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Journal of Autoimmunity, ISSN 0896-8411, 2011, Volume 37, Issue 3, pp. 190 - 197
Abstract One way for intravenous Ig (IVIg) to affect responses of the B cells might be to operate through their TLR7 and TLR9. We confirm the ability of TLR... 
Allergy and Immunology | Intravenous immunoglobulin | B lymphocyte | TLR9 | Autoimmune diseases | SYSTEMIC-LUPUS-ERYTHEMATOSUS | ACTIVATION | KAPPA-B | IMMUNOLOGY | PROINFLAMMATORY CYTOKINE PRODUCTION | FC-GAMMA-RIIB | IN-VITRO | NEGATIVE REGULATION | TOLL-LIKE RECEPTORS | UP-REGULATION | BACTERIAL-DNA | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - immunology | Humans | Toll-Like Receptor 9 - genetics | Molecular Sequence Data | Sialic Acid Binding Ig-like Lectin 2 - immunology | Toll-Like Receptor 9 - agonists | Toll-Like Receptor 9 - immunology | Signal Transduction - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Sialic Acid Binding Ig-like Lectin 2 - metabolism | Toll-Like Receptor 7 - immunology | Myeloid Differentiation Factor 88 - immunology | Autoimmune Diseases - metabolism | Autoimmune Diseases - pathology | Autoimmune Diseases - drug therapy | B-Lymphocytes - metabolism | Interleukin-2 Receptor alpha Subunit - immunology | Toll-Like Receptor 7 - agonists | Toll-Like Receptor 7 - genetics | Amino Acid Sequence | B-Lymphocytes - cytology | Toll-Like Receptor 7 - metabolism | Autoimmune Diseases - immunology | Cells, Cultured | Reverse Transcriptase Polymerase Chain Reaction | Immunoglobulins, Intravenous - pharmacology | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Interleukin-2 Receptor alpha Subunit - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Lymphocyte Activation - drug effects | Interleukin-6 - biosynthesis | Interleukin-10 - biosynthesis | Oligodeoxyribonucleotides - pharmacology | Toll-Like Receptor 9 - metabolism | Myeloid Differentiation Factor 88 - metabolism | Employee recruitment | Enzymes | Phosphatases | Immunoglobulin G | Oligodeoxyribonucleotides | Interleukin-2 Receptor alpha Subunit | Signal Transduction | Interleukin-10 | Lymphocyte Activation | Toll-Like Receptor 9 | B-Lymphocytes | Myeloid Differentiation Factor 88 | Toll-Like Receptor 7 | Interleukin-6 | Life Sciences | Immunology | Autoimmune Diseases | Sialic Acid Binding Ig-like Lectin 2 | Protein Tyrosine Phosphatase, Non-Receptor Type 6 | Immunoglobulins, Intravenous
Journal Article
Immunology, ISSN 0019-2805, 11/2011, Volume 134, Issue 3, pp. 257 - 269
Summary Interferon‐α (IFN‐α) produced at high levels by human plasmacytoid dendritic cells (pDCs) can specifically regulate B‐cell activation to Toll‐like... 
