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Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2009, Volume 29, Issue 19, pp. 6088 - 6093
On bipolar cells are connected to photoreceptors via a sign-inverting synapse. At this synapse, glutamate binds to a metabotropic receptor which couples to the... 
CAPSAICIN RECEPTOR | RETINAL GANGLION-CELLS | METABOTROPIC GLUTAMATE-RECEPTOR | PATHWAY | DESENSITIZATION | MGLUR6 | MOUSE | TRP CHANNELS | G-PROTEIN | NEUROSCIENCES | STATIONARY NIGHT BLINDNESS | Propionates - pharmacology | Transient Receptor Potential Channels - antagonists & inhibitors | Synaptic Transmission - physiology | Capsaicin - pharmacology | Receptors, Metabotropic Glutamate - metabolism | Amino Acids - pharmacology | TRPV Cation Channels - metabolism | Retinal Bipolar Cells - physiology | TRPV Cation Channels - antagonists & inhibitors | Synaptic Transmission - drug effects | Ruthenium Red - metabolism | Transient Receptor Potential Channels - metabolism | Endocannabinoids | Xanthenes - pharmacology | Membrane Potentials - drug effects | Mice, Inbred C57BL | TRPV Cation Channels - agonists | TRPM Cation Channels - agonists | Capsaicin - analogs & derivatives | Excitatory Amino Acid Antagonists - pharmacology | Membrane Potentials - physiology | Cinnamates - pharmacology | Transient Receptor Potential Channels - agonists | Arachidonic Acids - pharmacology | Mice, Knockout | Animals | TRPM Cation Channels - antagonists & inhibitors | Anilides - pharmacology | Polyunsaturated Alkamides - pharmacology | Glutamic Acid - metabolism | Mice | TRPM Cation Channels - metabolism | Receptors, Metabotropic Glutamate - agonists | Index Medicus | Retina | TRPV | Patch Clamp | metabotropic glutamate receptor | Capsaicin | Knockout
Journal Article
Science, ISSN 0036-8075, 5/2012, Volume 336, Issue 6081, pp. 597 - 601
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 44, pp. 19084 - 19089
Reduced functional bladder capacity and concomitant increased micturition frequency (pollakisuria) are common lower urinary tract symptoms associated with... 
Edema | Receptors | Urinary bladder | Overactive bladder | Mice | Pharmacology | Urination | Genetic mutation | Cystitis | Vehicles | Transient receptor potential channels | Urothelium | ACTIVATION | NEUROGENIC INFLAMMATION | ANTAGONIST | URINARY-BLADDER | MULTIDISCIPLINARY SCIENCES | transient receptor potential channels | urothelium | LACKING TRPV4 | OVERACTIVE BLADDER | CONTRACTION | RECEPTOR POTENTIAL VANILLOID-4 | MUTATIONS | TRANSIENT | Urothelium - metabolism | Cystitis - metabolism | Rats, Wistar | Humans | Antineoplastic Agents, Alkylating - pharmacology | Cyclophosphamide - adverse effects | Cystitis - drug therapy | TRPV Cation Channels - metabolism | TRPV Cation Channels - antagonists & inhibitors | Membrane Transport Modulators - pharmacology | Urination - drug effects | Urinary Bladder - metabolism | Morpholines - pharmacology | Rats | Urothelium - physiopathology | TRPV Cation Channels - genetics | Mice, Knockout | Pyrroles - pharmacology | Animals | Cyclophosphamide - pharmacology | Urinary Bladder - physiopathology | Antineoplastic Agents, Alkylating - adverse effects | Cystitis - chemically induced | Cystitis - physiopathology | Complications and side effects | Care and treatment | Cyclophosphamide | Medical examination | Bladder | Ion channels | Diagnosis | Properties | Cysts | Medical disorders | Rodents | Cells | Quality of life | Index Medicus | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, pp. e0128242 - e0128242
Background Various transient receptor potential (TRP) channels in sensory neurons contribute to the transduction of mechanical stimuli in the colon. Recently,... 
