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International Immunopharmacology, ISSN 1567-5769, 09/2011, Volume 11, Issue 9, pp. 1166 - 1172
(AS), an herb used in traditional Chinese medicine, is thought to have anti-inflammatory activities. Ligustilide is its most abundant ingredient. This study... 
Mitogen-activated protein kinase | Activator protein-1 | Ligustilide | Nitric oxide | Nuclear factor κB | ACTIVATION | NITRIC-OXIDE SYNTHASE | DYSMENORRHEA | IMMUNOLOGY | ENDOMETRIOSIS | Nuclear factor kappa B | ISOBENZOFURANONE | PATHOGENESIS | ANTIOXIDANT | PHARMACOLOGY & PHARMACY | TNF-ALPHA | ANGELICA-SINENSIS | PROSTAGLANDIN E-2 | Nitric Oxide Synthase Type II - biosynthesis | Reactive Oxygen Species - metabolism | Transcription Factor RelA - antagonists & inhibitors | Dinoprostone - biosynthesis | Transcription Factor AP-1 - genetics | Transcription Factor AP-1 - metabolism | Transcription Factor RelA - genetics | Mitogen-Activated Protein Kinase Kinases - metabolism | I-kappa B Kinase - metabolism | I-kappa B Kinase - antagonists & inhibitors | Nitric Oxide Synthase Type II - antagonists & inhibitors | MAP Kinase Kinase 4 - antagonists & inhibitors | Phosphorylation - drug effects | Cell Line | Nitric Oxide - biosynthesis | Nitric Oxide - antagonists & inhibitors | 4-Butyrolactone - pharmacology | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | I-kappa B Kinase - genetics | Down-Regulation - drug effects | Macrophages - enzymology | Macrophages - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Transcription Factor AP-1 - antagonists & inhibitors | Animals | MAP Kinase Signaling System - drug effects | Nitric Oxide Synthase Type II - genetics | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | 4-Butyrolactone - analogs & derivatives | Dinoprostone - antagonists & inhibitors | Tumor Necrosis Factor-alpha - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus
Journal Article
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 03/2017, Volume 24, Issue 3, pp. 481 - 491
Peptido-mimetic inhibitor of apoptosis protein (IAP) antagonists (Smac mimetics (SMs)) can kill tumour cells by depleting endogenous IAPs and thereby inducing... 
TUMOR-NECROSIS-FACTOR | INTERFERON-GAMMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | ALPHA-DEPENDENT APOPTOSIS | RECEPTOR | FAS-MEDIATED APOPTOSIS | TNF-ALPHA | NF-KAPPA-B | SIGNALING COMPLEXES | EXPRESSION | CELL BIOLOGY | Protein Kinases - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Caspase Inhibitors - pharmacology | Apoptosis - drug effects | Humans | Caspase 8 - metabolism | Caspase 8 - chemistry | Cytokine TWEAK - pharmacology | Interferon-gamma - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Recombinant Proteins - biosynthesis | Caspase 8 - genetics | Recombinant Proteins - isolation & purification | Receptors, Tumor Necrosis Factor, Type I - deficiency | Inhibitor of Apoptosis Proteins - antagonists & inhibitors | Pentanoic Acids - pharmacology | Inhibitor of Apoptosis Proteins - metabolism | Protein Kinases - deficiency | Interferon-gamma - genetics | Cell Line | Caspase 10 - metabolism | Recombinant Proteins - pharmacology | Receptors, Tumor Necrosis Factor, Type I - genetics | CRISPR-Cas Systems - genetics | Mice, Knockout | Drug Synergism | HT29 Cells | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Caspase 10 - genetics | Caspase 10 - chemistry | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Interferon-gamma - pharmacology | Index Medicus | Original Paper
Journal Article
The Journal of Immunology, ISSN 0022-1767, 06/2002, Volume 168, Issue 12, pp. 6404 - 6411
Previous studies have shown that IL-10 can induce the expression of the suppressor of cytokine signaling 3 (SOCS-3) mRNA in human monocytes and neutrophils,... 
