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Journal Article
Scientific Reports, ISSN 2045-2322, 10/2015, Volume 5, Issue 1, p. 14989
Hepatic stem/progenitor cells, hepatoblasts, have a high proliferative ability and can differentiate into mature hepatocytes and cholangiocytes. Therefore,... 
PLURIPOTENT STEM-CELLS | LONG-TERM CULTURE | IN-VITRO | MESSENGER-RNA | GENE | ONCOSTATIN-M | MULTIDISCIPLINARY SCIENCES | HEPATOCYTE TRANSPLANTATION | BINDING-PROTEIN | LIVER DEVELOPMENT | EXPRESSION | Hepatocyte Nuclear Factor 1-alpha - genetics | Embryo, Mammalian | Cytochrome P-450 Enzyme System - metabolism | Mouse Embryonic Stem Cells - cytology | Hepatocytes - metabolism | SOXF Transcription Factors - metabolism | Hepatocyte Nuclear Factor 1-alpha - metabolism | Oncostatin M - pharmacology | Mouse Embryonic Stem Cells - drug effects | Mouse Embryonic Stem Cells - metabolism | Hepatocytes - cytology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Fetus | Transcription, Genetic | CCAAT-Enhancer-Binding Proteins - genetics | Hepatocytes - drug effects | Carbamoyl-Phosphate Synthase (Ammonia) - genetics | CCAAT-Enhancer-Binding Proteins - metabolism | SOX9 Transcription Factor - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Transcription Factors - genetics | Keratin-19 - genetics | Transcription Factors - metabolism | Animals | Carbamoyl-Phosphate Synthase (Ammonia) - metabolism | Cell Differentiation - drug effects | Cytochrome P-450 Enzyme System - genetics | Cell Proliferation - drug effects | Mice | Primary Cell Culture | Keratin-19 - metabolism | SOXF Transcription Factors - genetics | SOX9 Transcription Factor - genetics | Cell culture | Transcription factors | Liver diseases | Cytochrome P450 | Sox9 protein | Fetuses | CCAAT/enhancer-binding protein | Drug screening | Oncostatin M | Embryos | Regeneration | Hepatocytes | Rodents | Stem cells | Helix-loop-helix proteins
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 11/2001, Volume 155, Issue 4, pp. 519 - 530
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 04/2007, Volume 292, Issue 4, pp. G1123 - G1132
Several animal models have been developed to investigate the pathobiology of pancreatitis, but few studies have examined the effects that altered pancreatic... 
Growth arrest and DNA damage inducible 34 | Endoplasmic reticulum stress response | Activating transcription factor 3 | Apoptosis | PHYSIOLOGY | UNFOLDED-PROTEIN RESPONSE | ER STRESS | apoptosis | ENDOPLASMIC-RETICULUM STRESS | growth arrest and DNA damage inducible 34 | SEROUS EXOCRINE CELLS | ACINAR-CELL | IN-VITRO | PERK EIF2-ALPHA KINASE | ACUTE NECROTIZING PANCREATITIS | C-JUN | GASTROENTEROLOGY & HEPATOLOGY | activating transcription factor 3 | endoplasmic reticulum stress response | Amylases - blood | Apoptosis - drug effects | Pancreas, Exocrine - pathology | Endoplasmic Reticulum - metabolism | Male | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Immediate-Early Proteins - metabolism | Pancreatitis - genetics | Stress, Physiological - chemically induced | Cholecystokinin - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Time Factors | Stress, Physiological - metabolism | Cell Cycle Proteins - genetics | Eukaryotic Initiation Factor-2 - metabolism | Antigens, Differentiation - metabolism | Pancreatitis - physiopathology | Protein Phosphatase 1 | Basic Helix-Loop-Helix Transcription Factors - deficiency | Activating Transcription Factor 3 - genetics | Disease Models, Animal | Severity of Illness Index | Acute Disease | Gene Expression | Basic Helix-Loop-Helix Transcription Factors - genetics | Stress, Physiological - genetics | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Cells, Cultured | Cholecystokinin - pharmacology | Stress, Physiological - physiopathology | Pancreatitis - chemically induced | Activating Transcription Factor 3 - metabolism | Mice, Knockout | Regeneration | Animals | Immediate-Early Proteins - genetics | Eukaryotic Initiation Factor-2 - genetics | Stress, Physiological - pathology | Pancreatitis - pathology | Antigens, Differentiation - genetics | Mice | Pancreas, Exocrine - metabolism | Pancreatitis - metabolism | Ceruletide
Journal Article
Physiological Genomics, ISSN 1094-8341, 02/2011, Volume 43, Issue 3, pp. 174 - 186
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 2016, Volume 36, Issue 23, pp. 2931 - 2944
Transcriptional networks that govern secretory cell specialization, including instructing cells to develop a unique cytoarchitecture, amass extensive protein... 