PAIN | INFLAMMATORY RESPONSES | MULTIDISCIPLINARY SCIENCES | CATION CHANNEL | MENTHOL | MOUSE COLON | VR-OAC | RECEPTOR POTENTIAL VANILLOID-4 | ION CHANNELS | SUBSTANCE-P | HYPERSENSITIVITY | Transient Receptor Potential Channels - antagonists & inhibitors | Calcitonin Gene-Related Peptide - secretion | Colon - drug effects | Mechanoreceptors - physiology | Nociception - physiology | Male | TRPV Cation Channels - antagonists & inhibitors | Colon - physiopathology | Female | Transient Receptor Potential Channels - physiology | Benzamides - pharmacology | Acetanilides - pharmacology | TRPV Cation Channels - physiology | Purines - pharmacology | Mice, Inbred C57BL | Thiophenes - pharmacology | Sulfonamides - pharmacology | TRPM Cation Channels - physiology | Colon - metabolism | TRPV Cation Channels - genetics | Mice, Knockout | Animals | Nociceptive Pain - physiopathology | TRPM Cation Channels - antagonists & inhibitors | Ruthenium Red - pharmacology | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | TRPM Cation Channels - genetics | Gadolinium | Neuropeptides | Antagonists | Channels | Distension | Transient receptor potential proteins | Signal transduction | Amiloride | Pain | Rodents | Gastroenterology | Sensory neurons | Physiology | Menthol | Colon | Sodium channels | Pain perception | Ruthenium | Cold | Pharmacology | Pressure | Inflammatory bowel disease | Calcitonin gene-related peptide | Signaling | Ruthenium red | Irritable bowel syndrome | Mice | Mutation | Mechanical stimuli | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2013, Volume 33, Issue 13, pp. 5603 - 5611
Visceral afferents expressing transient receptor potential (TRP) channels TRPV1 and TRPA1 are thought to be required for neurogenic inflammation and... 
ACTIVATION | SUBPOPULATIONS | PHOSPHORYLATION | SENSITIZATION | PROTEIN-KINASE | HYPERALGESIA | EXPRESSION | NEUROSCIENCES | SENSORY NEURONS | ERK | Calcium - metabolism | Male | Nodose Ganglion - metabolism | RNA, Messenger - metabolism | Amidines - metabolism | Time Factors | TRPV Cation Channels - antagonists & inhibitors | Pancreatitis, Chronic - drug therapy | Exploratory Behavior - drug effects | Monocytes - pathology | Pain - etiology | Sensory Receptor Cells - metabolism | Transient Receptor Potential Channels - metabolism | Calcitonin Gene-Related Peptide - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Injections, Intraperitoneal | Pancreas - pathology | Analgesics, Opioid - therapeutic use | Pancreas - metabolism | Pain - pathology | Disease Progression | Pancreatitis, Chronic - pathology | Analysis of Variance | Transient Receptor Potential Channels - genetics | Ganglia, Spinal - pathology | Mice | Transient Receptor Potential Channels - antagonists & inhibitors | Oximes - therapeutic use | Pancreatitis, Chronic - chemically induced | Monocytes - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | TRPV Cation Channels - metabolism | Antigens, Differentiation - metabolism | Pyridines - therapeutic use | Sensory Receptor Cells - pathology | Pain - prevention & control | Mice, Inbred C57BL | Ceruletide - toxicity | Pancreas - drug effects | Pain Measurement - drug effects | TRPV Cation Channels - genetics | Gene Expression Regulation - drug effects | Nodose Ganglion - pathology | Animals | Sensory Receptor Cells - drug effects | Pancreatitis, Chronic - complications | TRPA1 Cation Channel | Morphine - therapeutic use | Ganglia, Spinal - metabolism | Peroxidase - metabolism | Index Medicus
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e77998 - e77998
Primary sensory afferents of the dorsal root and trigeminal ganglia constantly transmit sensory information depicting the individual's physical and chemical... 
