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Journal Article
PLoS ONE, ISSN 1932-6203, 10/2007, Volume 2, Issue 10, p. e1058
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 11, p. e0206823
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e102797
In diabetics, methylglyoxal (MG), a glucose-derived metabolite, plays a noxious role by inducing oxidative stress, which causes and exacerbates a series of... 
LRP5 GENE | HIGH-BONE-MASS | INDUCED OXIDATIVE STRESS | MULTIDISCIPLINARY SCIENCES | PROMOTER REGION | RECEPTOR-RELATED PROTEIN-5 | METHYLATION STATUS | HYDROGEN-PEROXIDE | MICE | TRANSCRIPTIONAL REGULATION | GLYCATION END-PRODUCTS | Diabetes Mellitus - genetics | Gene Expression - genetics | Methyl-CpG-Binding Protein 2 - metabolism | Wnt Proteins - metabolism | Promoter Regions, Genetic - genetics | Methyl-CpG-Binding Protein 2 - genetics | Pyruvaldehyde - metabolism | Wnt Proteins - genetics | Epigenomics - methods | Receptor Activator of Nuclear Factor-kappa B - metabolism | Proto-Oncogene Proteins - metabolism | Cell Line | Oxidative Stress - genetics | Diabetes Mellitus - metabolism | Mesenchymal Stromal Cells - metabolism | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | DNA Methylation - genetics | Signal Transduction - genetics | beta Catenin - metabolism | Down-Regulation - genetics | beta Catenin - genetics | Bone Diseases, Metabolic - metabolism | Animals | Epigenesis, Genetic - genetics | Receptor Activator of Nuclear Factor-kappa B - genetics | Mice | Bone Diseases, Metabolic - genetics | Oxidative stress | Bisulfite | Frizzled-related protein | Wnt protein | Frizzled protein | Genes | DNA damage | Inactivation | Osteoblasts | Gene sequencing | Osteoprotegerin | Proteins | Osteoporosis | β-catenin | Demethylation | Biomedical materials | Methyl-CpG binding protein | Rodents | DNA methylation | Bone marrow | Aging | Biocompatibility | Physiology | TRANCE protein | Deoxyribonucleic acid--DNA | Osteopenia | University graduates | CpG islands | NF-κB protein | Deactivation | Complications | Legal medicine | Diabetes mellitus | Stroma | Gene expression | Medicine | Pathology | Signaling | Deoxyguanosine | Sodium | MeCP2 protein | Derepression | Epigenetics | Biomarkers | Diabetes | Pyruvaldehyde | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2017, Volume 12, Issue 10, p. e0185426
Osteoclasts are large multinucleated cells responsible for bone resorption. Excessive inflammatory activation of osteoclasts leads to bony erosions, which are... 
ANTIINFLAMMATORY PHENOTYPE | C-FOS | PROTEIN-KINASE | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | RECEPTOR ACTIVATOR | MICE | DIFFERENTIATION | NF-KAPPA-B | EXPRESSION | BONE-RESORPTION | Cell Line | Osteogenesis - physiology | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Pyrimidines - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Gene Knockdown Techniques | Animals | RANK Ligand - physiology | Clustered Regularly Interspaced Short Palindromic Repeats | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Mice | Phenylurea Compounds - pharmacology | Protein Kinase Inhibitors - pharmacology | Real-Time Polymerase Chain Reaction | Care and treatment | DNA binding proteins | Rheumatoid arthritis | Analysis | Osteoclasts (Biology) | Osteoprogenitor cells | Salts | Transcription factors | Gene regulation | Arthritis | Activation | Kinases | Macrophages | Inflammatory diseases | Proteins | Bone resorption | Signal transduction | Immunology | Penicillin | Bone marrow | Biocompatibility | Inhibition | TRANCE protein | c-Fos protein | CRISPR | NF-κB protein | Cytokines | Internal medicine | Rheumatology | Extracellular signal-regulated kinase | JNK protein | Inflammation | Gene expression | Gelatinase B | Medicine | Osteoclastogenesis | Pathology | Hospitals | Inhibitors | Osteoclasts | Acid phosphatase (tartrate-resistant) | Bone | Differentiation
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e14073
The TGF family plays a key role in bone homeostasis. Systemic or topic application of proteins of this family apparently positively affects bone healing in... 