EXPERIMENTAL ENDOTOXEMIA | TUMOR-NECROSIS-FACTOR | CHRONIC ENTEROCOLITIS | HUMAN NEUTROPHILS | MESSENGER-RNA | INTERFERON-GAMMA | NEGATIVE REGULATION | INTERLEUKIN-10 RECEPTOR | TNF-ALPHA | IMMUNOLOGY | HUMAN MONOCYTES | Sialoglycoproteins - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Repressor Proteins | Tumor Necrosis Factor-alpha - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Nitric Oxide Synthase Type II | RNA, Messenger - biosynthesis | Sialoglycoproteins - biosynthesis | Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | Interleukin-6 - metabolism | Nitric Oxide - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Interleukin-6 - genetics | Macrophage Activation - immunology | Macrophages, Peritoneal - immunology | Signal Transduction - genetics | Mice, Knockout | Macrophages - metabolism | Up-Regulation - immunology | Lipopolysaccharides - pharmacology | Mice | Transcription Factors | Macrophages, Peritoneal - metabolism | Tumor Necrosis Factor-alpha - biosynthesis | Macrophage Activation - genetics | Nitric Oxide Synthase - antagonists & inhibitors | Nitric Oxide Synthase - genetics | MAP Kinase Signaling System - immunology | Interleukin-10 - physiology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Transfection | RNA, Messenger - antagonists & inhibitors | Nitric Oxide Synthase - biosynthesis | Macrophages - immunology | Cell Line | Proteins - physiology | Nitric Oxide - antagonists & inhibitors | Mice, Inbred C57BL | Cells, Cultured | Up-Regulation - genetics | Proteins - genetics | Suppressor of Cytokine Signaling 3 Protein | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - antagonists & inhibitors | Interleukin 1 Receptor Antagonist Protein | Trans-Activators - metabolism | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Receptors, Interleukin-1 - antagonists & inhibitors | Interleukin-10 - pharmacology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 12/2010, Volume 207, Issue 3, pp. 343 - 353
Visceral adipocytes and associated macrophages produce and release excessive amounts of biologically active inflammatory cytokines via the portal and systemic... 
TOLL-LIKE RECEPTOR-3 | SKELETAL-MUSCLE | NONCANONICAL WNT5A | TYROSINE PHOSPHORYLATION | ENDOCRINOLOGY & METABOLISM | NECROSIS-FACTOR-ALPHA | TNF-ALPHA | SATURATED FATTY-ACIDS | SERINE PHOSPHORYLATION | INSULIN-RECEPTOR SUBSTRATE-1 | ADIPOSE-TISSUE | Leptin - antagonists & inhibitors | Interleukin-6 - antagonists & inhibitors | Adipocytes - drug effects | Lipopolysaccharides - antagonists & inhibitors | Nitric Oxide Synthase Type II - antagonists & inhibitors | Palmitates - pharmacology | Inflammation Mediators - antagonists & inhibitors | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Methimazole - analogs & derivatives | Interleukin-1beta - antagonists & inhibitors | Insulin Receptor Substrate Proteins - analysis | NF-kappa B - antagonists & inhibitors | Methimazole - pharmacology | Insulin Resistance | Chemokine CXCL10 - antagonists & inhibitors | 3T3-L1 Cells | Interferon-beta - antagonists & inhibitors | Up-Regulation - drug effects | Suppressor of Cytokine Signaling 3 Protein | Insulin - metabolism | Macrophages - metabolism | Animals | Adipocytes - metabolism | Suppressor of Cytokine Signaling Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - analysis | Macrophages - drug effects | Mice | Palmitates - metabolism | Thiones - pharmacology | AKT protein | Index Medicus
Journal Article
Journal of Immunological Methods, ISSN 0022-1759, 10/2011, Volume 373, Issue 1-2, pp. 229 - 239
A cell-based assay has been developed for the quantification of the activity of TNFα antagonists based on human erythroleukemic K562 cells transfected with a... 
TNF-alpha antagonists | Crohn's disease | Neutralizing antibodies | Cell-based assays | Rheumatoid arthritis | Reporter-gene | RHEUMATOID-ARTHRITIS | CROHNS-DISEASE | BIOCHEMICAL RESEARCH METHODS | MONOCLONAL-ANTIBODY | IMMUNOLOGY | BIOASSAY | TUMOR-NECROSIS-FACTOR | IMMUNOGENICITY | THERAPEUTIC PROTEINS | BIOLOGICAL-ACTIVITY | INFLIXIMAB | INTERFERONS | Antibodies, Neutralizing - analysis | Gene Expression Regulation, Enzymologic - drug effects | Luciferases - metabolism | Luciferases, Renilla - genetics | Arthritis, Rheumatoid - blood | Humans | Recombinant Fusion Proteins - metabolism | Luciferases - genetics | Antibodies, Monoclonal - blood | Dose-Response Relationship, Drug | Serum - immunology | Antibodies, Neutralizing - immunology | Infliximab | Luciferases, Renilla - metabolism | Arthritis, Rheumatoid - drug therapy | Antibodies, Monoclonal, Humanized - blood | Antibodies, Monoclonal, Humanized - pharmacology | Tumor Necrosis Factor-alpha - immunology | Culture Media - chemistry | Antibodies, Monoclonal - immunology | Luciferases, Firefly - metabolism | Antibodies, Monoclonal - pharmacology | Regulatory Sequences, Nucleic Acid - genetics | Tumor Necrosis Factor-alpha - pharmacology | Animals | NF-kappa B - genetics | Crohn Disease - blood | Crohn Disease - drug therapy | Culture Media - pharmacology | K562 Cells | Recombinant Fusion Proteins - genetics | Luciferases, Firefly - genetics | Adalimumab | Antibodies, Monoclonal, Humanized - immunology | Antibodies, Neutralizing - blood | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Serum - chemistry | Index Medicus
Journal Article