dendritic cells | B cells | Type I interferon/toll‐like receptors | CpGDNA | adjuvants | plasmacytoid | Type I interferon/toll-like receptors | Adjuvants | Plasmacytoid | Dendritic cells | NATURAL-KILLER-CELLS | ACTIVATION | HIV-1 ENV TRIMERS | IMMUNOLOGY | VACCINE ADJUVANTS | BLOOD MONONUCLEAR-CELLS | I INTERFERON | MEMORY | TLR LIGANDS | CPG OLIGONUCLEOTIDES | INNATE IMMUNITY | Interferon-alpha - pharmacology | Dendritic Cells - immunology | Humans | Macaca mulatta - immunology | Immunoglobulin M - immunology | Quinolines - pharmacology | Toll-Like Receptor 9 - agonists | Immunoglobulin M - metabolism | Toll-Like Receptor 9 - immunology | Toll-Like Receptor 8 - immunology | Macaca mulatta - metabolism | Flow Cytometry | Toll-Like Receptor 7 - immunology | Dendritic Cells - drug effects | Toll-Like Receptors - metabolism | B-Lymphocytes - metabolism | Dendritic Cells - metabolism | Toll-Like Receptor 8 - agonists | Toll-Like Receptor 7 - agonists | Tumor Necrosis Factor Receptor Superfamily, Member 7 - immunology | Antigens, CD20 - immunology | Interferon-alpha - metabolism | Toll-Like Receptor 7 - metabolism | Enzyme-Linked Immunosorbent Assay | Toll-Like Receptors - immunology | Cells, Cultured | Imidazoles - pharmacology | Antigens, CD20 - metabolism | Toll-Like Receptors - agonists | Interferon-alpha - immunology | B-Lymphocytes - drug effects | Cell Differentiation - immunology | Animals | B-Lymphocytes - immunology | Cell Differentiation - drug effects | Tumor Necrosis Factor Receptor Superfamily, Member 7 - metabolism | Cell Proliferation - drug effects | Oligodeoxyribonucleotides - pharmacology | Toll-Like Receptor 9 - metabolism | Toll-Like Receptor 8 - metabolism | Primates | Immunoglobulins | Biological response modifiers | Analysis | Medical research | Ligands | T cell receptors | Immune system | Type I interferon | toll-like receptors | Original
Journal Article
Autoimmunity, ISSN 0891-6934, 2/2015, Volume 48, Issue 1, pp. 1 - 12
Abstract Ig class switch DNA recombination (CSR) in B cells is crucial to the maturation of antibody responses. It requires IgH germline IH-CH transcription... 
lipopolysaccharides | Antibody | T-independent antibody response | class switch DNA recombination | B cells | CpG | B cell receptor | Toll-like receptor | Class switch DNA recombination | Lipopolysaccharides | CROSS-REGULATION | ACTIVATION | RECOGNITION | INNATE | MICRORNAS | IMMUNOLOGY | AUTOIMMUNE | TOLL-LIKE RECEPTORS | AID | NF-KAPPA-B | EXPRESSION | Toll-Like Receptor 2 - genetics | Calcium - metabolism | Lipopeptides - pharmacology | Toll-Like Receptor 9 - genetics | Calcium - immunology | Immunoglobulin G - classification | Toll-Like Receptor 9 - immunology | Immunoglobulin G - biosynthesis | Recombination, Genetic | Toll-Like Receptor 7 - immunology | CD40 Antigens - genetics | Cell Differentiation | Immunoglobulin Class Switching - drug effects | Membrane Glycoproteins - immunology | Toll-Like Receptor 7 - genetics | B-Lymphocytes - cytology | Signal Transduction | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Toll-Like Receptor 4 - genetics | Imidazoles - pharmacology | Toll-Like Receptor 4 - immunology | Receptors, Antigen, B-Cell - immunology | Membrane Glycoproteins - genetics | Receptor Cross-Talk - immunology | Toll-Like Receptor 1 - genetics | B-Lymphocytes - drug effects | Animals | B-Lymphocytes - immunology | CD40 Ligand - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Receptors, Antigen, B-Cell - genetics | Mice | Toll-Like Receptor 1 - immunology | CD40 Antigens - immunology | Cytokines - biosynthesis | antibody, AID | cytokine | immunoglobulin | B cell receptor (BCR) | lipopolysaccharides (LPS) | germinal center | Toll-like Receptor (TLR) | class switch DNA recombination (CSR)
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6244 - 6250
High mobility group box-1 (HMGB1) protein was originally characterized as a nuclear DNA-binding protein, and was described to have an extracellular role when... 