Inflammation - pathology | Receptors, Transforming Growth Factor beta - genetics | RANK Ligand - metabolism | Bone Density - physiology | Bone Morphogenetic Protein 7 - genetics | Humans | Alkaline Phosphatase - metabolism | Male | Inflammation - blood | Time Factors | Bone Morphogenetic Protein 2 - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Dioxoles - pharmacology | Female | Benzamides - pharmacology | Phosphorylation - drug effects | Transforming Growth Factor beta1 - blood | Protein-Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - pharmacology | Bone Morphogenetic Protein 2 - genetics | Bone Resorption - physiopathology | Alkaline Phosphatase - genetics | Osteoblasts - drug effects | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Smad Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Transforming Growth Factor beta1 - physiology | Gene Expression Regulation - drug effects | RANK Ligand - genetics | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Bone Morphogenetic Protein 7 - metabolism | Aged | Cell Proliferation - drug effects | Osteoblasts - metabolism | Chronic Disease | Smad Proteins - metabolism | Cell proliferation | Cell culture | Surgical implants | Femur | Phosphorylation | Transplants & implants | Homeostasis | Cytotoxicity | Bone diseases | Kinases | Osteoblasts | Osteoprotegerin | Proteins | Biomedical materials | Mineralization | Smad2 protein | Biocompatibility | Bone density | Inhibition | TRANCE protein | Growth factors | Total hip arthroplasty | Osteocalcin | Cytokines | Bone morphogenetic protein 2 | Tissue engineering | Osteopontin | Bone healing | Bone turnover | Metabolism | Gene expression | Patients | Hip | Bone mineral content | Studies | Signaling | Ligands | Healing | Bone mineral density | Bone | Cancer
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 08/2018, Volume 119, Issue 8, pp. 6974 - 6985
Several growth factors in bone tissues are reported to be associated with osteoclastogenesis. Activin‐A, a member of the transforming growth factor‐β (TGF‐β)... 
c‐fos | activin‐A | osteoclast | nuclear factor of activated T cell cytoplasmic 1 (NFATc1) | SMAD2/3 | c-fos | activin-A | SMAD2 | SIGNALING PATHWAYS | RANKL | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE HOMEOSTASIS | RECEPTOR | KAPPA-B | BINDING PROTEIN | CELL BIOLOGY | DIFFERENTIATION | EXPRESSION | RANK Ligand - metabolism | Bone Marrow Cells - cytology | NFATC Transcription Factors - metabolism | Proto-Oncogene Proteins c-fos - biosynthesis | Smad2 Protein - metabolism | Male | Macrophage Colony-Stimulating Factor - metabolism | Smad3 Protein - metabolism | Osteoclasts - cytology | Osteoclasts - metabolism | Animals | Activins - pharmacology | Cathepsin K - metabolism | Follistatin - pharmacology | RAW 264.7 Cells | Mice | Bone Marrow Cells - metabolism | Physiological aspects | Bone morphogenetic proteins | T cells | Transforming growth factors | Gene expression | Protein binding | Cell culture | Smad protein | Immunoprecipitation | Transforming growth factor-a | Lymphocytes T | Kinases | Macrophages | Tissues | Blockage | Bone resorption | Proteins | Signal transduction | Bone growth | Biomedical materials | Actin | Smad2 protein | Acid resistance | Bone marrow | Biocompatibility | TRANCE protein | Growth factors | Cathepsin K | Follistatin | MAP kinase | Osteoclastogenesis | Signaling | Monocytes | Protein kinase | Activin | Acid phosphatase
Journal Article
Cell death & disease, ISSN 2041-4889, 09/2017, Volume 8, Issue 9, pp. e3037 - e3037
Postmenopausal osteoporosis (POMP) is a public health problem characterized by decreased bone density and increased fracture risk. Over-activated... 