SEROUS EXOCRINE CELLS | XBP1 | GENE | ER STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | TRANSCRIPTION FACTOR MIST1 | ENDOPLASMIC-RETICULUM STRESS | AUTOPHAGY | DIFFERENTIATION | EXPRESSION | CELL BIOLOGY
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 2016, Volume 36, Issue 23, pp. 2945 - 2955
Much remains unknown regarding the regulatory networks formed by transcription factors in mature, differentiated mammalian cells in vivo, despite many studies... 
ORGANIZATION | SEROUS EXOCRINE CELLS | DNA-BINDING | EXPRESSION ANALYSIS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | NETWORK MOTIFS | MOUSE | NEURAL-TUBE | TRANSCRIPTION FACTOR MIST1 | DIFFERENTIATION | CELL BIOLOGY
Journal Article
Physiological Genomics, ISSN 1094-8341, 01/2011, Volume 43, Issue 3, pp. 174 - 186
Despite their divergent developmental ancestry, plasma cells and gastric zymogenic (chief) cells share a common function: high-capacity secretion of protein.... 
Activating transcription factor 1
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e84182
Gene expression is affected by modifications to histone core proteins within chromatin. Changes in these modifications, or epigenetic reprogramming, can... 
METHYLATION | ACTIVATION | HISTONE H3 | MULTIDISCIPLINARY SCIENCES | REGIONS | TRANSCRIPTION FACTOR MIST1 | MECHANISMS | STRESS | ACTIVE GENES | EXPRESSION | EXOCRINE | Chromatin - metabolism | Carcinoma, Pancreatic Ductal - chemically induced | Pancreatic Neoplasms - metabolism | Epigenesis, Genetic | Carcinoma, Pancreatic Ductal - metabolism | Male | Carcinoma, Pancreatic Ductal - genetics | Pancreatitis - genetics | Pancreatic Neoplasms - chemically induced | Basic Helix-Loop-Helix Transcription Factors - deficiency | Acinar Cells - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Pancreatic Neoplasms - pathology | Pancreas - pathology | Pancreatic Neoplasms - genetics | Pancreas - metabolism | Pancreatitis - chemically induced | Carcinoma, Pancreatic Ductal - pathology | Mice, Knockout | Acinar Cells - pathology | Animals | Histones - genetics | Metabolic Networks and Pathways | Pancreatitis - pathology | Mice | Histones - metabolism | Pancreatitis - metabolism | Ceruletide | Methylation | Chromatin - genetics | Enrichment | Adenocarcinoma | Chromatin | Transcription factors | Immunoprecipitation | Disease | Transcription | Laboratories | Genes | Childrens health | Genomes | Gene sequencing | Proteins | Cell fate | Rodents | Gastroenterology | DNA methylation | Physiology | Pancreas | Acinar cells | Medical research | Pancreatitis | Gene expression | Ablation | Children & youth | Lysine | Pancreatic cancer | Epigenetics | Cellular stress response
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 6, pp. 2038 - 2049
Journal Article
Journal Article