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Journal Article
Nature, ISSN 0028-0836, 08/2012, Volume 488, Issue 7409, pp. 111 - 115
Antisense oligonucleotides (ASOs) hold promise for gene-specific knockdown in diseases that involve RNA or protein gain-of-function effects. In the hereditary... 
CTG REPEAT | 3' END | PROTEIN | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | TOXIC RNA | MODIFIED ANTISENSE OLIGONUCLEOTIDE | PHOSPHOROTHIOATE OLIGONUCLEOTIDES | MOUSE MODELS | TRINUCLEOTIDE REPEAT | Humans | Muscle, Skeletal - metabolism | RNA, Messenger - metabolism | RNA, Untranslated - genetics | Gene Knockdown Techniques | RNA - genetics | Myotonic Dystrophy - pathology | Base Sequence | Muscle, Skeletal - drug effects | Oligonucleotides, Antisense - therapeutic use | RNA, Messenger - antagonists & inhibitors | Mice, Inbred mdx | Myotonic Dystrophy - genetics | Disease Models, Animal | RNA - metabolism | Oligonucleotides, Antisense - pharmacology | Myotonin-Protein Kinase | RNA - antagonists & inhibitors | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Myotonic Dystrophy - therapy | Transcriptome - drug effects | Transcriptome - genetics | Myotonic Dystrophy - physiopathology | Animals | Oligonucleotides, Antisense - genetics | Cell Nucleus - genetics | RNA, Long Noncoding | Alleles | Trinucleotide Repeat Expansion - genetics | Mice | Mice, Inbred BALB C | Cell Nucleus - drug effects | Ribonuclease H - metabolism | Myotonic dystrophy | RNA | Oligonucleotides | Physiological aspects | Development and progression | Genetic aspects | Health aspects | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 09/2017, Volume 114, Issue 38, pp. 10232 - 10237
Journal Article
Journal of the American Chemical Society, ISSN 0002-7863, 04/2014, Volume 136, Issue 17, pp. 6355 - 6361
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, pp. e40395 - e40395
Anti-cancer therapy based on anthracyclines (DNA intercalating Topoisomerase II inhibitors) is limited by adverse effects of these compounds on the... 
ANTHRACYCLINE CARDIOTOXICITY | CELLS | REPLICATION | METASTASIS | GENE | RNA | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | MECHANISMS | CANCER | FAILURE | Antibiotics, Antineoplastic - toxicity | Antibiotics, Antineoplastic - pharmacology | Humans | Transcriptional Activation - drug effects | Transcriptome | Male | MicroRNAs - metabolism | GTPase-Activating Proteins - metabolism | RNA, Messenger - metabolism | Vacuoles - drug effects | RNA Interference | Myocardium - metabolism | HEK293 Cells | Muscle Proteins - metabolism | Nuclear Proteins - genetics | Biomarkers - metabolism | RNA, Messenger - genetics | Rats | Myocardium - pathology | Nuclear Proteins - metabolism | Rats, Sprague-Dawley | Doxorubicin - toxicity | Up-Regulation - drug effects | Animals | Cardiomyopathies - metabolism | Adaptor Proteins, Signal Transducing - genetics | MicroRNAs - genetics | GTPase-Activating Proteins - genetics | Adaptor Proteins, Signal Transducing - metabolism | Doxorubicin - pharmacology | Cardiomyopathies - chemically induced | Heart failure | Anthracyclines | Care and treatment | MicroRNA | Genes | Physiological aspects | Health aspects | Genetic translation | Cancer | Heart | Post-transcription | Biomedical research | Toxicity | Cardiomyopathy | Genomics | Science | Biology | Metastasis | Autophagy | Doxorubicin | Toxicology | Rodents | Cell cycle | Physiology | Bioindicators | Heart diseases | Drug dosages | Deoxyribonucleic acid--DNA | Cardiovascular system | Medical research | DNA topoisomerase (ATP-hydrolysing) | Environmental health | MiRNA | Etoposide | Cardiomyocytes | Breast cancer | Gene expression | Molecular modelling | Ribonucleic acids | Anthracycline | MicroRNAs | Biomarkers | mRNA stability | Guanosinetriphosphatase | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article