SIGNALING PATHWAYS | INHIBITION | RECEPTOR ACTIVATOR | PROLIFERATION | LIGAND | SUPPRESSES OSTEOCLASTOGENESIS | NF-KAPPA-B | MMP-9 | MESENCHYMAL STEM-CELLS | INDUCED BONE LOSS | CELL BIOLOGY | Humans | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Mesenchymal Stromal Cells - cytology | RANK Ligand - pharmacology | Macrophage Colony-Stimulating Factor - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Mesenchymal Stromal Cells - drug effects | NF-kappa B - antagonists & inhibitors | Osteoclasts - pathology | Signal Transduction | Macrophage Colony-Stimulating Factor - antagonists & inhibitors | Mesenchymal Stromal Cells - metabolism | Osteoporosis, Postmenopausal - genetics | Bone Density Conservation Agents - chemical synthesis | Ribosomal Proteins - agonists | Osteoporosis, Postmenopausal - drug therapy | Bone Density Conservation Agents - pharmacology | Mitogen-Activated Protein Kinases - genetics | Mice | Ovariectomy - adverse effects | Alkaloids - chemical synthesis | Osteoporosis, Postmenopausal - pathology | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Ribosomal Proteins - metabolism | Bone Marrow Cells - drug effects | Female | Benzopyrans - pharmacology | Osteogenesis - genetics | Osteoporosis, Postmenopausal - etiology | Alkaloids - pharmacology | Bone Marrow Cells - cytology | Ribosomal Proteins - genetics | Osteogenesis - drug effects | Gene Expression Regulation | Osteoclasts - metabolism | Phosphatidylinositol 3-Kinases - genetics | Bone Density - drug effects | Animals | Mitogen-Activated Protein Kinases - antagonists & inhibitors | NF-kappa B - genetics | RANK Ligand - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - agonists | Acetates - pharmacology | Primary Cell Culture | Bone Marrow Cells - metabolism | Osteoclasts - drug effects | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Mitogen-Activated Protein Kinases - metabolism | Mesenchyme | Ovariectomy | Ribosomal protein S5 | Menopause | Macrophage colony-stimulating factor | Stem cell transplantation | AKT protein | Post-menopause | 1-Phosphatidylinositol 3-kinase | Osteoclastogenesis | Osteoporosis | Allografts | Cellular biology | Bioactive compounds | Stem cells | Bone marrow | Hormone replacement therapy | Bone density | TRANCE protein | Bone loss | Public health | Osteogenesis | Adipogenesis | Original
Journal Article
by Meng, S and Zhang, L and Tang, Y and Tu, Q and Zheng, L and Yu, L and Murray, D and Cheng, J and Kim, S.H and Zhou, X and Chen, J
Journal of Dental Research, ISSN 0022-0345, 7/2014, Volume 93, Issue 7, pp. 657 - 662
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 02/2018, Volume 233, Issue 2, pp. 1481 - 1489
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3445 - 3452
Macrophage surface antigen‐1 (Mac‐1, CD11b/CD18) has been implicated in the regulation of osteoclastogenesis. In the synovial tissues of patients with aseptic... 
bone micro‐CT | cytokines | molecular pathways | osteoclasts | bone micro-CT | MEDICINE, RESEARCH & EXPERIMENTAL | OSTEOLYSIS | DAP12 | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | RANK Ligand - metabolism | Tumor Necrosis Factor-alpha - genetics | CD11b Antigen - genetics | RANK Ligand - pharmacology | Syk Kinase - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Female | Macrophage-1 Antigen - metabolism | Osteogenesis - genetics | Osteoclasts - pathology | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | CD18 Antigens - genetics | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Osteogenesis - drug effects | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | Osteoclasts - metabolism | CD18 Antigens - metabolism | RANK Ligand - genetics | Syk Kinase - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Macrophage-1 Antigen - genetics | Tartrate-Resistant Acid Phosphatase - metabolism | Proto-Oncogene Proteins c-fos - genetics | Mice | CD11b Antigen - metabolism | Primary Cell Culture | Tartrate-Resistant Acid Phosphatase - genetics | Osteoclasts - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | NFATC Transcription Factors - genetics | Tyrosine | Phenols | Osteoprogenitor cells | Polyethylenes | Prostheses | Surgical implants | Transcription factors | NF-AT protein | Macrophages | Osteolysis | Tissues | Molecular weight | Signal transduction | Mac1 protein | Lymphocytes | Acid resistance | Tumor necrosis factor-TNF | Biocompatibility | TRANCE protein | c-Fos protein | Protein-tyrosine kinase | Total hip arthroplasty | Spleen | Polyethylene | CD11b antigen | Alpha rays | Extracellular signal-regulated kinase | Hip | Osteoclastogenesis | Signaling | Bone implants | Tumor necrosis factor | Loosening | CD18 antigen | Syk protein | Osteoclasts | Acid phosphatase (tartrate-resistant) | Acid phosphatase | Tumors | Original
Journal Article