleukin-10 - biosynthesis | Macrophages - drug effects | RAW 264.7 Cells | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Substance P - administration & dosage | Analysis | Physiological aspects | Inflammation | Genetic aspects | Research | Macrophages | Gene expression | Oxidative stress | Transcription factors | Arginase | Kinases | Lipopolysaccharides | Prostaglandin endoperoxide synthase | Interleukin 6 | Proteins | Genotype & phenotype | Phagocytes | Interleukin 4 | Periodontal ligament | Rodents | Penicillin | Carbon monoxide | Infrared imaging systems | Immune system | NF-κB protein | Immune response | Cytokines | Substance P | Tumor necrosis factor-α | Nitric-oxide synthase | Nitric oxide | Interleukin 10 | Insulin resistance | Cyclooxygenase-2 | Viability | Cell migration | Cytoplasm | Apoptosis
Journal Article
Circulation Research, ISSN 0009-7330, 08/2009, Volume 105, Issue 4, pp. 353 - 364
Journal Article
Environmental Toxicology and Pharmacology, ISSN 1382-6689, 2015, Volume 39, Issue 3, pp. 1081 - 1090
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 12/2017, Volume 113, pp. 304 - 310
Journal Article