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Cytokine and Growth Factor Reviews, ISSN 1359-6101, 2012, Volume 23, Issue 1-2, pp. 15 - 24
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 7, p. e69252
Objectives: delta-opioid receptor (DOR) activation reduced brain ischemic infarction and attenuated neurological deficits, while DOR inhibition aggravated the ischemic damage... 
CREB PHOSPHORYLATION | TRUNCATED TRKB | CORTICAL-NEURONS | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | DEATH | HYPOXIA | K+ HOMEOSTASIS | EXPRESSION | Naltrexone - analogs & derivatives | Receptors, Opioid, delta - metabolism | Reperfusion Injury - etiology | Brain Ischemia - metabolism | Male | Infarction, Middle Cerebral Artery - metabolism | Quinolines - pharmacology | Brain - metabolism | Brain - blood supply | Naltrexone - pharmacology | Infarction, Middle Cerebral Artery - complications | Receptors, Opioid, delta - antagonists & inhibitors | Brain-Derived Neurotrophic Factor - metabolism | Reperfusion Injury - metabolism | Brain Ischemia - complications | Cerebrovascular Circulation - drug effects | Activating Transcription Factor 1 - metabolism | Rats | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Animals | Cyclic AMP Response Element-Binding Protein - metabolism | CD11b Antigen - metabolism | Receptor, trkB - metabolism | Receptors, Opioid, delta - agonists | Occlusion | Brain | Neurosciences | Phosphorylation | Traumatic brain injury | Neurobiology | Fluorescence | Reversing | Activation | Neurosurgery | Cerebrospinal fluid | Kinases | Proteins | Head injuries | Reperfusion | Ischemia | Cerebral blood flow | Rodents | Neostriatum | Inhibition | Cerebral infarction | CD11b antigen | Narcotics | Opioid receptors | Cyclic AMP response element-binding protein | Blood flow | Neurological diseases | Medicine | Studies | Signaling | Injury prevention | Brain research | Brain-derived neurotrophic factor | Hypoxia | Naltrindole | TrkB receptors | Infarction | Receptor mechanisms | Laboratory animals | Hippocampus | Brain injury | Integrative medicine
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 5, p. e34844
.... Many antidepressants increase expression of brain-derived neurotrophic factor (BDNF) in brain or activate, via phosphorylation, its receptor, TrkB... 
SIGNAL-TRANSDUCTION | ANTIDEPRESSANT ACTION | TREATMENT-RESISTANT DEPRESSION | SEROTONIN TRANSPORTER | TRUNCATED RECEPTORS | TYROSINE KINASE | BIOLOGY | GENE-EXPRESSION | GROWTH-FACTOR | HIPPOCAMPAL-NEURONS | FACTOR BDNF | Fluoxetine - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Vagus Nerve - drug effects | Male | Hippocampus - drug effects | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | Brain - metabolism | Hippocampus - metabolism | Vagus Nerve - physiology | Desipramine - pharmacology | Tyrosine - metabolism | Animals | Time Factors | Indole Alkaloids - pharmacology | Adrenergic Uptake Inhibitors - pharmacology | Phosphorylation - drug effects | Carbazoles - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Serotonin Uptake Inhibitors - pharmacology | Vagus Nerve Stimulation - methods | Receptor, trkB - metabolism | Tyrosine | Brain | Fluoxetine | Depression, Mental | Antidepressants, Tricyclic | Drugs | Phosphorylation | Desipramine | Extracellular signal-regulated kinase | Stimulation | AKT protein | Mental depression | Kinases | Sertraline | Brain-derived neurotrophic factor | Antidepressants | Rodents | TrkB receptors | Vagus nerve | Pretreatment | Acute effects | Protein-tyrosine kinase | Hippocampus | Chronic effects | Depression | Western blotting | Brain-derived neurotrophic factor receptors | sertraline
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2012, Volume 33, Issue 6, pp. 1122.e23 - 1122.e39
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