DOMAIN POTASSIUM CHANNELS | CAPSAICIN RECEPTOR | CALCIUM-IONS | PAIN | MULTIDISCIPLINARY SCIENCES | CATION CHANNEL | TRP CHANNELS | ION-CHANNEL | BACKGROUND K+ CHANNEL | DIRECT ACTIVATION | SENSORY NEURONS | Transient Receptor Potential Channels - antagonists & inhibitors | Humans | Terpenes - pharmacology | TRPV Cation Channels - metabolism | TRPV Cation Channels - antagonists & inhibitors | Neurons - metabolism | Transient Receptor Potential Channels - metabolism | Neurons - drug effects | TRPC Cation Channels - genetics | Potassium Channels, Tandem Pore Domain - metabolism | Trigeminal Ganglion - cytology | Cells, Cultured | Potassium Channels, Tandem Pore Domain - genetics | Rats | Benzaldehydes - pharmacology | TRPC Cation Channels - antagonists & inhibitors | TRPV Cation Channels - genetics | Volatile Organic Compounds - pharmacology | Animals | TRPM Cation Channels - antagonists & inhibitors | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | Potassium Channels, Tandem Pore Domain - antagonists & inhibitors | TRPC Cation Channels - metabolism | TRPM Cation Channels - genetics | TRPM Cation Channels - metabolism | Neurons | Brain | Anosmia | Capsaicin receptors | Activation | Antagonists | Experiments | Channels | Odorants | Oocytes | Depolarization | Transient receptor potential proteins | Toxicology | Pain | Trigeminal nerve | Rodents | Sensory neurons | Physiology | Membrane potential | Subpopulations | Cold | Potassium channels | Ganglia | Chemicals | Environmental effects | Smell | Heat | Olfaction | Ligands | Volatile compounds | Vanillin | Trigeminal ganglion | Potassium | Stimuli | Acetone | Index Medicus
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 03/2013, Volume 168, Issue 6, pp. 1430 - 1444
Background and Purpose Palmitoylethanolamide (PEA) is an endogenous fatty acid amide displaying anti-inflammatory and analgesic actions. To investigate the... 
intracellular calcium concentrations | transcription factor | pain | palmitoylethanolamide | PPAR | transient receptor potential vanilloid type‐1 channels | capsaicin | bradykinin | PPAR transcription factor | transient receptor potential vanilloid type-1 channels | N-PALMITOYLETHANOLAMIDE | CA2+ INFLUX | RAT | ROOT GANGLION NEURONS | VR1 RECEPTORS | ANANDAMIDE | HUMAN VANILLOID RECEPTORS | CANNABINOID RECEPTOR | CALCIUM-CHANNELS | PHARMACOLOGY & PHARMACY | CELL-LINE | Ethanolamines - pharmacology | Cricetulus | Endocannabinoids - pharmacology | Membrane Transport Modulators - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | TRPV Cation Channels - metabolism | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | TRPV Cation Channels - antagonists & inhibitors | Membrane Transport Modulators - pharmacology | Sensory Receptor Cells - metabolism | Palmitic Acids - pharmacology | Action Potentials - drug effects | CHO Cells | Recombinant Proteins - metabolism | Tachyphylaxis | Cell Line | Nerve Tissue Proteins - antagonists & inhibitors | Cricetinae | PPAR alpha - antagonists & inhibitors | Recombinant Proteins - antagonists & inhibitors | Palmitic Acids - antagonists & inhibitors | Nerve Tissue Proteins - agonists | TRPV Cation Channels - agonists | Rats | Ethanolamines - antagonists & inhibitors | Recombinant Proteins - agonists | Nerve Tissue Proteins - genetics | TRPV Cation Channels - genetics | Nerve Tissue Proteins - metabolism | Animals | Sensory Receptor Cells - cytology | Sensory Receptor Cells - drug effects | Drug Antagonism | Calcium Signaling - drug effects | PPAR alpha - agonists | Mice | PPAR alpha - metabolism | Endocannabinoids - antagonists & inhibitors | Index Medicus | Research Papers
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2013, Volume 288, Issue 18, pp. 12818 - 12827
The sensations of pain, itch, and cold often interact with each other. Pain inhibits itch, whereas cold inhibits both pain and itch. TRPV1 and TRPA1 channels... 
G-ALPHA(Q/11) | PAIN | NOCICEPTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ION-CHANNEL | RECEPTOR | SENSATION | COLD | HISTAMINE | Transient Receptor Potential Channels - antagonists & inhibitors | Sensation - physiology | Amebicides - pharmacology | Humans |