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Apoptosis, ISSN 1360-8185, 1/2018, Volume 23, Issue 1, pp. 65 - 78
Sp1 is a ubiquitous transcription factor that regulates many genes involved in apoptosis and senescence. Sp1 also has a role in the DNA damage response; at low... 
Biochemistry, general | Caspase cleavage | Intrinsic pathway | DNA damage | Oncology | Protein stability | Cell Biology | Virology | Biomedicine | Sp1 | Extrinsic pathway | Cancer Research | Transcription factor | Apoptosis | ACTIVATION | STRAND BREAK REPAIR | CANCER CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | O-GLYCOSYLATION | DNA-DAMAGE | DOWN-REGULATION | MEDIATED CLEAVAGE | CELL BIOLOGY | PREMATURE SENESCENCE | DIHYDROFOLATE-REDUCTASE | GENE-TRANSCRIPTION | Cell Cycle - genetics | RNA, Small Interfering - genetics | Apoptosis - drug effects | Apoptosis - radiation effects | Humans | Caspase 3 - metabolism | Apoptosis - genetics | Sp1 Transcription Factor - metabolism | TNF-Related Apoptosis-Inducing Ligand - pharmacology | MCF-7 Cells | Proteolysis | Caspase 3 - genetics | HEK293 Cells | Madin Darby Canine Kidney Cells | Protein Stability | Fibroblasts - metabolism | Cell Line | Osteoblasts - radiation effects | Signal Transduction | Cell Cycle - radiation effects | Osteoblasts - drug effects | Gene Expression Regulation | Ultraviolet Rays - adverse effects | Osteoblasts - pathology | Animals | Fibroblasts - radiation effects | Sp1 Transcription Factor - genetics | Fibroblasts - drug effects | Dogs | Cell Line, Tumor | Bleomycin - pharmacology | Fibroblasts - cytology | DNA Damage | Mutation | Cell Cycle - drug effects | Osteoblasts - metabolism | Camptothecin - pharmacology | Doxorubicin - pharmacology | RNA, Small Interfering - metabolism | Proteins | Medical colleges | Analysis | Aspartate | DNA repair | Senescence | Alanine | Transcription factors | Epithelial cells | Caspase | Amino acids | Activation | Double-strand break repair | Sp1 protein | Caspase-3 | Damage localization | Cleavage | Aspartic acid | Deoxyribonucleic acid--DNA | Stimuli
Journal Article
Nucleic Acids Research, ISSN 0305-1048, 02/2012, Volume 40, Issue 4, pp. 1499 - 1508
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2013, Volume 288, Issue 47, pp. 33927 - 33938
Pancreatic cancer, the fourth most prevalent cancer-related cause of death in the United States, is a disease with a dismal survival rate of 5% 5 years after... 
PATHWAYS | APOPTOSIS | ACTIVATION | ADENOCARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | Pancreatic Cancer | KAPPA-B | O-GlcNAc | N-ACETYLGLUCOSAMINE TRANSFERASE | Heat Shock Protein | Minnelide | IN-VITRO | INHIBITION | Sp1 | Triptolide | Pancreas | TUMOR-GROWTH | EXPRESSION | Pancreatic Neoplasms - metabolism | Humans | Cell Survival - genetics | N-Acetylglucosaminyltransferases - genetics | Antineoplastic Agents, Alkylating - pharmacology | NF-kappa B - metabolism | Sp1 Transcription Factor - metabolism | Glycosylation - drug effects | Neoplasm Proteins - metabolism | Acetylglucosamine - metabolism | Heat Shock Transcription Factors | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Cell Nucleus - metabolism | Epoxy Compounds - pharmacology | Cell Death - genetics | Cell Nucleus - pathology | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Active Transport, Cell Nucleus - genetics | Gene Expression Regulation, Neoplastic - genetics | HSP70 Heat-Shock Proteins - biosynthesis | Diterpenes - pharmacology | Cell Survival - drug effects | Pancreatic Neoplasms - pathology | HSP70 Heat-Shock Proteins - genetics | Pancreatic Neoplasms - genetics | Signal Transduction - genetics | Transcription Factors - genetics | Acetylglucosamine - genetics | DNA-Binding Proteins - genetics | Down-Regulation - drug effects | Down-Regulation - genetics | N-Acetylglucosaminyltransferases - metabolism | Transcription Factors - metabolism | Phenanthrenes - pharmacology | Cell Nucleus - genetics | NF-kappa B - genetics | Signal Transduction - drug effects | Sp1 Transcription Factor - genetics | Active Transport, Cell Nucleus - drug effects | Cell Line, Tumor | N-Acetylglucosaminyltransferases - antagonists & inhibitors | Molecular Bases of Disease
Journal Article