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Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2014, Volume 34, Issue 10, pp. 2310 - 2320
OBJECTIVE—Vascular endothelial growth factor (VEGF) has been identified as a crucial regulator of physiological and pathological angiogenesis. Among the... 
Plasma membrane calcium-transporting ATPase | Angiogenesis effect | Calcium | Nuclear factors of activated T cells | Calcineurin | calcium | angiogenesis effect | VEGF | calcineurin | PUMP | plasma membrane calcium-transporting ATPase | BREAST-CANCER CELLS | SMOOTH-MUSCLE | ACTIVATED T-CELLS | PATHWAY | nuclear factors of activated T cells | DEPENDENT ATPASE | PERIPHERAL VASCULAR DISEASE | NUCLEAR FACTOR | HEMATOLOGY | EXPRESSION | KNOCKOUT MOUSE | Neovascularization, Physiologic - drug effects | Hindlimb | Cell Proliferation | Plasma Membrane Calcium-Transporting ATPases - genetics | Humans | Ischemia - enzymology | Intracellular Signaling Peptides and Proteins - metabolism | Calcium-Transporting ATPases - genetics | Plasma Membrane Calcium-Transporting ATPases - metabolism | Transfection | RNA Interference | Time Factors | HEK293 Cells | Muscle Proteins - metabolism | Calcium-Transporting ATPases - metabolism | Angiogenesis Inducing Agents - pharmacology | Vascular Endothelial Growth Factor A - pharmacology | Disease Models, Animal | Muscle, Skeletal - blood supply | Calcium-Transporting ATPases - deficiency | Human Umbilical Vein Endothelial Cells - drug effects | Signal Transduction | NFATC Transcription Factors - metabolism | Ischemia - physiopathology | Mice, Knockout | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Cyclooxygenase 2 - metabolism | Mice | Endothelial Cells - enzymology | Calcineurin - metabolism | Cell Movement | Endothelial Cells - drug effects | NFATC Transcription Factors - genetics
Journal Article
The FEBS Journal, ISSN 1742-464X, 11/2013, Volume 280, Issue 21, pp. 5419 - 5429
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2017, Volume 127, Issue 8, pp. 3065 - 3074
The lack of mechanistic explanations for many genotype-phenotype associations identified by GWAS precludes thorough assessment of their impact on human health.... 
MEDICINE, RESEARCH & EXPERIMENTAL | TRANSCRIPTION FACTOR-BINDING | BCL11A | SATURATING MUTAGENESIS | FETAL-HEMOGLOBIN | DISEASE | GENE-EXPRESSION | LOCI | ATPASE 4 | REGULATORY ELEMENTS | VARIANT | Plasma Membrane Calcium-Transporting ATPases - genetics | Calcium - metabolism | Humans | Erythroblasts - metabolism | Male | Gene Expression Profiling | Calcium-Transporting ATPases - genetics | Gene Regulatory Networks | Plasma Membrane Calcium-Transporting ATPases - metabolism | HEK293 Cells | Calcium-Transporting ATPases - metabolism | Erythrocytes - cytology | Malaria - metabolism | Genetic Predisposition to Disease | Genome-Wide Association Study | Epigenomics | Mice, Transgenic | Chromosome Mapping | Phenotype | Animals | Enhancer Elements, Genetic | CRISPR-Cas Systems | Mice | Polymorphism, Single Nucleotide | Quantitative Trait Loci | Malaria - genetics | Quantitative trait loci | Malaria | Erythrocytes | Disease susceptibility | Genetic aspects | Research | Gene expression | Transcription factors | Infections | Genomes | Single-nucleotide polymorphism | Regulatory sequences | Blood | Ca2+-transporting ATPase | Consortia | Genotype & phenotype | Calcium (blood) | Clonal deletion | Rodents | Hemoglobin | CRISPR | Calcium (intracellular) | Hydration | Blood cells | Principal components analysis | Erythroid cells | Mutagenesis | Erythroblasts | Gene mapping | Binding sites
Journal Article
Journal Article