SIGNALING PATHWAYS | DANGER SIGNALS | PROTEIN | DNA | TOLL-LIKE RECEPTOR | GROUP BOX CHROMOSOMAL-PROTEIN-1 | LIVER ISCHEMIA | RAGE | IMMUNOLOGY | ARTHRITIS | BINDING | Toll-Like Receptor 2 - agonists | Toll-Like Receptor 2 - genetics | Arthritis, Experimental - metabolism | Arthritis, Experimental - pathology | HMGB1 Protein - pharmacology | Receptors, Immunologic - agonists | Toll-Like Receptor 4 - agonists | Female | Mice, Inbred DBA | Flavonoids - pharmacology | Phosphorylation - immunology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Receptors, Immunologic - immunology | Glycation End Products, Advanced - immunology | Macrophages, Peritoneal - drug effects | Receptor for Advanced Glycation End Products | Cytokines - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Macrophages, Peritoneal - immunology | Toll-Like Receptor 4 - genetics | Up-Regulation - genetics | Arthritis, Experimental - immunology | Imidazoles - pharmacology | Toll-Like Receptor 4 - immunology | p38 Mitogen-Activated Protein Kinases - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Anthracenes - pharmacology | Mice, Knockout | Animals | Cytokines - drug effects | Up-Regulation - immunology | Glycation End Products, Advanced - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Pyridines - pharmacology | Macrophages, Peritoneal - metabolism | Receptors, Immunologic - metabolism
Journal Article
2008, Handbook of experimental pharmacology, ISBN 9783540721673, Volume 183, xi, 240
Book
Molecular Immunology, ISSN 0161-5890, 2004, Volume 40, Issue 11, pp. 773 - 783
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2012, Volume 32, Issue 3, pp. 669 - 676
OBJECTIVE—Nicotinic acid (NA) treatment has been associated with benefits in atherosclerosis that are usually attributed to effects on plasma lipoproteins. The... 
atherosclerosis | macrophages | vascular biology | receptors | cholesterol-lowering drugs | LONG-TERM | INTEGRIN VLA-4 | ACTIVATION | DENDRITIC CELLS | KAPPA-B | FOAM CELLS | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | EXTENDED-RELEASE NIACIN | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Integrin alpha4beta1 - metabolism | Toll-Like Receptor 2 - agonists | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Human Umbilical Vein Endothelial Cells - immunology | Monocytes - metabolism | Monocytes - immunology | Niacin - pharmacology | Receptors, Prostaglandin - metabolism | Receptors, Immunologic - antagonists & inhibitors | Inflammation - metabolism | Transfection | Chemotaxis, Leukocyte - drug effects | I-kappa B Kinase - metabolism | RNA Interference | Inflammation Mediators - metabolism | Toll-Like Receptor 4 - agonists | Chemokine CCL2 - metabolism | Receptors, G-Protein-Coupled - drug effects | Interleukin-6 - metabolism | Receptors, Prostaglandin - antagonists & inhibitors | Receptors, Nicotinic - drug effects | Receptors, Nicotinic - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Cyclooxygenase 2 Inhibitors - pharmacology | Anti-Inflammatory Agents - pharmacology | Cells, Cultured | Inflammation - immunology | Toll-Like Receptor 2 - metabolism | Cell Adhesion - drug effects | Toll-Like Receptor 4 - metabolism | Monocytes - drug effects | Transcription Factor RelA - metabolism | Lipopolysaccharides - pharmacology | Inflammation - genetics | Receptors, G-Protein-Coupled - genetics | Pyrazines - pharmacology | Vascular Cell Adhesion Molecule-1 - metabolism | Receptors, Nicotinic - genetics | Receptors, Immunologic - metabolism
Journal Article
2005, Progress in inflammation research, ISBN 3764372850, xii, 244
A repertoire of 10 TLRs mediate the first response to all microbes that infect mammals. They are the long sought receptors for a wide range of microbial... 
Immunological aspects | Toll-Like Receptors | Cell receptors | Signal Transduction | Mediators | Inflammation | immunology | etiology | Other branches of medicine | Medical Microbiology | Infectious Diseases | Biomedicine | Immunology | Pharmacology/Toxicology | Cell Biology
Book
Journal Article